Search results for "REACTIVE OXYGEN SPECIES"

showing 10 items of 879 documents

Isoindolo[2,1-a]quinoxaline derivatives, novel potent antitumor agents with dual inhibition of tubulin polymerization and topoisomerase I.

2008

Isoindoloquinoxalines 4 and 5 were obtained by refluxing 2-(2'-aminoaryl)-1-cyanoisoindoles 3a- e in acetic or formic acid. All derivatives were screened by the National Cancer Institute (Bethesda, MD) for the in vitro one dose primary anticancer assay against a 3-cell line panel. Compounds 4a- e, screened against a panel of about 60 human tumor cell lines, showed remarkable antineoplastic activity; they had GI 50 values in the low micromolar or submicromolar range and reached, in the case of 4c, nanomolar concentrations on 88% of the 59 tested cell lines. Flow cytometric analysis of cell cycle after treatment with 4c demonstrated an arrest of the cell cycle in G2/M phase. This effect was a…

Mitotic indexMagnetic Resonance SpectroscopySpectrophotometry InfraredPolymersFLUORESCENT-PROBELIGAND-DNA SYSTEMSMitosisCELL-LINESAntineoplastic AgentsACRIDINE-ORANGETopoisomerase-I InhibitorMITOCHONDRIATubulinCell Line TumorQuinoxalinesDrug DiscoveryHumansCytotoxicitybiologyChemistryTopoisomeraseB-DNACell CycleCell cycleAPOPTOSISCDEnzyme ActivationMICROTUBULESBiochemistryMicroscopy FluorescenceCell cultureApoptosisEnzyme inhibitorLINEAR DICHROISM SPECTROSCOPYCaspasesbiology.proteinMolecular MedicineDrug Screening Assays AntitumorTopoisomerase I InhibitorsReactive Oxygen SpeciesJournal of medicinal chemistry
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Comparison of Protective Effects against Reactive Oxygen Species of Mononuclear and Dinuclear Cu(II) Complexes with N-Substituted Benzothiazolesulfon…

2005

Copper(II) complexes of N-benzothiazolesulfonamides (HL1=N-2-(4-methylphenylsulfamoyl)-6-nitro-benzothiazole, HL2=N-2-(phenylsulfamoyl)-6-chloro-benzothiazole, and HL3=N-2-(4-methylphenylsulfamoyl)-6-chloro-benzothiazole) with ammonia have been synthesized and characterized. The crystal structures of the [Cu(L1)2(NH3)2].2MeOH, [Cu(L2)2(NH3)2], and [Cu(L3)2(NH3)2] compounds have been determined. Compounds and present a distorted square planar geometry. In both compounds the metal ion is coordinated by two benzothiazole N atoms from two sulfonamidate anions and two NH3 molecules. Complex is distorted square-pyramidal. The Cu(II) ion is linked to the benzothiazole N and sulfonamidate O atoms o…

Models MolecularFree RadicalsStereochemistrychemistry.chemical_elementSaccharomyces cerevisiaeCrystal structureIn Vitro TechniquesCrystallography X-RayMedicinal chemistryIonInorganic ChemistryMetalSuperoxide dismutaseStructure-Activity RelationshipAmmoniachemistry.chemical_compoundOrganometallic CompoundsMoleculeBenzothiazolesPhysical and Theoretical ChemistrySulfonamidesMolecular StructurebiologySuperoxide DismutaseCopperchemistryBenzothiazolevisual_artvisual_art.visual_art_mediumbiology.proteinCrystallizationReactive Oxygen SpeciesCopperInorganic Chemistry
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Characterization and metal-induced gene transcription of two new copper zinc superoxide dismutases in the solitary ascidian Ciona intestinalis

2013

Antioxidant enzymes are known to protect living organisms against the oxidative stress risk, also induced by metals. In the present study, we describe the purification and molecular characterization of two Cu,Zn superoxide dismutases (SODs), referred to as Ci-SODa and Ci-SODb, from Ciona intestinalis, a basal chordate widely distributed in temperate shallow seawater. The putative amino acid sequences were compared with Cu,Zn SODs from other metazoans and phylogenetic analyses indicate that the two putative Ci-SODs are more related to invertebrate SODs than vertebrate ones. Both phylogenetic and preliminary homology modeling analyses suggest that Ci-SODa and Ci-SODb are extracellular and int…

