Search results for "REACTIVE OXYGEN SPECIES"

showing 10 items of 879 documents

Water Extract of Cryphaea heteromalla (Hedw.) D. Mohr Bryophyte as a Natural Powerful Source of Biologically Active Compounds

2019

Bryophytes comprise of the mosses, liverworts, and hornworts. Cryphaea heteromalla, (Hedw.) D. Mohr, is a non-vascular lower plant belonging to mosses group. To the date, the most chemically characterized species belong to the liverworts, while only 3.2% and 8.8% of the species belonging to the mosses and hornworts, respectively, have been investigated. In this work, we present Folin–Ciocalteu and oxygen radical absorbance capacity (ORAC) data related to crude extracts of C. heteromalla obtained by three di erent extraction solvents: pure water (WT), methanol:water (80:20 v/v) (MET), and ethanol:water (80:20 v/v) (ETH). The water extract proved to be the best solvent showing the highest con…

Oxygen radical absorbance capacityBryophytaCoumaric acidMass spectrometryCatalysisArticleInorganic ChemistryExtract HPLC-ESI-TOF-MS analysilcsh:Chemistrychemistry.chemical_compoundCryphaea heteromalla; bryophytes; extract HPLC-ESI-TOF-MS analysis; oxidative stress evaluationMiceCryphaea heteromallabryophytesAnimalsPhysical and Theoretical ChemistryMolecular Biologylcsh:QH301-705.5SpectroscopyExtract HPLC-ESI-TOF-MS analysisChromatographyEthanolChemistryPlant ExtractsOxidative stress evaluationOrganic ChemistryExtraction (chemistry)Bryophytesoxidative stress evaluationGeneral MedicineFree Radical Scavengersextract hplc-esi-tof-ms analysisComputer Science ApplicationsSolventOxidative Stresslcsh:Biology (General)lcsh:QD1-999<i>cryphaea heteromalla</i>BryophyteSettore BIO/03 - Botanica Ambientale E ApplicataNIH 3T3 CellsBryophyteMethanolReactive Oxygen SpeciesInternational Journal of Molecular Sciences
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SYNTHESIS OF PYRROLO[3,2-H]QUINOLINONES WITH GOOD PHOTOCHEMOTHERAPEUTIC ACTIVITY AND NO DNA DAMAGE

2010

In the search for new photochemotherapeutic agents, a series of derivatives of the ring system pyrrolo[3,2-h]quinoline--bioisosters of the angular furocoumarin angelicin--were synthesized through a four-step synthetic approach, in reasonable overall yields. Eight of the synthesized derivatives showed a remarkable phototoxicity against a panel of four human tumor cell lines and a great dose UV-A dependence, reaching IC₅₀ values at submicromolar level. The mode of cellular death photoinduced by pyrrolo[3,2-h]quinolines was evaluated through a series of flow cytometric analysis and other tests were performed to clarify their mechanism of action.

PYRROLO[32-H]QUINOLINONESStereochemistryDNA damageClinical BiochemistryPharmaceutical SciencePhosphatidylserinesBiochemistryChemical synthesischemistry.chemical_compoundCell Line TumorFurocoumarinsDrug Discovery2-H]QUINOLINONESmedicineHumansPyrrolesPhotosensitizerMolecular BiologyMembrane Potential MitochondrialPhotosensitizing AgentsPYRROLO[3; 2-H]QUINOLINONES; ANGELICIN HETEROANALOGUES; PHOTOCHEMOTHERAPY; PHOTOTOXICITYFurocoumarinOrganic ChemistryBiological activitySettore CHIM/08 - Chimica FarmaceuticaPHOTOCHEMOTHERAPYPHOTOTOXICITYPYRROLO[3ANGELICIN HETEROANALOGUESMechanism of actionchemistryQuinolinesLactamMolecular Medicinemedicine.symptomReactive Oxygen SpeciesPhototoxicityDNA Damage
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An in vitro procedure for evaluation of early stage oxidative stress in an established fish cell line applied to investigation of PHAH and pesticide …

