Search results for "RAPA"

showing 10 items of 471 documents

Dietary protein restriction reduces circulating VLDL triglyceride levels via CREBH-APOA5-dependent and -independent mechanisms

2018

Hypertriglyceridemia is an independent risk factor for cardiovascular disease. Dietary interventions based on protein restriction (PR) reduce circulating triglycerides (TGs), but underlying mechanisms and clinical relevance remain unclear. Here, we show that 1 week of a protein-free diet without enforced calorie restriction significantly lowered circulating TGs in both lean and diet-induced obese mice. Mechanistically, the TG-lowering effect of PR was due, in part, to changes in very low-density lipoprotein (VLDL) metabolism both in liver and peripheral tissues. In the periphery, PR stimulated VLDL-TG consumption by increasing VLDL-bound APOA5 expression and promoting VLDL-TG hydrolysis and…

0301 basic medicineMalemedicine.medical_specialtyVery low-density lipoproteinDietary proteinFGF21Calorie restrictionmTORC1Lipoproteins VLDLMechanistic Target of Rapamycin Complex 1Protein Serine-Threonine Kinases03 medical and health sciencesMice0302 clinical medicineRisk FactorsInternal medicinemedicineDiet Protein-RestrictedIntegrated stress responseAnimalsHumansCyclic AMP Response Element-Binding ProteinTriglyceridesRandomized Controlled Trials as TopicHypertriglyceridemiaChemistryHydrolysisHypertriglyceridemianutritional and metabolic diseasesGeneral Medicinemedicine.diseaseLipid Metabolism030104 developmental biologyEndocrinologyApolipoproteinsHypotriglyceridemiaLiverApolipoprotein A-Vlipids (amino acids peptides and proteins)Female030217 neurology & neurosurgeryLipoproteinResearch Article
researchProduct

In vitro evaluation of a biomaterial-based anticancer drug delivery system as an alternative to conventional post-surgery bone cancer treatment

2018

Patients diagnosed with osteosarcoma are currently treated with intravenous injections of anticancer agents after tumor resection. However, due to remaining neoplastic cells at the site of tumor removal, cancer recurrence often occurs. Successful bone regeneration combined with the control of residual cancer cells presents a challenge for tissue engineering. Cyclodextrins loaded with chemotherapeutic drugs reversibly release the drugs over time. Hydroxyapatite bone biomaterials coated with doxorubicin-loaded cyclodextrin should release the drug with time after implantation directly at the original tumor site and may be a way to eliminate residual neoplastic cells. In the present study, we h…

0301 basic medicineMaterials scienceBone NeoplasmsBioengineeringBiomaterials03 medical and health sciencesDrug Delivery Systems0302 clinical medicineTissue engineeringHuman Umbilical Vein Endothelial Cellspolycyclic compoundsmedicineHumansCytotoxic T cellDoxorubicinBone regenerationPostoperative CareCyclodextrinsOsteosarcomaAntibiotics AntineoplasticOsteoblastsBone cancermedicine.diseaseDurapatite030104 developmental biologyDoxorubicinMechanics of Materials030220 oncology & carcinogenesisCancer cellDrug deliveryCancer researchOsteosarcomaDrug Screening Assays Antitumormedicine.drugMaterials Science and Engineering: C
researchProduct

Cell-to-Cell Communication in Learning and Memory: From Neuro- and Glio-Transmission to Information Exchange Mediated by Extracellular Vesicles

2019

Most aspects of nervous system development and function rely on the continuous crosstalk between neurons and the variegated universe of non-neuronal cells surrounding them. The most extraordinary property of this cellular community is its ability to undergo adaptive modifications in response to environmental cues originating from inside or outside the body. Such ability, known as neuronal plasticity, allows long-lasting modifications of the strength, composition and efficacy of the connections between neurons, which constitutes the biochemical base for learning and memory. Nerve cells communicate with each other through both wiring (synaptic) and volume transmission of signals. It is by now…

