Search results for "Pyramid"

showing 10 items of 410 documents

Lateral habenula and hippocampus: A complex interaction raphe cells-mediated

1997

The study has shown an excitatory influence exerted by lateral habenula (LH) on hippocampal pyramidal cells. The modulatory influence is paradoxically serotonine-mediated; in fact all LH stimulation effects were abolished by intrahippocampal iontophoretic methysergide application. The data suggest the involvement of dorsal raphe nucleus. In fact, the dorsal raphe nucleus stimulation caused on hippocampus an expected inhibitory effect antagonized by intrahippocampal iontophoretic methysergide application. In the context of this neural structure we have highlighted a disinhibitory relation between two types of cells: slow serotonergic efferent neurones and fast GABAergic interneurones. The di…

MaleN-MethylaspartateMethysergideCell CommunicationBicucullineGABA AntagonistsDorsal raphe nucleusmedicineAnimalsRats WistarBiological PsychiatryNeuronsHabenulaRapheChemistryPyramidal CellsIontophoresisBicucullineGABA receptor antagonistElectric StimulationRatsPsychiatry and Mental healthHabenula2-Amino-5-phosphonovaleratenervous systemNeurologyRaphe NucleiGABAergicNeurology (clinical)Raphe nucleiNeurosciencemedicine.drugJournal of Neural Transmission
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PSA-NCAM expression in the rat medial prefrontal cortex

2005

The rat medial prefrontal cortex, an area considered homologous to the human prefrontal cortex, is a region in which neuronal structural plasticity has been described during adulthood. Some plastic processes such as neurite outgrowth and synaptogenesis are known to be regulated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM). Since PSA-NCAM is present in regions of the adult CNS which are undergoing structural remodeling, such as the hypothalamus or the hippocampus, we have analyzed the expression of this molecule in the medial prefrontal cortex of adult rats using immunohistochemistry. PSA-NCAM immunoreactivity was found both in cell bodies and in the neuropil of…

MaleNeuropilNeuriteInterneuronAntimetabolitesCell SurvivalSynaptophysinSynaptogenesisPrefrontal CortexHippocampusNeural Cell Adhesion Molecule L1BiologyRats Sprague-DawleyNeuroplasticityNeuropilmedicineAnimalsFluorescent Antibody Technique IndirectPrefrontal cortexNeuronsNeuronal PlasticityGlutamate DecarboxylasePyramidal CellsGeneral NeuroscienceImmunohistochemistryRatsPhenotypemedicine.anatomical_structureBromodeoxyuridinenervous systemSialic AcidsNeural cell adhesion moleculeNeuroscienceNeuroscience
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Optical release of caged glutamate for stimulation of neurons in the in vitro slice preparation

2005

Optical stimulation techniques prove useful to map func- tional inputs in the in vitro brain slice preparation: Glutamate released by a focused beam of UV light induces action potentials, which can be detected in postsynaptic neurons. The direct activation effect is influenced by factors such as compound concentration, focus depth, light absorption in the tissue, and sensitivity of different neuronal do- mains. We analyze information derived from direct stimulation ex- periments in slices from rat barrel cortex and construct a computa- tional model of a layer V pyramidal neuron that reproduces the experimental findings. The model predictions concerning the influ- ence of focus depth on inpu…

MalePatch-Clamp TechniquesUltraviolet RaysModels NeurologicalBiomedical EngineeringAction PotentialsStimulationIn Vitro TechniquesCaged glutamateBrain mappingBiomaterialsOpticsSlice preparationGlutamatesPostsynaptic potentialmedicineAnimalsComputer SimulationRats WistarMicroscopy VideoPhotolysisbusiness.industryChemistryPyramidal CellsGlutamate receptorEquipment DesignSomatosensory CortexBarrel cortexAtomic and Molecular Physics and OpticsRatsElectronic Optical and Magnetic Materialsmedicine.anatomical_structureLens (anatomy)SynapsesBiophysicsbusinessJournal of Biomedical Optics
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Sporadic Parkinson disease and amyotrophic lateral sclerosis complex (Brait-Fahn-Schwartz disease).

