Search results for "Remodeling"

showing 10 items of 357 documents

'Up-regulation of histone acetylation induced by social defeat mediates the conditioned rewarding effects of cocaine

2016

Social defeat (SD) induces a long-lasting increase in the rewarding effects of psychostimulants measured using the self-administration and conditioned place procedures (CPP). However, little is known about the epigenetic changes induced by social stress and about their role in the increased response to the rewarding effects of psychostimulants. Considering that histone acetylation regulates transcriptional activity and contributes to drug-induced behavioral changes, we addressed the hypothesis that SD induces transcriptional changes by histone modifications associated with the acquisition of place conditioning. After a fourth defeat, H3(K9) acetylation was decreased in the hippocampus, whil…

Dominance-SubordinationMaleCurcuminHippocampusSpatial BehaviorPharmacologyHippocampusChromatin remodelingEpigenesis GeneticSocial defeatHistone H4Histones03 medical and health sciencesMice0302 clinical medicineRewardCocaineConditioning PsychologicalValproic acidAnimalsEpigeneticsBiological PsychiatryHistone AcetyltransferasesPharmacologySocial stressCerebral CortexbiologyValproic AcidAcetylation030227 psychiatryUp-RegulationHistone Deacetylase InhibitorsDisease Models AnimalHistoneHistone acetylationAcetylationbiology.proteinCentral Nervous System StimulantsPsychologySocial defeat stress030217 neurology & neurosurgeryStress Psychological
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Atrial Fibrosis Hampers Non-invasive Localization of Atrial Ectopic Foci From Multi-Electrode Signals: A 3D Simulation Study

2018

[EN] Introduction: Focal atrial tachycardia is commonly treated by radio frequency ablation with an acceptable long-term success. Although the location of ectopic foci tends to appear in specific hot-spots, they can be located virtually in any atrial region. Multi-electrode surface ECG systems allow acquiring dense body surface potential maps (BSPM) for non-invasive therapy planning of cardiac arrhythmia. However, the activation of the atria could be affected by fibrosis and therefore biomarkers based on BSPM need to take these effects into account. We aim to analyze the effect of fibrosis on a BSPM derived index, and its potential application to predict the location of ectopic foci in the …

Ectopic focus locationmedicine.medical_specialtyFocus (geometry)Physiologymedicine.medical_treatment0206 medical engineeringAtrial tachycardiaStructural remodelingBody surface potential map02 engineering and technologyOptimal electrode location030204 cardiovascular system & hematology3d simulationlcsh:PhysiologyTECNOLOGIA ELECTRONICA03 medical and health sciences0302 clinical medicineFibrosisPhysiology (medical)Internal medicineMedicineMachine-learningAtrial tachycardiaOriginal Researchlcsh:QP1-981business.industryCardiac electrophysiologyCardiac arrhythmiaTorsoAblationmedicine.disease020601 biomedical engineeringmedicine.anatomical_structureCardiologymedicine.symptombusinessFrontiers in Physiology
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Effects of SGLT2 inhibitors on cardiac structure and function.

2022

SGLT2 inhibitors reduce cardiovascular death or hospitalization for heart failure, regardless of the presence or absence of diabetes in patients at high cardiovascular risk and in those with heart failure and reduced ejection fraction (HFrEF). In patients with HF and preserved EF, empagliflozin also showed favorable effects mainly related to the reduction of hospitalization for heart failure. These favorable effects are beyond the reduction of glycemic levels and mainly related to beneficial hemodynamic and anti-inflammatory effects of these drugs and improved cardiac energy metabolism. In this review, we aimed to evaluate the effects of SGLT2 inhibitor on cardiac remodeling and function, w…

Ejection fractionDiastolic functionSystolic functionHeart failureLeft ventricleCardiology and Cardiovascular MedicineSGLT2 inhibitorsCardiac remodelingHeart failure reviews
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De novo SMARCA2 variants clustered outside the helicase domain cause a new recognizable syndrome with intellectual disability and blepharophimosis di…

2020

International audience; Purpose: Nontruncating variants in SMARCA2, encoding a catalytic subunit of SWI/SNF chromatin remodeling complex, cause Nicolaides-Baraitser syndrome (NCBRS), a condition with intellectual disability and multiple congenital anomalies. Other disorders due to SMARCA2 are unknown.Methods: By next-generation sequencing, we identified candidate variants in SMARCA2 in 20 individuals from 18 families with a syndromic neurodevelopmental disorder not consistent with NCBRS. To stratify variant interpretation, we functionally analyzed SMARCA2 variants in yeasts and performed transcriptomic and genome methylation analyses on blood leukocytes.Results: Of 20 individuals, 14 showed…

Foot DeformitiesFoot Deformities Congenital[SDV]Life Sciences [q-bio]BiologyBlepharophimosisSettore MED/03 - GENETICA MEDICAHypotrichosisChromatin remodeling03 medical and health sciencesCongenital0302 clinical medicineNeurodevelopmental disorderIntellectual DisabilityIntellectual disabilitySMARCA2medicineHumansGeneGenetics (clinical)030304 developmental biologyGenetics0303 health sciencesBISFaciesmedicine.diseaseBlepharophimosisPhenotypeneurodevelopmental disorderPhenotypeNicolaides–Baraitser syndromeintellectual disabilityDNA methylationNicolaides–Baraitser syndrome030217 neurology & neurosurgeryTranscription FactorsGenetics in medicine : official journal of the American College of Medical Genetics
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Ascl1 Coordinately Regulates Gene Expression and the Chromatin Landscape during Neurogenesis

