Search results for "REACTIVE OXYGEN SPECIES"

showing 10 items of 879 documents

Mitochondrial G protein coupled receptor kinase 2 regulates proinflammatory responses in macrophages.

2013

G-protein-coupled receptor kinase 2 (GRK2) levels are elevated in inflammation but its role is not clear yet. Here we show that GRK2 expression is dependent on NFκB transcriptional activity. In macrophages, LPS induces GRK2 accumulation in mitochondria increasing biogenesis. The overexpression of the carboxy-terminal domain of GRK2 (βARK-ct), known to displace GRK2 from plasma membranes, in macrophages induces earlier localization of GRK2 in mitochondria in response to LPS leading to increased mt-DNA transcription, reduced ROS production and cytokines expression. Our study shows the relevance of GRK2 subcellular localization in macrophage’s biology and its potential therapeutic properties i…

LipopolysaccharidesG-Protein-Coupled Receptor Kinase 2BiophysicsβARK-ctGRK2InflammationMitochondrionBiochemistryArticleProinflammatory cytokineMiceStructural BiologyGeneticsmedicineAnimalsHumansReceptorMolecular BiologyInflammationG protein-coupled receptor kinasebiologyKinaseSubcellular localizationBeta adrenergic receptor kinaseMacrophagesCell BiologySubcellular localizationCell biologyMitochondriabiology.proteinmedicine.symptomReactive Oxygen SpeciesSignal Transduction
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Flow cytometric investigation of neutrophil oxidative burst and apoptosis in physiological and pathological situations

2009

Flow cytometric analysis provides a rapid screen for abnormalities of polymorphonuclear neutrophils (PMN) function and reflect their behavior in vivo more accurately. This review summarizes the major fluorescent probes used to study PMN oxidative burst and apoptosis using flow cytometry (FCM). We also provide examples of FCM studies in physiological and pathological situations, illustrating the advantages of FCM for assessment of PMN oxidative burst and PMN apoptosis. These data point to the role of FCM in detecting primary immunodeficiencies such as IRAK4 deficiency and support the use of the assessment of the PMN oxidative burst for routine testing in patients with bacterial infections. W…

LipopolysaccharidesHistologyNeutrophilsSimian Acquired Immunodeficiency SyndromeApoptosisBiologyPathology and Forensic MedicineFlow cytometryAdjuvants ImmunologicIn vivomedicineAnimalsHumansReceptorNeutrophil oxidative burstPathologicalRespiratory Burstmedicine.diagnostic_testToll-Like ReceptorsImidazolesNADPH OxidasesCell BiologyFlow CytometryPhenotypeRespiratory burstInterleukin-1 Receptor-Associated KinasesOligodeoxyribonucleotidesApoptosisImmunologyReactive Oxygen SpeciesCytometry Part A
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Mitochondrial Function in the Kidney and Heart, but Not the Brain, is Mainly Altered in an Experimental Model of Endotoxaemia

2019

Significant impairments in mitochondrial function are associated with the development of multi-organ failure in sepsis/endotoxaemia, but the data on the dynamics of simultaneous mitochondrial impairment in multiple organs are limited. The aim of this study was to evaluate the changes in heart, brain and kidney mitochondrial function in an experimental model of lipopolysaccharide (LPS)-induced endotoxaemia.Samples were collected 4 and 24 h after single injection of LPS (10 mg/kg) in mice. Marked increases in inflammation-related gene expression were observed in all studied tissues 4 h after LPS administration. At 24 h post LPS administration, this expression of inflammation-related genes rem…

LipopolysaccharidesMaleCardiac function curvemedicine.medical_specialtyOxidative phosphorylation030204 cardiovascular system & hematologyMitochondrionKidneyCritical Care and Intensive Care MedicineMitochondria HeartMice03 medical and health sciences0302 clinical medicineInternal medicineRespirationmedicineAnimalsHeart metabolismchemistry.chemical_classificationMice Inbred ICRKidneyReactive oxygen speciesChemistryMyocardiumBrainKidney metabolism030208 emergency & critical care medicineEndotoxemiaDisease Models AnimalEndocrinologymedicine.anatomical_structureEmergency MedicineShock
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Responses of retinal arterioles and ciliary arteries in pigs with acute respiratory distress syndrome (ARDS)

2019

Abstract Acute respiratory distress syndrome (ARDS) is a clinical syndrome of acute lung failure in critically sick patients, which severely compromises the function of multiple organs, including the brain. Although, the optic nerve and the retina are a part of the central nervous system, the effects of ARDS on these ocular structures are completely unknown. Thus, the major goal of this study was to test the hypothesis that ARDS affects vascular function in the eye. ARDS was induced in anesthetized pigs by intratracheal injection of lipopolysaccharide (LPS). Sham-treated animals served as controls. Pigs were monitored for 8 h and then sacrificed. Subsequently, retinal arterioles and short p…