0301 basic medicineNervous systemCell CommunicationReviewSynaptic Transmissiontetrapartite synapseRegulatory moleculesmemorylcsh:Chemistry0302 clinical medicineCell to cell communicationSettore BIO/10 - BiochimicaSettore BIO/06 - Anatomia Comparata E Citologialcsh:QH301-705.5SpectroscopyNeuronsDrug CarriersNeuronal PlasticitylearningBrainGeneral Medicineglial cellsComputer Science ApplicationsCrosstalk (biology)medicine.anatomical_structureNerve cellsextracellular vesiclesNeurogliavolume transmissionBiologytripartite synapsisExtracellular vesiclesCatalysisInorganic Chemistry03 medical and health sciencesNeuroplasticitymedicineAnimalsHumansPhysical and Theoretical ChemistryMolecular BiologyMemory Disorderssynaptic plasticityOrganic Chemistryglial cellwiring transmissionBiological Transport030104 developmental biologylcsh:Biology (General)lcsh:QD1-999nervous systemAstrocytesSynapsesSynaptic plasticitytripartite synapsiextracellular vesiclesynaptic plasticity.NeuroscienceBiomarkers030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
researchProduct

Efficacy and safety of everolimus in extrapancreatic neuroendocrine tumor: a comprehensive review of literature

2016

BACKGROUND Everolimus, an oral mTOR (mammalian target of rapamycin) inhibitor, is currently approved for the treatment of progressive pancreatic neuroendocrine tumors (NETs). Although promising, only scattered data, often from nondedicated studies, are available for extrapancreatic NETs. PATIENTS AND METHODS A systematic review of the published data was performed concerning the use of everolimus in extrapancreatic NET, with the aim of summarizing the current knowledge on its efficacy and tolerability. Moreover, the usefulness of everolimus was evaluated according to the different sites of the primary. RESULTS The present study included 22 different publications, including 874 patients and 4…

0301 basic medicineOncologyCancer ResearchLung NeoplasmsAdrenal Gland NeoplasmsColorectal NeoplasmNeuroendocrine tumorsSettore MED/13 - EndocrinologiaAntineoplastic Agent0302 clinical medicineEndocrinologyNeuroendocrine tumors; everolimus; extrapancreatic; efficacy; safetyProspective cohort studyNeuroendocrine TumorsEverolimuOncologyTolerability030220 oncology & carcinogenesisIleal NeoplasmSafetyColorectal Neoplasmsmedicine.drugHumanmedicine.medical_specialtyEfficacyAntineoplastic AgentsPheochromocytomaExtrapancreatic neuroendocrine tumorDisease-Free Survival03 medical and health sciencesNeuroendocrine tumorStomach NeoplasmsStomach NeoplasmInternal medicinemedicineHumansEverolimusThyroid NeoplasmsAdverse effectEverolimusbusiness.industryRetrospective cohort studymedicine.diseaseDiscontinuationCarcinoma NeuroendocrineClinical trialIleal NeoplasmsAdrenal Gland NeoplasmLung Neoplasm030104 developmental biologyEndocrinologybusiness
researchProduct

Compassionate use of everolimus for refractory epilepsy in a patient with MTOR mosaic mutation

2020

Abstract The MTOR gene encodes the mechanistic target of rapamycin (mTOR), which is a core component of the PI3K-AKT-mTOR signaling pathway. Postzygotic MTOR variants result in various mosaic phenotypes, referred to in OMIM as Smith-Kinsgmore syndrome or focal cortical dysplasia. We report here the case of a patient, with an MTOR mosaic gain-of-function variant (p.Glu2419Lys) in the DNA of 41% skin cells, who received compassionate off-label treatment with everolimus for refractory epilepsy. This 12-year-old-girl presented with psychomotor regression, intractable seizures, hypopigmentation along Blaschko's lines (hypomelanosis of Ito), asymmetric regional body overgrowth, and ocular anomali…