2012

Clinical evidence for parkinsonism may accompany Amyotrophic Lateral Sclerosis with a frequency ranging from 5% to 17%. The concurrence of Amyotrophic Lateral Sclerosis and Parkinson's disease, outside the known Guam and Kii Peninsula foci, is instead rare, but this raises the possibility of a common pathogenesis. Clinically this complex presents with a levodopa-responsive parkinsonism and Amyotrophic Lateral Sclerosis and has been termed Brait-Fahn-Schwartz disease. Here we describe two patients with this uncommon neurodegenerative complex. Both presented with Parkinson disease and progressed to a full blown Amyotrophic Lateral Sclerosis. We further suggest that the association of Parkinso…

MalePathologymedicine.medical_specialtyExtrapyramidal signsbusiness.industryParkinsonismAmyotrophic Lateral SclerosisParkinson DiseaseDiseaseMiddle Agedmedicine.diseasenervous system diseasesPathogenesisNeurologyKii peninsulaClinical evidencemedicineHumansFemaleNeurology (clinical)Sporadic Parkinson diseaseAmyotrophic lateral sclerosisbusinessAgedJournal of the neurological sciences
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Stage-dependent and sector-specific neuronal loss in hippocampus during Alzheimer's disease

2000

Recent stereological studies documented a severe loss of hippocampal neurons in end-stage Alzheimer's disease. The development of the disease, however, is progressive and slow, over clinically inconspicuous decades. The Braak-staging system distinguishes six histopathological stages some of which are not accompanied by clinical symptoms. We analyzed hippocampal cell loss in correlation to Braak stages. Neuron numbers were determined with unbiased stereological principles in a defined subportion of the hippocampus of 28 subjects. There were no age-dependent neuronal losses in any of the hippocampal subdivisions examined. Compared to stage I, pyramidal cell loss in CA1 was reduced by 33% in s…

MalePathologymedicine.medical_specialtyHippocampusCell CountStereologyHippocampal formationBiologyHippocampusPathology and Forensic MedicineCentral nervous system diseaseCellular and Molecular NeuroscienceAlzheimer DiseasemedicineHumansAgedAged 80 and overNeuronsSubiculumMiddle Agedmedicine.diseasemedicine.anatomical_structurenervous systemFemaleNeurology (clinical)NeuronPyramidal cellAlzheimer's diseaseActa Neuropathologica
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Clinical and neuroradiological findings in classic infantile and late-onset globoid-cell leukodystrophy (Krabbe disease)

1996

In the present study the clinical course and imaging of early and late-onset forms of Krabbe disease are analyzed. We report on 11 patients with a biochemical diagnosis of galactosyl ceramide β-galactoside deficiency. Two presented as the classic infantile form and died within the second year of life. In 9 children the first clinical signs, such as gait difficulties and visual failure, started after age 2 years. All these patients developed slow regression of motor and mental capacities, and most of them died within their first decade. In patients of both groups computed tomography (CT) and magnetic resonance imaging (MRI) were performed. In the late-onset form, hypodensities of the central…

MalePediatricsmedicine.medical_specialtyGalactosylceramidesDiseaseWhite matterCentral nervous system diseasemedicineHumansAge of OnsetGenetics (clinical)Pyramidal tractsmedicine.diagnostic_testbusiness.industryBrainInfantMagnetic resonance imagingmedicine.diseaseMagnetic Resonance ImagingLeukodystrophy Globoid Cellmedicine.anatomical_structureEl NiñoSpinal CordChild PreschoolKrabbe diseaseFemaleAge of onsetbusinessTomography X-Ray Computed
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Effects of caffeine on neuromuscular function in a non‐fatigued state and during fatiguing exercise

2020

New findings What is the central question of the study? What are the effects of caffeine on neuromuscular function in a non-fatigued state and during fatiguing exercise? What is the main finding and its importance? In a non-fatigued state, caffeine decreased the duration of the silent period evoked by transcranial magnetic stimulation. Caffeine-induced reduction of inhibitory mechanisms in the central nervous system before exercise was associated with an increased performance. Individuals who benefit from caffeine ingestion may experience lower perception of effort during exercise and an accelerated recovery of M-wave amplitude postfatigue. This study elucidates the mechanisms of action of …