LipopolysaccharidesMalePathologymedicine.medical_specialtyARDSEndotheliumRetinal ArterySwineNitric Oxide Synthase Type IIEnzyme-Linked Immunosorbent AssayVasodilationReal-Time Polymerase Chain ReactionCiliary ArteriesCellular and Molecular Neurosciencechemistry.chemical_compoundGlutathione Peroxidase GPX1medicine.arterymedicineAnimalsRNA MessengerEndothelial dysfunctionGlutathione PeroxidaseRespiratory Distress SyndromeRetinaMicroscopy Videobusiness.industryInterleukinsRetinalShort posterior ciliary arteriesCatalasemedicine.diseaseSensory SystemsCiliary arteriesArteriolesDisease Models AnimalOphthalmologymedicine.anatomical_structurechemistryEndothelium VascularHypoxia-Inducible Factor 1Reactive Oxygen SpeciesbusinessExperimental Eye Research
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Oxidative stress and innate immunity responses in cigarette smoke stimulated nasal epithelial cells

2013

Cigarette smoke extracts (CSE) may play a significant role in diseases of the upper airway including chronic rhinosinusitis. Even short term exposure of cigarette smoke has adverse effects on mitochondrial functions and redox homeostasis in tissues which may progress to further complications associated with chronic smoking. Cigarette smoke alters toll-like receptor 4 (TLR4) expression and activation in bronchial epithelial cells. Carbocysteine is an anti-oxidant and mucolytic agent. The effects of carbocysteine on CSE induced oxidative stress and on associated innate immune and inflammatory responses in nasal epithelial cells are largely unknown. The present study was aimed to assess in CSE…

LipopolysaccharidesNecrosisNeutrophilsPhalloidineCARB CSE Cigarette smoke LPS Nasal epithelial cells ROS Reactive oxygen species TLR4 carbocysteine cigarette smoke extracts lipolysaccharide reactive oxygen species toll like receptor 4Fluorescent Antibody TechniqueApoptosisMucous membrane of noseCell SeparationBiologyToxicologymedicine.disease_causeCell LineNecrosisSmokeTobaccomedicineHumansExpectorantschemistry.chemical_classificationReactive oxygen speciesInnate immune systemCarbocysteineEpithelial CellsCarbocysteineTobacco ProductsGeneral MedicineActinsImmunity InnateToll-Like Receptor 4Nasal MucosaOxidative StresschemistryApoptosisImmunologyTLR4medicine.symptomReactive Oxygen SpeciesOxidative stressToxicology in Vitro
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Conversion of biliverdin to bilirubin by biliverdin reductase contributes to endothelial cell protection by heme oxygenase-1—evidence for direct and …

2009

Heme oxygenase-1 (HO-1) is highly protective in various pathophysiological states such as cardiovascular and neurodegenerative diseases. HO-1-derived bilirubin is an efficient scavenger of reactive oxygen and nitrogen species (RONS). It remains to determine whether conversion of biliverdin to bilirubin is an essential step for HO-1-conferred protection of endothelial cells. RONS scavenging activities of biliverdin versus bilirubin were assessed by different RONS generating systems and detection techniques. We also silenced the biliverdin reductase (BVR) or HO-1 gene in cultured primary human endothelial cells (HUVECs) and measured the effect on RONS formation upon stimulation with lipopolys…

LipopolysaccharidesOxidoreductases Acting on CH-CH Group DonorsUmbilical VeinsXanthine OxidaseNeutrophilsBilirubinNitrosationModels BiologicalAntioxidantschemistry.chemical_compoundPeroxynitrous AcidLeukocytespolycyclic compoundsHumansGene SilencingMolecular BiologyHemeReactive nitrogen speciesRespiratory BurstBiliverdinAngiotensin IIBiliverdineBiliverdin reductaseEndothelial CellsBilirubinFree Radical ScavengersAngiotensin IIMitochondriaEndothelial stem cellHeme oxygenasechemistryBiochemistryCytoprotectionGene Knockdown TechniquesTyrosineReactive Oxygen SpeciesCardiology and Cardiovascular MedicineHeme Oxygenase-1Journal of Molecular and Cellular Cardiology
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Acute exercise activates nuclear factor (NF)-kappaB signaling pathway in rat skeletal muscle.

2004

Two studies were performed to investigate the effects of an acute bout of physical exercise on the nuclear protein kappaB (NF-kappaB) signaling pathway in rat skeletal muscle. In Study 1, a group of rats (n=6) was run on the treadmill at 25 m/min, 5% grade, for 1 h or until exhaustion (Ex), and compared with a second group (n=6) injected with two doses of pyrrolidine dithiocarbamate (PDTC, 100 mg/kg, i.p.) 24 and 1 h prior to the acute exercise bout. Three additional groups of rats (n=6) were injected with either 8 mg/kg (i.p.) of lipopolysaccharide (LPS), 1 mmol/kg (i.p.) t-butylhydroperoxide (tBHP), or saline (C) and killed at resting condition. Ex rats showed higher levels of NF-kappaB b…

