Search results for "REGULATION"

showing 10 items of 4463 documents

BRAF(V600E) mutation influences hypoxia-inducible factor-1alpha expression levels in papillary thyroid cancer

2010

Abstract Hypoxia-inducible factor-1alpha is found frequently overexpressed in solid tumors cells, exerting an important role in angiogenesis, glucose metabolism, cell proliferation, survival and invasion. In thyroid carcinomas, hypoxia-inducible factor-1alpha expression was found increased in differentiated, poorly differentiated, medullary and anaplastic variants. Hypoxia represents the principal stimulus responsible for hypoxia-inducible factor-1alpha induction. Other nonhypoxic stimuli increase hypoxia-inducible factor-1alpha synthesis through the activation of phosphatidylinositol 3-kinase and mitogen-activated protein kinase pathways in a cell-type-specific manner. We have previously s…

AdultMaleProto-Oncogene Proteins B-rafAdolescentSettore BIO/11 - Biologia MolecolareSettore MED/08 - Anatomia PatologicaTransfectionhypoxia mutation carcinomaMixed Function OxygenasesSettore MED/13 - EndocrinologiaYoung AdultCell Line TumorBiomarkers TumorHumansGene SilencingThyroid NeoplasmsRNA Small InterferingAgedMiddle AgedGene Expression Regulation NeoplasticRepressor ProteinsAdenocarcinoma PapillaryGene Knockdown TechniquesMutationThyroidectomyFemale
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BRAFV600E Mutation and p27kip1 Expression in Papillary Carcinomas of the Thyroid ≤1 cm and Their Paired Lymph Node Metastases

2007

BACKGROUND. BRAFV600E mutation and p27kip1 expression have been introduced as novel indicators that may predict prognosis in different tumors, as well as in papillary thyroid carcinomas. METHODS. Tissue samples from 214 consecutive patients who underwent total or near-total thyroidectomy with histological diagnosis of papillary thyroid carcinoma (PTC) ≤1 cm were analyzed for BRAFV600E mutation by a real-time, allele-specific amplification and for p27kip1 expression by immunohistochemistry. RESULTS. The BRAFV600E mutation was detected in 88 of the tumors examined, with significant differences between groups with and without lymph node (LN) metastases; the mean age of patients with BRAFV600E …

AdultMaleProto-Oncogene Proteins B-rafCancer ResearchPathologymedicine.medical_specialtymedicine.medical_treatmentDown-RegulationGlutamic AcidSettore MED/08 - Anatomia PatologicaPolymerase Chain ReactionMetastasisSettore MED/13 - EndocrinologiaThyroid carcinomaPredictive Value of TestsCarcinomaBiomarkers TumorMedicineHumansThyroid NeoplasmsLymph nodebusiness.industryThyroidThyroidectomyCancerValineSequence Analysis DNAMiddle Agedmedicine.diseasePrognosispapillary thyroid carcinoma BRAF p27 cell cycle.ImmunohistochemistryCarcinoma PapillaryGene Expression Regulation Neoplasticmedicine.anatomical_structureOncologyLymphatic MetastasisMutation (genetic algorithm)MutationFemalebusinessCyclin-Dependent Kinase Inhibitor p27
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Subchronic haloperidol downregulates dopamine synthesis capacity in the brain of schizophrenic patients in vivo

2003

Udgivelsesdato: 2003-Apr The antipsychotic effect of neuroleptics cannot be attributed entirely to acute blockade of postsynaptic D(2)-like dopamine (DA) receptors, but may arise in conjunction with the delayed depolarization block of the presynaptic neurons and reduced DA synthesis capacity. Whereas the phenomenon of depolarization block is well established in animals, it is unknown if a similar phenomenon occurs in humans treated with neuroleptics. We hypothesized that haloperidol treatment should result in decreased DA synthesis capacity. We used 6-[(18)F]fluoro-L-dopa (FDOPA) and positron emission tomography (PET) in conjunction with compartmental modeling to measure the relative activi…