0301 basic medicineOncologyCompassionate Use Trialsmedicine.medical_specialty[SDV]Life Sciences [q-bio]030105 genetics & heredityMuscle hypertrophyCraniofacial Abnormalities03 medical and health sciencesInternal medicineGeneticsmedicineHumansEverolimusChildMechanistic target of rapamycinProtein Kinase InhibitorsGenetics (clinical)PI3K/AKT/mTOR pathwayHypopigmentationEverolimusbiologybusiness.industryMosaicismTOR Serine-Threonine KinasesNeuropsychologyGeneral MedicineCortical dysplasiamedicine.disease3. Good healthClinical trialMalformations of Cortical Development[SDV] Life Sciences [q-bio]030104 developmental biologyPhenotypeGain of Function Mutationbiology.proteinFemaleEpilepsies Partialmedicine.symptombusinessmedicine.drug
researchProduct

Substrate Specificity of Aglaia loheri Active Isolate towards P-glycoprotein in Multidrug-Resistant Cancer Cells

2016

Multidrug resistance (MDR) is a major contributory factor in the failure of chemotherapy. Concrete interpretation of P-glycoprotein (P-gp) substrate specificity, whether a substance is a substrate or an inhibitor, represents an important feature of a compound's pharmaceutical profiling in drug design and development. In this work, the P-gp substrate specificity of Maldi 531.2[M+H]+, a phenol ester from Aglaia loheri Blanco leaves was investigated. This study focuses on the effect of Maldi 531.2[M+H]+ on P-gp ATPase activity, which was examined by measuring the amount of inorganic phosphates (Pi) released as a result of ATP hydrolysis. To test the effects of Maldi 531.2[M+H]+ on MDR activit…

0301 basic medicinePharmacologybiologyChemistryStereochemistryStimulationPlant ScienceGeneral MedicineMultiple drug resistance03 medical and health sciences030104 developmental biology0302 clinical medicineNon-competitive inhibitionComplementary and alternative medicineATP hydrolysis030220 oncology & carcinogenesisDrug DiscoveryCancer cellmedicinebiology.proteinPiVerapamilmedicine.drugP-glycoproteinNatural Product Communications
researchProduct

Amorphous polyphosphate–hydroxyapatite: A morphogenetically active substrate for bone-related SaOS-2 cells in vitro

2015

There is increasing evidence that inorganic calcium-polyphosphates (polyP) are involved in human bone hydroxyapatite (HA) formation. Here we investigated the morphology of the particles, containing calcium phosphate (CaP) with different concentrations of various Na-polyP concentrations, as well as their effects in cell culture. We used both SaOS-2 cells and human mesenchymal stem cells. The polymeric phosphate readily binds calcium ions under formation of insoluble precipitates. We found that addition of low concentrations of polyP (10wt.%, referred to the CaP deposits) results in an increased size of the HA crystals. Surprisingly, at higher polyP concentrations (10wt.%) the formation of cr…

0301 basic medicinePolymersBiocompatible Materials02 engineering and technologyBone tissueBiochemistryApatitechemistry.chemical_compoundX-Ray DiffractionOsteogenesisPolyphosphatesSpectroscopy Fourier Transform InfraredTissue ScaffoldsBiomaterialGeneral Medicine021001 nanoscience & nanotechnologyMicrospheresGene Expression Regulation Neoplasticmedicine.anatomical_structureBiochemistryvisual_artvisual_art.visual_art_mediumAlkaline phosphataseHydroxyapatites0210 nano-technologyBiotechnologyMaterials scienceBiocompatibilityBiomedical Engineeringchemistry.chemical_elementCalciumCollagen Type IBiomaterials03 medical and health sciencesCalcification PhysiologicMicroscopy Electron TransmissionCell Line TumormedicineHumansBone regenerationMolecular BiologyCell ProliferationIonsOsteoblastsTissue EngineeringSodiumMesenchymal Stem CellsAlkaline PhosphatasePhosphateMicroscopy ElectronDurapatite030104 developmental biologychemistryBiophysicsCalciumActa Biomaterialia
researchProduct

The morphogenetically active polymer, inorganic polyphosphate complexed with GdCl 3 , as an inducer of hydroxyapatite formation in vitro

