Search results for "Reactive nitrogen"

showing 10 items of 74 documents

Hydralazine is a powerful inhibitor of peroxynitrite formation as a possible explanation for its beneficial effects on prognosis in patients with con…

2005

The hemodynamic and anti-ischemic effects of nitroglycerin (GTN) are rapidly blunted as a result of the development of nitrate tolerance. Hydralazine has been shown to prevent tolerance in experimental and clinical studies, all of which may be at least in part secondary to antioxidant properties of this compound. The antioxidant effects of hydralazine were tested in cell free systems, cultured smooth muscle cells, isolated mitochondria, and isolated vessels. Inhibitory effects on the formation of superoxide and/or peroxynitrite formation were tested using lucigenin and L-012 enhanced chemiluminescence as well as DHE-fluorescence. The peroxynitrite scavenging properties were also assessed by…

Malemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentBiophysicsMitochondrionBiochemistryAntioxidantsMitochondrial ProteinsNitroglycerinchemistry.chemical_compoundPeroxynitrous AcidInternal medicinemedicineAnimalsHumansLucigeninRats WistarMolecular BiologyHeart FailureSuperoxideAldehyde Dehydrogenase MitochondrialMicrofilament ProteinsDrug ToleranceFree Radical ScavengersCell BiologyAldehyde DehydrogenaseHydralazineHydralazinePhosphoproteinsPrognosismedicine.diseaseReactive Nitrogen SpeciesMitochondriaRatsOxidative StressEndocrinologychemistryHeart failureIsosorbide dinitrateReactive Oxygen SpeciesCell Adhesion MoleculesPeroxynitritemedicine.drugBiochemical and Biophysical Research Communications
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17β-Estradiol Reduces Nitric Oxide Production in the Guinea Pig Cochlea

2013

Intense noise exposure and the application of ototoxic substances result in increased levels of reactive oxygen species (ROS) and reactive nitrogen species (RNS), such as nitric oxide (NO). In order to reduce the free NO concentration in the inner ear under pathological conditions, the use of natural cytoprotective substances such as 17β-estradiol is a promising therapeutic concept. In male guinea pigs the organ of Corti and the lateral wall were isolated from the cochlea and afterwards incubated for 6 h in cell-culture medium. 17β-Estradiol was adjusted in 2 concentrations to organ cultures of the right ears (12 animals per concentration). The left ears were used as controls. The NO produc…

Malemedicine.medical_specialtyEndocrinology Diabetes and MetabolismGuinea PigsClinical BiochemistryDown-RegulationBiologyNitric OxideCell morphologyOrgan cultureBiochemistryNitric oxidechemistry.chemical_compoundOrgan Culture TechniquesEndocrinologyInternal medicineotorhinolaryngologic diseasesmedicineAnimalsInner earCell ShapeNitritesCochleaReactive nitrogen specieschemistry.chemical_classificationReactive oxygen speciesEstradiolBiochemistry (medical)General MedicineCochleaUp-Regulationmedicine.anatomical_structureEndocrinologychemistryOrgan of Cortisense organsHormone and Metabolic Research
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Silibinin modulates lipid homeostasis and inhibits nuclear factor kappa B activation in experimental nonalcoholic steatohepatitis.

2012

Nonalcoholic steatohepatitis (NASH) is associated with increased liver-related mortality. Disturbances in hepatic lipid homeostasis trigger oxidative stress and inflammation (ie, lipotoxicity), leading to the progression of NASH. This study aimed at identifying whether silibinin may influence the molecular events of lipotoxicity in a mouse model of NASH. Eight-week-old db/db mice were fed a methionine-choline deficient (MCD) diet for 4 weeks and treated daily with silibinin (20 mg/kg intraperitoneally) or vehicle. Liver expression and enzyme activity of stearoyl-CoA desaturase-1 and acyl-CoA oxidase, and expression of liver fatty acid-binding protein were assessed. Hepatic levels of reactiv…