AdultMalePsychosismedicine.medical_specialtyPatientsDopamineDown-RegulationStatistics NonparametricDopamineInternal medicinemedicineHaloperidolHumansPharmacologybusiness.industryPutamenDopamine antagonistBrainDepolarizationHuman brainMiddle Agedmedicine.diseasePsychiatry and Mental healthEndocrinologymedicine.anatomical_structureCerebral cortexAnesthesiaSchizophreniaHaloperidolbusinessTomography Emission-Computedmedicine.drug
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Budesonide increases TLR4 and TLR2 expression in Treg lymphocytes of allergic asthmatics

2015

Abstract Background Reduced innate immunity responses as well as reduced T regulatory activities characterise bronchial asthma. Objectives In this study the effect of budesonide on the expression of TLR4 and TLR2 in T regulatory lymphocyte sub-population was assessed. Methods TLR4 and TLR2 expression in total peripheral blood mononuclear cells (PBMC), in CD4+/CD25+ and in CD4+/CD25− was evaluated, by flow cytometric analysis, in mild intermittent asthmatics (n = 14) and in controls (n = 11). The in vitro effects of budesonide in modulating: TLR4 and TLR2 expression in controls and in asthmatics; IL-10 expression and cytokine release (IL-6 and TNF-α selected by a multiplex assay) in asthmati…

AdultMalePulmonary and Respiratory MedicineBudesonidemedicine.medical_treatmentLymphocyteIn Vitro TechniquesCorticosteroids; Immunoregulation; T lymphocytes; TLR; Adult; Asthma; Budesonide; Cytokines; Female; Flow Cytometry; Glucocorticoids; Humans; In Vitro Techniques; Interleukin-10; Leukocytes Mononuclear; Male; T-Lymphocytes Regulatory; Toll-Like Receptor 2; Toll-Like Receptor 4; Young Adult; Pulmonary and Respiratory Medicine; Pharmacology (medical); Biochemistry (medical); Medicine (all)T-Lymphocytes RegulatoryPeripheral blood mononuclear cellYoung AdultGlucocorticoidTLRT lymphocytemedicineCorticosteroidHumansPharmacology (medical)IL-2 receptorBudesonideCytokineGlucocorticoidsIn Vitro Techniquebusiness.industryMedicine (all)Biochemistry (medical)ImmunoregulationFlow CytometryAsthmaToll-Like Receptor 2Interleukin-10Toll-Like Receptor 4Interleukin 10TLR2Cytokinemedicine.anatomical_structureImmunologyLeukocytes MononuclearTLR4CytokinesFemalebusinessHumanmedicine.drugPulmonary Pharmacology & Therapeutics
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Increased expression of the homeostatic chemokines CCL19 and CCL21 in clinical and experimental Rickettsia conorii infection

2013

Background: Based on their essential role in concerting immunological and inflammatory responses we hypothesized that the homeostatic chemokines CCL19 and CCL21 may play a pathogenic role in rickettsiae infection. Methods: Serum levels of CCL19 and CCL21 in patients with R. africae and R. conorii infection were analyzed by enzyme immunoassays. Lungs from R. conorii infected mice were examined for CCL19, CCL21 and CCR7 expression by immunohistochemistry. Results: We found that patients with R. africae infection (n = 15) and in particular those with R. conorii infection (n = 16) had elevated serum levels of CCL19 on admission, with a decline during follow-up. While a similar pattern was seen …

AdultMaleReceptors CCR7Chemokineendocrine systemR. africaeInflammationC-C chemokine receptor type 7ImmunofluorescencePathogenesisMiceYoung AdultmedicineAnimalsHomeostasisHumansAgedInflammationMice Inbred C3HR. conoriiChemokine CCL21biologymedicine.diagnostic_testCCL19Rickettsia InfectionsMiddle Agedbiology.organism_classificationUp-RegulationRickettsia conoriiInfectious DiseasesImmunologybiology.proteinChemokine CCL19FemaleChemokinesmedicine.symptomRickettsia conoriiImmunostainingResearch ArticleCCR7
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Repeated muscle biopsies through a single skin incision do not elicit muscle signaling, but IL-6 mRNA and STAT3 phosphorylation increase in injured m…

