Search results for "Pyridine"

showing 10 items of 2516 documents

Kinetic experiments on the synergistic effect of NADH on microsomal drug oxidation.

1974

Abstract1. The synergistic effect of NADH on the NADPH-dependent mixed function oxidation of p-nitroanisole and hexobarbital can be measured both photometrically and by following the substrate-induced oxygen consumption. The increase in reaction rate is about 50% and lasts as long as NADH is present in the microsomal suspension.2. The oxidation of added NADH is increased by hexobarbital, ethylmorphine and SKF 525-A. Lineweaver-Burk transformation of the NADH oxidation rates yields straight lines for xenobiotic substrates suggesting Michaelis constants similar to those obtained from metabolic experiments. NADH oxidation in the absence of NADPH is about half as rapid as in its presence.3. Som…

MalePyridinesHealth Toxicology and MutagenesisCyanidechemistry.chemical_elementHexobarbitalAnisolesToxicologyPhotochemistryBiochemistryOxygenMixed Function Oxygenaseschemistry.chemical_compoundOxygen ConsumptionPhenolsmedicineOrganic chemistryAnimalsPharmacologyCarbon MonoxideAniline CompoundsChemistryProadifenDrug SynergismGeneral MedicineMetyraponeEthylmorphineNADNitro CompoundsRatsHexobarbitalPharmaceutical PreparationsSpectrophotometryReagentPhenobarbitalMicrosomeMicrosomes LiverXenobioticChloromercuribenzoatesOxidation-Reductionmedicine.drugCarbon monoxideXenobiotica; the fate of foreign compounds in biological systems
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Indicaxanthin from Opuntia ficus-indica Crosses the Blood–Brain Barrier and Modulates Neuronal Bioelectric Activity in Rat Hippocampus at Dietary-Con…

2015

Indicaxanthin is a bioactive and bioavailable betalain pigment from the Opuntia ficus-indica fruits. In this in vivo study, kinetic measurements showed that indicaxanthin is revealed in the rat brain within 1 h from oral administration of 2 μmol/ kg, an amount compatible with a dietary consumption of cactus pear fruits in humans. A peak (20 ± 2.4 ng of indicaxanthin per whole brain) was measured after 2.5 h; thereafter the molecule disappeared with first order kinetics within 4 h. The potential of indicaxanthin to affect neural activities was in vivo investigated by a microiontophoretic approach. Indicaxanthin, administered in a range between 0.085 ng and 0.34 ng per neuron, dose-dependentl…

MalePyridinesHippocampusPharmacologyBiologyHippocampal formationBlood–brain barrierInhibitory postsynaptic potentialHippocampuschemistry.chemical_compoundSettore BIO/10 - BiochimicamedicineAnimalsRats WistarNeuronsGlutamate receptorOpuntiaGeneral Chemistryindicaxanthin phytochemicals BBB electrophysiology hippocampus microiontophoresis molecular modelingBetaxanthinsElectrophysiologymedicine.anatomical_structureReceptors GlutamateBiochemistrychemistryBlood-Brain BarrierNMDA receptorNeuronGeneral Agricultural and Biological SciencesIndicaxanthinJournal of Agricultural and Food Chemistry
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The hMTH1 paradox: antioxidants recommended in cancer?

2014

Summary Activated Ras GTPase signalling is a critical driver of oncogenic transformation and malignant disease. Cellular models of RAS-dependent cancers have been used to identify experimental small-molecules, such as SCH51344, but their molecular mechanism of action remains generally enigmatic. Here, using a chemical proteomic approach we identify the target of SCH51344 as the human mutT homologue MTH1, a nucleotide pool sanitising enzyme. Loss-of-function of MTH1 impaired growth of KRAS tumour cells whereas MTH1 overexpression mitigated sensitivity toward SCH51344. Searching for more drug-like inhibitors, we identified the kinase inhibitor crizotinib as a nanomolar suppressor of MTH1 acti…