Lipopolysaccharidesmedicine.medical_specialtyP50PyrrolidinesElectrophoretic Mobility Shift AssayIκB kinaseBiologyProtein Serine-Threonine Kinasesmedicine.disease_causeBiochemistryRats Sprague-Dawleychemistry.chemical_compoundPyrrolidine dithiocarbamateNF-KappaB Inhibitor alphatert-ButylhydroperoxideThiocarbamatesInternal medicinePhysical Conditioning AnimalGeneticsmedicineAnimalsMuscle SkeletalMolecular Biologychemistry.chemical_classificationReactive oxygen speciesNF-kappa BSkeletal muscleI-kappa B KinaseRatsCytosolOxidative Stressmedicine.anatomical_structureEndocrinologychemistryFemaleI-kappa B ProteinsSignal transductionOxidative stressBiotechnologySignal TransductionFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Induction of stress resistance and extension of lifespan in Chaenorhabditis elegans serotonin-receptor knockout strains by withanolide A

2021

Abstract Introduction Approximately 300 million people worldwide suffer from depression. The COVID-19 crisis may dramatically increase these numbers. Severe side effects and resistance development limit the use of standard antidepressants. The steroidal lactone withanolide A (WA) from Withania somnifera may be a promising alternative. Caenorhabditis elegans was used as model to explore WA's anti-depressive and anti-stress potential. Methods C. elegans wildtype (N2) and deficient strains (AQ866, DA1814, DA2100, DA2109 and MT9772) were used to assess oxidative, osmotic or heat stress as measured by generation of reactive oxygen species (ROS), determination of lifespan, and mRNA expression of …

LongevityPharmaceutical SciencePharmacologyWithaniamedicine.disease_cause03 medical and health sciencesGene Knockout Techniques0302 clinical medicineFluoxetineDrug DiscoverymedicineAnimalsCaenorhabditis elegansCaenorhabditis elegans ProteinsWithanolidesCaenorhabditis elegansSerotonin transporter5-HT receptor030304 developmental biologyPharmacology0303 health sciencesFluoxetinebiologyPlant ExtractsWild typebiology.organism_classificationMolecular Docking SimulationOxidative StressComplementary and alternative medicine030220 oncology & carcinogenesisReceptors Serotoninbiology.proteinAntidepressantMolecular MedicineSerotoninReactive Oxygen SpeciesOxidative stressmedicine.drugPhytomedicine
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Dual Cherenkov Radiation-Induced Near-Infrared Luminescence Imaging and Photodynamic Therapy toward Tumor Resection

2020

International audience; Cherenkov radiation (CR), the blue light seen in nuclear reactors, is emitted by some radiopharmaceuticals. This study showed that (1) a portion of CR could be transferred in the region of the optical spectrum, where biological tissues are most transparent: as a result, upon radiance amplification in the near-infrared window, the detection of light could occur twice deeper in tissues than during classical Cherenkov luminescence imaging and (2) Cherenkov-photodynamic therapy (CR-PDT) on cells could be achieved under conditions mimicking unlimited depth using the CR-embarked light source, which is unlike standard PDT, where light penetration depth is limited in biologi…

LuminescenceLightInfrared RaysInfraredPhthalocyaninesmedicine.medical_treatmentBodipyPhotodynamic therapy01 natural sciencesEnergy-transferMice03 medical and health sciencesOpticsUnresectedCell Line TumorQuantum DotsDrug DiscoverymedicineAnimalsCherenkov radiation030304 developmental biology0303 health sciencesChemistrybusiness.industryOptical ImagingCerenkov Radiation[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciences0104 chemical sciencesRecognition010404 medicinal & biomolecular chemistryPhotochemotherapyBright EnoughRadianceNanoparticlesMolecular MedicineAntenna (radio)Reactive Oxygen SpeciesLuminescencebusinessVisible spectrumJournal of Medicinal Chemistry
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Cajaninstilbene acid (CSA) exerts cytoprotective effects against oxidative stress through the Nrf2-dependent antioxidant pathway.

2013

Cajaninstilbene acid (CSA), an active compound separated from pigeon pea leaves, possesses the highly efficient antioxidant activities. Transcription factor nuclear factor-erythroid 2-related factor 2 (Nrf2) is an important regulator of cellular oxidative stress. This study examined the role of Nrf2 in CSA-mediated antioxidant effects on human hepatocarcinoma (HepG2) cell line. The generation of reactive oxygen species (ROS) upon H2O2 and CSA treatment was lower than that of H2O2 alone. CSA activated Nrf2 as evaluated by Western blotting. A luciferase reporter assay also demonstrated that CSA-activated signaling resulted in the increased transcriptional activity of Nrf2 through binding to t…

MAPK/ERK pathwayAntioxidantNF-E2-Related Factor 2medicine.medical_treatmentBlotting WesternBiologyToxicologymedicine.disease_causeenvironment and public healthAntioxidantsStilbenesmedicineNAD(P)H Dehydrogenase (Quinone)HumansProtein kinase BTranscription factorPI3K/AKT/mTOR pathwaychemistry.chemical_classificationReactive oxygen speciesGeneral MedicineHep G2 Cellsrespiratory systemAntioxidant Response ElementsSalicylatesOxidative StressBiochemistrychemistryCytoprotectionNAD+ kinaseOxidative stressHeme Oxygenase-1Signal TransductionToxicology letters
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