Search results for "Race"

showing 10 items of 4458 documents

Adaptation of neuronal cells to chronic oxidative stress is associated with altered cholesterol and sphingolipid homeostasis and lysosomal function

2009

Chronic oxidative stress has been causally linked to several neurodegenerative disorders. As sensitivity for oxidative stress greatly differs between brain regions and neuronal cell types, specific cellular mechanisms of adaptation to chronic oxidative stress should exist. Our objective was to identify molecular mechanisms of adaptation of neuronal cells after applying chronic sublethal oxidative stress. We demonstrate that cells resistant to oxidative stress exhibit altered cholesterol and sphingomyelin metabolisms. Stress-resistant cells showed reduced levels of molecules involved in cholesterol trafficking and intracellular accumulation of cholesterol, cholesterol precursors, and metabol…

medicine.medical_specialtyCell typeCerebellumLipid metabolismBiologymedicine.disease_causeBiochemistrySphingolipidCellular and Molecular Neurosciencemedicine.anatomical_structureEndocrinologyCell cultureInternal medicinemedicineIntracellularOxidative stressHomeostasisJournal of Neurochemistry
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The Homeostasis of Brain Choline

1993

The interest in the homeostasis of brain choline is reinforced by the role of choline as immediate precursor of acetylcholine, phosphatidylcholine and other phospholipids in the brain. In order to obtain a comprehensive view of the mochanisms of homeostasis it appeared necessary to elucidate the negative arteriovenous difference of choline across the brain (net release), a phenomenon that has been known for 20 years and is present in mammals and in man. This finding prompted an intense search for a de novo synthesis of choline in the brain. We detected in anaesthetized rats a reversal of the net release into a net uptake (positive arterio-venous difference), when the plasma level of choline…

medicine.medical_specialtyChemistryDe novo synthesischemistry.chemical_compoundEndocrinologyPhosphatidylcholineInternal medicinemedicineExtracellularPhosphorylationCholineAcetylcholineHomeostasismedicine.drugPhosphocholine
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Role of IP3 Receptors in Shaping the Carotid Chemoreceptor Response to Hypoxia But Not to Hypercapnia in the Rat Carotid Body: An Evidence Review

2020

This article addresses the disparity in the transduction pathways for hypoxic and hypercapnic stimuli in carotid body glomus cells. We investigated and reviewed the experimental evidence showing that the response to hypoxia, but not to hypercapnia, is mediated by 1,4,5-inositol triphosphate receptors (IP3R/s) regulating the intracellular calcium content [Ca2+]c in glomus cells. The rationale was based on the past observations that inhibition of oxidative phosphorylation leads to the explicit inhibition of the hypoxic chemoreflex. [Ca2+]c changes were measured using cellular Ca2+-sensitive fluorescent probes, and carotid sinus nerve (CSN) sensory discharge was recorded with bipolar electrode…

medicine.medical_specialtyChemistryInositol trisphosphate receptorHypoxia (medical)Calcium in biologymedicine.anatomical_structureGlomus cellEndocrinologyInternal medicinemedicineCarotid bodymedicine.symptomReceptorHypercapniaIntracellular
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Intra-Articular Injection of Fructus Ligustri Lucidi Extract Attenuates Pain Behavior and Cartilage Degeneration in Mono-Iodoacetate Induced Osteoart…

2018

Fructus Ligustri Lucidi (FLL) has been widely used as a traditional Chinese medicine (TCM) for treating soreness and weakness of waist and knees. It has potential for treating OA owing to its kidney-tonifying activity with bone-strengthening effects, but there is so far no report of its anti-OA effect. This study established a rat OA model by intra-articular (IA) injection of mono-iodoacetate (1.5 mg) and weekly treated by IA administration of FLL at 100 μg/mL for 4 weeks. Thermal withdrawal latency, mechanical withdrawal threshold, and spontaneous activity were tested for evaluation of pain behavior, and histopathological (HE, SO, and ABH staining) and immunohistochemical (Col2, Col10, and…

medicine.medical_specialtyChondrocyte hypertrophyintra-articularChondrocyteMuscle hypertrophymono-iodoacetateExtracellular matrix03 medical and health sciences0302 clinical medicineInternal medicinemedicinePharmacology (medical)MTT assayAggrecanFructus Ligustri LucidiOriginal Research030203 arthritis & rheumatologyPharmacologyChemistryCartilagelcsh:RM1-950medicine.anatomical_structureEndocrinologylcsh:Therapeutics. Pharmacology030220 oncology & carcinogenesisJoint painchondrocytemedicine.symptomhypertrophyFrontiers in Pharmacology
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Getting the Fat out of Met and Fas

2008

Lack of Fas antagonism by Met in human fatty liver disease. Zou C, Ma J, Wang X, Guo L, Zhu Z, Stoops J, Eaker AE, Johnson CJ, Strom S, Michalopoulos GK, DeFrances MC, Zarnegar R. Hepatocytes in fatty livers are hypersensitive to apoptosis and undergo escalated apoptotic activity via death receptor-mediated pathways, particularly that of Fas–FasL, causing hepatic injury that can eventually proceed to cirrhosis and end-stage liver disease. Here we report that the hepatocyte growth factor receptor, Met, plays an important part in preventing Fas-mediated apoptosis of hepatocytes by sequestering Fas. We also show that Fas antagonism by Met is abrogated in human fatty liver disease (FLD). Throug…

medicine.medical_specialtyCirrhosisHepatologyFatty liverBiologymedicine.diseaseMolecular biologyFas ligandLiver diseaseEndocrinologyHepatocyte Growth Factor ReceptorApoptosisInternal medicinemedicineExtracellularAntagonismJournal of Hepatology
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Intestinal filtration as a consequence of increased mucosal hydraulic permeability