MalePyridinesMEDLINEDNA repairAntineoplastic AgentsAntioxidantesSaludBiologyBioinformaticsstereoselectivityBiochemistryArticleText miningNeoplasmsmedicineAnimalsHumanscancerMolecular BiologyProtein Kinase Inhibitorscrizotinibbusiness.industryNucleotidesCancerdrugCell BiologyCáncermedicine.diseasePhosphoric Monoester HydrolasesMTH1DNA Repair EnzymesPyrazolesFemalebusiness
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NEPA (netupitant/palonosetron) for the antiemetic prophylaxis of nausea and vomiting induced by chemotherapy (CINV) with Folfirinox and Folfoxiri eve…

2021

OBJECTIVE: The outbreak of coronavirus disease 2019 (COVID-19) has affected the treatment of cancer patients, with particular regard to the management of both chemotherapy and side effects. Chemotherapy-induced nausea and vomiting (CINV) are amongst the most troublesome side effects that impair patients’ adherence to treatments and their quality of life (QoL). NEPA (Akynzeo®), is an oral fixed-dose combination of netupitant [a neurokinin-1 receptor antagonist (NK1RA), 300 mg] and palonosetron [(5-hydroxytryptamine (serotonin or 5HT) type3 receptor antagonist (5HT3RA), 0.5 mg] which has been shown to be effective in preventing CINV. PATIENTS AND METHODS: This prospective study started before…

MalePyridinesVomitingCINVLeucovorinCOVID-19NauseaMiddle AgedIrinotecanColorectal cancerBevacizumabOxaliplatinPalonosetronNetupitantAntineoplastic Combined Chemotherapy ProtocolsAntiemeticsHumansEmetogenic chemotherapyFemaleFluorouracilProspective StudiesColorectal NeoplasmsPandemicsAdvanced pancreatic cancerAgedEuropean review for medical and pharmacological sciences
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Vorapaxar in the secondary prevention of atherothrombotic events

2012

BACKGROUND: Thrombin potently activates platelets through the protease-activated receptor PAR-1. Vorapaxar is a novel antiplatelet agent that selectively inhibits the cellular actions of thrombin through antagonism of PAR-1. METHODS: We randomly assigned 26,449 patients who had a history of myocardial infarction, ischemic stroke, or peripheral arterial disease to receive vorapaxar (2.5 mg daily) or matching placebo and followed them for a median of 30 months. The primary efficacy end point was the composite of death from cardiovascular causes, myocardial infarction, or stroke. After 2 years, the data and safety monitoring board recommended discontinuation of the study treatment in patients …

MalePyridines[SDV]Life Sciences [q-bio]Myocardial InfarctionMedizinKaplan-Meier Estimate030204 cardiovascular system & hematologyBrain IschemiaLactones0302 clinical medicineMESH: Peripheral Arterial DiseaseSecondary PreventionMESH: Double-Blind Method030212 general & internal medicineMyocardial infarctionStrokeVorapaxarMESH: AgedAspirinMESH: Middle AgedMESH: RiskCardiovascular diseases [NCEBP 14]MESH: Secondary PreventionHazard ratioMESH: Brain IschemiaGeneral MedicineMiddle AgedClopidogrel3. Good healthStrokeMESH: Receptor PAR-1MESH: Myocardial Infarctionvorapaxar secondary prevention atherothrombotic eventsCardiovascular DiseasesMESH: Platelet Aggregation InhibitorsAnesthesiaRetreatmentPlatelet aggregation inhibitorFemaleIntracranial HemorrhagesMESH: HemorrhageMESH: Intracranial HemorrhagesMESH: Lactonescirculatory and respiratory physiologymedicine.drugRiskISQUEMIA CEREBRALHemorrhagePlaceboMESH: StrokePeripheral Arterial Disease03 medical and health sciencesDouble-Blind Method[INFO.INFO-IM]Computer Science [cs]/Medical ImagingmedicineHumansReceptor PAR-1MESH: RetreatmentMESH: Kaplan-Meier EstimateAgedMESH: Humansbusiness.industryMESH: PyridinesMESH: Cardiovascular Diseasesmedicine.diseaseSettore MED/11 - Malattie Dell'Apparato CardiovascolareMESH: MalebusinessMESH: FemalePlatelet Aggregation Inhibitors
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Inhibition of the mechanical activity of mouse ileum by cactus pear (Opuntia Ficus Indica, L, Mill.) fruit extract and its pigment indicaxanthin.

