Search results for "RECESS"

showing 10 items of 379 documents

Homozygous Familial Hypercholesterolemia in Spain Prevalence and Phenotype-Genotype Relationship

2016

Background— Homozygous familial hypercholesterolemia (HoFH) is a rare disease characterized by elevated plasma levels of low-density lipoprotein cholesterol (LDL-C) and extremely high risk of premature atherosclerotic cardiovascular disease. HoFH is caused by mutations in several genes, including LDL receptor ( LDLR ), apolipoprotein B ( APOB ), proprotein convertase subtilisin/kexin type 9 ( PCSK9 ), and LDL protein receptor adaptor 1 ( LDLRAP1 ). No epidemiological studies have assessed HoFH prevalence or the clinical and molecular characteristics of this condition. Here, we aimed to characterize HoFH in Spain. Methods and Results— Data were collected from the Spanish Dyslipidemia Regist…

Male0301 basic medicineOncologyLdl receptor geneApolipoprotein BLipid-lowering therapyFamilial hypercholesterolemia030204 cardiovascular system & hematologyCompound heterozygosity0302 clinical medicineAutosomal-dominant hypercholesterolemiaRisk FactorsEpidemiologyPrevalenceDiseaseRegistriesGenetics (clinical)Molecular EpidemiologybiologyhypercholesterolemiaHomozygoteDouble-blindMiddle AgedPhenotypeCardiovascular DiseasesApolipoprotein B-100allelesFemalelipids (amino acids peptides and proteins)Proprotein Convertase 9Cardiology and Cardiovascular MedicineMutationsAdultGenetic MarkersHeterozygotemedicine.medical_specialtyInhibitorAdolescentPlacebo-controlled trialHyperlipoproteinemia Type IIlipidsYoung Adult03 medical and health sciencesInternal medicineGeneticsmedicineHumansGenetic Predisposition to DiseaseAlleleAdaptor Proteins Signal TransducingRecessive hypercholesterolemiaPCSK9registriesCholesterol LDLApolipoprotein-bmedicine.disease030104 developmental biologyEndocrinologyReceptors LDLSpainMutationLDL receptorbiology.proteinmutationDyslipidemia
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Proprotein Convertase Subtilisin Kexin Type 9 Inhibition for Autosomal Recessive Hypercholesterolemia—Brief Report

2016

Objective— Proprotein convertase subtilisin kexin type 9 (PCSK9) inhibitors lower low-density lipoprotein (LDL) cholesterol in the vast majority of patients with autosomal dominant familial hypercholesterolemia. Will PCSK9 inhibition with monoclonal antibodies, in particular alirocumab, be of therapeutic value for patients with autosomal recessive hypercholesterolemia (ARH)? Approach and Results— Primary lymphocytes were obtained from 28 genetically characterized ARH patients and 11 controls. ARH lymphocytes treated with mevastatin were incubated with increasing doses of recombinant PCSK9 with or without saturating concentrations of alirocumab. Cell surface LDL receptor expression measured…

Male0301 basic medicineSettore MED/09 - Medicina Interna[SDV]Life Sciences [q-bio]receptorsalirocumabFamilial hypercholesterolemia030204 cardiovascular system & hematologyproprotein convertase subtilisin kexin type 90302 clinical medicinetherapeuticsLymphocytesCells CulturedhypercholesterolemiaAnticholesteremic AgentsPCSK9 InhibitorsAntibodies MonoclonalMiddle Aged3. Good healthPhenotypeAutosomal Recessive HypercholesterolemiaKexinDrug Therapy CombinationFemalelipids (amino acids peptides and proteins)LovastatinProprotein Convertase 9Cardiology and Cardiovascular Medicinemedicine.drugAdultmedicine.medical_specialtySerine Proteinase InhibitorsAdolescentBiologyAntibodies Monoclonal HumanizedLDLYoung Adult03 medical and health sciencesInternal medicinemedicineHumansGenetic Predisposition to DiseaseLovastatinAdaptor Proteins Signal TransducingAlirocumabPCSK9receptors LDLCholesterol LDLmedicine.diseaseProprotein convertasetherapeutic030104 developmental biologyEndocrinologyCase-Control Studiesalirocumab; hypercholesterolemia; proprotein convertase subtilisin kexin type 9; receptors LDL; therapeutics; Cardiology and Cardiovascular MedicineMutationLDL receptorHydroxymethylglutaryl-CoA Reductase InhibitorsArteriosclerosis, Thrombosis, and Vascular Biology
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Differences in physical activity at recess and school-related social factors in four Finnish lower secondary schools

