Search results for "RATS"

showing 10 items of 3537 documents

Proteomic analysis of the pinworm Syphacia muris (Nematoda: Oxyuridae), a parasite of laboratory rats

2012

Syphacia muris (Nematoda: Oxyuridae) is a ubiquitous nematode that commonly infects rats in the laboratory which can interfere in the development of biological assays. The somatic extract of S. muris adults collected from infected rats was investigated using a proteomic approach. A shot-gun liquid chromatography/tandem mass spectrometry procedure was used. We used the MASCOT search engine (Matrix-Science) and ProteinPilot software v2.0 (Applied Biosystems) for the database search. A total of 359 proteins were accurately identified from the worms. The largest protein families consisted of metabolic enzymes and those involved in the nucleic metabolism and cell cycle. Proteins of transmembrane…

ProteomicsProtein familyNematodaSyphaciaProteomicsOxyuridaeRodentsMicrobiologyTranscriptomeRodent DiseasesLaboratory Animal ScienceOxyuroideaParasite hostingAnimalsDatabase search engineRats WistarNematode InfectionsOxyuridaebiologyHelminth ProteinsSyphacia murisbiology.organism_classificationMolecular biologyRatsInfectious DiseasesNematodeGene Expression RegulationProteomeParasitologyTranscriptome
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Correlation of Crystallin Expression and RGC Susceptibility in Experimental Glaucoma Rats of Different Ages.

2018

Glaucoma is one of the leading causes of blindness worldwide with age being an important risk factor. However, the pathogenesis remains poorly understood. Aim of this study was to focus on age-dependent molecular changes in an experimental animal model of glaucoma.Intraocular pressure was elevated in Sprague-Dawley rats aged 3, 14, and 47 weeks for a period of 7 weeks by episcleral vein cauterization. Ganglion cell loss was monitored by an immunohistochemical staining of the Brain-specific homeobox/POU (Pit-1, Oct-2, Unc-86) domain protein 3A positive cells in retinal flat-mounts and spectral-domain optical coherence tomography measuring the retinal nerve fiber layer thickness. Molecular pr…

ProteomicsRetinal Ganglion CellsIntraocular pressureAgingGlaucomaCell CountBioinformaticsMass SpectrometryRetinaCorrelationPathogenesisRats Sprague-Dawley03 medical and health sciencesCellular and Molecular NeuroscienceTonometry Ocular0302 clinical medicineNerve FibersCrystallinMedicineAnimalsRisk factorIntraocular PressureBlindnessbusiness.industryGlaucomamedicine.diseaseCrystallinsSensory SystemsOphthalmologyDisease Models AnimalAgeing030221 ophthalmology & optometryFemalebusiness030217 neurology & neurosurgeryTomography Optical CoherenceCurrent eye research
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Serum and antibodies of glaucoma patients lead to changes in the proteome, especially cell regulatory proteins, in retinal cells.

2012

PURPOSE: Previous studies show significantly specifically changed autoantibody reactions against retinal antigens in the serum of glaucoma and ocular hypertension (OHT) patients in comparison to healthy people. As pathogenesis of glaucoma still is unknown the aim of this study was to analyze if the serum and antibodies of glaucoma patients interact with neuroretinal cells. METHODS: R28 cells were incubated with serum of patients suffering from primary open angle glaucoma (POAG), normal tension glaucoma (NTG) or OHT, POAG serum after antibody removal and serum from healthy people for 48 h under a normal or an elevated pressure of 15000 Pa (112 mmHg). RGC5 cells were additionally incubated wi…

