Search results for "Reactive"

showing 10 items of 1469 documents

Endotoxin accelerates atherosclerosis independent of complement activation

2008

a Central Laboratory Animal Facility b Institute of Clinical Chemistry and Laboratory Medicine c Institute of Medical Microbiology and Hygiene, Johannes Gutenberg University, Mainz, Germany d Klinik fur Gefaschirurgie und Nierentransplantation, Heinrich Heine Universitatsklinik, Dusseldorf, Germany e Zentrum fur Medizin und Biowissenschaften, Forschungszentrum, Borstel, Germany f Centre Hospitalier Universitaire Vaudois, Lausanne, Switzerland g Department of Molecular Pathology, Graduate School of Medicine and Engineering, University of Yamanashi, Japan

MaleTime FactorsLibrary scienceHyperlipidemiasHematologyBiologyAtherosclerosisComplement C6Central laboratoryEndotoxinsC-Reactive ProteinCholesterolImmunologyAnimalsHumansFemaleRabbitsComplement ActivationTriglyceridesAnimal facilityThrombosis Research
researchProduct

Incremental predictive value of mean platelet volume/platelet count ratio in in-hospital stroke after acute myocardial infarction.

2017

IF 2.558; International audience; Stroke is a serious complication after acute myocardial infarction (AMI) and is associated with an increased risk of death. Though the pathophysiological mechanisms are not exactly known, increased inflammation and platelet reactivity could play an important role in the occurrence of stroke during AMI. We aimed to investigate the relationship between both mean platelet volume (MPV), a parameter of platelet function, and C-reactive protein (CRP) and the occurrence of in-hospital ischemic stroke (IHS) after AMI. Data were obtained from a French regional survey for AMI that included 5976 patients admitted to an intensive care unit (ICU) between 2001 and 2010. …

MaleTime FactorsMESH : StrokeMyocardial InfarctionMESH : AgedMESH: ComorbidityComorbidityMESH: Hospitalization030204 cardiovascular system & hematologyMESH : Platelet Countlaw.inventionMESH: Proportional Hazards Models0302 clinical medicineMESH: Aged 80 and overRisk FactorslawMESH: Risk FactorsOdds Ratio[ SDV.MHEP.HEM ] Life Sciences [q-bio]/Human health and pathology/HematologyMESH : FemaleMyocardial infarctionMESH : BiomarkersStrokeMESH: Blood PlateletsAged 80 and overMESH: AgedEjection fractionMESH: Middle AgedMESH : PrognosisbiologyMESH : Mean Platelet Volume[SDV.MHEP.HEM]Life Sciences [q-bio]/Human health and pathology/HematologyHematologyGeneral MedicineMiddle AgedPrognosisIntensive care unitstrokeMESH : Risk Factors3. Good healthHospitalizationMESH: Myocardial Infarctionrisk factorplateletsMESH : ComorbidityCardiologyMESH : HospitalizationFemaleMean Platelet VolumeMESH : Time FactorsBlood Plateletsmedicine.medical_specialtyMESH : Male[SDV.BC]Life Sciences [q-bio]/Cellular BiologyAcute myocardial infarctionMESH: PrognosisMESH: StrokeC-reactive protein03 medical and health sciencesInternal medicinemedicineHumansMESH : Middle AgedMESH: Platelet Countcardiovascular diseasesRisk factorMean platelet volumeMESH : Aged 80 and overSurvival analysisAgedProportional Hazards ModelsMESH: HumansPlatelet Countbusiness.industry[ SDV.BC ] Life Sciences [q-bio]/Cellular BiologyC-reactive proteinMESH: Time FactorsMESH : HumansMESH : Blood Plateletsmedicine.diseaseMESH : Proportional Hazards ModelsMESH: Odds RatioMESH: MaleMESH: Mean Platelet Volumebiology.proteinMESH: BiomarkersMESH : Odds RatioMESH : Myocardial InfarctionbusinessMESH: FemaleBiomarkers030217 neurology & neurosurgery
researchProduct

Replenishment of glutathione levels improves mucosal function in experimental acute colitis.