2017

This study investigated the differences in physical activity (PA) at recess and school-related social factors, and described school PA promotion processes and staff experiences at four lower secondary schools from the Finnish Schools on the Move programme. Recess PA, peer relationships at school, relatedness to school, and school climate were assessed via surveys with eighth-grade students in spring 2011 (n ¼ 385) and spring 2013 (n ¼ 373). Local contact people in the school projects (n ¼ 6), school staff (n ¼ 83) and principals (n ¼ 3) provided information on the PA promotion process via telephone interviews and surveys. Differences in student-level data in years 2011 and 2013 were analyse…

MaleAdolescentmedia_common.quotation_subjecteducationphysical activityOrganizational culturekouluyhteisöSocial EnvironmentPeer GroupEducation03 medical and health sciencesInterpersonal relationship0302 clinical medicinePromotion (rank)Sex FactorsSurveys and QuestionnairesEmployee engagementHumansta516lower secondary schoolsNarrativeInterpersonal Relations030212 general & internal medicineta315ChildStudentssocial factorsExerciseFinlandSocial influencemedia_commonMedical educationSchoolsrecessPublic Health Environmental and Occupational HealthSocial environmentPeer group030229 sport sciencesOriginal ArticlesvälitunnitOrganizational CultureFemaleyläkouluPsychologySocial psychologyfyysinen aktiivisuusHealth Education Research
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Optimizing clinical exome design and parallel gene-testing for recessive genetic conditions in preconception carrier screening: Translational researc…

2019

Limited translational genomic research data have been reported on the application of exome sequencing and parallel gene testing for preconception carrier screening (PCS). Here, we present individual-level data from a large PCS program in which exome sequencing was routinely performed on either gamete donors (5,845) or infertile patients (8,280) undergoing in vitro fertilization (IVF) treatment without any known family history of inheritable genetic conditions. Individual-level data on pathogenic variants were used to define conditions for PCS based on criteria for severity, penetrance, inheritance pattern, and age of onset. Fetal risk was defined based on actual carrier frequency data accou…

MaleCancer ResearchGenetic ScreensHeredityGenetic LinkageMolecular biologyGenetic Carrier ScreeningGene Identification and AnalysisGene SequencingQH426-470BioinformaticsPathology and Laboratory MedicineTranslational Research Biomedical0302 clinical medicineSequencing techniquesMedicine and Health SciencesExomeDNA sequencingGenome SequencingChildExomeGenetics (clinical)Exome sequencing0303 health scienceseducation.field_of_studymedicine.diagnostic_testGenetic Carrier ScreeningGenomicsPenetranceX-Linked TraitsSex LinkageChild PreschoolMedical geneticsFemalePathogensResearch ArticleAdultmedicine.medical_specialtyHeterozygotePopulationGenes RecessiveBiology03 medical and health sciencesGenomic MedicineDirected Tissue DonationExome SequencingmedicineGeneticsHumansGenetic Predisposition to DiseaseGenetic TestingeducationEcology Evolution Behavior and Systematics030304 developmental biologyGenetic testingClinical GeneticsGenome HumanInfant NewbornBiology and Life SciencesInfantHuman geneticsResearch and analysis methodsMolecular biology techniquesInfertilityGenetics of DiseaseMutation030217 neurology & neurosurgeryPLoS Genetics
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Periodontal clinical and microbiological data in desquamative gingivitis patients.