ProteomicsRetinal Ganglion CellsSerumProteomegenetic structuresOcular hypertensionGlaucomalcsh:MedicineAutoimmunityPathogenesischemistry.chemical_compoundMolecular Cell Biologylcsh:ScienceCellular Stress ResponsesMultidisciplinarySpectrometric Identification of ProteinsbiologyNeurodegenerative DiseasesBlood proteinsSignaling CascadesNeurologyMedicineRetinal DisordersElectrophoresis Polyacrylamide GelAntibodyGlaucoma Open-AngleRetinal NeuronsSignal TransductionResearch ArticleSpectrometry Mass Electrospray IonizationImmunologyImmunoglobulinsPeptide MappingAntibodiesStress Signaling CascadeCell LineAntigenmedicinePressureAnimalsHumansBiologylcsh:RAutoantibodyRetinalGlaucomamedicine.diseaseeye diseasesRatsOphthalmologychemistrySpectrometry Mass Matrix-Assisted Laser Desorption-IonizationImmunologybiology.proteinOcular HypertensionClinical Immunologylcsh:Qsense organsChromatography LiquidPLoS ONE
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Morphological, biochemical, and molecular biological characterization of a rat rhabdomyosarcoma cell line during differentiation induction in vitro.

1990

BA-HAN-1C is a clonal rat rhabdomyosarcoma cell line consisting of proliferating mononuclear tumor cells, some of which spontaneously fuse to form terminally differentiated postmitotic myotubelike giant cells. Exposure to retinoic acid resulted in an inhibition of proliferation and a marked increase in cellular differentiation. The number of myotubelike giant cells significantly increased, and about 30% of the mononuclear tumor cells exhibited morphological features of rhabdomyogenic differentiation which were not observed in the mononuclear cells of untreated cultures. Morphological differentiation was paralleled by an increase in total creatine kinase activity as a biochemical marker of d…

Proto-OncogenesCell divisionHealth Toxicology and MutagenesisCellular differentiationTretinoinBiologyCell LineTretinoinProto-OncogenesRhabdomyosarcomaTumor Cells CulturedmedicineAnimalsRhabdomyosarcomaPublic Health Environmental and Occupational HealthCell Differentiationmedicine.diseaseMolecular biologyIn vitroRatsGene Expression Regulation NeoplasticCell cultureRat RhabdomyosarcomaCell DivisionResearch Articlemedicine.drugEnvironmental Health Perspectives
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Hypoxia-induced dysfunction of rat diaphragm

2004

Contains fulltext : 47331.pdf (Publisher’s version ) (Closed access) Oxidants may play a role in hypoxia-induced respiratory muscle dysfunction. In the present study we hypothesized that hypoxia-induced impairment in diaphragm contractility is associated with elevated peroxynitrite generation. In addition, we hypothesized that strenuous contractility of the diaphragm increases peroxynitrite formation. In vitro force-frequency relationship, isotonic fatigability, and nitrotyrosine levels were assessed under hypoxic (Po(2) approximately 6.5 kPa) and hyperoxic (Po(2) approximately 88.2 kPa) control conditions and also in the presence of authentic peroxynitrite (60 min), ebselen (60 min), and t…

Pulmonary and Respiratory MedicineAzolesMalemedicine.medical_specialtyPhysiologyDiaphragmAetiology screening and detection [ONCOL 5]In Vitro TechniquesIsoindolesNitric oxideContractilitychemistry.chemical_compoundTranslational research [ONCOL 3]Physiology (medical)Internal medicineOrganoselenium CompoundsPeroxynitrous AcidmedicineRespiratory muscleAnimalsRespiratory systemEnzyme InhibitorsRats WistarHypoxiaHeart lung and circulation [UMCN 2.1]Renal disorder [IGMD 9]omega-N-MethylarginineNitrotyrosineCell BiologyHypoxia (medical)Tissue engineering and pathology [NCMLS 3]musculoskeletal systemRatsPathogenesis and modulation of inflammation [N4i 1]EndocrinologychemistryBiochemistryMuscle FatigueTyrosineRat DiaphragmLipid Peroxidationmedicine.symptomPeroxynitriteMuscle ContractionAmerican Journal of Physiology - Lung Cellular and Molecular Physiology
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The Adult Sprague-Dawley Sugen-Hypoxia Rat Is Still "the One:" A Model of Group 1 Pulmonary Hypertension: Reply to Le Cras and Abman

