Search results for "RED"

showing 10 items of 23890 documents

Muscle deficits persist after unilateral knee replacement and have implications for rehabilitation.

2009

BackgroundKnee joint arthritis causes pain, decreased range of motion, and mobility limitation. Knee replacement reduces pain effectively. However, people with knee replacement have decreases in muscle strength (“force-generating capacity”) of the involved leg and difficulties with walking and other physical activities.Objective and DesignThe aim of this cross-sectional study was to determine the extent of deficits in knee extensor and flexor muscle torque and power (ability to perform work over time) and in the extensor muscle cross-sectional area (CSA) after knee joint replacement. In addition, the association of lower-leg muscle deficits with mobility limitations was investigated.Methods…

musculoskeletal diseasesMalemedicine.medical_specialtymedicine.medical_treatmentKnee replacementArthritisPhysical Therapy Sports Therapy and RehabilitationOsteoarthritisMuscle Strength DynamometerKnee JointWeight-BearingPhysical medicine and rehabilitationmedicineHumansMuscle StrengthMobility LimitationRange of Motion ArticularArthroplasty Replacement KneeMuscle SkeletalAgedRehabilitationbusiness.industryWork (physics)Middle AgedOsteoarthritis Kneemusculoskeletal systemmedicine.diseasePreferred walking speedTreatment OutcomePhysical therapyFemaleRange of motionbusinesshuman activitiesMuscle ContractionPhysical therapy
researchProduct

Influence of the Screw Positioning on the Stability of Locking Plate for Proximal Tibial Fractures: A Numerical Approach

2020

Tibial fractures are common injuries in people. The proper treatment of these fractures is important in order to recover complete mobility. The aim of this work was to investigate if screw positioning in plates for proximal tibial fractures can affect the stability of the system, and if it can consequently influence the patient healing time. In fact, a more stable construct could allow the reduction of the non-weight-bearing period and consequently speed up the healing process. For that purpose, virtual models of fractured bone/plate assemblies were created, and numerical simulations were performed to evaluate the reaction forces and the maximum value of the contact pressure at the screw/bo…

musculoskeletal diseasesMaterials sciencemedicine.medical_treatmentHealing timelcsh:TechnologyStability (probability)Locking platelcsh:Chemistry03 medical and health sciences0302 clinical medicinemedicineCADGeneral Materials Science030212 general & internal medicineTibial fractureSettore ING-IND/15 - Disegno E Metodi Dell'Ingegneria Industrialelcsh:QH301-705.5InstrumentationReduction (orthopedic surgery)implant stabilityFluid Flow and Transfer ProcessesOrthodonticsFEM030222 orthopedicslcsh:TProcess Chemistry and TechnologyWork (physics)General Engineeringtibial fracturemusculoskeletal systemlocking plateslcsh:QC1-999Finite element methodComputer Science Applicationsreverse engineeringlcsh:Biology (General)lcsh:QD1-999lcsh:TA1-2040Proper treatmentlcsh:Engineering (General). Civil engineering (General)lcsh:PhysicsLocking plateApplied Sciences
researchProduct

(i) Comminuted patellar fractures

2006

Summary Purpose of review This article reviews current best practice for the diagnosis and treatment of comminuted patellar fractures. Recent findings Patellar fractures make up about 1% of all fractures. As a rule, fractures of the patella are caused by direct trauma to the knee. A transverse fracture is the most common fracture type. Open reduction and internal fixation is the treatment of choice for the majority of displaced patellar fractures. Treatment must achieve anatomic reduction of the articular surface and reestablish the continuity of the extensor mechanism. Tension-band wiring, interfragmentary screw fixation and a combination of cerclage wiring and screw fixation are the most …

musculoskeletal diseasesOrthodonticsmedicine.medical_specialtybusiness.industryTension band wiringmedicine.medical_treatmentDirect traumamusculoskeletal systemmedicine.diseaseSurgerymedicineFracture (geology)Internal fixationOrthopedics and Sports MedicinePatellaDisplacement (orthopedic surgery)Patella fracturebusinessReduction (orthopedic surgery)Current Orthopaedics
researchProduct

Quasi-Fractional Models of Human Tendons Hereditariness

2018

In this study, the authors, after collecting a series of experimental evidences following a creep and relaxation tendon campaign, propose a non-linear model of the viscoelastic behavior of the tendons. The ligaments investigated are the patellars and the hamstrings. The analytical model proposed by the authors aims to explain the non-linear hereditary behavior of these tissues and proposes an approach with which to develop a hereditary fractional-order non-linear model.