2013

Objectives: A series of patients affected by desquamative gingivitis (DG) was investigated in order to evaluate relation patterns among clinical parameters relevant to plaque induced periodontitis, periodontal microbiological data and the presence of DG lesions. Patients and methods: Eight oral lichen planus (OLP) and four mucous membrane pemphigoid (MMP) patients were examined. Periodontal measurements (performed at six sites per tooth on all teeth) included probing depth (PD), gingival recession (REC), clinical attachment loss (CAL) and full mouth plaque (FMPS) and bleeding (FMBS) scores; the presence and the exact location (site by site) of DG lesions were carefully recorded. Sub-gingiva…

MaleDesquamative gingivitiMucous membrane pemphigoidSettore MED/50 - Scienze Tecniche Mediche ApplicateDentistrySettore MED/28 - Malattie OdontostomatologicheMedicineHumansGeneral DentistryGingival recessionAgedPeriodontitisbiologybusiness.industryPeriodontal pathogensSignificant differenceAggregatibacter actinomycetemcomitansMiddle Agedmedicine.diseasebiology.organism_classificationGingivitisDesquamative gingivitisstomatognathic diseasesPeriodontal statuOral lichen planuClinical attachment lossMucous membrane pemphigoidOral lichen planusFemalemedicine.symptombusinessClinical oral investigations
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Economic cycles and inequalities in alcohol‐related mortality in the Baltic countries and Finland in 2000–2015: a register‐based study

2021

Publisher Copyright: © 2021 The Authors. Addiction published by John Wiley & Sons Ltd on behalf of Society for the Study of Addiction. Aim: To estimate whether large macroeconomic fluctuations in the 2000s affected inequalities in alcohol-related mortality in the Baltic countries and Finland. Design: Longitudinal register-based follow-up study. Setting: Estonia, Latvia, Lithuania and Finland. Participants: General population in the 35–74 age group. Measurements: Socioeconomic status was measured by the highest achieved educational level and was categorised using the International Standard Classification of Education 2011 as low (included categories 0–2), middle (3–4), and high (5–8). Educat…

MaleEconomic expansionInequalitymedia_common.quotation_subjectPopulation030508 substance abuseMedicine (miscellaneous)Recession03 medical and health sciences0302 clinical medicineinequalitiesHumansregister-based030212 general & internal medicineMortalityeconomic expansioneducationSocioeconomic statusFinlandmedia_commoneducationeducation.field_of_studyMortality rate1. No povertyInternational Standard Classification of EducationLatvian3142 Public health care science environmental and occupational healthlanguage.human_language3. Good healthPsychiatry and Mental healthGeographySocial ClassSocioeconomic FactorslanguageAlcohol-related mortalityEducational StatusFemalerecession0305 other medical scienceFollow-Up StudiesDemographyAddiction
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Next-generation sequencing confirms the implication of SLC24A1 in autosomal-recessive congenital stationary night blindness.

2015

Congenital stationary night blindness (CSNB) is a clinically and genetically heterogeneous retinal disorder which represents rod photoreceptor dysfunction or signal transmission defect from photoreceptors to adjacent bipolar cells. Patients displaying photoreceptor dysfunction show a Riggs-electroretinogram (ERG) while patients with a signal transmission defect show a Schubert-Bornschein ERG. The latter group is subdivided into complete or incomplete (ic) CSNB. Only few CSNB cases with Riggs-ERG and only one family with a disease-causing variant in SLC24A1 have been reported. Whole-exome sequencing (WES) in a previously diagnosed icCSNB patient identified a homozygous nonsense variant in SL…