2004

Oxidative stress by increased production of reactive oxygen species such as superoxide has been implicated in the toxicity of PCB's and non-target toxicity of many pesticides. We report the development of a microplate-based method for determination of early stage oxidative stress using an established cell line (EPC) from a skin tumour of carp Cyprinus carpio L. and 2',7'-dichlorodihydrofluorescein diacetate (H(2)-DCFDA) as a fluorescent probe for detection of reactive oxygen species (ROS) formation. Sublethal concentrations of the herbicide Paraquat, an established redox cycling agent and a crude PCB mixture, Arochlor 1254 elicited a linear increase in ROS formation over 2 h exposure which …

ParaquatAroclorsCarpsTime FactorsAquatic ScienceBiologyIn Vitro TechniquesOceanographymedicine.disease_causePesticide toxicityToxicologychemistry.chemical_compoundParaquatToxicity TestsmedicineTumor Cells CulturedAnimalsPesticidesFluorescent Dyeschemistry.chemical_classificationReactive oxygen speciesSuperoxideGeneral MedicineFluoresceinsPollutionpeople.cause_of_deathAcute toxicityOxidative StresschemistryBiochemistryToxicityMalathionpeopleReactive Oxygen SpeciesOxidative stressMarine environmental research
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Effects of pharmacological agents on the lifespan phenotype of Drosophila DJ-1beta mutants.

2010

Mutations in the DJ-1 gene cause autosomal recessive, early-onset Parkinsonism. The DJ-1 protein exerts a protective role against oxidative stress damage, working as a cellular oxidative stress sensor, and it seems to regulate gene expression at different levels. In Drosophila, two DJ-1 orthologs have been identified: DJ-1β and DJ-1β. Several studies have shown that loss of DJ-1β function causes Parkinson's disease (PD)-like phenotypes in flies such as age-dependent locomotor defects, reduced lifespan, and enhanced sensitivity to toxins that induce oxidative stress, like the herbicide paraquat. However, no dopaminergic neurodegeneration is observed. These results suggested that both locomot…

ParaquatDopamineMutantOxidative phosphorylationBiologymedicine.disease_causeAntioxidantsLipid peroxidationchemistry.chemical_compoundParkinsonian DisordersGeneticsmedicineAnimalsGeneticschemistry.chemical_classificationReactive oxygen speciesNeurodegenerationDopaminergicBrainParkinson DiseaseGeneral Medicinemedicine.diseasePhenotypeCell biologyOxidative StressPhenotypechemistryMutationDrosophilaReactive Oxygen SpeciesOxidative stressGene
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Hypothesis: can N-acetylcysteine be beneficial in Parkinson's disease?

1999

Based on the finding of decreased mitochondrial complex I activity in the substantia nigra of patients with Parkinson's disease, we propose that the consequent reduction of ATP synthesis and increased generation of reactive oxygen species may be a possible cause of nigrostriatal cell death. Since sulfhydryl groups are essential in oxidative phosphorylation, thiolic antioxidants may contribute to the preservation of these proteins against oxidative damage. In the present paper, we hypothesize that treatment with a sulfur-containing antioxidant such as N-acetylcysteine may provide a new neuroprotective therapeutic strategy for Parkinson's disease.

Parkinson's diseaseAntioxidantmedicine.medical_treatmentModels NeurologicalSubstantia nigraOxidative phosphorylationPharmacologyBiologyMitochondrionNeuroprotectionGeneral Biochemistry Genetics and Molecular BiologyOxidative PhosphorylationAcetylcysteineAdenosine TriphosphatemedicineNAD(P)H Dehydrogenase (Quinone)HumansGeneral Pharmacology Toxicology and Pharmaceuticschemistry.chemical_classificationReactive oxygen speciesParkinson DiseaseGeneral Medicinemedicine.diseaseCorpus StriatumAcetylcysteineMitochondriaSubstantia NigraNeuroprotective AgentschemistryReactive Oxygen SpeciesNeurosciencemedicine.drugLife sciences
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From clinical description, to in vitro and animal studies, and backward to patients: Oxidative stress and mitochondrial dysfunction in Fanconi anemia

