Search results for "RIO"

showing 10 items of 19845 documents

7-ketocholesterol and 7β-hydroxycholesterol: in vitro and animal models used to characterize their activities and to identify molecules preventing th…

2020

International audience; Oxysterols are molecules derived by the oxidation of cholesterol and can be formed either by auto-oxidation, enzymatically or by both processes. Among the oxysterols formed by auto-oxidation, 7-ketocholesterol and 7beta-hydroxycholesterol are the main forms generated. These oxysterols, formed endogenously and brought in large quantities by certain foods, have major cytotoxic properties. They are powerful inducers of oxidative stress, inducing dysfunction of organelles (mitochondria, lysosomes and peroxisomes) that can cause cell death. These molecules are often identified in increased amounts in common pathological states such as cardiovascular diseases, certain eye …

0301 basic medicine[SDV]Life Sciences [q-bio]CellmicrofluidicMitochondrionPharmacologiemedicine.disease_causeBiochemistry0302 clinical medicineanimal modèleKetocholesterolsComputingMilieux_MISCELLANEOUSCells CulturedsignalingpathwaysCell DeathChemistry7β-hydroxycholesterolNeurodegenerative DiseasesPeroxisomeanimal models3. Good healthmedicine.anatomical_structureBiochemistryCardiovascular Diseases030220 oncology & carcinogenesisToxicity[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]modèle cellulaireSignal transductionProgrammed cell deathCataractCell Line03 medical and health sciencesPharmaceutical sciencesCell Line TumormedicineAnimalsHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biologyhydroxycholestérol7-ketocholesterolPharmacologyOrganelles7-ketocholesterol;7β-hydroxycholesterol;cell models;animal models;microfluidic;signalingpathwaysInflammatory Bowel DiseasesIn vitroHydroxycholesterolscell modelsDisease Models Animal030104 developmental biologyvoie de signalisationSciences pharmaceutiques[SDV.AEN]Life Sciences [q-bio]/Food and NutritionOxidative stress
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GDF11 exhibits tumor suppressive properties in hepatocellular carcinoma cells by restricting clonal expansion and invasion.

2019

Growth differentiation factor 11 (GDF11) has been characterized as a key regulator of differentiation in cells that retain stemness features, despite some controversies in age-related studies. GDF11 has been poorly investigated in cancer, particularly in those with stemness capacity, such as hepatocellular carcinoma (HCC), one of the most aggressive cancers worldwide. Here, we focused on investigating the effects of GDF11 in liver cancer cells. GDF11 treatment significantly reduced proliferation, colony and spheroid formation in HCC cell lines. Consistently, down-regulation of CDK6, cyclin D1, cyclin A, and concomitant upregulation of p27 was observed after 24 h of treatment. Interestingly,…

0301 basic medicine[SDV]Life Sciences [q-bio]Cyclin ACellChick EmbryoChorioallantoic Membrane0302 clinical medicineCell MovementCyclin D1HCCbiologyNeovascularization PathologicCell DifferentiationHep G2 CellsCell cycleCadherinsHuh7 cells3. Good health[SDV] Life Sciences [q-bio]Gene Expression Regulation NeoplasticGrowth Differentiation Factorsmedicine.anatomical_structure030220 oncology & carcinogenesisBone Morphogenetic ProteinsMolecular MedicineLiver cancerCyclin-Dependent Kinase Inhibitor p27Signal Transduction[SDV.CAN]Life Sciences [q-bio]/CancerCyclin ACell cycleHep3B cells03 medical and health sciencesCyclin D1Downregulation and upregulation[SDV.CAN] Life Sciences [q-bio]/CancerAntigens CDCell Line TumorOccludinSpheroids CellularmedicineAnimalsHumansViability assayMolecular BiologyCell Proliferation[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and GastroenterologyCyclin-Dependent Kinase 6[SDV.MHEP.HEG] Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology030104 developmental biologyCell cultureGDF11biology.proteinCancer researchCyclin-dependent kinase 6Snail Family Transcription FactorsBiochimica et biophysica acta. Molecular basis of disease
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Transcriptome analysis revealed that a quorum sensing system regulates the transfer of the pAt megaplasmid in Agrobacterium tumefaciens.