2010

We investigated, using an organ bath technique, the effects of a hydrophilic extract from Opuntia ficus indica fruit pulp (cactus fruit extract, CFE) on the motility of mouse ileum, and researched the extract component(s) responsible for the observed responses. CFE (10-320 mg of fresh fruit pulp equivalents/mL of organ bath) reduced dose-dependently the spontaneous contractions. This effect was unaffected by tetrodotoxin, a neuronal blocker, N(omega)-nitro-l-arginine methyl ester, a nitric oxide synthase blocker, tetraethylammonium, a potassium channel blocker, or atropine, a muscarinic receptor antagonist. CFE also reduced the contractions evoked by carbachol, without affecting the contrac…

MalePyridineschemistry.chemical_elementindicaxanthinPharmacologyBiologyCalciumintestinal smooth musclechemistry.chemical_compoundMiceIleumBotanymedicineAnimalsTetraethylammoniumVoltage-dependent calcium channelPlant ExtractsOpuntiaPotassium channel blockerantispasmodic effectGeneral ChemistryAscorbic acidPotassium channelBetaxanthinsMice Inbred C57BLchemistryFruitopuntia ficus indicaAntispasmodicGeneral Agricultural and Biological SciencesGastrointestinal MotilityIndicaxanthinmedicine.drugJournal of agricultural and food chemistry
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Effects of CNQX and MPEP on sensitization to the rewarding effects of morphine

2011

The present study was conducted to evaluate the influence of the glutamatergic receptors α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) and metabotropic glutamate 5 (mGlu5) receptors on sensitization to the rewarding effects of morphine. The effects of pre-treatment with saline or 20 mg/kg morphine plus the AMPA receptor antagonist 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX) (5 or 10 mg/kg) or the metabotropic Glu5 receptor antagonist 6-methyl-2-(phenylethynyl)-pyridine (MPEP) (5 or 10 mg/kg) on the place conditioning induced by a low dose of morphine (2 mg/kg) were assessed. The 2 mg/kg dose of morphine was ineffective in animals pre-treated with saline but induced a clear con…

MalePyridinesmedicine.drug_classReceptor Metabotropic Glutamate 5AMPA receptorPharmacologyReceptors Metabotropic GlutamateMicechemistry.chemical_compoundRewardmedicineAnimalsReceptors AMPASensitization6-Cyano-7-nitroquinoxaline-23-dionePharmacologyDose-Response Relationship DrugMorphineAntagonistReceptor antagonistConditioned place preferencemedicine.anatomical_structureMetabotropic receptorchemistryAnesthesiaCNQXMorphineConditioning OperantExcitatory Amino Acid Antagonistsmedicine.drugEuropean Journal of Pharmacology
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Preliminary experience on safety of regorafenib after sorafenib failure in recurrent hepatocellular carcinoma after liver transplantation

2019

Regorafenib is one option for second-line treatment of hepatocellular carcinoma (HCC), improving overall survival (OS) of sorafenib-tolerant patients who develop progression. We aim to evaluate the safety and outcomes of regorafenib as second-line treatment for HCC recurrence after liver transplantation (LT). This is a retrospective, multicenter, international study including regorafenib-treated LT patients (2015-2018), with analysis of baseline characteristics and evolutionary events during sorafenib/regorafenib treatment. Twenty-eight LT patients (57 years, 7% cirrhotics, 54% performance status 1) were included. Median time from LT to regorafenib initiation was 3.9 (1.1-18.5) years; media…

