Search results for "Pyroptosi"

showing 3 items of 13 documents

Routes to cell death in animal and plant kingdoms: from classic apoptosis to alternative ways to die—a review

2018

Programmed cell death is fundamental for multicellular organisms either in animal or plant kingdom. Classic apoptosis, which represents the best studied form of cell death, is dependent on caspase protease activity in animals. These proteases are not present in plants, where caspase-like activities, including metacaspases, are involved in the execution of plant cell death. Beyond apoptosis, various non-apoptotic forms of cell death also exist, including autophagy, necroptosis, pyroptosis, and ferroptosis. These types of cell death can be activated independently of apoptosis and sometimes occur when apoptosis is inhibited. Non-apoptotic forms of cell death are best characterized in animals, …

Necroptosi0301 basic medicineProteasesProgrammed cell deathNecroptosisCancer cellAnimal and plant cell death03 medical and health sciencesComparative death pathwaySettore BIO/10 - BiochimicaAutophagySettore BIO/04 - Fisiologia VegetaleCaspaseGeneral Environmental SciencebiologyAutophagyPyroptosiPyroptosisApoptosifood and beveragesCell biologyMulticellular organism030104 developmental biologyApoptosisbiology.proteinGeneral Earth and Planetary SciencesGeneral Agricultural and Biological SciencesFerroptosiRendiconti Lincei. Scienze Fisiche e Naturali
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Cellular Models and Assays to Study NLRP3 Inflammasome Biology

2020

The NLRP3 inflammasome is a multi-protein complex that initiates innate immunity responses when exposed to a wide range of stimuli, including pathogen-associated molecular patterns (PAMPs) and danger-associated molecular patterns (DAMPs). Inflammasome activation leads to the release of the pro-inflammatory cytokines interleukin (IL)-1β and IL-18 and to pyroptotic cell death. Over-activation of NLRP3 inflammasome has been associated with several chronic inflammatory diseases. A deep knowledge of NLRP3 inflammasome biology is required to better exploit its potential as therapeutic target and for the development of new selective drugs. To this purpose, in the past few years, several tools have…

Programmed cell death2019-20 coronavirus outbreakInflammasomesInterleukin-1betaReviewBiologyBiochemical assaysModels BiologicalCatalysisInflammasomelcsh:ChemistryInorganic ChemistryNLRP3NLR Family Pyrin Domain-Containing 3 ProteinPyroptosismedicineDeep knowledgeAlarminsAnimalsHumansPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopyInnate immune systemintegumentary systemCell modelsPathogen-Associated Molecular Pattern MoleculesOrganic ChemistryInterleukin-18InterleukinInflammasomeGeneral MedicineBiophysical assaysImmunity InnateComputer Science ApplicationsCell biologyNLRP3 inhibitorslcsh:Biology (General)lcsh:QD1-999Mechanism of actionRead-outsmedicine.symptomInflammasome complexSignal Transductionmedicine.drugInternational Journal of Molecular Sciences
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Bacterial and viral infections and related inflammatory responses in chronic obstructive pulmonary disease

2021

Abstract In chronic obstructive pulmonary disease (COPD) patients, bacterial and viral infections play a relevant role in worsening lung function and, therefore, favour disease progression. The inflammatory response to lung infections may become a specific indication of the bacterial and viral infections. We here review data on the bacterial–viral infections and related airways and lung parenchyma inflammation in stable and exacerbated COPD, focussing our attention on the prevalent molecular pathways in these different clinical conditions. The roles of macrophages, autophagy and NETosis are also briefly discussed in the context of lung infections in COPD. Controlling their combined response…

Review ArticleNK cells030204 cardiovascular system & hematologyAdaptive Immunitymedicine.disease_causeAutoimmunityPulmonary Disease Chronic Obstructive0302 clinical medicineNETosiPulmonary Medicine030212 general & internal medicineLungRespiratory Tract InfectionsT-lymphocytesCOPDB cellpyroptosisautoimmunityPyroptosisNETosisGeneral Medicinerespiratory systemAcquired immune systemmacrophagesmedicine.anatomical_structureautoimmunity; autophagy; B cells; dendritic cells; disability; ILCs; macrophages; NETosis; NK cells; outcome; pyroptosis; T-lymphocytesDisease Progressionoutcomemedicine.symptomSignal Transductionautophagydendritic cellILCsContext (language use)Inflammationmacrophage03 medical and health sciencesImmune systemmedicineHumansNK celldendritic cellsB cellsLungbusiness.industrymedicine.diseaseImmunity Innaterespiratory tract diseasespyroptosiILCdisabilityImmunologybusiness
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