Malemedicine.medical_specialtyMice ObeseSilibininmedicine.disease_causeAntioxidantsTranslational Research BiomedicalMicechemistry.chemical_compoundMethionineNon-alcoholic Fatty Liver DiseasePhysiology (medical)Internal medicineNonalcoholic fatty liver diseasemedicineTBARSAnimalsHomeostasisNASH MCD Silibinin lipotoxicity.Reactive nitrogen speciesLiver injurychemistry.chemical_classificationReactive oxygen speciesAnti-Inflammatory Agents Non-SteroidalBiochemistry (medical)NF-kappa BPublic Health Environmental and Occupational HealthGeneral MedicineLipid Metabolismmedicine.diseaseCholine DeficiencyFatty LiverDisease Models AnimalOxidative StressEndocrinologyLiverchemistryLipotoxicitySilybinOxidative stressSilymarin
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Redox signaling (cross-talk) from and to mitochondria involves mitochondrial pores and reactive oxygen species

2010

This review highlights the important role of redox signaling between mitochondria and NADPH oxidases. Besides the definition and general importance of redox signaling, the cross-talk between mitochondrial and Nox-derived reactive oxygen species (ROS) is discussed on the basis of 4 different examples. In the first model, angiotensin-II is discussed as a trigger for NADPH oxidase activation with subsequent ROS-dependent opening of mitochondrial ATP-sensitive potassium channels leading to depolarization of mitochondrial membrane potential followed by mitochondrial ROS formation and respiratory dysfunction. This concept was supported by observations that ethidium bromide-induced mitochondrial d…

Mitochondrial ROSAgingPotassium ChannelsMyocytes Smooth MuscleBiophysicsIn Vitro TechniquesMitochondrionmedicine.disease_causeMitochondrial Membrane Transport ProteinsModels BiologicalMitochondrial apoptosis-induced channelBiochemistryPeroxynitritechemistry.chemical_compoundmedicineAnimalsHumansMitochondrionFeedback PhysiologicalNADPH oxidasebiologyNADPH oxidaseMitochondrial Permeability Transition PoreSuperoxideAngiotensin IINADPH OxidasesSuperoxideNitric oxideCell BiologyReactive Nitrogen SpeciesMitochondriaCell biologyOxidative StressOxidative protein modificationchemistryMitochondrial permeability transition poreRedox regulationNOX1Hypertensionbiology.proteinReactive Oxygen SpeciesOxidation-ReductionOxidative stressSignal TransductionBiochimica et Biophysica Acta (BBA) - Bioenergetics
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Nitric oxide synthase inhibition and oxidative stress in cardiovascular diseases: Possible therapeutic targets?

2013

International audience; Nitric oxide (• NO) is synthetized enzymatically from L-arginine (L-Arg) by three NO synthase isoforms, iNOS, eNOS and nNOS. The synthesis of NO is selectively inhibited by guanidino-substituted analogs of L-Arg or methylarginines such as asymmetric dimethylarginine (ADMA), which results from protein degradation in cells. Many disease states, including cardiovascular diseases and diabetes, are associated with increased plasma levels of ADMA. The N-terminal catalytic domain of these NOS isoforms binds the heme prosthetic group as well as the redox cofactor, tetrahydrobiopterin (BH 4) associated with a regulatory protein, calmodulin (CaM). The enzymatic activity of NOS…

NO inhibitorsfree radicals030204 cardiovascular system & hematologyProtein degradationPharmacologyNitric OxideNitric oxide03 medical and health scienceschemistry.chemical_compoundBH 40302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemcardiovascular diseaseEnosmedicineAnimalsHumansPharmacology (medical)Enzyme InhibitorsEndothelial dysfunctionReactive nitrogen species030304 developmental biologyPharmacologyNO synthases0303 health sciencesbiologyTetrahydrobiopterinbiology.organism_classificationmedicine.disease[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular system3. Good healthADMANitric oxide synthaseOxidative StresschemistryBiochemistryCardiovascular Diseasesbiology.proteinNitric Oxide SynthaseAsymmetric dimethylargininemedicine.drugPharmacology & Therapeutics
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Redox signaling in acute pancreatitis

