Search results for "Relationship"

showing 10 items of 3616 documents

Role of Ca2+-activated K+ channels and Na+,K+-ATPase in prostaglandin E1- and E2-induced inhibition of the adrenergic response in human vas deferens

2011

We studied the role of K(+) channels and Na(+),K(+)-ATPase in the presynaptic inhibitory effects of prostaglandin E(1) (PGE(1)) and PGE(2) on the adrenergic responses of human vas deferens. Furthermore, we determined the effects of increasing extracellular K(+) concentrations ([K(+)](o)) and inhibition of Na(+),K(+)-ATPase on neurogenic and norepinephrine-induced contractile responses. Ring segments of the epididymal part of the vas deferens were taken from 45 elective vasectomies and mounted in organ baths for isometric recording of tension. The neuromodulatory effects of PGEs were tested in the presence of K(+) channel blockers. PGE(1) and PGE(2) (10(-8) to 10(-6)M) induced inhibition of …

AdultMalemedicine.medical_specialtyBioquímica clínicaAdrenergicApaminSynaptic TransmissionBiochemistryDinoprostoneOuabainPotassium Channels Calcium-Activatedchemistry.chemical_compoundOrgan Culture TechniquesVas DeferensInternal medicinePotassium Channel BlockersmedicineHumansAlprostadilNa+/K+-ATPasePharmacologyDose-Response Relationship DrugChemistryVas deferensPotassium channel blockerIberiotoxinElectric StimulationPotassium channelReceptors AdrenergicEndocrinologymedicine.anatomical_structurelipids (amino acids peptides and proteins)Sodium-Potassium-Exchanging ATPaseMuscle Contractionmedicine.drugBiochemical Pharmacology
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[123I]IBZM SPECT in patients treated with typical and atypical neuroleptics: relationship to drug plasma levels and extrapyramidal side effects

1997

[123I]Iodobenzamide (IBZM) is an iodine-labeled dopamine receptor ligand and can be used to visualize brain D2 receptors in humans with single photon emission computerized tomography (SPECT). The ratio of striatal IBZM uptake to uptake in frontal cortex (ST/FC ratio) represents a semiquantitative measure of D2 receptor binding in the striatum. Our study sample included six patients treated with haloperidol (3.0-8.0 mg/day orally; one patient with an average of 0.9 mg/day intramuscularly), five patients with benperidol (9.0-15.0 mg/day orally) and nine patients treated with clozapine (200.0-600.0 mg/day orally). Typical neuroleptics (TNs) and atypical neuroleptics (ANs) were significantly di…

AdultMalemedicine.medical_specialtyBipolar DisorderPyrrolidinesNeuroscience (miscellaneous)Benperidolchemistry.chemical_compoundIodobenzamideBasal Ganglia DiseasesDopamineInternal medicineDopamine receptor D2medicineHaloperidolHumansRadiology Nuclear Medicine and imagingChlorpromazineClozapineClozapineAgedNeurologic ExaminationPsychiatric Status Rating ScalesTomography Emission-Computed Single-PhotonDepressive Disorder MajorSchizophrenia ParanoidDose-Response Relationship DrugReceptors Dopamine D2business.industryBenperidolBrainMiddle AgedCorpus StriatumFrontal LobePsychiatry and Mental healthEndocrinologychemistryDopamine receptorBenzamidesDopamine AntagonistsHaloperidolFemalebusinessAntipsychotic Agentsmedicine.drugPsychiatry Research: Neuroimaging
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Long-term follow-up (42 months) of chronic anal fissure after healing with botulinum toxin.

2002

Abstract Background & Aims: Botulinum toxin is an effective treatment in idiopathic chronic anal fissure, but the long-term outcome after healing is not well documented. We analyzed the long-term outcome of patients in whom an anal fissure had healed after botulinum toxin injection and the factors contributing to recurrence. Methods: Fifty-seven patients who had completely healed 6 months after injection of botulinum toxin were reassessed every 6 months. The follow-up was 42 months in all patients. Clinical and manometric differences between the permanently healed and the relapsed group were statistically analyzed. Results: Four patients were lost to follow-up. A fissure recurrence was show…

AdultMalemedicine.medical_specialtyBotulinum ToxinsLong term follow upmedicine.medical_treatmentChronic anal fissureInjectionsRecurrenceLate RecurrencemedicinePressureEffective treatmentHumansAgedAnal fissureChemotherapyWound HealingHepatologyDose-Response Relationship Drugbusiness.industryGastroenterologyMiddle Agedmedicine.diseaseBotulinum toxinSurgeryTotal doseChronic DiseaseRetreatmentFemaleFissure in Anobusinessmedicine.drugFollow-Up StudiesGastroenterology
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Botulinum toxin A (Botox) and sweating-dose efficacy and comparison to other BoNT preparations.

