Search results for "Pyrimidine"

showing 10 items of 589 documents

Communication: Electronic UV-Vis transient spectra of the ·OH reaction products of uracil, thymine, cytosine, and 5,6-dihydrouracil by using the comp…

2013

Addition of ∙OH radicals to pyrimidine nucleobases is a common reaction in DNA/RNA damage by reactive oxygen species. Among several experimental techniques, transient absorption spectroscopy has been during the last decades used to characterize such compounds. Discrepancies have however appeared in the assignment of the adduct or adducts responsible for the reported transient absorption UV-Vis spectra. In order to get an accurate assignment of the transient spectra and a unified description of the absorption properties of the ∙OH reaction products of pyrimidines, a systematic complete active space self-consistent field second-order perturbation (CASPT2//CASSCF) theory study has been carried…

Molecular StructurePyrimidineHydroxyl RadicalGeneral Physics and AstronomyDihydrouracilElectronsUracilHydrogen atom abstractionPhotochemistryThymineAdductNucleobaseCytosinechemistry.chemical_compoundchemistryComputational chemistryQuantum TheorySpectrophotometry UltravioletPhysical and Theoretical ChemistryUracilThymineCytosineThe Journal of Chemical Physics
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Photoinduced DNA Lesions in Dormant Bacteria: The Peculiar Route Leading to Spore Photoproducts Characterized by Multiscale Molecular Dynamics

2020

International audience; Some bacterial species enter a dormant state in the form of spores to resist to unfavorable external conditions. Spores are resistant to a wide series of stress agents, including UV radiation, and can last for tens to hundreds of years. Due to the suspension of biological functions, such as DNA repair, they accumulate DNA damage upon exposure to UV radiation. Differently from active organisms, the most common DNA photoproducts in spores are not cyclobutane pyrimidine dimers, but rather the so‐called spore photoproducts. This noncanonical photochemistry results from the dry state of DNA and its binding to small, acid‐soluble proteins that drastically modify the struct…

Molecular modelDNA repairDNA damageUltraviolet RaysPyrimidine dimerMolecular Dynamics Simulation010402 general chemistry01 natural sciencesMolecular mechanicsCatalysischemistry.chemical_compound[SDV.BBM.BC]Life Sciences [q-bio]/Biochemistry Molecular Biology/Biochemistry [q-bio.BM]Spores Bacterial010405 organic chemistryChemistryOrganic ChemistryfungiGeneral ChemistryDNA0104 chemical sciencesSporePyrimidine DimersBiophysicsNucleic acidDNADNA Damage
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2-(2,6-Dihalophenyl)-3-(pyrimidin-2-yl)-1,3-thiazolidin-4-ones as non-nucleoside HIV-1 reverse transcriptase inhibitors.

2004

Several 1,3-thiazolidin-4-ones bearing a 2,6-dihalophenyl group at C-2 and a substituted pyrimidin-2-yl ring at the N-3 were synthesised and evaluated as anti-HIV agents. The results of the in vitro tests showed that some of them were highly effective inhibitors of human immunodeficiency virus type-1 (HIV-1) replication at 10–40 nM concentrations with minimal cytotoxicity. Structure–activity relationship studies revealed that the nature of the substituents at the 2 and 3 positions of the thiazolidinone nucleus had a significant impact on the in vitro anti-HIV activity of this class of potent antiretroviral agents. The compounds had significantly reduced activity against the characteristic N…

NNRTI3-Thiazolidin-4-onesAnti-HIV activity13-Thiazolidin-4-oneNNRTIs; 1; 3-Thiazolidin-4-ones; anti-HIVAnti-HIV Agents1Drug Evaluation PreclinicalMutation MissenseBiologyVirus ReplicationVirusStructure-Activity RelationshipVirologyDrug Resistance ViralmedicineStructure–activity relationshipCytotoxicityPharmacologyReverse-transcriptase inhibitorMolecular Structurevirus diseasesanti-HIVSettore CHIM/08 - Chimica FarmaceuticaMolecular biologyIn vitroReverse transcriptaseThiazolesPyrimidinesViral replicationAmino Acid SubstitutionNNRTIsHIV-1Reverse Transcriptase InhibitorsNucleosidemedicine.drugAntiviral research
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The azide/tetrazole equilibrium: an investigation in the series of furo- and thieno[2,3-e]tetrazolo[3,2-d]pyrimidine derivatives

2016

Abstract Starting from 7(8)-chlorofuro(thieno)[3,2- d ]pyrimidines 1 , the corresponding 7(8)-hydrazinofuro(thieno)[3,2- d ]pyrimidines 2 were obtained by reaction with hydrazine. By treatment with nitrous acid, compounds 2 gave fused furo- and thieno[2,3- e ]tetrazolo[1,5- c ]pyrimidines 3 T and 4 T in excellent yields, which in solution exist in equilibrium with 7(8)-azidofuro(thieno)[3,2- d ]pyrimidines 3 A and 4 A . Compounds 3 T / 3 A and 4 T / 4 A represent useful substrates for investigating the azide/tetrazole equilibrium: it has been examined as a function of solvent, temperature, and their structure. We have observed that, in solution, in both 3 and 4 the equilibrium was strongly …