2000

Because reactive oxygen species (ROS) have been implicated as mediators of inflammatory bowel disease (IBD), the purpose of the present work was to determine the functional role of mucosal GSH in the trinitrobenzenesulfonic acid in 50% ethanol (TNBS+ethanol)-induced colitis in rats. Mucosal samples were taken to evaluate the temporal relationship between the extent of injury, the levels of glutathione (GSH) during acute colitis induced by TNBS+ethanol, and the effect of N-acetylcysteine (NAC) administration. In vitro assays revealed the interaction of TNBS with GSH leading to the almost instantaneous disappearance of GSH, while the reductive metabolism of TNBS by GSSG reductase generated RO…

MaleTime Factorsanimal diseasesPharmacologymedicine.disease_causedigestive systemInflammatory bowel diseasePathology and Forensic MedicineAcetylcysteineRats Sprague-Dawleychemistry.chemical_compoundOral administrationmedicineAnimalsColitisIntestinal MucosaMolecular BiologyAcute colitischemistry.chemical_classificationReactive oxygen speciesEthanolCell BiologyGlutathionemedicine.diseaseColitisInflammatory Bowel DiseasesGlutathionedigestive system diseasesAcetylcysteineRatschemistryTrinitrobenzenesulfonic AcidImmunologyAcute DiseaseReactive Oxygen SpeciesOxidative stressmedicine.drugLaboratory investigation; a journal of technical methods and pathology
researchProduct

Endothelial Dysfunction in Tristetraprolin-deficient Mice Is Not Caused by Enhanced Tumor Necrosis Factor-α Expression

2014

Cardiovascular events are important co-morbidities in patients with chronic inflammatory diseases like rheumatoid arthritis. Tristetraprolin (TTP) regulates pro-inflammatory processes through mRNA destabilization and therefore TTP-deficient mice (TTP(-/-) mice) develop a chronic inflammation resembling human rheumatoid arthritis. We used this mouse model to evaluate molecular signaling pathways contributing to the enhanced atherosclerotic risk in chronic inflammatory diseases. In the aorta of TTP(-/-) mice we observed elevated mRNA expression of known TTP targets like tumor necrosis factor-α (TNF-α) and macrophage inflammatory protein-1α, as well as of other pro-atherosclerotic mediators, l…

MaleVasculitismedicine.medical_specialtyMRNA destabilizationRNA StabilityTristetraprolinInflammationBiochemistryNitric oxideMicechemistry.chemical_compoundOrgan Culture TechniquesTristetraprolinhemic and lymphatic diseasesInternal medicinemedicineAnimalsEndothelial dysfunctionMolecular BiologyAortaReactive nitrogen speciesMice KnockoutMembrane GlycoproteinsNADPH oxidasebiologyTumor Necrosis Factor-alphaEndothelial CellsNADPH OxidasesMolecular Bases of DiseaseCell Biologyrespiratory systemAtherosclerosismedicine.diseaseReactive Nitrogen SpeciesMice Inbred C57BLOxidative StressCholesterolEndocrinologychemistryMice Inbred DBAChronic DiseaseNADPH Oxidase 2biology.proteinFemaleTumor necrosis factor alphamedicine.symptomReactive Oxygen SpeciesJournal of Biological Chemistry
researchProduct

Effect of a two-year supplementation with low doses of antioxidant vitamins and/or minerals in elderly subjects on levels of nutrients and antioxidan…

1997

Eighty-one elderly hospitalized subjects (65 years) were recruited for a double-blind placebo-controlled study to examine low dose supplementation of antioxidant vitamins and minerals on biological and functional parameters of free radical metabolism. Subjects were randomly assigned to one of the four treatment groups, daily receiving for 2 years: placebo group; mineral group: 20 mg zinc, 100 micrograms selenium; vitamin group: 120 mg vitamin C (Vit C), 6 mg beta-carotene (beta CA), 15 mg vitamin E (Vit E); mineral and vitamin group: Zn 20 mg, Se 100 micrograms, Vit C 120 mg, beta CA 6 mg, Vit E 15 mg.Fifty-seven subjects completed the study. A large frequency of Vit C, Zn and Se deficienci…

MaleVitaminmedicine.medical_specialtyErythrocytesAntioxidantmedicine.medical_treatmentNutritional StatusMedicine (miscellaneous)chemistry.chemical_elementAscorbic AcidThiobarbituric Acid Reactive SubstancesAntioxidantsSeleniumchemistry.chemical_compoundOral administrationInternal medicinemedicineHumansVitamin ENutritional Physiological PhenomenaAgedAged 80 and overGlutathione PeroxidaseMineralsNutrition and DieteticsVitamin CSuperoxide DismutaseVitamin EVitaminsMetabolismbeta CaroteneAscorbic acidGlutathioneZincEndocrinologychemistryBiochemistryFemaleSeleniumJournal of the American College of Nutrition
researchProduct

Complex I dysfunction and tolerance to nitroglycerin: an approach based on mitochondrial-targeted antioxidants.