MaleGenes RecessiveSodium-Calcium ExchangerNight BlindnessElectroretinographyMyopiaHumansExomeGenetic Predisposition to DiseaseAmino Acid SequenceSLC24A1Family HealthHigh-throughput sequencingBase SequenceSequence Homology Amino AcidSettore MED/30 - Malattie Apparato VisivoHomozygoteHigh-Throughput Nucleotide SequencingEye Diseases HereditaryGenetic Diseases X-LinkedPedigreeNight BlindneMutationFemaleCongenital stationary night blindneHumanClinical genetics
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ALDH1A3 Mutations Cause Recessive Anophthalmia and Microphthalmia

2013

Anophthalmia and microphthalmia (A/M) are early-eye-development anomalies resulting in absent or small ocular globes, respectively. A/M anomalies occur in syndromic or nonsyndromic forms. They are genetically heterogeneous, some mutations in some genes being responsible for both anophthalmia and microphthalmia. Using a combination of homozygosity mapping, exome sequencing, and Sanger sequencing, we identified homozygosity for one splice-site and two missense mutations in the gene encoding the A3 isoform of the aldehyde dehydrogenase 1 (ALDH1A3) in three consanguineous families segregating A/M with occasional orbital cystic, neurological, and cardiac anomalies. ALDH1A3 is a key enzyme in the…

MaleGenetic LinkageRetinoic acidGenes RecessiveBiologymedicine.disease_causeMicrophthalmiachemistry.chemical_compoundsymbols.namesakeChromosome SegregationReportmedicineGeneticsFood and NutritionHumansMicrophthalmosMissense mutationGenetics(clinical)Genetics (clinical)Exome sequencingSanger sequencingGeneticsMutationAnophthalmiaHomozygoteAnophthalmosExonsSequence Analysis DNAAldehyde DehydrogenaseDisease gene identificationmedicine.diseaseAldehyde OxidoreductasesMolecular biologyIntronseye diseasesPedigreeHEK293 CellschemistryAlimentation et NutritionMutationsymbolsFemaleMutant Proteinssense organsThe American Journal of Human Genetics
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Esr, a second locus in the house mouse controlling esterase-5

1982

Electrophoretic variation characterized by the presence (ES-5B+) or absence (ES-5B−) of esterase-5B in the plasma of the house mouse has been observed. It is suggested that the expression of esterase-5B is controlled by an autosomal locus, Esr, linked to Ldr-1 on chromosome 6, in addition to the presumptive structural locus Es-5, which is located on chromosome 8. A gene order of Lyt-3-Esr-Ldr-1 was determined by two crosses.

MaleGeneticsGenetic LinkageEsterasesChromosome MappingGenes RecessiveLocus (genetics)General MedicineBiologybiology.organism_classificationBiochemistryMolecular biologyEsteraseHouse mouseMiceGene Expression RegulationGenesGenes RegulatorGeneticsAnimalsFemaleStructural locusMolecular BiologyGeneEcology Evolution Behavior and SystematicsBiochemical Genetics
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Sedaghatian congenital lethal metaphyseal chondrodysplasia—observations in a second Iranian family and histopathological studies

1987

In 1980, Sedaghatian described in two brothers and one sister a neonatally lethal disorder associated with slight rhizomelic limb shortness, mild platyspondyly, and severe metaphyseal dysplasia. Here data are presented on another Iranian infant with the Sedaghatian syndrome who died on day 4 and was found to have histologic evidence of severe epimetaphyseal dysplasia. The occurrence in children of both sexes in one instance, born to normal parents who were first cousins, and currently apparent confinement of the disorder to Iranians suggests that the Sedaghatian syndrome is an autosomal recessive trait with high gene frequency in Iranians. This may be a more complexly pleiotropic syndrome t…

MaleGeneticsmedicine.medical_specialtyDepressed nasal bridgeMetaphyseal chondrodysplasiabusiness.industryInfant NewbornShort neckConsanguinityIranOsteochondrodysplasiasmedicine.diseaseMetaphyseal dysplasiaDermatologyMicrophthalmiaBone and BonesAutosomal recessive traitCartilageDysplasiaotorhinolaryngologic diseasesmedicineHumansbusinessGenetics (clinical)American Journal of Medical Genetics
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