Search results for "Resistance"

showing 10 items of 3641 documents

Combined aerobic and resistance training decreases inflammation markers in healthy men

2017

Our primary aim was to study the effects of 24 weeks of combined aerobic and resistance training performed on the same day or on different days on inflammation markers. Physically active, healthy young men were randomly divided into three groups that performed: aerobic and resistance training consecutively in the same training session (SS) 2-3 days wk-1 or on alternating days (AD) 4-6 days wk-1 as well as control (C). The total training volume was matched in the training groups. The control group was asked to maintain their habitual physical activity and exercise level. Maximal leg press strength (1RM) and peak oxygen uptake (VO2peak ) were measured. Abdominal fat mass was estimated with du…

AdultLeptinMalemedicine.medical_specialtytulehdusarvotAdipokinePhysical Therapy Sports Therapy and RehabilitationPhysical exercise030204 cardiovascular system & hematologyliikunta03 medical and health sciences0302 clinical medicineOxygen Consumptionphysical exerciseInternal medicinemedicinelow-grade inflammationHumansOrthopedics and Sports MedicineResistinLeg pressta315ExerciseadipokinesChemokine CCL2InflammationbiologyAdiponectinbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaLeptinC-reactive proteinabdominal fatVO2 maxResistance Training030229 sport sciencesEndocrinologyC-Reactive Proteinbiology.proteinBody CompositionResistinAdiponectinbusinessBiomarkersScandinavian Journal of Medicine and Science in Sports
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Circulating levels of adipose products and differences in fat distribution in the ovulatory and anovulatory phenotypes of polycystic ovary syndrome.

2009

Central fat distribution is increased in anovulatory women with polycystic ovary syndrome (PCOS) compared with ovulatory PCOS and matched controls. Among secreted adipocytokines, this is reflected mainly in lower levels of adiponectin.

AdultLeptinOvulationmedicine.medical_specialtySettore MED/09 - Medicina Internaendocrine system diseasesmedia_common.quotation_subjectAdipokineAdipose tissueBiologyAnovulationInsulin resistanceAdipokinesInternal medicinemedicineBody Fat DistributionHumansNicotinamide PhosphoribosyltransferaseOvulationmedia_commonAdiponectinLeptinnutritional and metabolic diseasesObstetrics and Gynecologymedicine.diseasePolycystic ovaryfat distribution; polycystic ovary syndromeLipidsfemale genital diseases and pregnancy complicationsEndocrinologyC-Reactive ProteinPhenotypeReproductive Medicinefat distributionCase-Control StudiesFemaleAdiponectinInsulin ResistanceWaist CircumferenceRetinol-Binding Proteins PlasmaAnovulationPolycystic Ovary SyndromeFertility and sterility
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Inflammatory markers and cardiovascular risk in the metabolic syndrome

2011

Elevated blood glucose, obesity, high blood pressure, elevated triglycerides and low high density lipoprotein (HDL) cholesterol are well accepted risk factors in the development of coronary artery disease. Clustering of at least three of these factors in an individual is defined as metabolic syndrome (MetS). Obesity is a central pathological mechanism in the disease and it is expected that the incidence of this condition will increase dramatically within the next years. The visceral adipose tissue is not only an energy depot but also an endocrine organ which produces a large number of bioactive molecules, the so called adipokines. In the setting of obesity, the over-production of proinflamm…

AdultLeptinRiskAdolescentAngiotensinogenAdipokineAdipose tissueDiseaseIntra-Abdominal FatBioinformaticsCoronary artery diseasePathogenesisInsulin resistancePlasminogen Activator Inhibitor 1medicineHumansResistinObesityLife StyleChemokine CCL2InflammationMetabolic SyndromeInterleukin-6Tumor Necrosis Factor-alphabusiness.industryAtherosclerosisPrognosismedicine.diseaseObesityPlaque AtheroscleroticCardiovascular DiseasesAdiponectinEndothelium VascularInsulin ResistanceMetabolic syndromebusinessBiomarkersFrontiers in Bioscience
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Evidence for altered adipocyte function in polycystic ovary syndrome.

2005

Background: Adipocytokines are produced by adipose tissue and have been thought to be related to insulin resistance and other health consequences. We measured leptin, adiponectin, and resistin simultaneously in women with polycystic ovary syndrome (PCOS) and age- and weight-matched controls. Our hypothesis was that these simultaneous measurements would help determine whether adipocytokine secretion is abnormal in PCOS independent of body mass and whether these levels are related to insulin resistance as well as other hormonal changes. Methods: Fifty-two women with PCOS and 45 normal ovulatory women who were age- and weight-matched were studied. Blood was obtained for adipocytokines (leptin,…

AdultLeptinmedicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and MetabolismAdipose tissueAdipokineBiologyBody Mass Indexchemistry.chemical_compoundresistin polycystic ovary syndromeEndocrinologyInsulin resistanceRETN protein humanInternal medicineAdipocytemedicineIntercellular Signaling Peptides and ProteinAdipocytesHumansResistinAdiponectinLeptinnutritional and metabolic diseasesGeneral Medicinemedicine.diseasePolycystic ovaryleptin; adiponectin; resistin polycystic ovary syndromeEndocrinologychemistryCase-Control StudiesHormones EctopicIntercellular Signaling Peptides and ProteinsResistinFemaleAdiponectinInsulin Resistancehormones hormone substitutes and hormone antagonistsPolycystic Ovary SyndromeEuropean journal of endocrinology
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Insulin resistance in postmenopausal women with metabolic syndrome and the measurements of adiponectin, leptin, resistin, and ghrelin.

2005

Objective: Metabolic syndrome (MBS) is a significant health care problem in postmenopausal women and is driven largely by obesity. We wished to assess the prevalence of insulin resistance (IR), diagnosed using practical methods, and whether several adipocyte factors (adiponectin, leptin, resistin) or the gastric peptide ghrelin, associated with cardiovascular risk, might be abnormal and may relate to IR. Study design: We evaluated 37 obese postmenopausal women with MBS and 34 matched obese premenopausal controls, as well as 14 non-obese premenopausal controls. We measured fasting glucose and insulin, performed 75g 2 hr oral glucose tolerance and intravenous insulin tolerance tests to assess…

AdultLeptinmedicine.medical_specialtymedicine.medical_treatmentPeptide HormonesBody Mass Indexchemistry.chemical_compoundInsulin resistanceInternal medicineAdipocytemedicinePostmenopausal Insulin resistance Metabolic syndrome Adiponectin Leptin Resistin GhrelinHumansResistinObesityMetabolic SyndromeAdiponectinbusiness.industryLeptinInsulinnutritional and metabolic diseasesObstetrics and GynecologyMiddle Agedmedicine.diseaseGhrelinPostmenopauseEndocrinologychemistryPremenopauseCase-Control StudiesResistinGhrelinFemaleAdiponectinMetabolic syndromeInsulin Resistancebusinesshormones hormone substitutes and hormone antagonistsAmerican journal of obstetrics and gynecology
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Circulatory response to volume expansion and transjugular intrahepatic portosystemic shunt in refractory ascites: Relationship with diastolic dysfunc…

2015

Abstract Background Cirrhotic cardiomyopathy may lead to heart failure in stressful circumstances, such as after transjugular intrahepatic portosystemic shunt (TIPS) placement. Aim To examine whether acute volume expansion predicts haemodynamic changes after TIPS and elicits signs of impending heart failure. Methods We prospectively evaluated refractory ascites patients (group A) and compensated cirrhotics (group B), who underwent echocardiography, NT-proBNP measurement, and heart catheterization before and after volume load; group A repeated measurements after TIPS. Results 15 patients in group A (80% male; 54 ± 12.4 years) and 8 in group B (100% male; 56 ± 6.2 years) were enrolled. Echoca…

AdultLiver CirrhosisMaleCardiac Catheterizationmedicine.medical_specialtymedicine.medical_treatmentCardiac indexDiastoleElectrocardiographyInternal medicineNatriuretic Peptide BrainmedicineHumansProspective StudiesPulmonary wedge pressureAgedCardiac catheterizationHeart Failure DiastolicHepatologybusiness.industryHemodynamicsGastroenterologyAscitesMiddle Agedmedicine.diseasePeptide Fragmentsmedicine.anatomical_structureEchocardiographySpainHeart failureHeart catheterizationVascular resistanceCardiologyFemaleVascular ResistancePortasystemic Shunt Transjugular IntrahepaticbusinessTransjugular intrahepatic portosystemic shuntDigestive and Liver Disease
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High risk of hepatocellular carcinoma in anti-HBe positive liver cirrhosis patients developing lamivudine resistance

2004

The emergence of drug-resistant virus in hepatitis B virus patients treated with lamivudine is well documented. However. its clinical impact in the long-term treatment of anti-HBe positive compensated cirrhotic patients is not well known. In this study, we treated 22 consecutive patients with anti-HBe compensated cirrhosis with lamivudine for a median period of 42 months. All patients responded to lamivudine, but viral breakthrough occurred in 13 patients (59%) between 9 and 42 months of therapy due to the emergence of a mutant strain. During the follow-up, 11 developed hepatocellular carcinoma. Of these, 10 occurred soon after the emergence of viral resistance, generally showing aggressive…