1980

Two mechanisms have been proposed to explain the secretory action of laxative compounds in the intestine: 1. increase of the intracellular amount of cyclic adenosine monophosphate due to stimulation of the adenylate cyclase system and 2. inhibition of intestinal transfer processes, in particular the Na,K-ATPase activated sodium absorption. In a set of in vivo and in vitro experiments in rat colon it could be demonstrated that dihydroxy bile acids (deoxycholate) and diphenolic laxatives (oxyphenisatin) enhance the hydraulic permeability of the mucosal tissue. The permeability changes take place--and there is good experimental evidence--at the zonulae occludentes which bind the epithelial cel…

medicine.medical_specialtyColonSodiummedicine.medical_treatmentHydrostatic pressureLaxativechemistry.chemical_elementStimulationPermeabilitychemistry.chemical_compoundInternal medicineDrug DiscoveryElectrochemistrymedicineStarling equationAnimalsCyclic adenosine monophosphateIntestinal MucosaGenetics (clinical)Adenosine TriphosphatasesTight junctionCatharticsSodiumOxyphenisatin AcetateGeneral MedicineRatsEndocrinologychemistryPotassiumBiophysicsMolecular MedicineFiltrationIntracellularDeoxycholic AcidKlinische Wochenschrift
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Magnesium metabolism in type 2 diabetes mellitus, metabolic syndrome and insulin resistance

2007

Type 2 diabetes is characterized by cellular and extracellular Mg depletion. Epidemiologic studies showed a high prevalence of hypomagnesaemia and lower intracellular Mg concentrations in diabetic subjects. Insulin and glucose are important regulators of Mg metabolism. Intracellular Mg plays a key role in regulating insulin action, insulin-mediated-glucose uptake and vascular tone. Reduced intracellular Mg concentrations result in a defective tyrosine-kinase activity, post-receptorial impairment in insulin action, and worsening of insulin resistance in diabetic patients. Mg deficit has been proposed as a possible underlying common mechanism of the "insulin resistance" of different metabolic…

medicine.medical_specialtyDiabetes riskmedicine.medical_treatmentBiophysicsType 2 diabetesBiochemistryInsulin resistanceDiabetes mellitusInternal medicineHumansInsulinMedicineMagnesiumMolecular BiologyMetabolic Syndromebusiness.industryInsulinType 2 Diabetes Mellitusmedicine.diseaseGlucoseEndocrinologyDiabetes Mellitus Type 2Insulin ResistanceMetabolic syndromebusinessIntracellularArchives of Biochemistry and Biophysics
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The incidence of psychotic disorders among migrants and minority ethnic groups in Europe: findings from the multinational EU-GEI study

2022

AbstractBackgroundIn Europe, the incidence of psychotic disorder is high in certain migrant and minority ethnic groups (hence: ‘minorities’). However, it is unknown how the incidence pattern for these groups varies within this continent. Our objective was to compare, across sites in France, Italy, Spain, the UK and the Netherlands, the incidence rates for minorities and the incidence rate ratios (IRRs, minorities v. the local reference population).MethodsThe European Network of National Schizophrenia Networks Studying Gene–Environment Interactions (EU-GEI) study was conducted between 2010 and 2015. We analyzed data on incident cases of non-organic psychosis (International Classification of …

medicine.medical_specialtyDopamineRegion of originEthnic groupmigrationpsychosistress03 medical and health sciencesRace (biology)0302 clinical medicineEpidemiologymedicineHumanspsychosis10. No inequalityraceMinority GroupsApplied PsychologyTransients and MigrantsHigh rateIncidenceIncidence (epidemiology)Social environmentPREVALENCE030227 psychiatry3. Good healthEuropeschizophreniaPsychiatry and Mental healthGeographyPsychotic DisordersMultinational corporation1ST-CONTACT INCIDENCEethnicityepidemiology030217 neurology & neurosurgerySOCIAL DEFEAT HYPOTHESISDemographyPsychological Medicine
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Efavirenz induces alterations in lipid metabolism through AMPK activation

2008

Summary of results EFV produced an immediate reduction of mitochondrialfunction, evident by the significant and dose-dependentinhibition of mitochondrial O2 consumption and thedecrease of intracellular ATP and Δψm. This metabolicstress promoted the activation of AMPK, triggering severalof its signalling pathways, as EFV induced an increment inCD36 mRNA expression and in intracellular lipid content,which could have been a result of the formation of lipiddroplets. This intracellular lipid increase was not presentin cells treated with Compound C, which points to a keyrole for AMPK in these mechanisms. Conclusion Given that EFV treatment is usually prolonged, thesemechanisms may effect the gene…

medicine.medical_specialtyEfavirenzbusiness.industryPublic Health Environmental and Occupational HealthAMPKLipid metabolismmedicine.diseasechemistry.chemical_compoundInfectious DiseasesEndocrinologychemistryInternal medicinemedicineLipodystrophybusinessProtein kinase ALipoatrophyIntracellularCompound cJournal of the International AIDS Society
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Influence of different CyA formulations and calcium channel blocker phenyhidine regimens on intracellular (erythrocyte) calcium levels after kidney t…

1997

medicine.medical_specialtyErythrocytesmedicine.drug_classPrednisolonechemistry.chemical_elementAdministration OralCalcium channel blockerCalciumInternal medicineAzathioprinemedicineHumansDrug InteractionsKidney transplantationDosage FormsTransplantationKidneybusiness.industryCiclosporinmedicine.diseaseCalcium Channel BlockersKidney TransplantationTransplantationEndocrinologymedicine.anatomical_structurechemistryToxicityCyclosporineSurgeryCalciumEmulsionsbusinessIntracellularImmunosuppressive Agentsmedicine.drugTransplantation proceedings
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