2006

Nitroglycerin (GTN) tolerance was induced in vivo (rats) and in vitro (rat and human vessels). Electrochemical detection revealed that the incubation dose of GTN (5×10 −6 mol/L) did not release NO or modify O 2 consumption when administered acutely. However, development of tolerance produced a decrease in both mitochondrial O 2 consumption and the K m for O 2 in animal and human vessels and endothelial cells in a noncompetitive action. GTN tolerance has been associated with impairment of GTN biotransformation through inhibition of aldehyde dehydrogenase (ALDH)-2, and with uncoupling of mitochondrial respiration. Feeding rats with mitochondrial-targeted antioxidants (mitoquinone [MQ]) and i…

MaleantioxidantAntioxidantPhysiologyUbiquinonemedicine.medical_treatmentMuscle RelaxationVasodilator AgentsAldehyde dehydrogenasePharmacologyMitochondrionmedicine.disease_causeAntioxidantsMuscle Smooth VascularRats Sprague-Dawleychemistry.chemical_compoundNitroglycerinDrug toleranceoxidative stressCyclic GMPchemistry.chemical_classificationbiologyAldehyde Dehydrogenase MitochondrialDrug ToleranceGlutathioneMitochondriamitochondriaBiochemistrycardiovascular systemCardiology and Cardiovascular Medicinecirculatory and respiratory physiologyMuscle ContractionendotheliumIn Vitro TechniquesMitochondrial ProteinsOrganophosphorus CompoundsOxygen ConsumptionRespirationmedicineAnimalsHumansReactive oxygen speciesElectron Transport Complex IDose-Response Relationship DrugEndothelial CellsGlutathioneAldehyde DehydrogenasenitroglycerinRatsOxidative Stresschemistrybiology.proteinReactive Oxygen SpeciesOxidative stressCirculation research
researchProduct

Increased interleukin-6 and C-reactive protein levels after instrumented lumbar spine fusion in older patients

2019

Purpose: Interleukin 6 (IL-6) and the acute phase C-reactive protein (CRP) blood concentrations after lumbar spine fusion may be affected by age. The purpose of this prospective observational study was to assess postoperative serum levels of pro-inflammatory IL-6 and CRP after instrumented lumbar spine fusion surgery. We hypothesized that older patients would have increased levels of IL-6 and CRP after surgery. Methods: IL-6 and high-sensitive CRP biochemical marker levels were measured before instrumented spinal fusion, and postoperatively at 1 and 3 days, 6 weeks, and 3 months. The 49 patients in this sample were divided into two groups: age ≤ 60 years (n = 23) and age > 60 years (n = 26)…

MalefusionGastroenterologyleikkaushoitoDISEASEsurgery0302 clinical medicinePostoperative Complicationslcsh:Orthopedic surgeryOlder patientsMARKERSMedicinePostoperative PeriodProspective Studieskirurgiata315Aged 80 and over030222 orthopedicsLumbar VertebraebiologyAge FactorsMiddle AgedRECOVERYFemaleSpinal Diseasesmedicine.symptommusculoskeletal diseasesAdultmedicine.medical_specialtyLumbar spine fusionInflammationspinelannerankaC-reactive protein03 medical and health sciencesLumbarAGEselkärankaINFLAMMATIONc-reaktiivinen proteiiniInternal medicineINJURYHumansQUALITYInterleukin 6lumbarAgedbusiness.industryinterleukin-6DISABILITYC-reactive protein030229 sport sciences3126 Surgery anesthesiology intensive care radiologylcsh:RD701-811Spinal Fusionbiology.proteinbusinessBiomarkers
researchProduct

Reactive hyperemia index (RHI) and cognitive performance indexes are associated with histologic markers of liver disease in subjects with non-alcohol…