2015

Acute pancreatitis is an inflammatory process of the pancreatic gland that eventually may lead to a severe systemic inflammatory response. A key event in pancreatic damage is the intracellular activation of NF-κB and zymogens, involving also calcium, cathepsins, pH disorders, autophagy, and cell death, particularly necrosis. This review focuses on the new role of redox signaling in acute pancreatitis. Oxidative stress and redox status are involved in the onset of acute pancreatitis and also in the development of the systemic inflammatory response, being glutathione depletion, xanthine oxidase activation, and thiol oxidation in proteins critical features of the disease in the pancreas. On th…

NecrosisGSH reduced glutathioneSTAT3 signal transducer and activator of transcription 3ERK extracellular signal-regulated kinasesClinical BiochemistryCCK cholecystokininTRAFs TNF receptor associated factorsReview ArticleIκB kinasePharmacologymedicine.disease_causeBiochemistrySHP small heterodimer partnerSTIM1 stromal interaction molecule 1chemistry.chemical_compoundHATs histone acetyltransferasesMedicineASK1GCL glutamate cysteine ligaseTNF-α tumor necrosis factor alphaIKK IκB kinaseNOS nitric oxide synthaseAcute inflammationHIF hypoxia inducible factorlcsh:QH301-705.5NF-κB nuclear factor kappa BDAMPs damage-associated molecular pattern moleculeslcsh:R5-920biologyGSSG oxidized glutathioneNF-kappa BNLRs nucleotide-binding oligomerization domain (NOD) like receptorsTRADD tumor necrosis factor receptor type 1-associated DEATH domain proteinTRPC3 transient receptor potential channel 3VEGF vascular endothelial growth factorGlutathioneTNFR tumor necrosis factor receptorHMGB1 high-mobility group Box 1 proteinIP3R inositol 145-trisphosphate receptor type 3VCAM-1 Vascular Cell adhesion protein 1Acute DiseaseJNK c-Jun N-terminal kinaseAcute pancreatitisTLRs toll-like receptorsmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionAP-1 activator protein-1Signal TransductionmRNA messenger ribonucleic acidHMGB1ASC apoptosis-associated speck-like protein containing a carboxy-terminal CARDRNS reactive nitrogen speciesPTPs protein tyrosine phosphatasesROS reactive oxygen speciesNADH nicotinamide adenine dinucleotidepHe extracellular pHFAEE fatty acid ethyl estersAP acute pancreatitisHumansXanthine oxidaseCBP CREB-binding proteinRyR endoplasmic reticulum membrane ryanodine receptorsMDA malondialdehydeNO nitric oxideXO xanthine oxidaseASK1 apoptosis signal-regulating kinase-1business.industryOrganic ChemistryAutophagyNADPH nicotinamide adenine dinucleotide phosphateHDACs histone deacetylasesmedicine.diseaseCARS compensatory anti-inflammatory response syndromeXDH xanthine dehydrogenaseIL interleukinIκB inhibitor of kappa BAcute pancreatitisETC Electron transport chainPancreatitisMKPs MAPK phosphatasesSAP severe acute pancreatitischemistrylcsh:Biology (General)DTT dithiothreitolOxidative stressNAC N-acetyl cysteineImmunologybiology.proteinCalciumLysosomesReactive Oxygen SpeciesbusinessMAPK mitogen-activated protein kinaseOxidative stressERCP endoscopic retrograde cholangiopancreatographyRedox Biology
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DNA strand breaks induced by nuclear hijacking of neuronal NOS as an anti-cancer effect of 2-methoxyestradiol

2015

2-Methoxyestradiol (2-ME) is a physiological metabolite of 17β-estradiol. At pharmacological concentrations, 2-ME inhibits colon, breast and lung cancer in tumor models. Here we investigated the effect of physiologically relevant concentrations of 2-ME in osteosarcoma cell model. We demonstrated that 2-ME increased nuclear localization of neuronal nitric oxide synthase, resulting in nitro-oxidative DNA damage. This in turn caused cell cycle arrest and apoptosis in osteosarcoma cells. We suggest that 2-ME is a naturally occurring hormone with potential anti-cancer properties.