2004

Abstract Background Botulinum toxin type A (BoNT/A) is 20–50 times more effective than Botulinum toxin type B (BoNT/B) concerning the treatment of muscular hypercontractions [Sloop, R.R., Cole, B.A., Escutin, R.O., 1997. Human response to botulinum toxin injection: type B compared with type A. Neurology 49, 189–194]. Botulinum toxins block motor nerves as well as autonomic fibres [Rand, M.J., Whaler, B.C., 1965. Impairment of sympathetic transmission by botulinum toxin. Nature 206, 588–591]. Objective Purpose of this study was to analyse the dose dependent reduction of sweating using the BoNT/A preparation Botox® and to compare the results with our earlier results analysing Dysport® [Braune…

AdultMalemedicine.medical_specialtyBotulinum ToxinsTime FactorsInjections SubcutaneousSweatingBotulinum toxin aCellular and Molecular NeuroscienceMedicineHumansBotulinum toxin type BAnhidrosisBotulinum Toxins Type AHypohidrosisDose-Response Relationship DrugEndocrine and Autonomic Systemsbusiness.industryStarchBotulinum toxinSurgerySudomotorDose–response relationshipThreshold doseNeuromuscular AgentsAnesthesiaFemaleNeurology (clinical)medicine.symptombusinessBotulinum toxin typemedicine.drugFollow-Up StudiesAutonomic neuroscience : basicclinical
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Dose-dependent effects of decaffeinated coffee on endothelial function in healthy subjects

2009

ackground/Objectives: Coffee is known to contain antioxidant substances whose effects may be blunted because of caffeine that may unfavorably affect the cardiovascular system. This study was designed to investigate the acute dose-dependent effects of decaffeinated coffee (DC) on endothelial function measured by the brachial artery flow-mediated dilation (FMD). Subjects/Methods: A total of 15 (8 men and 7 women) healthy nonobese subjects underwent a single-blind, crossover study. Subjects ingested one and two cups of decaffeinated Italian espresso coffee in random order at 5- to 7-day intervals. Results: In the hour following the ingestion of two cups of DC, FMD increased (mean±s.e.m.): 0 mi…

AdultMalemedicine.medical_specialtyBrachial ArteryEndotheliumMedicine (miscellaneous)Blood PressureCoffeeGastroenterologychemistry.chemical_compoundHeart Ratemedicine.arteryInternal medicineHeart ratemedicineHumansIngestionSingle-Blind MethodBrachial arteryCross-Over StudiesNutrition and DieteticsDose-Response Relationship Drugbusiness.industryCrossover studyVasodilationEndocrinologyBlood pressuremedicine.anatomical_structurechemistryRegional Blood FlowArmFemaleEndothelium VascularAnalysis of varianceEndothelial function FMD coffeeCaffeinebusinessBlood Flow VelocityEuropean Journal of Clinical Nutrition
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Endothelium-dependent responses in human isolated thyroid arteries from donors

2004

The functional properties of the endothelium of human thyroid arteries remain unexplored. We investigated the intervention of nitric oxide (NO), prostacyclin (PGI(2)) and endothelium-derived hyperpolarizing factor (EDHF) in the responses to acetylcholine and noradrenaline in isolated thyroid arteries obtained from multi-organ donors. Artery rings were suspended in organ baths for isometric recording of tension. The contribution of NO, PGI(2) and EDHF to endothelium-dependent relaxation was determined by the inhibitory effects of N(G)-monomethyl-L-arginine (L-NMMA), indomethacin, and K(+) channel inhibitors respectively. Acetylcholine induced concentration-dependent relaxation; this effect w…

AdultMalemedicine.medical_specialtyCharybdotoxinCharybdotoxinEndotheliumEndocrinology Diabetes and MetabolismIndomethacinThyroid GlandProstacyclinNitric OxideApaminOuabainNitric oxideBiological FactorsNorepinephrinechemistry.chemical_compoundEndocrinologyCulture TechniquesInternal medicinePotassium Channel BlockersmedicineHumansomega-N-MethylarginineDose-Response Relationship DrugArteriesMiddle AgedEpoprostenolAcetylcholineEndocrinologymedicine.anatomical_structureApaminchemistryVasoconstrictioncardiovascular systemFemaleEndothelium VascularNitric Oxide SynthaseSodium-Potassium-Exchanging ATPasePeptidesAcetylcholinemedicine.drugArteryJournal of Endocrinology
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Dose-response relationships and plasma concentrations of digitalis glycosides in man.

1978

An inter-individual, randomized, double-blind study of digitoxin (Dt) and β-acetyl digoxin (D) was performed in 120 healthy male volunteers. Groups of 10 persons each received orally D 0, 0.1, 0.2, 0.3, 0.4, 0.5 or 0.6 mg and Dt 0.04, 0.08, 0.12, or 0.16 mg daily for 7 days; Loading doses were given for the first three days. Plasma levels were measured with an86Rb-erythrocyte assay 24 h after the last dose. ECG, carotid artery pulse and phonocardiogram were recorded prior to (b) and 24 h after (a) the last dose. QTc, amplitude of T-waves in V2 to V6, electromechanical systole (QS2c) and left ventricular ejection time (LVETc) were measured. The differences between a and b (Δ-values) reflect …

AdultMalemedicine.medical_specialtyDigoxinTime FactorsDigoxinDigitoxinQT intervalElectrocardiographyDigitoxinDouble-Blind MethodInternal medicinemedicineHumansPharmacology (medical)Pharmacologychemistry.chemical_classificationClinical Trials as TopicDose-Response Relationship DrugChemistryPulse (signal processing)GlycosideDigitalis GlycosidesHeartGeneral MedicinePlasma levelsEndocrinologySystolic time intervalsPlasma concentrationmedicine.drugEuropean journal of clinical pharmacology
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Therapeutic drug monitoring for optimizing amisulpride therapy in patients with schizophrenia.