2015

Inorganic polyphosphate (polyP) is a physiological polymer composed of tens to hundreds of phosphate units linked together via phosphoanhydride bonds. Here we compared the biological activity of polyP (chain length of 40 phosphate units), complexed with Gd(3+) (polyP·Gd), with the one caused by polyP (as calcium salt) and by GdCl3 alone, regarding their potencies to induce hydroxyapatite (HA) formation in SaOS-2 cells in vitro. The three compounds, GdCl3, polyP and polyP·Gd were found to be non-toxic at concentrations up to at least 30μM. Selecting a low, 5μM, concentration it was found that polyP·Gd significantly induced HA formation, as determined by Alizarin Red S staining and by quantit…

0301 basic medicinePolymerschemistry.chemical_elementGadolinium02 engineering and technologyCalciumBiochemistry03 medical and health scienceschemistry.chemical_compoundPolyphosphatesCell Line Tumorotorhinolaryngologic diseasesHumansneoplasmsSaos-2 cellsPharmacologychemistry.chemical_classificationDose-Response Relationship DrugChemistryPolyphosphateBiological activitypathological conditions signs and symptoms021001 nanoscience & nanotechnologyPhosphatedigestive system diseasesIn vitroDurapatitesurgical procedures operative030104 developmental biologyEnzymeBiochemistryAlkaline phosphatase0210 nano-technologyBiochemical Pharmacology
researchProduct

Characterisation of microsatellite and SNP markers from Miseq and genotyping-by-sequencing data among parapatric Urophora cardui (Tephritidae) popula…

2017

Phylogeographic analyses of the gall flyUrophora carduihave in earlier studies based on allozymes and mtDNA identified small-scale, parapatrically diverged populations within an expanding Western Palearctic population. However, the low polymorphism of these markers prohibited an accurate delimitation of the evolutionary origin of the parapatric divergence.Urophora carduifrom the Western Palearctic have been introduced into Canada as biological control agents of the host plantCirsium arvense. Here, we characterise 12 microsatellite loci with hexa-, penta- and tetra-nucleotide repeat motifs and report a genotyping-by-sequencing SNP protocol. We test the markers for genetic variation among thr…

0301 basic medicinePopulationlcsh:MedicineLocus (genetics)Single-nucleotide polymorphismGBSBiologyParapatric speciationParapatryGeneral Biochemistry Genetics and Molecular BiologyEcoR103 medical and health sciencesGenetic variationGenetic clineAlleleeducationGeneticseducation.field_of_studyUrophora carduiGeneral Neurosciencelcsh:RAlternative sex-linked locusBiodiversityGenomicsGeneral MedicineSSREvolutionary StudiesGenome-wide differentiationPhylogeography030104 developmental biologyEvolutionary biologyMicrosatelliteGeneral Agricultural and Biological SciencesEntomologyPeerJ
researchProduct

Regulation of Autophagic Signaling by Mechanical Loading and Inflammation in Human PDL Fibroblasts

2020

Autophagy (cellular self-consumption) is a crucial adaptation mechanism during cellular stress conditions. This study aimed to examine how this important process is regulated in human periodontal ligament (PDL) fibroblasts by mechanical and inflammatory stress conditions and whether the mammalian target of rapamycin (mTOR) signaling pathway is involved. Autophagy was quantified by flow cytometry. Qualitative protein phosphorylation profiling of the mTOR pathway was carried out. Effects of mTOR regulation were assessed by quantification of important synthesis product collagen 1, cell proliferation and cell death with real-time PCR and flow cytometry. Autophagy as a response to mechanical or …

0301 basic medicineProgrammed cell deathautophagyInflammationCatalysisArticlelcsh:ChemistryInorganic Chemistry03 medical and health sciences0302 clinical medicineinflammatory conditionsmedicineHumansProtein phosphorylationPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopyPI3K/AKT/mTOR pathwayCells CulturedInflammationCell DeathCell growthChemistryOrganic ChemistryAutophagymechanical stress030206 dentistryGeneral MedicineFibroblastsComputer Science ApplicationsCell biologyorthodontic tooth movement030104 developmental biologylcsh:Biology (General)lcsh:QD1-999mammalian target of rapamycin (mTOR) signaling pathwayPhosphorylationStress Mechanicalmedicine.symptomSignal transductionSignal TransductionInternational Journal of Molecular Sciences
researchProduct