MalePhysiologyväsymysmedicine.medical_treatmentliikunta030204 cardiovascular system & hematologyRC1200H-Reflexchemistry.chemical_compound0302 clinical medicineTriceps surae muscletranscranial magnetic stimulationMedicinerasitusMotor NeuronsNutrition and DieteticsMotor CortexGeneral MedicineTranscranial Magnetic Stimulationhermo-lihastoimintamedicine.anatomical_structureNeuromuscular AgentsMuscle FatigueCaffeineMuscle ContractionAdultmedicine.medical_specialtyQP301.H75_Physiology._Sport.Postureperipheral fatiguePlacebo03 medical and health sciencesPhysical medicine and rehabilitationDouble-Blind MethodCaffeinePhysiology (medical)Humansrate of perceived exertionExercisesoleussuorituskykySoleus musclePyramidal tractsbusiness.industryEvoked Potentials MotorQPCrossover studycentral fatigueTranscranial magnetic stimulationchemistrykofeiiniSilent periodbusiness030217 neurology & neurosurgeryExperimental Physiology
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Switching between persistent firing and depolarization block in individual rat CA1 pyramidal neurons

2018

The hippocampal formation plays a role in mnemonic tasks and epileptic discharges in vivo. In vitro, these functions and malfunctions may relate to persistent firing (PF) and depolarization block (DB), respectively. Pyramidal neurons of the CA1 field have previously been reported to engage in either PF or DB during cholinergic stimulation. However, it is unknown whether these cells constitute disparate populations of neurons. Furthermore, it is unclear which cell-specific peculiarities may mediate their diverse response properties. However, it has not been shown whether individual CA1 pyramidal neurons can switch between PF and DB states. Here, we used whole cell patch clamp in the current …

MalePotassium ChannelsPatch-Clamp Techniquesantagonists & inhibitors [TRPC Cation Channels]physiology [Electrophysiological Phenomena]Cognitive Neurosciencepharmacology [Muscarinic Agonists]metabolism [TRPC Cation Channels]drug effects [Pyramidal Cells]HippocampusStimulationMuscarinic AgonistsIn Vitro TechniquesHippocampal formation050105 experimental psychologyMembrane Potentialspharmacology [Carbachol]03 medical and health sciences0302 clinical medicineCurrent clampAnimalsRats Long-Evans0501 psychology and cognitive sciencesddc:610Patch clampCA1 Region HippocampalTRPC Cation Channelsphysiology [CA1 Region Hippocampal]Dose-Response Relationship Drugphysiology [Pyramidal Cells]ChemistryPyramidal Cells05 social sciencescytology [CA1 Region Hippocampal]drug effects [Membrane Potentials]Depolarizationmetabolism [Potassium Channels]drug effects [Electrophysiological Phenomena]Potassium channelElectrophysiological PhenomenaRatsdrug effects [CA1 Region Hippocampal]CholinergicCarbacholFemaleNeuroscience030217 neurology & neurosurgeryHippocampus
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Intrinsically determined cell death of developing cortical interneurons.

2009

The cell death of inhibitory neurons, which originate far from the cortical areas to which they migrate during embryonic development, is determined autonomously rather than by competition for trophic signals from other cell types. It has long been known that apoptosis, a form of programmed cell death, eliminates young cells from developing tissues. In the field of neurobiology, it is widely believed that developmental neuronal-cell death results from cellular competition for environmentally derived survival signals that selects for an optimally sized and properly wired population of neurons. This study of developmental cell death in the mouse cortex in vivo, in vitro and after transplantati…

MaleProgrammed cell deathInterneurongenetic structuresCell SurvivalPopulationApoptosisCell CountNeocortexBiologyArticle03 medical and health sciencesMice0302 clinical medicineNeural Stem CellsInterneuronsmedicineAnimalseducationCellular Senescence030304 developmental biologybcl-2-Associated X Protein0303 health scienceseducation.field_of_studyMultidisciplinaryNeocortexMembrane GlycoproteinsCaspase 3musculoskeletal neural and ocular physiologyPyramidal CellsfungiProtein-Tyrosine KinasesCell biologyTransplantationMice Inbred C57BLmedicine.anatomical_structurenervous systemAnimals NewbornInhibitory Postsynaptic PotentialsCerebral cortexbiology.proteinFemaleCell aging030217 neurology & neurosurgeryNeurotrophinNature
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Overlap between emotional blunting, depression, and extrapyramidal symptoms in schizophrenia

2002

MalePsychiatric Status Rating Scalesmedicine.medical_specialtybusiness.industryEmotional bluntingMiddle Agedmedicine.diseasePsychiatry and Mental healthBasal Ganglia DiseasesExtrapyramidal symptomsSchizophreniaGermanySchizophreniaHumansMedicineFemaleSchizophrenic Psychologymedicine.symptombusinessPsychiatryBiological PsychiatryDepression (differential diagnoses)Schizophrenia Research
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