Models MolecularGene isoformHemocytesCiona intestinaliHealth Toxicology and MutagenesisIn silicoSettore BIO/05 - ZoologiaSuperoxide dismutaseAquatic ScienceGene Expression Regulation EnzymologicSuperoxide dismutaseOvarian FollicleConsensus sequenceAnimalsSUPEROXIDE-DISMUTASECiona intestinalisGeneCiona intestinalis; SUPEROXIDE-DISMUTASE; ascidiansPhylogenybiologyEcologyCadmium; Ciona intestinalis; Copper; Reactive oxygen species; Superoxide dismutase; ZincPromoterbiology.organism_classificationProtein Structure TertiaryCiona intestinalisascidiansCionaZincBiochemistryMetalsbiology.proteinReactive oxygen specieFemaleWater Pollutants ChemicalCopperCadmium
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Preparation of two sets of 5,6,7-trioxygenated dihydroflavonol derivatives as free radical scavengers and neuronal cell protectors to oxidative damag…

2009

An unusual class of 5,6,7-trioxygenated dihydroflavonols (3a-e and 4a-j) were designed and prepared. Their antioxidative properties were assessed by examining their capacities in several in vitro models, including superoxide anion and 1,1-diphenyl-2-picrylhydrazyl (DPPH) radical scavenging, rat liver homogenate lipid peroxidation inhibition, PC12 cells protection from oxidative damage, and xanthine oxidase inhibition. These dihydroflavonols displayed positive quenching abilities towards O(2)(-) and DPPH free radicals, in which the majority exhibited superior antioxidant properties to Vitamin C. cis-Configurated compound (+/-)-3e demonstrated remarkable inhibition to LPO with an IC(50) value…

Models MolecularXanthine OxidaseAntioxidantFlavonolsmedicine.drug_classDPPHmedicine.medical_treatmentClinical BiochemistryMolecular ConformationPharmaceutical Sciencemedicine.disease_causeBiochemistryPC12 CellsAntioxidantsLipid peroxidationRats Sprague-Dawleychemistry.chemical_compoundStructure-Activity RelationshipDrug DiscoverymedicineAnimalsXanthine oxidaseMolecular BiologyXanthine oxidase inhibitorNeuronsSuperoxideOrganic ChemistryFree Radical ScavengersFree radical scavengerRatschemistryBiochemistryMolecular MedicineLipid PeroxidationReactive Oxygen SpeciesOxidative stressBioorganicmedicinal chemistry
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Oxidative stress upregulates the NMDA receptor on cerebrovascular endothelium.

2009

N-methyl-d-aspartate receptor (NMDA-R)-mediated oxidative stress has been implicated in blood-brain barrier (BBB) disruption in a variety of neuropathological diseases. Although some interactions between both phenomena have been elucidated, possible influences of reactive oxygen species (ROS) on the NMDA-R itself have so far been neglected. The objective of this study was to examine how the cerebroendothelial NMDA-R is affected by exposure to oxidative stress and to assess possible influences on BBB integrity. RT-PCR confirmed several NMDA-R subunits (NR1, NR2B-D) expressed in the bEnd3 cell line (murine cerebrovascular endothelial cells). NR1 protein expression after exposure to ROS was ob…

N-MethylaspartateEndotheliumBlotting WesternGlutamic AcidStimulationApoptosismedicine.disease_causeBlood–brain barrierBiochemistryReceptors N-Methyl-D-AspartateImmunoenzyme Techniqueschemistry.chemical_compoundMicePhysiology (medical)medicineAnimalsRNA MessengerCells Culturedchemistry.chemical_classificationReactive oxygen speciesChemistrySuperoxideReverse Transcriptase Polymerase Chain ReactionCell biologyOxidative Stressmedicine.anatomical_structurenervous systemBiochemistryBlood-Brain BarrierCerebrovascular CirculationNMDA receptorEndothelium VascularReactive Oxygen SpeciesPeroxynitriteOxidative stressFree radical biologymedicine
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β-Aminobutyric Acid Primes an NADPH Oxidase–Dependent Reactive Oxygen Species Production During Grapevine-Triggered Immunity

2010

International audience; The molecular mechanisms underlying the process of priming are poorly understood. In the present study, we investigated the early signaling events triggered by β-aminobutyric acid (BABA), a well-known priming-mediated plant resistance inducer. Our results indicate that, in contrast to oligogalacturonides (OG), BABA does not elicit typical defense-related early signaling events nor defense-gene expression in grapevine. However, in OG-elicited cells pretreated with BABA, production of reactive oxygen species (ROS) and expression of the respiratory-burst oxidase homolog RbohD gene were primed. In response to the causal agent of downy mildew Plasmopara viticola, a strong…