MalePyridinesmedicine.medical_treatment030230 surgeryLiver transplantationchemotherapyGastroenterologychemistry.chemical_compound0302 clinical medicineAntineoplastic Combined Chemotherapy ProtocolsImmunology and AllergyPharmacology (medical)Liver NeoplasmsMiddle AgedSorafenibPrognosisRecurrent Hepatocellular Carcinomaside effectsHepatocellular carcinomaFemalemedicine.drugSorafenibAdultmedicine.medical_specialtyCarcinoma Hepatocellularcancer/malignancy/neoplasiaclinical research/practice03 medical and health sciencesYoung AdultInternal medicineRegorafenibmedicineHumansAdverse effectAgedRetrospective StudiesTransplantationdrug interactionPerformance statusbusiness.industryPhenylurea Compoundsmedicine.diseaseDiscontinuationLiver TransplantationchemistryDrug Resistance NeoplasmNeoplasm Recurrence Localpharmacologybusinessliver transplantation/hepatologyFollow-Up Studies
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Indicaxanthin from cactus pear fruit exerts anti-inflammatory effects in carrageenin-induced rat pleurisy.

2014

Nutritional research has shifted recently from alleviating nutrient deficiencies to chronic disease prevention. We investigated the activity of indicaxanthin, a bioavailable phytochemical of the betalain class from the edible fruit of Opuntia ficus-indica (L. Miller) in a rat model of acute inflammation. Rat pleurisy was achieved by injection of 0.2 mL of l-carrageenin in the pleural cavity, and rats were killed 4, 24, and 48 h later; exudates were collected to analyze inflammatory parameters, such as nitric oxide (NO), prostaglandin E2 (PGE2), interleukin-1b (IL-1b), and tumor necrosis factor-a (TNF-a); cells recruited in pleura were analyzed for cyclooxygenase-2 (COX-2), inducible nitric …

MalePyridinesmedicine.medical_treatmentPyridineAnti-Inflammatory AgentsMedicine (miscellaneous)PharmacologyCarrageenanchemistry.chemical_compoundLeukocytesInflammation MediatorPleural CavityNutrition and DieteticsbiologyOpuntiaBetaxanthinsNitric oxide synthaseAnti-Inflammatory AgentTumor necrosis factor alphamedicine.symptomInflammation MediatorsIndicaxanthinProstaglandin Emedicine.drug_classInflammationAnti-inflammatoryNitric oxidePlant ExtractmedicineAnimalsRNA MessengerRats WistarPleurisyInflammationIndicaxanthin phytochemicals inflammation pleurisy redox state.business.industryAnimalPlant ExtractsLeukocyteCarrageenanRatsDisease Models AnimalchemistryBetaxanthinFruitImmunologybiology.proteinRatbusinessPhytotherapyThe Journal of nutrition
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Selective action of two aporphines at alpha 1-adrenoceptors and potential-operated Ca2+ channels.

1993

Abstract Contractions evoked by noradrenaline (1 μM) or a depolarizing solution of 60 mM KCI were concentration dependently depressed by the aporphine alkaloids (S)-boldine and (R)-apomorphine in rataorta. Both drugs had a greater inhibitory potency on the contraction elicited by noradrenaline. Dose-response curves for noradrenaline were shifted to the right in presence of (S)-boldine. (R)-Apomorphine acted by a complex mechanism at α 1 -adrenoceptors and its inhibitory effects was irreversible. The conformational features of these alkaloids may explain their different behaviour at α 1 -adrenoceptors. In Ca 2+ -free solution, the alkaloids inhibited the contraction evoked by noradrenaline b…

MaleReceptor complexAporphinesApomorphineStereochemistryPhosphodiesterase InhibitorsMolecular ConformationIn Vitro TechniquesMuscle Smooth VascularPotassium Chloridechemistry.chemical_compoundNorepinephrineRadioligand AssaymedicinePrazosinBoldineAnimalsAporphineRats WistarEvoked PotentialsPharmacologyMembranesAlkaloidDihydropyridinePhosphodiesteraseReceptors Adrenergic alphaRatsAntitussive AgentsMechanism of actionchemistryCattleCalcium Channelsmedicine.symptommedicine.drugMuscle ContractionEuropean journal of pharmacology
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