AdultLiver CirrhosisMaleHepatitis B virusmedicine.medical_specialtyCarcinoma HepatocellularCirrhosisLAMIVUDINEmedicine.disease_causeRisk AssessmentGastroenterologyVirusVirologyInternal medicineDrug Resistance ViralHumansMedicineHepatitis B e AntigensHEPATOCELLULAR CARCINOMAHepatitis B AntibodiesCIRRHOISIS; HEPATOCELLULAR CARCINOMA; LAMIVUDINE; HEPATITIS B; PRE-CORE MUTANTHepatitis B virusCirrhosiHepatologybusiness.industryCIRRHOISISLamivudineMiddle AgedHepatitis Bmedicine.diseaseHepatitis B Core AntigensViral BreakthroughPRE-CORE MUTANTInfectious DiseasesHepatocellular carcinomaRelative riskMutationHEPATITIS BReverse Transcriptase InhibitorsFemalePrecore mutantbusinessmedicine.drugJournal of Viral Hepatitis
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The impact of insulin resistance, serum adipocytokines and visceral obesity on steatosis and fibrosis in patients with chronic hepatitis C.

2007

Aims To assess whether host metabolic factors influence the degree of hepatic steatosis and fibrosis in patients infected with hepatitis C virus, and to evaluate the impact of anti-viral therapy on insulin resistance and serum levels of adipocytokines. Methods Clinical and biochemical features, anthropometrical characteristics, and levels of fasting insulin, leptin, adiponectin and resistin were measured in ‘naı¨ve’ patients with chronic hepatitis C, before, during and after therapy with Peg-Interferon-alpha 2a plus Ribavirin. Results Forty-eightpatientswereincluded(M/F28/20;meanage50.0 12.6years; 62.5% genotype-1). Body mass index was 26.4 4.0 kg/m2 , and visceral obesity was present in 24…

AdultLiver CirrhosisMaleInterferon-alphahcv steatosisHepatitis C ChronicInterferon alpha-2Intra-Abdominal FatMiddle AgedAntiviral AgentsRecombinant ProteinsFatty LiverRibavirinAdipocytesHumansFemaleObesityInsulin ResistanceAlimentary pharmacologytherapeutics
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Insulin resistance is associated with steatosis in nondiabetic patients with genotype 1 chronic hepatitis C.

2005

Conflicting data exist regarding the relationship between hepatitis C virus genotype 1 and hepatic steatosis as well as the latter's role in the progression of fibrosis and treatment response. We assessed factors associated with hepatic steatosis in genotype 1 chronic hepatitis C and the impact of hepatic fat on fibrosis development and interferon responsiveness. Two hundred ninety-one non-diabetic patients with genotype 1 chronic hepatitis C were examined for the presence of steatosis and its correlation with clinical, virological, and biochemical data, including insulin resistance (IR), evaluated by the homeostasis model assessment (HOMA) score. Steatosis was graded as mild (1%-20% of hep…

AdultLiver CirrhosisMalemedicine.medical_specialtyAdolescentGenotypeHepacivirusGastroenterologyBody Mass IndexInsulin resistanceSex FactorsFibrosisRisk FactorsInternal medicinemedicineHumansRisk factorAgedHepatologybusiness.industryHCV steatosi insulinoresistenzaHepatitis COdds ratiogamma-GlutamyltransferaseHepatitis C ChronicMiddle Agedmedicine.diseaseFatty LiverEndocrinologyMultivariate AnalysisFemaleSteatosisMetabolic syndromeInsulin ResistancebusinessBody mass indexHepatology (Baltimore, Md.)
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Further evidence for an association between non-insulin-dependent diabetes mellitus and chronic hepatitis C virus infection

1999

Non-insulin-dependent diabetes mellitus (NIDDM) may be associated with chronic hepatitis C virus (HCV) infection. This was studied further in two parts. First, 1,151 patients with HCV-related cirrhosis and 181 patients with hepatitis B virus (HBV)-related cirrhosis, well matched for age, sex, and severity of cirrhosis, were reviewed retrospectively. The prevalence of diabetes mellitus was higher in HCV-related cirrhosis (23.6%) than in HBV-related cirrhosis (9.4%; odds ratio [OR], 2.78; 95% confidence interval [CI], 1.6-4.79; P =.0002). The prevalence of diabetes mellitus was associated closely with the Child-Pugh score (OR, 3.83; 95% CI, 2. 38-6.17; P.0001) and increasing age (OR, 1.02; 95…

AdultLiver CirrhosisMalemedicine.medical_specialtyCirrhosisHepatitis Viral Humanmedicine.medical_treatmentmedicine.disease_causeGastroenterologyInsulin resistanceInternal medicineDiabetes mellitusmedicineHumansInsulinProspective StudiesAgedRetrospective StudiesHepatitis B virusGlucose tolerance testHepatologymedicine.diagnostic_testbusiness.industryInsulinFlaviviridaeOdds ratioHepatitis C ChronicMiddle AgedHepatitis Bmedicine.diseasedigestive system diseasesEndocrinologyDiabetes Mellitus Type 2FemalebusinessViral hepatitisHepatology
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