2013

Fanconi anemia (FA) is a rare genetic disease associated with deficiencies in DNA repair pathways. A body of literature points to a pro-oxidant state in FA patients, along with evidence for oxidative stress (OS) in the FA phenotype reported by in vitro, molecular, and animal studies. A highlight arises from the detection of mitochondrial dysfunction (MDF) in FA cell lines of complementation groups A, C, D2, and G. As yet lacking, in vivo studies should focus on FA-associated MDF, which may help in the understanding of the mitochondrial basis of OS detected in cells and body fluids from FA patients. Beyond the in vitro and animal databases, the available analytical devices may prompt the dir…

Pathologymedicine.medical_specialtyDNA RepairFree RadicalsDNA repairmitochondrial nutrientsCell Cycle ProteinsFree radicalsDiseaseBiologymedicine.disease_causeBioinformaticsBiochemistryChemopreventionPathogenesis03 medical and health sciencesMice0302 clinical medicineIn vivoFanconi anemiaPhysiology (medical)medicineAnimalsHumans030304 developmental biology0303 health sciencesMitochondrial nutrientNuclear ProteinsFanconi anemia Mitochondrial dysfunction Mitochondrial nutrients Chemoprevention Free radicalsmedicine.diseasePhenotype3. Good healthMitochondriaOxidative StressFanconi Anemia030220 oncology & carcinogenesisFanconi anemiaAnimal studiesReactive Oxygen SpeciesMitochondrial dysfunctionOxidative stress
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Role of mitochondria in parvovirus pathology.

2014

Proper functioning of the mitochondria is crucial for the survival of the cell. Viruses are able to interfere with mitochondrial functions as they infect the host cell. Parvoviruses are known to induce apoptosis in infected cells, but the role of the mitochondria in parvovirus induced cytopathy is only partially known. Here we demonstrate with confocal and electron microscopy that canine parvovirus (CPV) associated with the mitochondrial outer membrane from the onset of infection. During viral entry a transient depolarization of the mitochondrial transmembrane potential and increase in ROS level was detected. Subsequently, mitochondrial homeostasis was normalized shortly, as detected by rep…

PathologyvirusesCelllcsh:MedicineMitochondrionSignal transductionERK signaling cascadeMolecular cell biologyInner mitochondrial membraneExtracellular Signal-Regulated MAP Kinaseslcsh:SciencepatologiaCellular Stress ResponsesMembrane Potential MitochondrialMultidisciplinarybiologyCell DeathCanine parvovirusapoptosisSignaling cascadesCellular StructuresCell biologyMitochondriaHost-Pathogen Interactionmedicine.anatomical_structureMitochondrial MembranesResearch Articlemedicine.medical_specialtyViral EntryParvovirus CanineMAP Kinase Signaling SystemmitokondriotMicrobiologyCell LineParvoviridae InfectionsDogsViral entryVirologymedicineAnimalsBiologysoluviestintäParvovirusta1183parvoviruslcsh:Rta1182biology.organism_classificationMolecular biologyEnzyme ActivationViral replicationSubcellular OrganellesApoptosisCatsCalciumlcsh:QReactive Oxygen SpeciesViral Transmission and InfectionPLoS ONE
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Exercise acts as a drug; the pharmacological benefits of exercise.

2012

The beneficial effects of regular exercise for the promotion of health and cure of diseases have been clearly shown. In this review, we would like to postulate the idea that exercise can be considered as a drug. Exercise causes a myriad of beneficial effects for health, including the promotion of health and lifespan, and these are reviewed in the first section of this paper. Then we deal with the dosing of exercise. As with many drugs, dosing is extremely important to get the beneficial effects of exercise. To this end, the organism adapts to exercise. We review the molecular signalling pathways involved in these adaptations because understanding them is of great importance to be able to pr…

Pharmaceutical PreparationsLongevityAnimalsHumansReviewsMuscle SkeletalReactive Oxygen SpeciesAdaptation PhysiologicalExerciseBritish journal of pharmacology
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Activity of three cytotoxic isoflavonoids from Erythrina excelsa and Erythrina senegalensis (neobavaisoflavone, sigmoidin H and isoneorautenol) towar…