Nitrous acidPyrimidine010405 organic chemistryStereochemistryOrganic Chemistry010402 general chemistry01 natural sciencesBiochemistryMedicinal chemistryTautomer0104 chemical sciencesSolventchemistry.chemical_compoundchemistryDrug DiscoveryTetrazoleAzideSolvent effectsEquilibrium constantTetrahedron
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Synthesis, characterization, and in vitro antimicrobial activity of organotin(IV) complexes with triazolo-pyrimidine ligands containing exocyclic oxy…

2005

Abstract Tri-organotin(IV) complexes of the triazolo-pyrimidine derivatives 4,5-dihydro-5-oxo-[1,2,4]triazolo-[1,5 a ]pyrimidine (5HtpO), 4,7-dihydro-5-methyl-7-oxo-[1,2,4]triazolo-[1,5 a ]pyrimidine (HmtpO), and 4,5,6,7-tetrahydro-5,7-dioxo-[1,2,4]triazolo-[1,5 a ]pyrimidine (H 2 tpO 2 ), and the diorganotin derivative n -Bu 2 Sn(tpO 2 ), were synthesized and characterized by means of infrared and 119 Sn Mossbauer spectroscopy. In all the complexes obtained the triazolopyrimidines act as multidentate ligands producing polymeric structures. A trigonal bipyramidal arrangement of the ligands around the tin atom is proposed for triorganotin(IV) derivatives, with organic groups on the equatoria…

ONIOMDenticityPyrimidineStereochemistryLigandOrganic ChemistryDFT calculationTriazolopyrimidine; Organotin(IV); Mossbauer; DFT calculations; Antimicrobial activityOrganotin(IV)Antimicrobial activityBiochemistryMedicinal chemistryMossbauerInorganic Chemistrychemistry.chemical_compoundTrigonal bipyramidal molecular geometrychemistryMössbauer spectroscopyOctahedral molecular geometryMaterials ChemistryTriazolopyrimidineChelationPhysical and Theoretical ChemistryJournal of Organometallic Chemistry
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Functional polymorphisms in SOCS1 and PTPN22 genes correlate with the response to imatinib treatment in newly diagnosed chronic-phase chronic myeloid…

2011

a b s t r a c t The function of the natural modulators of BCR-ABL-induced signaling pathways could influence the results to imatinib treatment. We assessed the association between single nucleotide polymorphisms (SNPs) on genes of the phosphatase family and the suppressors of cytokine signaling and the response to imatinib in 105 patients newly diagnosed with chronic-phase CML. SNPs in SOCS1 (rs243327) and PTPN22 (rs2476601) genes correlated with the risk of primary resistance to imatinib. A high-risk Sokal score, the T allele in PTPN22 SNP, and each copy of the C allele in SOCS1 SNP were adverse prognostic factors for failure-free survival (FFS). Based on such parameters, three risk groups…

OncologyAdultMaleCancer Researchmedicine.medical_specialtyAdolescentGenotypeSingle-nucleotide polymorphismAntineoplastic AgentsSuppressor of Cytokine Signaling ProteinsBiologyReal-Time Polymerase Chain ReactionPolymorphism Single NucleotidePiperazinesPTPN22Young AdultSuppressor of Cytokine Signaling 1 Proteinhemic and lymphatic diseasesInternal medicineGenotypemedicineSNPHumansAlleleAgedSuppressor of cytokine signaling 1ImatinibProtein Tyrosine Phosphatase Non-Receptor Type 22HematologyDNAMiddle AgedPrognosisPyrimidinesOncologyCase-Control StudiesImmunologyBenzamidesLeukemia Myeloid Chronic-PhaseImatinib MesylateFemaleSokal Scoremedicine.drugLeukemia research
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Health-related quality of life data from a phase 3, international, randomized, open-label, multicenter study in patients with previously treated mant…

2017

Mantle cell lymphoma (MCL) is a rare, aggressive, incurable B-cell malignancy. Ibrutinib has been shown to be highly active for patients with relapsed/refractory (R/R) MCL. The RAY trial (MCL3001) was a phase 3, randomized, open-label, multicenter study that compared ibrutinib with temsirolimus in patients with R/R MCL. Active disease is frequently associated with impaired functional status and reduced well-being. Therefore, the current study employed two patient-reported outcome instruments, the Functional Assessment of Cancer Therapy-Lymphoma (FACT-Lym) and the EQ-5D-5L, to assess symptoms, well-being, health status, and health-related quality of life of patients on treatment within the R…