2011

To determine if muscle biopsies can be repeated using a single small (5–6 mm) skin incision without inducing immediate MAPK activation or inflammation in the noninjured areas, the phosphorylation of ERK1/2, p38-MAPK, c-Jun NH2-terminal kinases (JNKs), IκBα, IKKα, and signal transducer and activator of transcription 3 (STAT3) was examined concurrent with IL-6 mRNA in six muscle biopsies obtained from the vastus lateralis of five men. Four biopsies were obtained through the same incision (5–6 mm) from the right leg (taken at 0, 30, 123, and 126 min) and another two each from new incisions performed in the left leg (at 31 and 120 min), while the subjects rested supine. The first three biopsie…

AdultMaleSTAT3 Transcription FactorMAPK/ERK pathwaymedicine.medical_specialtyPathologyTime FactorsPhysiologyBiopsyInflammationp38 Mitogen-Activated Protein KinasesQuadriceps MuscleMuscular DiseasesNF-KappaB Inhibitor alphaPhysiology (medical)Internal medicinemedicineHumansRNA MessengerPhosphorylationSTAT3Interleukin 6Mitogen-Activated Protein Kinase 1Analysis of VarianceWound HealingMitogen-Activated Protein Kinase 3Skin incisionbiologyInterleukin-6JNK Mitogen-Activated Protein KinasesIl 6 mrnaI-kappa B KinaseUp-RegulationEndocrinologybiology.proteinSTAT proteinPhosphorylationI-kappa B Proteinsmedicine.symptomSignal TransductionJournal of Applied Physiology
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Cytokine Gene Transcription By NF-kappaB Family Members in Patients with Inflammatory Bowel Disease

1998

We examined the expression of the transcription factor NF-kappa B, a nuclear trans-acting factor known to play a key role in cytokine gene regulation, in patients with inflammatory bowel disease (IBD). It was found that LP macrophages in Crohn's disease (CD) and ulcerative colitis (UC) display high levels of NF-kappa B DNA-binding activity accompanied by an increased production of interleukin (IL)-1, IL-6, and tumor necrosis factor (TNF) alpha. Western blot studies showed an increased expression of the p50 and c-rel subunits of NF-kappa B; however, the most striking finding was an increased expression level of NF-kappa B p65 in patients with CD and UC. Selective downregulation of p65 in IBD…

AdultMaleShort Bowel SyndromeAdolescentTranscription GeneticColonBiologyInflammatory bowel diseaseGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokinechemistry.chemical_compoundCrohn DiseaseHistory and Philosophy of ScienceDownregulation and upregulationmedicineHumansIntestinal MucosaTranscription factorCells CulturedRegulation of gene expressionMacrophagesGeneral NeuroscienceNF-kappa BInterleukinNF-κBMiddle Agedmedicine.diseaseGene Expression RegulationchemistryImmunologyCancer researchCytokinesColitis UlcerativeFemaleTumor necrosis factor alphaAnnals of the New York Academy of Sciences
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NF-κB protects Behçet's disease T cells against CD95-induced apoptosis up-regulating antiapoptotic proteins

2005

Objective To determine whether prolongation of the inflammatory reaction in patients with Behcet's disease (BD) is related to apoptosis resistance and is associated with the up-regulation of antiapoptotic factors. Methods The percentage of cell death was evaluated by flow cytometry in peripheral blood mononuclear cells from 35 patients with BD and 30 healthy volunteers. The expression levels of antiapoptotic factors and NF-κB regulatory proteins were measured using Western blotting and immunohistochemical analyses. To down-regulate NF-κB nuclear translocation, BD T lymphocytes were exposed in vitro to thalidomide and subjected to transfection with NF-κB small interfering RNA. Results Althou…