2020

To the editor: Kojonazarov et al. recently reported severe emphysema in the SU5416/Hypoxia (SuHx) rat model of pulmonary hypertension (1). The authors found that adult male Wistar Kyoto (WKY) rats had increased air-to-tissue ratio as judged by non-gated in vivo micro-computed tomography (CT), and an increased mean linear intercept (MLI) as surrogate of emphysema (1, 2). Le Cras and Abman now responded to the Kojonazarov report by underlining the “important role of the developmental timing of disrupted VEGF signaling” (3). They cite earlier studies conducted on the ovine fetus showing that VEGF inhibition caused vascular remodeling, reduction in vascular/airway growth, and neonatal pulmonary…

Pulmonary and Respiratory MedicineEmphysema0303 health sciencesIndolesbusiness.industryHypertension PulmonaryHypoxia (medical)Critical Care and Intensive Care Medicinemedicine.diseasePulmonary hypertensionRatsSprague dawleyRats Sprague-Dawley03 medical and health sciences0302 clinical medicine030228 respiratory systemAnesthesiaCorrespondenceMedicineAnimalsPyrrolesmedicine.symptombusinessHypoxia030304 developmental biology
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Theophylline suppresses the release of tumour necrosis factor-alpha by blood monocytes and alveolar macrophages.

1994

The purpose of this study was to evaluate the effect of theophylline on tumour necrosis factor-alpha (TNF-alpha) release by human blood monocytes (BMo), and rat BMo and alveolar macrophages (AM). BMo and AM were incubated in the absence or presence of theophylline, and the cell-free supernatants were harvested and tested for TNF-alpha activity by bioassay. Theophylline dose-dependently reduced TNF-alpha release by human BMo: significant inhibition was observed at 100 microns (41 +/- 5.9% of controls) and at 50 microns (59 +/- 4.8% of controls), while the inhibitory activity of theophylline at 10 microns (71 +/- 8.9% of controls) was not statistically significant. This activity was maximal a…

Pulmonary and Respiratory MedicineLipopolysaccharidesMalemedicine.medical_specialtyNecrosismedicine.drug_classmedicine.medical_treatmentGene ExpressionIn Vitro TechniquesTheophyllineBronchodilatorInternal medicineMacrophages AlveolarmedicineAnimalsHumansTheophyllineRats WistarDose-Response Relationship Drugbusiness.industryTumor Necrosis Factor-alphaMonocytemedicine.diseaseBlotting NorthernRatsmedicine.anatomical_structureEndocrinologyCytokineBronchial hyperresponsivenessLeukocytes MononuclearTumor necrosis factor alphaPulmonary alveolusmedicine.symptombusinessmedicine.drugThe European respiratory journal
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Effects of bacterial lipopolysaccharides (LPS) and tumour necrosis factor-alpha (TNF alpha) on rat tracheal epithelial cells in culture: morphology, …

1996

Rat tracheal epithelial cells were cultured and the effects of LPS and TNF alpha on cell morphology, rate of proliferation and NO synthase activity were studied. NO synthase activity was determined by measuring the accumulation of 3H-L-citrulline during incubation of confluent monolayer with 3H-L-arginine. In untreated cells no significant 3H-L-citrulline formation was detected, and bradykinin and the calcium ionophore A 23187 failed to stimulate 3H-L-citrulline formation excluding a constitutively expressed, calcium-dependent NO synthase activity. After culturing the cells for 18 h in the presence of LPS (10 micrograms/ml) and TNF alpha (500 U/ml) a marked formation of 3H-L-citrulline coul…