2013

Abstract Introduction Resistance of cancer cells to chemotherapy has become a worldwide concern. Naturally occuring isoflavonoids possess a variety of biological activities including anti-cancer effects. The present study was aimed at investigating the cytotoxicity and the modes of action of three naturally occuring isoflavonoids, neobavaisoflavone ( 1 ), sigmoidin H ( 2 ) and a pterocarpan that is a special type of isoflavonoid, isoneorautenol ( 3 ) against a panel of nine cancer cell lines, including various sensitive and drug-resistant phenotypes. Methods The cytotoxicity of the compounds was determined using a resazurin reduction assay, whereas the caspase-Glo assay was used to detect t…

Pharmaceutical ScienceApoptosisIsoflavonoidDrug DiscoveryHumansCytotoxic T cellBenzopyransCytotoxicityCaspaseBenzofuransErythrinaMembrane Potential MitochondrialPharmacologybiologyCell CyclePterocarpanHCT116 CellsAntineoplastic Agents PhytogenicIsoflavonesMolecular biologyComplementary and alternative medicineBiochemistryDrug Resistance NeoplasmCell cultureApoptosisCaspasesCancer cellbiology.proteinMolecular MedicineDrug Screening Assays AntitumorReactive Oxygen SpeciesPhytomedicine
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Synthesis and photocytotoxic activity of [1,2,3]triazolo[4,5-h][1,6]naphthyridines and [1,3]oxazolo[5,4-h][1,6]naphthyridines

2018

Abstract [1,2,3]Triazolo[4,5-h][1,6]naphthyridines and [1,3]oxazolo[5,4-h][1,6]naphthyridines were synthesized with the aim to investigate their photocytotoxic activity. Upon irradiation, oxazolo-naphtapyridines induced light-dependent cell death at nanomolar/low micromolar concentrations (EC50 0.01–6.59 μM). The most photocytotoxic derivative showed very high selectivity and photocytotoxicity indexes (SI = 72–86, PTI>5000), along with a triplet excited state with exceptionally long lifetime (18.0 μs) and high molar absorptivity (29781 ± 180 M−1cm−1 at λmax 315 nm). The light-induced production of ROS promptly induced an unquenchable apoptotic process selectively in tumor cells, with mitoch…

Pharmaceutical ScienceApoptosisMitochondrionPhotochemiotherapy; Photosensitizing agents; Reactive oxygen species; [123]Triazolo[45-h][16]naphthyridines; [13]oxazolo[54-h][16]naphthyridines; Pharmacology; Drug Discovery; Pharmaceutical Science; Organic Chemistry01 natural sciencesMedicinal chemistry[13]oxazolo[54-h][16]naphthyridinechemistry.chemical_compoundDrug Discovery6]naphthyridineschemistry.chemical_classification0303 health sciencesTumorPhotosensitizing AgentsCell DeathSinglet OxygenSinglet oxygenPhotochemiotherapy; Photosensitizing agents; Reactive oxygen species; [1; 2; 3]Triazolo[4; 5-h][1; 6]naphthyridines; [1; 3]oxazolo[5; 4-h][1; 6]naphthyridines; Apoptosis; Cell Death; Cell Line; Tumor; Humans; Lysosomes; Mitochondria; Naphthyridines; Photochemotherapy; Photosensitizing Agents; Reactive Oxygen Species; Singlet OxygenGeneral MedicineLysosomeMitochondriaExcited stateReactive oxygen specie5-h][1HumanProgrammed cell death2NaphthyridinePhotochemiotherapy3]Triazolo[4Cell Line03 medical and health sciences4-h][1Cell Line TumorHumansNaphthyridines030304 developmental biologyPharmacologyReactive oxygen speciesPhotosensitizing agent010405 organic chemistryOrganic ChemistryApoptosi0104 chemical sciences3]oxazolo[5chemistryPhotochemotherapyCell cultureApoptosis[123]Triazolo[45-h][16]naphthyridine[1LysosomesReactive Oxygen SpeciesDerivative (chemistry)
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