AdultMaleSmall interfering RNAProgrammed cell deathT-LymphocytesT cellImmunologyCASP8 and FADD-Like Apoptosis Regulating Proteinbcl-X ProteinApoptosisCaspase 3TransfectionCaspase 8RheumatologyHumansImmunology and AllergyMedicinePharmacology (medical)fas ReceptorRNA Small InterferingCells CulturedDose-Response Relationship Drugbusiness.industryBehcet SyndromeIntracellular Signaling Peptides and ProteinsNF-kappa BTransfectionFlow CytometryFas receptorThalidomideUp-Regulationmedicine.anatomical_structureGene Expression RegulationProto-Oncogene Proteins c-bcl-2ApoptosisImmunologyLeukocytes MononuclearCancer researchFemalebusinessArthritis & Rheumatism
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Tas1R1-Tas1R3 taste receptor variants in human fungiform papillae.

2009

International audience; Monosodium glutamate as well as metabotropic and ionotropic glutamate receptor agonists have been reported to be perceived as umami by humans. In spite of the fact that Tas1R1-Tas1R3 has been shown to mediate most of the glutamate taste sensation in mice other candidate receptors have been put forward for which a clear role in detection is still lacking. This work was aimed at investigating the molecular determinants underlying umami taste detection in humans. First, we show evidence supporting expression of Tas1R1 and Tas1R3 but not mGluRs in the fungiform papillae of several individuals. Next, we report a number of naturally occurring l-glutamate taste receptor var…

AdultMaleTasteTASTE RECEPTORSGlutamic AcidSNPFUNGIFORM PAPILLAEUmamiBiologyLigandsReceptors Metabotropic GlutamatePolymorphism Single NucleotideReceptors G-Protein-CoupledTAS1R103 medical and health sciencesGLUTAMATE0302 clinical medicineTAS1R3Allosteric RegulationTongueTaste receptorHumansProtein IsoformsMSG030304 developmental biologyAgedGenetics0303 health sciencesBinding SitesGeneral Neuroscience[SCCO.NEUR]Cognitive science/NeuroscienceGenetic VariationHUMANMiddle AgedTaste BudsProtein Structure TertiaryTAS2R38BiochemistryTasteTaste Threshold[ SCCO.NEUR ] Cognitive science/NeuroscienceMetabotropic glutamate receptor 1Ionotropic glutamate receptorFemaleUNAMI030217 neurology & neurosurgery
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PD-1, PD-L1 and PD-L2 Gene Expression on T-Cells and Natural Killer Cells Declines in Conjunction with a Reduction in PD-1 Protein during the Intensi…

2015

Background The PD-1 axis is a cell intrinsic immunoregulatory pathway that mediates T cell exhaustion in chronic infection particularly in some viral infections. We hypothesized that PD-1, PD-L1 and PD-L2 would be highly expressed in untreated tuberculosis patients compared to controls due to their chronic infection and would decrease with successful TB treatment. Materials and Methods Untreated tuberculosis patients (n = 26) were recruited at diagnosis and followed up during treatment. Household contacts (n = 24) were recruited to establish baseline differences. Blood gene expression ex vivo was investigated using qRT-PCR. Flow cytometry was performed to establish protein expression patter…

AdultMaleTuberculosisT cellProgrammed Cell Death 1 ReceptorAntitubercular Agentslcsh:MedicineDown-RegulationB7-H1 AntigenImmunophenotypingMycobacterium tuberculosisYoung AdultImmunophenotypingT-Lymphocyte SubsetsPD-L1medicineCytotoxic T cellHumansTuberculosisLymphocyte Countlcsh:ScienceAgedMultidisciplinarybiologylcsh:RMycobacterium tuberculosisMiddle Agedbiology.organism_classificationmedicine.diseaseProgrammed Cell Death 1 Ligand 2 ProteinGranzyme BKiller Cells Naturalmedicine.anatomical_structureGene Expression RegulationImmunologybiology.proteinlcsh:QFemaleCD8Research Article
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