Pathologymedicine.medical_specialtyneuronal nitric oxide synthaseCell cycle checkpoint2-methoxyestradiolDNA damageAntineoplastic AgentsApoptosisBone NeoplasmsNitric Oxide Synthase Type Imedicine.disease_causeNitric OxideNitric oxidechemistry.chemical_compoundReactive nitrogen specieCell Line TumormedicineHumans2-MethoxyestradiolReactive nitrogen speciesCytokinesisOsteosarcomaEstradiolbusiness.industryDNA BreaksIntracellular Signaling Peptides and ProteinsCancermedicine.diseaseReactive Nitrogen SpeciesG2 Phase Cell Cycle CheckpointsOxidative StressOncologychemistryApoptosis2-methoxyestradiol; Neuronal nitric oxide synthase; Nitric oxide; Osteosarcoma; Reactive nitrogen species; OncologyCancer researchM Phase Cell Cycle CheckpointsbusinessTumor Suppressor p53-Binding Protein 1Oxidative stressmedicine.drugResearch Paper
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Drugs modulating the biological effects of peroxynitrite and related nitrogen species

2006

The term “reactive nitrogen species” includes nitrogen monoxide, commonly called nitric oxide, and some other remarkable chemical entities (peroxynitrite, nitrosoperoxycarbonate, etc.) formed mostly from nitrogen monoxide itself in biological environments. Regardless of the specific mechanisms implicated in their effects, however, it is clear that an integrated pharmacological approach to peroxynitrite and related species is only just beginning to take shape. The array of affected chemical and pathological processes is extremely broad. One of the most conspicuous mechanisms observed thus far has been the scavenging of the peroxynitrite anion by molecules endowed with antioxidant activity. T…

PharmacologyAntioxidantmedicine.medical_treatmentOxidative phosphorylationLung injuryIn vitroNitric oxidechemistry.chemical_compoundchemistryBiochemistryIn vivoDrug DiscoverymedicineMolecular MedicineReactive nitrogen speciesPeroxynitriteMedicinal Research Reviews
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Oxidative stress and pulmonary inflammation: pharmacological intervention with antioxidants.

1999

Reactive oxygen and nitrogen species are generated by several inflammatory and structural cells of the airways. These oxidant species may have important effects on different lung cells as regulators of signal transduction, activators of key transcription factors, and modulators of gene expression and apoptosis. Thus, an increased oxidative stress accompanied by reduced endogenous antioxidant defences may have a role in the pathogenesis of a number of inflammatory pulmonary diseases including asthma. Although antioxidant drugs could play a useful role in the therapy of inflammatory lung diseases, their clinical impact is relatively modest at present. Rigorous clinical investigation with the …

Pharmacologychemistry.chemical_classificationReactive oxygen speciesAntioxidantLungmedicine.medical_treatmentPneumoniaBiologymedicine.disease_causeAntioxidantsPathogenesischemistry.chemical_compoundOxidative Stressmedicine.anatomical_structurechemistryApoptosisImmunologymedicineAnimalsHumansSignal transductionLungReactive nitrogen speciesOxidative stressPharmacological research
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What is the function of neuroglobin?

2009

SUMMARY For a long time, haemoglobin and myoglobin had been assumed to represent the only globin types of vertebrates. In 2000, however, we discovered a third globin type by mining the genome sequence data. Based on a preferential expression in the nervous system, this globin is referred to as neuroglobin. Despite nine years of research, its function is still uncertain and a number of hypotheses have been put forward. Neuroglobin enhances cell viability under hypoxia and under various types of oxidative stress in transgenic systems, but does not appear to be strongly upregulated in response to stress. A close phylogenetic relationship with invertebrate nerve globins and its positive correla…

PhysiologyTransgeneCellNeuroglobinNerve Tissue ProteinsAquatic ScienceMitochondrionBiologymedicine.disease_causeNervous Systemchemistry.chemical_compoundmedicineAnimalsGlobinHypoxiaMolecular BiologyEcology Evolution Behavior and SystematicsReactive nitrogen speciesNeuronsCytoglobinReactive Nitrogen SpeciesCell biologyGlobinsOxygenmedicine.anatomical_structurechemistryBiochemistryGene Expression RegulationInsect ScienceNeuroglobinVertebratesAnimal Science and ZoologyReactive Oxygen SpeciesOxidative stressThe Journal of experimental biology
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