Pulmonary and Respiratory MedicineLipopolysaccharidesNecrosisEndogenyBiologyCell morphologyNitric OxideTritiumEpitheliumNitric oxidechemistry.chemical_compoundmedicineAnimalsPharmacology (medical)General Pharmacology Toxicology and PharmaceuticsDexamethasoneCells CulturedCell growthTumor Necrosis Factor-alphaEpithelial CellsDNAMolecular biologyRatsTracheaBiochemistrychemistryEnzyme InductionRespiratory epitheliumTumor necrosis factor alphamedicine.symptomNitric Oxide SynthaseCell Divisionmedicine.drugThymidinePulmonary pharmacology
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Cardioprotective effects of the serine protease inhibitor aprotinin after regional ischemia and reperfusion on the beating heart.

2002

AbstractObjective: Early coronary reperfusion of the ischemic myocardium is a desired therapeutic goal to preserve myocardium. However, reperfusion itself contributes to an additional myocardial injury (ie, reperfusion injury), which has been attributed to neutrophil infiltration with subsequent release of proteases and oxygen-derived radicals. We studied the effects of the serine protease inhibitor aprotinin (Trasylol) on myocardial ischemia and reperfusion in a rat model. Methods: The effects of aprotinin (5000 and 20,000 U/kg) were examined in vivo in a rat model of regional myocardial ischemia (20 minutes) and long-term reperfusion (24 hours). Cardioprotecive effects were determined by …

Pulmonary and Respiratory MedicineMaleProteasesCardiotonic AgentsSerine Proteinase InhibitorsNeutrophilsHeart VentriclesIschemiaMyocardial IschemiaApoptosisMyocardial ReperfusionMyocardial Reperfusion InjuryPharmacologyRats Sprague-DawleyLeukocyte CountAprotininIn vivoMedicineAnimalsAprotininCreatine KinasePeroxidaseCardioprotectionbiologyDose-Response Relationship Drugbusiness.industryMyocardiumModels Cardiovascularmedicine.diseaseCombined Modality TherapyRatsDisease Models AnimalTreatment OutcomeEnzyme inhibitorAnesthesiabiology.proteinSurgeryCreatine kinaseCardiology and Cardiovascular MedicinebusinessReperfusion injuryBiomarkersmedicine.drugThe Journal of thoracic and cardiovascular surgery
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Oral N-acetylcysteine attenuates the rat pulmonary inflammatory response to antigen.

2003

Oxidative stress is involved in the pathophysiology of inflammatory airway diseases including asthma; therefore, antioxidants might be of clinical benefit in asthma treatment. In the present study, the effects of N-acetylcysteine on sensitised brown Norway rats were examined. N-Acetylcysteine (3 mmol kg body weight(-1) administered orally) was given daily for 1 week before challenge and various antigen-induced pulmonary responses were studied. Antigen exposure increased lipid peroxidation in bronchoalveolar lavage fluid (BALF) and oxidised glutathione levels in lung tissue 2 h after challenge. Lung nuclear transcription factor-KB-binding activity was increased 2 h after challenge, and BALF …

Pulmonary and Respiratory MedicineMalemedicine.medical_treatmentMolecular Sequence DataAdministration OralNitric Oxide Synthase Type IIInflammationPharmacologyBronchial Provocation TestsAcetylcysteinechemistry.chemical_compoundMedicineAnimalsEvans BlueProbabilityAnalysis of VarianceLungmedicine.diagnostic_testBase Sequencebusiness.industryReverse Transcriptase Polymerase Chain ReactionAirway Resistancerespiratory systemEosinophilAllergensIntercellular Adhesion Molecule-1ExtravasationAsthmarespiratory tract diseasesAcetylcysteineRatsDisease Models AnimalCytokinemedicine.anatomical_structureBronchoalveolar lavagechemistryImmunologyLipid Peroxidationmedicine.symptomBronchial HyperreactivityInflammation MediatorsNitric Oxide SynthasebusinessBronchoalveolar Lavage Fluidmedicine.drugThe European respiratory journal
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