OncologyAdultMaleCancer Researchmedicine.medical_specialtyLymphoma Mantle-CellMalignancy03 medical and health scienceschemistry.chemical_compound0302 clinical medicineRefractoryQuality of lifePiperidinesInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumans030212 general & internal medicineDisease burdenAgedNeoplasm StagingAged 80 and overSirolimusbusiness.industryAdenineCancerHematologyMiddle Agedmedicine.diseaseTemsirolimusSurgeryPyrimidinesTreatment OutcomeOncologychemistryDrug Resistance Neoplasm030220 oncology & carcinogenesisIbrutinibRetreatmentQuality of LifePyrazolesMantle cell lymphomaFemalebusinessmedicine.drug
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Imatinib combined with mitoxantrone/etoposide and cytarabine is an effective induction therapy for patients with chronic myeloid leukemia in myeloid …

2007

BACKGROUND Despite advances in drug therapy and allogeneic stem cell transplantation (allo-SCT), the prognosis of patients with chronic myeloid leukemia (CML) in blast crisis remains poor. Imatinib has demonstrated synergistic effects in vitro with mitoxantrone, etoposide, and cytarabine. METHODS A Phase I/II trial was performed in patients with CML myeloid blast crisis. Patients were treated with imatinib + mitoxantrone/etoposide in four cohorts: mitoxantrone 10 mg/m2/day and etoposide 100 mg/m2/day for 2 or 3 consecutive days and imatinib 600 mg/day from Day 15 (cohorts 1 and 2) or from Day 1 (cohorts 3 and 4). After hematologic reconstitution after the cytopenic phase, cytarabine was giv…

OncologyAdultMaleCancer Researchmedicine.medical_specialtymedicine.medical_treatmentPharmacologyPiperazineshemic and lymphatic diseasesInternal medicineLeukemia Myelogenous Chronic BCR-ABL PositiveAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansEtoposideAgedEtoposideMitoxantroneChemotherapybusiness.industryCytarabineMyeloid leukemiaImatinibMiddle AgedSurvival AnalysisTransplantationImatinib mesylatePyrimidinesTreatment OutcomeOncologyBenzamidesCytarabineImatinib MesylateFemaleMitoxantronebusinessBlast Crisismedicine.drugCancer
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Imatinib compared with interferon and low-dose cytarabine for newly diagnosed chronic-phase chronic myeloid leukemia.

2003

Imatinib, a selective inhibitor of the BCR-ABL tyrosine kinase, produces high response rates in patients with chronic-phase chronic myeloid leukemia (CML) who have had no response to interferon alfa. We compared the efficacy of imatinib with that of interferon alfa combined with low-dose cytarabine in newly diagnosed chronic-phase CML.We randomly assigned 1106 patients to receive imatinib (553 patients) or interferon alfa plus low-dose cytarabine (553 patients). Crossover to the alternative group was allowed if stringent criteria defining treatment failure or intolerance were met. Patients were evaluated for hematologic and cytogenetic responses, toxic effects, and rates of progression.Afte…

OncologyAdultMalemedicine.medical_specialtyAdolescentAlpha interferonAntineoplastic AgentsPiperazineschemistry.chemical_compoundhemic and lymphatic diseasesInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansProspective StudiesInterferon alfaAgedbusiness.industryPonatinibCytarabineInterferon-alphaImatinibGeneral MedicineMiddle AgedDasatinibSurvival RateImatinib mesylatePyrimidineschemistryNilotinibImmunologyBenzamidesLeukemia Myeloid Chronic-PhaseCytarabineDisease ProgressionImatinib MesylateFemalebusinessmedicine.drugThe New England journal of medicine
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Beyond the comfort zone of deep molecular response: discontinuation in major molecular response chronic myeloid leukemia.

2019

Discontinuation of tyrosine kinase inhibitors (TKIs) therapy is now feasible for patients with chronic myeloid leukemia (CML) with deep and longstanding molecular response (MR 4/4.5); around 40–60%...

OncologyDrugAdultMaleCancer Researchmedicine.medical_specialtymedia_common.quotation_subjectAntineoplastic AgentsDisease-Free Survival03 medical and health sciencesMyelogenous0302 clinical medicinehemic and lymphatic diseasesInternal medicineLeukemia Myelogenous Chronic BCR-ABL PositivemedicineHumansProtein Kinase Inhibitorsmedia_commonWithholding TreatmentDose-Response Relationship Drugbusiness.industryMyeloid leukemiaHematologyProtein-Tyrosine Kinasesmedicine.diseaseDiscontinuationLeukemiaPyrimidinesOncologyWithholding Treatment030220 oncology & carcinogenesisMolecular ResponseImatinib MesylateFemalebusinessTyrosine kinase030215 immunologyFollow-Up StudiesLeukemialymphoma
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