2017

BACKGROUND: No study evaluated vascular health markers in subjects with non-alcoholic fatty liver disease (NAFLD) through a combined analysis of reactive hyperemia peripheral arterial tonometry (RH-PAT) and arterial stiffness indexes. AIM OF THE STUDY: We aimed to assess whether NAFLD and its histological severity are associated with impairment of arterial stiffness and RH-PAT indexes in a mixed cohort of patients with biopsy-proven NAFLD. MATERIALS AND METHODS: The Kleiner classification was used to grade NAFLD grade. Pulse wave velocity (PWV) and augmentation index (Aix) were used as markers of arterial stiffness, whereas endothelial function was assessed using reactive hyperemia index (R…

Malelcsh:Diseases of the circulatory (Cardiovascular) systemSettore MED/09 - Medicina InternaBiopsyEndocrinology Diabetes and MetabolismHistopathology; Liver fibrosis; Non-alcoholic fatty liver disease030204 cardiovascular system & hematologySeverity of Illness IndexLiver diseaseCognition0302 clinical medicineRisk FactorsPrevalencePulse wave velocityOriginal InvestigationFatty liverMiddle AgedMental Status and Dementia TestsItalyLiverCardiovascular DiseasesCardiologyFemaleCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtyManometryLiver fibrosisHistopathologyHyperemiaPulse Wave Analysis03 medical and health sciencesVascular StiffnessDiabetes mellitusInternal medicineInternal MedicinemedicineHumansReactive hyperemiaAgedbusiness.industryLiver fibrosinutritional and metabolic diseasesmedicine.diseasedigestive system diseasesCross-Sectional StudiesBlood pressurelcsh:RC666-701Case-Control StudiesArterial stiffnessSteatohepatitisCognition Disordersbusiness030217 neurology & neurosurgeryNon-alcoholic fatty liver diseaseCardiovascular Diabetology
researchProduct

[Relationship of C-reactive protein levels with angiographic findings and markers of necrosis in non-ST-segment elevation acute coronary syndrome].

2004

The mechanism responsible for elevated C-reactive protein levels (inflammation of the ruptured atherosclerotic plaque or myocardial necrosis) in acute coronary syndromes is controversial. The aim of this study was to investigate the relationship between C-reactive protein levels and angiographic complexity of the culprit lesion and troponin elevation in patients with non-ST elevation acute coronary syndromes.The study group consisted of 125 patients with single-vessel disease. Troponin-I and C-reactive protein were measured, and the complexity of the culprit lesion was analyzed (TIMI flow and thrombus). Information on age, sex, smoking habit, hypertension, hypercholesterolemia and diabetes …

Malemedicine.medical_specialtyAcute coronary syndromeCoronary DiseaseCoronary AngiographyElectrocardiographyRisk FactorsInternal medicineDiabetes mellitusmedicineST segmentHumansThrombusbiologyRupture SpontaneousUnstable anginabusiness.industryC-reactive proteinTroponin IGeneral MedicineSyndromeMiddle Agedmedicine.diseaseTroponinC-Reactive ProteinAcute Diseasebiology.proteinCardiologyFemalebusinessTIMIBiomarkersRevista espanola de cardiologia
researchProduct

Insulin resistance and acute coronary syndrome

2010

BACKGROUND: Insulin resistance (IR), which can be quantified by HOMA index (fasting glucose X fasting insulin/22.5), is considered the "primum movens" for the development of Metabolic Syndrome. Many authors have suggested that insulin resistance could raise both incidence and mortality of coronary heart disease (CHD). IR is also associated with important predictors of cardiovascular disease, as increased concentration of LDL or triglyceride, decreased concentration of HDL, high systolic blood pressure, visceral obesity. There is accumulating evidence that chronic sub-clinical inflammation, as measured by inflammatory markers as C-reactive protein (CRP) and fibrinogen, is related with insuli…

Malemedicine.medical_specialtyAcute coronary syndromemedicine.medical_treatmentBlood PressureCoronary DiseaseCoronary artery diseaseInsulin resistanceInternal medicinemedicineHyperinsulinemiaHumansObesityAcute Coronary SyndromeAgedInflammationType 1 diabetesbusiness.industryInsulinCholesterol HDLFibrinogenCholesterol LDLMiddle Agedmedicine.diseaseSettore MED/11 - Malattie Dell'Apparato CardiovascolareC-Reactive ProteinEndocrinologyCardiologyFemaleInsulin ResistanceMetabolic syndromeCardiology and Cardiovascular MedicinebusinessINSULIN RESISTANCE ACUTE CORONARY SYNDROMEDyslipidemiaAtherosclerosis
researchProduct