NADPH OXIDASE-DEPENDENTPhytophthora0106 biological sciencesACIDE β-AMINOBUTYRIQUEPhysiologyArabidopsisBiology01 natural sciencesAminobutyric acid03 medical and health sciencesImmunityTobaccoGene expression[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyVitisDNA Primers030304 developmental biologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesOxidase testNADPH oxidaseReverse Transcriptase Polymerase Chain ReactionAminobutyratesNADPH OxidasesHydrogen PeroxideGeneral MedicineKineticsEnzymeBiochemistrychemistryBABAbiology.proteinCalciumSignal transductionReactive Oxygen SpeciesAgronomy and Crop ScienceRESISTANCE010606 plant biology & botanyMolecular Plant-Microbe Interactions®
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Loss of Nrf2 in bone marrow-derived macrophages impairs antigen-driven CD8+ T cell function by limiting GSH and Cys availability

2015

NF-E2-related factor 2 (Nrf2), known to protect against reactive oxygen species, has recently been reported to resolve acute inflammatory responses in activated macrophages. Consequently, disruption of Nrf2 promotes a proinflammatory macrophage phenotype. In the current study, we addressed the impact of this macrophage phenotype on CD8(+) T cell activation by using an antigen-driven coculture model consisting of Nrf2(-/-) and Nrf2(+/+) bone marrow-derived macrophages (BMDMΦ) and transgenic OT-1 CD8(+) T cells. OT-1 CD8(+) T cells encode a T cell receptor that specifically recognizes MHC class I-presented ovalbumin OVA(257-264) peptide, thereby causing a downstream T cell activation. Interes…

NF-E2-Related Factor 2OvalbuminAntiporterT cellBlotting WesternReceptors Antigen T-CellApoptosisMice TransgenicCD8-Positive T-LymphocytesBiologyReal-Time Polymerase Chain Reactionenvironment and public healthBiochemistryAntioxidantsImmunoenzyme TechniquesMicechemistry.chemical_compoundBone MarrowPhysiology (medical)MHC class ImedicineAnimalsCytotoxic T cellRNA MessengerCells CulturedCell ProliferationMice KnockoutReverse Transcriptase Polymerase Chain ReactionGCLMMacrophagesHistocompatibility Antigens Class IGlutathionerespiratory systemFlow CytometryGlutathioneMolecular biologyMice Inbred C57BLOxidative Stressmedicine.anatomical_structurechemistrybiology.proteinCystineReactive Oxygen SpeciesIntracellularCD8Signal TransductionFree Radical Biology and Medicine
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Redox signaling in acute pancreatitis

2015

Acute pancreatitis is an inflammatory process of the pancreatic gland that eventually may lead to a severe systemic inflammatory response. A key event in pancreatic damage is the intracellular activation of NF-κB and zymogens, involving also calcium, cathepsins, pH disorders, autophagy, and cell death, particularly necrosis. This review focuses on the new role of redox signaling in acute pancreatitis. Oxidative stress and redox status are involved in the onset of acute pancreatitis and also in the development of the systemic inflammatory response, being glutathione depletion, xanthine oxidase activation, and thiol oxidation in proteins critical features of the disease in the pancreas. On th…