musculoskeletal diseasesPatellarComputer scienceQuantitative Biology::Tissues and OrgansEnergy Engineering and Power TechnologyViscoelasticityIndustrial and Manufacturing EngineeringArtificial IntelligencemedicineLigamentInstrumentationbusiness.industryRenewable Energy Sustainability and the EnvironmentComputer Science Applications1707 Computer Vision and Pattern RecognitionStructural engineeringFractional-order hereditarinemusculoskeletal systemTendonmedicine.anatomical_structureComputer Networks and CommunicationCreepEvolution biologyRelaxation (approximation)businessSettore ICAR/08 - Scienza Delle CostruzioniHamstring
researchProduct

Sicilian pistachio (Pistacia vera L.) nut inhibits expression and release of inflammatory mediators and reverts the increase of paracellular permeabi…

2014

Background Dietary approaches to control inflammatory bowel diseases (IBD) may include proanthocyanidin-rich foods. Our previous research showed that a hydrophilic extract from Sicilian pistachio nut (HPE) contains sub- stantial amounts of proanthocyanidins and possesses anti- inflammatory activities. Purpose We studied the effects of HPE and of its poly- meric proanthocyanidin fraction (PPF) in a cell model that simulated some conditions of IBD, consisting of interleukin (IL)-1b-stimulated Caco-2 cells. Methods HPE was prepared by Pistacia vera L. nuts, and PPF was isolated from HPE by adsorbance chromatogra- phy. Proanthocyanidins were quantified as anthocyanidins after acidic hydrolysis.…

musculoskeletal diseasesPistachio nut Inflammation Intestinal epithelium Polyphenols Proanthocyanidinscongenital hereditary and neonatal diseases and abnormalitiesCellInterleukin-1betaAnti-Inflammatory AgentsMedicine (miscellaneous)BiologyPharmacologyPermeabilityCell membraneSettore BIO/10 - BiochimicamedicineHumansNutsProanthocyanidinsViability assayIntestinal MucosaCell ProliferationNutrition and DieteticsPistaciaInterleukin-6Interleukin-8NF-kappa BEpithelial Cellsbiology.organism_classificationIntestinal epitheliumIntestinesmedicine.anatomical_structureProanthocyanidinBiochemistryCaco-2Cyclooxygenase 2Paracellular transportPistaciaCaco-2 Cells
researchProduct

The revised ghent nosology; reclassifying isolated ectopia lentis

2014

Inherited ectopia lentis (EL) is most commonly caused by Marfan syndrome (MFS), a multisystemic disorder caused by mutations in FBN1. Historically the diagnosis for patients with EL who have no systemic features of MFS is isolated EL (IEL). However, the Ghent nosology for MFS was updated in 2010 and made some important alterations. In particular, patients with EL and a FBN1 mutation are now categorically diagnosed with MFS, if their mutation has previously been described with aortic dilation/dissection. This carries significant systemic implications, as many patients previously diagnosed with IEL are now reclassified. We provide a review of all published cases of IEL caused by FBN1 mutation…

musculoskeletal diseasesProbandMarfan syndromeNosologycongenital hereditary and neonatal diseases and abnormalitiesPediatricsmedicine.medical_specialtybusiness.industrymedicine.disease3. Good healthDissectionGeneticsMedicineIn patientChinese familyAortic dilationbusinessEctopia lentisGenetics (clinical)Clinical Genetics
researchProduct

Successful treatment of primary chronic osteomyelitis in SAPHO syndrome with bisphosphonates.

2008

Summary The treatment of the painful osteomyelitis in patients with SAPHO syndrome (Synovitis, Acne, Pustulosis, Hyperostosis, Osteitis) is often a problem. A 53-year-old woman had experienced palmo-plantar pustular skin lesions for four years, and in the past two years complained about progressive breath-and movement-dependent pain of the sternum. On examination she had extensive palmoplantar pustules and a painful swelling in the area of the right sternoclavicular joint. The three-phase bone scintigraphy showed a strong focal enrichment in the right sternoclavicular joint and at the transition from the manubrium to the corpus sterni suggesting active osteo-chondritis. Initially prednisolo…

musculoskeletal diseasesSAPHO syndromemedicine.medical_specialtyHyperostosisDermatologySynovitismedicineHumansAcnemedicine.diagnostic_testBone Density Conservation AgentsDiphosphonatesbusiness.industryOsteomyelitisAcquired Hyperostosis SyndromeOsteomyelitisMiddle Agedmedicine.diseasePustulosisDermatologySurgeryTreatment OutcomeBone scintigraphyChronic DiseaseFemaleOsteitismedicine.symptombusinessJournal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG
researchProduct