2015

Summary The proneural transcription factor Ascl1 coordinates gene expression in both proliferating and differentiating progenitors along the neuronal lineage. Here, we used a cellular model of neurogenesis to investigate how Ascl1 interacts with the chromatin landscape to regulate gene expression when promoting neuronal differentiation. We find that Ascl1 binding occurs mostly at distal enhancers and is associated with activation of gene transcription. Surprisingly, the accessibility of Ascl1 to its binding sites in neural stem/progenitor cells remains largely unchanged throughout their differentiation, as Ascl1 targets regions of both readily accessible and closed chromatin in proliferatin…

Genetics0303 health sciencesNeurogenesisNeurogenesisDNABiologyGeneral Biochemistry Genetics and Molecular BiologyChromatin remodelingArticleCell biologyChromatin03 medical and health sciences0302 clinical medicinelcsh:Biology (General)[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular Biology[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyAscl1Scaffold/matrix attachment regionEnhancerlcsh:QH301-705.5Transcription factor030217 neurology & neurosurgeryChIA-PET030304 developmental biologyBivalent chromatin
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A Gene-Specific Requirement for FACT during Transcription Is Related to the Chromatin Organization of the Transcribed Region

2006

The FACT complex stimulates transcription elongation on nucleosomal templates. In vivo experiments also involve FACT in the reassembly of nucleosomes traversed by RNA polymerase II. Since several features of chromatin organization vary throughout the genome, we wondered whether FACT is equally required for all genes. We show in this study that the in vivo depletion of Spt16, one of the subunits of Saccharomyces cerevisiae FACT, strongly affects transcription of three genes, GAL1, PHO5, and Kluyveromyces lactis LAC4, which exhibit positioned nucleosomes at their transcribed regions. In contrast, showing a random nucleosome structure, YAT1 and Escherichia coli lacZ are only mildly influenced …

GeneticsChromatin ImmunoprecipitationSaccharomyces cerevisiae ProteinsTranscription GeneticbiologyHigh Mobility Group ProteinsRNA polymerase IIPromoterArticlesSaccharomyces cerevisiaeCell BiologyFACT complexChromatinChromatin remodelingChromatinDNA-Binding ProteinsHistone methylationProtein FACTEscherichia colibiology.proteinTranscriptional Elongation FactorsTranscription factor II DMolecular BiologyRNA polymerase II holoenzymePlasmidsMolecular and Cellular Biology
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Chromatin structure of yeast genes.

1989

GeneticsDeoxyribonucleasesBioengineeringSaccharomyces cerevisiaeBiologyApplied Microbiology and BiotechnologyBiochemistryChromatin remodelingYeastChromatinChromatinCell biologyHistoneGeneticsbiology.proteinNucleosomeDNA FungalGeneChIA-PETBiotechnologyBivalent chromatinYeast (Chichester, England)
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Deciphering the histone code to build the genome structure

2017

Histones are punctuated with small chemical modifications that alter their interaction with DNA. One attractive hypothesis stipulates that certain combinations of these histone modifications may function, alone or together, as a part of a predictive histone code to provide ground rules for chromatin folding. We consider four features that relate histone modifications to chromatin folding: charge neutralisation, molecular specificity, robustness and evolvability. Next, we present evidence for the association among different histone modifications at various levels of chromatin organisation and show how these relationships relate to function such as transcription, replication and cell division…

GeneticsHistone-modifying enzymesHistonebiologyHistone H1Histone methylationbiology.proteinHistone codeRobustness (evolution)Computational biologyChromatin remodelingChromatin
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Abstract 4479: Unveiling the relationship between the SWI/SNF chromatin remodeling complex and noncoding RNAs

2016

Abstract Chromatin remodeling complexes are crucial for the viability of the cells due to their role in regulating interactions between DNA and histones and, therefore, modifying the accessibility of the genetic information to the transcriptional machinery. This relevance can also been seen in the SWI/SNF complex that has been associated with cancer in the last deep-sequencing efforts on tumoral genomes. BRG1 is the helicase/ATPase catalytic subunit of the SWI/SNF complex and it is frequently lost in NSCLC cell lines with a high mutation rate. In primary tumors, the loss of expression of BRG1 is also frequent, however it cannot be explained by mutations or by promoter hypermethylation. In t…

GeneticsRegulation of gene expressionCancer ResearchSmall RNABiologymedicine.disease_causeChromatin remodelingSWI/SNFHistoneOncologymicroRNAmedicinebiology.proteinCoding regionCarcinogenesisCancer Research
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Genetic and Cytological Analysis of Drosophila Chromatin-Remodeling Factors

2003

GeneticsSettore BIO/18 - GeneticabiologyRNA-POLYMERASE-IIDrosophila melanogasterDrosophila (subgenus)biology.organism_classificationDrosophila ProteinChromatin remodelingCell biologyHISTONE MODIFICATIONS
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