2005

Amisulpride is a clinically effective antipsychotic drug in a broad dose range with low propensity for extrapyramidal symptoms (EPS). Daily doses and plasma levels of amisulpride were analyzed within a large-scale therapeutic drug monitoring (TDM) survey to find plasma level ranges for optimized treatment under naturalistic conditions. Data of 378 schizophrenic patients treated with amisulpride (100-1550 mg) were included (40% female). Amisulpride plasma levels were analyzed at steady state; assessment comprised improvement (CGI-I) and side-effects, particularly EPS. For detection of cut-off values regarding non-response or EPS, receiver operating characteristics (ROC) curves were applied a…

AdultMalemedicine.medical_specialtyDyskinesia Drug-InducedAdolescentmedicine.drug_classStatistics as TopicAtypical antipsychoticPharmacologyGastroenterologyExtrapyramidal symptomsInternal medicineGermanymedicineHumansAmisulprideBiological PsychiatryAgedRetrospective StudiesAged 80 and overNeurologic ExaminationPsychiatric Status Rating ScalesReceiver operating characteristicmedicine.diagnostic_testDose-Response Relationship Drugbusiness.industryDopamine antagonistMiddle Agedmedicine.diseasePsychiatry and Mental healthDose–response relationshipROC CurveSchizophreniaTherapeutic drug monitoringSchizophreniaFemalemedicine.symptomAmisulprideDrug MonitoringSulpiridebusinessmedicine.drugAntipsychotic AgentsJournal of psychiatric research
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Tolerance to nitroglycerin-induced preconditioning of the endothelium: a human in vivo study

2009

Damage and dysfunction of the vascular endothelium critically influence clinical outcomes after ischemia and reperfusion (I/R). Brief exposure to organic nitrates can protect the vascular endothelium from I/R injury via a mechanism that is similar to ischemic preconditioning and is independent of hemodynamic changes. The clinical relevance of these protective effects clearly depends on whether they can be sustained over time. Twenty-four healthy (age 25–32) male volunteers were randomized to receive 1) transdermal nitroglycerin (GTN; 0.6 mg/h) administered for 2 h on 1 day only, 2) transdermal GTN for 2 h/day for 7 days, or 3) continuous therapy with transdermal GTN for 7 days. Eight volunt…

AdultMalemedicine.medical_specialtyEndotheliumPhysiologyVasodilator AgentsIschemiaAscorbic AcidAdministration CutaneousAntioxidantsNitroglycerinIn vivoPhysiology (medical)Internal medicinemedicineHumansInfusions Intra-ArterialIschemic PreconditioningNitroglycerinDose-Response Relationship Drugbusiness.industryDrug Tolerancemedicine.diseaseAcetylcholineOrganic nitratesPlethysmographyVascular endotheliummedicine.anatomical_structureReperfusion InjuryAnesthesiaCirculatory systemcardiovascular systemCardiologyIschemic preconditioningEndothelium VascularCardiology and Cardiovascular Medicinebusinesscirculatory and respiratory physiologymedicine.drugAmerican Journal of Physiology-Heart and Circulatory Physiology
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Chemically and electrically induced sweating and flare reaction

2004

Both thin afferent (nociceptors) and efferent (sympathetic sudomotor) nerve fibers can be activated electrically and chemically, resulting in neurogenic erythema and sweating. These reactions have been used before to assess the impairment of sympathetic and nociceptor fibers in humans. In this study, electrically induced sweating and erythema were assessed simultaneously in the foot dorsum and thigh, and were compared to chemically induced activation. Reproducible intensity-response relations (stimulation intensities 0-30 mA, 1 Hz) were obtained from 32 subjects. The steepest increase of the sweat response was induced at lower intensities as compared to that of the erythema (18.3 mA vs. 25.…

AdultMalemedicine.medical_specialtyErythemaEfferentSweatingStimulationFunctional LateralityCellular and Molecular NeuroscienceSex FactorsInternal medicineReflexLaser-Doppler FlowmetrymedicineHumansAgedSkinAnalysis of VarianceNeurogenic inflammationintegumentary systemFootEndocrine and Autonomic SystemsChemistryAge FactorsReproducibility of ResultsDose-Response Relationship RadiationMiddle AgedAcetylcholineElectric StimulationStimulation ChemicalSudomotorAutonomic nervous systemEndocrinologyThighErythemaAnesthesiaNociceptorFemaleNeurology (clinical)medicine.symptomAcetylcholinemedicine.drugAutonomic Neuroscience
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