NecrosisGSH reduced glutathioneSTAT3 signal transducer and activator of transcription 3ERK extracellular signal-regulated kinasesClinical BiochemistryCCK cholecystokininTRAFs TNF receptor associated factorsReview ArticleIκB kinasePharmacologymedicine.disease_causeBiochemistrySHP small heterodimer partnerSTIM1 stromal interaction molecule 1chemistry.chemical_compoundHATs histone acetyltransferasesMedicineASK1GCL glutamate cysteine ligaseTNF-α tumor necrosis factor alphaIKK IκB kinaseNOS nitric oxide synthaseAcute inflammationHIF hypoxia inducible factorlcsh:QH301-705.5NF-κB nuclear factor kappa BDAMPs damage-associated molecular pattern moleculeslcsh:R5-920biologyGSSG oxidized glutathioneNF-kappa BNLRs nucleotide-binding oligomerization domain (NOD) like receptorsTRADD tumor necrosis factor receptor type 1-associated DEATH domain proteinTRPC3 transient receptor potential channel 3VEGF vascular endothelial growth factorGlutathioneTNFR tumor necrosis factor receptorHMGB1 high-mobility group Box 1 proteinIP3R inositol 145-trisphosphate receptor type 3VCAM-1 Vascular Cell adhesion protein 1Acute DiseaseJNK c-Jun N-terminal kinaseAcute pancreatitisTLRs toll-like receptorsmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionAP-1 activator protein-1Signal TransductionmRNA messenger ribonucleic acidHMGB1ASC apoptosis-associated speck-like protein containing a carboxy-terminal CARDRNS reactive nitrogen speciesPTPs protein tyrosine phosphatasesROS reactive oxygen speciesNADH nicotinamide adenine dinucleotidepHe extracellular pHFAEE fatty acid ethyl estersAP acute pancreatitisHumansXanthine oxidaseCBP CREB-binding proteinRyR endoplasmic reticulum membrane ryanodine receptorsMDA malondialdehydeNO nitric oxideXO xanthine oxidaseASK1 apoptosis signal-regulating kinase-1business.industryOrganic ChemistryAutophagyNADPH nicotinamide adenine dinucleotide phosphateHDACs histone deacetylasesmedicine.diseaseCARS compensatory anti-inflammatory response syndromeXDH xanthine dehydrogenaseIL interleukinIκB inhibitor of kappa BAcute pancreatitisETC Electron transport chainPancreatitisMKPs MAPK phosphatasesSAP severe acute pancreatitischemistrylcsh:Biology (General)DTT dithiothreitolOxidative stressNAC N-acetyl cysteineImmunologybiology.proteinCalciumLysosomesReactive Oxygen SpeciesbusinessMAPK mitogen-activated protein kinaseOxidative stressERCP endoscopic retrograde cholangiopancreatographyRedox Biology
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The role of oxidative stress in pro-inflammatory activation of human endothelial cells on Ti6Al4V alloy

2013

Inflammation is an important step in the early phase of tissue regeneration around an implanted metallic orthopaedic device. However, prolonged inflammation, which can be induced by metallic corrosion products, can lead to aseptic loosening and implant failure. Cells in peri-implant tissue as well as metal corrosion can induce reactive oxygen species (ROS) formation, thus contributing to an oxidative microenvironment around an implant. Understanding cellular reactions to implant-induced oxidative stress and inflammatory activation is important to help prevent an adverse response to metallic materials. In an earlier study we have shown that endothelial cells grown on Ti6Al4V alloy are subjec…

NecrosisMaterials scienceBiophysicsEnzyme-Linked Immunosorbent AssayBioengineeringInflammationOxidative phosphorylationmedicine.disease_causeAntioxidantsCell LineBiomaterialsTissue cultureAlloysmedicineHumansTitaniumchemistry.chemical_classificationReactive oxygen speciesReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaInterleukin-8Endothelial CellsIntercellular Adhesion Molecule-1Cell biologyEndothelial stem cellOxidative StresschemistryMechanics of MaterialsCell cultureImmunologyCeramics and Compositesmedicine.symptomReactive Oxygen SpeciesOxidative stressBiomaterials
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Reactive oxygen species induced by beauvericin, patulin and zearalenone in CHO-K1 cells

2008

The cytotoxic effects of mycotoxins, induction of reactive oxygen species (ROS) and generation of lipid peroxidation products in CHO-K1 cells were determined as function of increasing time of exposure and concentrations of beauvericin (BEA), patulin (PAT) and zearalenone (ZEA). The end points were evaluated after 24h of exposure, by the tetrazolium salt (MTT) and neutral red (NR) assays. The IC(50) values obtained on the MTT and NR assays ranged from 0.69 to 79.40 microM and 4.40 to 108.76 microM, respectively. To determine the intracellular production of ROS, the intensity of fluorescence emitted from the probe H(2)-DCFDA was measured. The relative intensity of fluorescence from cells incu…

Neutral redThiobarbituric acidCHO CellsToxicologyPatulinLipid peroxidationchemistry.chemical_compoundCricetulusCricetinaeDepsipeptidesMalondialdehydeTBARSAnimalschemistry.chemical_classificationReactive oxygen speciesfood and beveragesGeneral MedicineMycotoxinsMalondialdehydeBeauvericinPatulinchemistryBiochemistryZearalenoneLipid PeroxidationReactive Oxygen SpeciesToxicology in Vitro
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