Muscleblind, BSF and TBPH are mislocalized in the muscle sarcomere of a Drosophila myotonic dystrophy model

2012

SummaryMyotonic dystrophy type 1 (DM1) is a genetic disease caused by the pathological expansion of a CTG trinucleotide repeat in the 3' UTR of the DMPK gene. In the DMPK transcripts, the CUG expansions sequester RNA-binding proteins into nuclear foci, including transcription factors and alternative splicing regulators such as MBNL1. MBNL1 sequestration has been associated with key features of DM1. However, the basis behind a number of molecular and histological alterations in DM1 remain unclear. To help identify new pathogenic components of the disease, we carried out a genetic screen using a Drosophila model of DM1 that expresses 480 interrupted CTG repeats, i(CTG)480, and a collection of…

musculoskeletal diseasesSarcomerescongenital hereditary and neonatal diseases and abnormalitiesNeuroscience (miscellaneous)lcsh:MedicineMedicine (miscellaneous)RNA-binding proteinGenes InsectBiologyMyotonic dystrophyGeneral Biochemistry Genetics and Molecular BiologyAnimals Genetically Modifiedchemistry.chemical_compoundImmunology and Microbiology (miscellaneous)RNA interferencelcsh:PathologymedicineMBNL1AnimalsDrosophila ProteinsHumansMyotonic DystrophyGeneticsMuscleslcsh:RAlternative splicingNuclear ProteinsRNA-Binding ProteinsEpistasis Geneticmedicine.diseaseDisease Models AnimalchemistryGene Knockdown TechniquesDrosophilaFemaleRNA InterferenceTrinucleotide repeat expansionTrinucleotide Repeat ExpansionDrosophila Proteinlcsh:RB1-214Genetic screenResearch ArticleDisease Models & Mechanisms
researchProduct

Type V collagen counteracts osteo-differentiation of human mesenchymal stem cells

2014

In search of novel gene signatures for osteo-differentiation of mesenchymal stem cells (MSCs), we submitted cDNA preparations from undifferentiated and differentiating MSCs to differential display- and semiquantitative-PCR and found down-regulation of COL5A1 in osteo-induced cultures at days 21 and 28, when the mineralized matrix accumulates. We also cultured osteo-differentiating MSCs onto type V collagen substrates and found a decrease in the accumulation of extracellular calcium compared to those grown in uncoated flasks. To our knowledge, this is first evidence that type V collagen might represent a stromal component that impairs osteogenesis.

musculoskeletal diseasesStromal cellchemistry.chemical_elementDown-RegulationBioengineeringBiologyMatrix (biology)CalciumApplied Microbiology and BiotechnologyOsteogenesisGene expressionExtracellularHumansSettore BIO/06 - Anatomia Comparata E CitologiaCells CulturedPharmacologyDifferential displayOsteoblastsGeneral Immunology and MicrobiologyMesenchymal stem cellCell DifferentiationMesenchymal Stem CellsGeneral MedicineMolecular biologychemistryembryonic structurescollagen stem cells osteogenesis gene expressionStem cellCollagen Type VBiotechnology
researchProduct

Chromogenic detection of nerve agent mimics

2008

The current rise in international concern over criminal terrorist attacks via chemical warfare (CW) agents has resulted in an increasing interest in the detection of these lethal chemicals. Among CW species, nerve agents are extremely dangerous and their high toxicity and ease of production underscore the need to detect these deadly chemicals via quick and reliable procedures. A number of detection systems have been developed, most of them based on enzymatic and physical methodologies. However, these usually show limitations such as low selectivity, lack of portability and a certain complexity in their use. An alternative to these classical methods that has been gaining interest in recent y…

musculoskeletal diseasesTertiary amineUNESCO::QUÍMICANerve agent mimicsElectron donorBiosensing TechniquesUNESCO::ASTRONOMÍA Y ASTROFÍSICA:QUÍMICA [UNESCO]Reductive aminationCatalysischemistry.chemical_compoundOrganophosphorus CompoundsMaterials ChemistryMoietyChemical Warfare Agentsskin and connective tissue diseasesChromogenic protocol ; Nerve agent mimics ; Internationalchemistry.chemical_classificationChromogenic protocolMolecular StructureChromogenicChemistryfungiMetals and AlloysGeneral ChemistryElectron acceptorCombinatorial chemistrySurfaces Coatings and FilmsElectronic Optical and Magnetic MaterialsDiethyl chlorophosphatebody regionsKineticsstomatognathic diseasesChromogenic CompoundsInternationalCeramics and CompositesColorimetryHypsochromic shiftAzo Compounds:ASTRONOMÍA Y ASTROFÍSICA [UNESCO]
researchProduct