2016

Background Agrobacterium tumefaciens strain P4 is atypical, as the strain is not pathogenic and produces a for this species unusual quorum sensing signal, identified as N-(3-hydroxy-octanoyl)-homoserine lactone (3OH,C8-HSL). Results By sequence analysis and cloning, a functional luxI-like gene, named cinI, has been identified on the At plasmid of A. tumefaciens strain P4. Insertion mutagenesis in the cinI gene and transcriptome analyses permitted the identification of 32 cinI-regulated genes in this strain, most of them encoding proteins responsible for the conjugative transfer of pAtP4. Among these genes were the avhB genes that encode a type 4 secretion system (T4SS) involved in the forma…

0301 basic medicineacylhomoserime lactoneIdentification[SDV]Life Sciences [q-bio]AgrobacteriumPlasmidePlant Rootsfluids and secretionsPlasmidSolanum lycopersicumhttp://aims.fao.org/aos/agrovoc/c_16014Expression des gènesDynamique des populationsCloning MolecularPhylogenyGeneticsbiology000 - Autres thèmeshttp://aims.fao.org/aos/agrovoc/c_27583food and beveragesAgrobacterium tumefaciensLactonehttp://aims.fao.org/aos/agrovoc/c_768[SDV] Life Sciences [q-bio]Quorum sensingT4SSConjugation GeneticPropriété biologiquehttp://aims.fao.org/aos/agrovoc/c_35128PlasmidsResearch Articlehttp://aims.fao.org/aos/agrovoc/c_4145BiotechnologyDtr systemSéquence nucléotidiqueAgrobacteriumSequence analysisMutagenesis (molecular biology technique)At plasmid03 medical and health scienceshttp://aims.fao.org/aos/agrovoc/c_4891Bacterial Proteinsstomatognathic systemhttp://aims.fao.org/aos/agrovoc/c_3081Geneticshttp://aims.fao.org/aos/agrovoc/c_1501Acylhomoserine lactoneTranscriptomicsGenehttp://aims.fao.org/aos/agrovoc/c_6111H20 - Maladies des plantesCloning[ SDV ] Life Sciences [q-bio]Bactériologiehttp://aims.fao.org/aos/agrovoc/c_27444Sequence Analysis RNATranscription géniqueConjugationGene Expression ProfilingBiologie moléculaireGene Expression Regulation Bacterialbiochemical phenomena metabolism and nutritionQuorum sensing;Agrobacterïum;At plasmid;transcriptomics;conjugation;T4SS;Dtr system;Acylhomoserine lactonebiology.organism_classificationhttp://aims.fao.org/aos/agrovoc/c_27527Quorum sensinghttp://aims.fao.org/aos/agrovoc/c_3791030104 developmental biologyAgrobacterium tumefaciensbacteriaGenetic Fitness
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Oxidative Stress, Neuroinflammation and Mitochondria in the Pathophysiology of Amyotrophic Lateral Sclerosis

2020

Amyotrophic lateral sclerosis (ALS) is a progressive motor neuron (MN) disease. Its primary cause remains elusive, although a combination of different causal factors cannot be ruled out. There is no cure, and prognosis is poor. Most patients with ALS die due to disease-related complications, such as respiratory failure, within three years of diagnosis. While the underlying mechanisms are unclear, different cell types (microglia, astrocytes, macrophages and T cell subsets) appear to play key roles in the pathophysiology of the disease. Neuroinflammation and oxidative stress pave the way leading to neurodegeneration and MN death. ALS-associated mitochondrial dysfunction occurs at different le…

0301 basic medicineamyotrophic lateral sclerosisPhysiologyClinical BiochemistryReviewDiseaseMitochondrionmedicine.disease_causeBiochemistryneuroinflammationNeurologia03 medical and health sciences0302 clinical medicineoxidative stressMedicineAmyotrophic lateral sclerosisMolecular BiologyNeuroinflammationMicrogliabusiness.industrylcsh:RM1-950NeurodegenerationCell Biologymedicine.diseasePatologiaPathophysiologymitochondrialcsh:Therapeutics. Pharmacology030104 developmental biologymedicine.anatomical_structuremotor neuron diseasebusinessNeuroscience030217 neurology & neurosurgeryOxidative stressAntioxidants
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Therapeutic alternative of the ketogenic Mediterranean diet to improve mitochondrial activity in Amyotrophic Lateral Sclerosis (ALS): A Comprehensive…

2019

Abstract Amyotrophic lateral sclerosis (ALS) is an incurable neurodegenerative disease which is pathogenically based on the mitochondrial alteration of motor neurons, causing progressive neuron death. While ALS is characterized by enormous oxidative stress, the Mediterranean diet has been seen to have high antioxidant power. Therefore, the aim of this study is to determine how the Mediterranean diet can improve mitochondrial activity, establishing the specific nutrients and, in addition, observing the pathogenic mechanisms related to the disease that would achieve this improvement. To this end, a comprehensive review of the literature was performed using PubMed. KBs have been observed to ha…

0301 basic medicineamyotrophic lateral sclerosismitochondria ; mediterranean diet ; amyotrophic lateral sclerosis ; ketone bodiesMediterranean dietReviewslcsh:TX341-641ReviewDiseaseMitochondrionBioinformaticsmedicine.disease_causeNeuroprotection03 medical and health sciences0302 clinical medicineKetogenesisMedicineAmyotrophic lateral sclerosisbusiness.industryfood and beveragesmediterranean dietmedicine.diseasemitochondria030104 developmental biologyketone bodiesbusinessNeuron deathlcsh:Nutrition. Foods and food supply030217 neurology & neurosurgeryOxidative stressFood ScienceFood Science & Nutrition
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Herpes simplex virus 1 induces egress channels through marginalized host chromatin

2016

AbstractLytic infection with herpes simplex virus type 1 (HSV-1) induces profound modification of the cell nucleus including formation of a viral replication compartment and chromatin marginalization into the nuclear periphery. We used three-dimensional soft X-ray tomography, combined with cryogenic fluorescence, confocal and electron microscopy, to analyse the transformation of peripheral chromatin during HSV-1 infection. Our data showed an increased presence of low-density gaps in the marginalized chromatin at late infection. Advanced data analysis indicated the formation of virus-nucleocapsid-sized (or wider) channels extending through the compacted chromatin of the host. Importantly, co…

0301 basic medicineanalysisvirusesHerpesvirus 1 Humanmedicine.disease_causeVirus Replicationlaw.inventionRussia[ SDV.CAN ] Life Sciences [q-bio]/CancerMicelaw2.1 Biological and endogenous factorsAetiologynuclear organisationTomographyB-LymphocytesMicroscopyMultidisciplinaryMicroscopy ConfocalTomography X-Rayta3141Chromatin3. Good healthCell biologyChromatinOther Physical SciencesInfectious Diseasesmedicine.anatomical_structureLytic cycleConfocalHost-Pathogen InteractionsVirusesFranceInfectionHumanConfocal030106 microbiology[SDV.CAN]Life Sciences [q-bio]/CancerBiologyta3111ElectronTime-Lapse ImagingArticleCell Line03 medical and health sciencesMicroscopy Electron TransmissionmedicineHerpes virusTransmissionAnimalsHumansCell Nucleusta114Herpesvirus 1ta1182VirionHerpes SimplexCell nucleus030104 developmental biologyHerpes simplex virusViral replicationCell cultureX-RaySexually Transmitted InfectionsBiochemistry and Cell BiologyElectron microscopeLaboratories
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Burying beetles regulate the microbiome of carcasses and use it to transmit a core microbiota to their offspring

2017

Necrophagous beetles utilize carrion, a highly nutritious resource that is susceptible to intense microbial competition, by treating it with antimicrobial anal and oral secretions. However, how this regulates the carcass microbiota remains unclear. Here, we show that carcasses prepared by the burying beetle Nicrophorus vespilloides undergo significant changes in their microbial communities subsequent to their burial and ‘preparation’. Prepared carcasses hosted a microbial community that was more similar to that of beetles’ anal and oral secretions than to the native carcass community or the surrounding soil, indicating that the beetles regulated the carcass microbiota. A core microbial comm…

0301 basic medicineanimal diseasesmedia_common.quotation_subjectEnterococcaceaeCompetition (biology)03 medical and health sciencesfluids and secretionsEnterococcaceaeGeneticsAnimalsCarrionMicrobiomeSymbiosisEcology Evolution Behavior and Systematicsmedia_commonbiologyEcologyMicrobiotadigestive oral and skin physiologyXanthomonadaceaetechnology industry and agriculturefood and beveragesbiology.organism_classificationNicrophorus vespilloidesColeoptera030104 developmental biologyMicrobial population biologyLarvaBurying beetleDigestive SystemMolecular Ecology
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The Challenging Riddle about the Janus-Type Role of Hsp60 and Related Extracellular Vesicles and miRNAs in Carcinogenesis and the Promises of Its Sol…

2021

Hsp60 is one of the most ancient and evolutionarily conserved members of the chaperoning system. It typically resides within mitochondria, in which it contributes to maintaining the organelle’s proteome integrity and homeostasis. In the last few years, it has been shown that Hsp60 also occurs in other locations, intracellularly and extracellularly, including cytosol, plasma-cell membrane, and extracellular vesicles (EVs). Consequently, non-canonical functions and interacting partners of Hsp60 have been identified and it has been realized that it is a hub molecule in diverse networks and pathways and that it is implicated, directly or indirectly, in the development of various pathological co…

0301 basic medicineanimal structuresBiologyMitochondrionmedicine.disease_causechaperonopathieslcsh:TechnologyChaperoninlcsh:Chemistry03 medical and health sciences0302 clinical medicinemicroRNAmedicineExtracellularGeneral Materials ScienceInstrumentationlcsh:QH301-705.5CarcinogenesichaperonotherapymiRNAFluid Flow and Transfer Processeslcsh:TProcess Chemistry and Technologyextracellular vesicle (EV)fungiGeneral EngineeringHsp60lcsh:QC1-999Computer Science ApplicationsCell biologyCytosol030104 developmental biologylcsh:Biology (General)lcsh:QD1-999lcsh:TA1-2040030220 oncology & carcinogenesisProteomeChaperonopathieHSP60Carcinogenesislcsh:Engineering (General). Civil engineering (General)carcinogenesislcsh:PhysicsApplied Sciences
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Wnt1 Promotes Cementum and Alveolar Bone Growth in a Time-Dependent Manner

2021

The WNT/β-catenin signaling pathway plays a central role in the biology of the periodontium, yet the function of specific extracellular WNT ligands remains poorly understood. By using a Wnt1-inducible transgenic mouse model targeting Col1a1-expressing alveolar osteoblasts, odontoblasts, and cementoblasts, we demonstrate that the WNT ligand WNT1 is a strong promoter of cementum and alveolar bone formation in vivo. We induced Wnt1 expression for 1, 3, or 9 wk in Wnt1Tg mice and analyzed them at the age of 6 wk and 12 wk. Micro–computed tomography (CT) analyses of the mandibles revealed a 1.8-fold increased bone volume after 1 and 3 wk of Wnt1 expression and a 3-fold increased bone volume aft…

0301 basic medicineanimal structuresCementoblastmineralized tissue/development03 medical and health sciences0302 clinical medicinestomatognathic systemmedicineCementumGeneral DentistryDental alveolusperiodontal ligament (PDL)Chemistrybone biologyWnt signaling pathwayResearch ReportsPeriodontiumBiologicalCementogenesisCell biologycementogenesis030104 developmental biologyOdontoblastmedicine.anatomical_structure030220 oncology & carcinogenesisembryonic structuresPulp (tooth)signal transductionWnt/β-catenin signalingJournal of Dental Research
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Heat Shock Protein 60 in Cardiovascular Physiology and Diseases.

2020

Heat shock protein 60 (HSP60) is a highly conserved protein abundantly expressed in both prokaryotic and eukaryotic cells. In mammals, HSP60 has been primarily considered to reside in the mitochondria, where HSP60 and HSP10 form a complex and facilitate mitochondrial protein folding. However, HSP60 is also observed in the cytoplasm, the plasma membrane, and the extracellular space. HSP60 regulates a broad spectrum of cellular events including protein trafficking, peptide hormone signaling, cell survival, cell proliferation, inflammation, and immunization. In the cardiovascular system, growing evidence indicates that HSP60 could not only play an important role under physiological conditions,…

0301 basic medicineanimal structuresMini Reviewheat shock proteinheart failureInflammationchemical and pharmacologic phenomenacardiomyocyteBiologyMitochondrionBiochemistry Genetics and Molecular Biology (miscellaneous)Biochemistrycomplex mixtures03 medical and health sciences0302 clinical medicineHeat shock proteinmedicineMolecular Bioscienceslcsh:QH301-705.5Molecular BiologyCell growthfungiCardiovascular physiologyCell biology030104 developmental biologylcsh:Biology (General)Cytoplasm030220 oncology & carcinogenesisHSP60medicine.symptomSignal transductionatherosclerosisHSP60Frontiers in molecular biosciences
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