Search results for "Rule"

showing 10 items of 1403 documents

Seasonal recovery ofEdwardsiella piscicidafrom wild European eels and natural waters: Isolation methods, virulence and reservoirs

2018

A total of 127 wild eels caught in the L'Albufera Lake (Spain) and 24 samples of lagoon freshwater were analysed for 1-year period. Edwardsiella strains were isolated from liver/kidney on TSA-1 plates in 31.9% of total diseased specimens, and the edwardsiellosis prevalence in the fishery was of 11.8%. The use of double-strength Salmonella-Shigella (DSSS) broth and SS agar yielded Edwardsiella isolation from intestine in 100% of those edwardsiellosis-diseased eels, but also in 40.4% of other sick fish with vibriosis or aeromonosis and in 28.8% of healthy eels, as well as from freshwater in 8.3% of samples. Pure cultures were isolated on SS agar from the former, but motile Aeromonas, Plesiomo…

0301 basic medicineVeterinary medicineanimal structuresfood.ingredientVeterinary (miscellaneous)030106 microbiologyVirulenceAquatic ScienceBiologyFish Diseases03 medical and health sciencesfoodPrevalenceAnimalsAgarVirulenceEdwardsiella piscicidaIncidenceEnterobacteriaceae InfectionsAnguillabiology.organism_classificationIsolation (microbiology)LakesEdwardsiellaAeromonasSpainPlesiomonas shigelloidesEdwardsiellaBacteriaJournal of Fish Diseases
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High Nutrient Concentration Can Induce Virulence Factor Expression and Cause Higher Virulence in an Environmentally Transmitted Pathogen

2016

Environmentally transmitted opportunistic pathogens shuttle between two substantially different environments: outside-host and within-host habitats. These environments differ from each other especially with respect to nutrient availability. Consequently, the pathogens are required to regulate their behavior in response to environmental cues in order to survive, but how nutrients control the virulence in opportunistic pathogens is still poorly understood. In this study, we examined how nutrient level in the outside-host environment affects the gene expression of putative virulence factors of the opportunistic fish pathogen Flavobacterium columnare. The impact of environmental nutrient concen…

0301 basic medicineVirulence Factors030106 microbiologyvirulence factorsSoil ScienceVirulenceBiologyReal-Time Polymerase Chain ReactionFlavobacteriumVirulence factorflavobacterium columnareMicrobiologyFish Diseases03 medical and health sciencesMicrobial ecologynutrientscolony typeAnimalsCollagenasesPathogenGeneEcology Evolution Behavior and SystematicsChondroitin LyasesEcologyHost (biology)RT-qPCREnvironmental Exposurechondroitinasebiology.organism_classificationcollagenase030104 developmental biologyFoodOncorhynchus mykissFlavobacterium columnareWater MicrobiologyBacteriaMicrobial Ecology
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Selective α-synuclein knockdown in monoamine neurons by intranasal oligonucleotide delivery: potential therapy for parkinson’s disease

2018

Progressive neuronal death in brainstem nuclei and widespread accumulation of α-synuclein are neuropathological hallmarks of Parkinson’s disease (PD). Reduction of α-synuclein levels is therefore a potential therapy for PD. However, because α-synuclein is essential for neuronal development and function, α-synuclein elimination would dramatically impact brain function. We previously developed conjugated small interfering RNA (siRNA) sequences that selectively target serotonin (5-HT) or norepinephrine (NE) neurons after intranasal administration. Here, we used this strategy to conjugate inhibitory oligonucleotides, siRNA and antisense oligonucleotide (ASO), with the triple monoamine reuptake …

0301 basic medicineanimal diseasesDopamineOligonucleotidesGene ExpressionPharmacologySynaptic TransmissionPrefrontal cortexMiceDA neurotransmission0302 clinical medicineDrug DiscoveryMonoaminergicNeural PathwaysRNA Small InterferingCells Cultured5-HT neurotransmissionChemistryGene Transfer TechniquesParkinson DiseaseVentral tegmental areaSubstantia Nigramedicine.anatomical_structureCaudate putamenGene Knockdown Techniquesalpha-SynucleinMolecular MedicineRNA InterferenceOriginal ArticleMonoamine reuptake inhibitormedicine.drugSignal TransductionSerotoninSubstantia nigraASO03 medical and health sciencesProsencephalonα-synucleinDopamineIntranasal administrationGeneticsmedicineAnimalsHumansMolecular BiologyAdministration IntranasalPharmacologyPars compactaDopaminergic NeuronsGenetic TherapyCorpus Striatumnervous system diseases030104 developmental biologyMonoamine neurotransmitterGene Expression Regulationnervous systemsiRNAParkinson’s diseaseLocus coeruleus030217 neurology & neurosurgery
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Erwinia amylovoracatalases KatA and KatG are virulence factors and delay the starvation-induced viable but non-culturable (VBNC) response

2017

The life cycle of the plant pathogen Erwinia amylovora comprises periods inside and outside the host in which it faces oxidative stress caused by hydrogen peroxide (H2 O2 ) and other compounds. The sources of this stress are plant defences, other microorganisms and/or exposure to starvation or other environmental challenges. However, the functional roles of H2 O2 -neutralizing enzymes, such as catalases, during plant-pathogen interactions and/or under starvation conditions in phytopathogens of the family Erwiniaceae or closely related families have not yet been investigated. In this work, the contribution of E. amylovora catalases KatA and KatG to virulence and survival in non-host environm…

0301 basic medicinechemistry.chemical_classificationbiology030106 microbiologyMutantSoil ScienceVirulencePlant ScienceErwiniabiology.organism_classificationmedicine.disease_causeVirulence factorMicrobiology03 medical and health sciencesEnzymechemistryCatalasebiology.proteinmedicineAgronomy and Crop ScienceMolecular BiologyPathogenOxidative stressMolecular Plant Pathology
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Importance of Sequence and Timing in Parasite Coinfections

2019

Coinfections by multiple parasites predominate in the wild. Interactionsbetween parasites can be antagonistic, neutral, or facilitative, and they canhave significant implications for epidemiology, disease dynamics, and evolu-tion of virulence. Coinfections commonly result from sequential exposure ofhosts to different parasites. We argue that the sequential nature of coinfectionsis important for the consequences of infection in both natural and man-madeenvironments. Coinfections accumulate during host lifespan, determining thestructure of the parasite infracommunity. Interactions within the parasite com-munity and their joint effect on the host individual potentially shape evolution ofparasi…

0301 basic medicineinfection dynamicsTime Factors030231 tropical medicineDisease epidemiology2405 ParasitologyVirulenceBiologyinfektiotHost-Parasite Interactions03 medical and health sciencessequential infection10127 Institute of Evolutionary Biology and Environmental Studies0302 clinical medicineloisetParasitic DiseasesParasite hostingAnimalsHumansParasitesepidemiologiaSequence (medicine)Transmission (medicine)Host (biology)Coinfectiondisease epidemiologymultiple infection2725 Infectious Diseasesvirulence evolutionPlantsMultiple infections030104 developmental biologyInfectious DiseasesParasitologyconcomitant infectionEvolutionary biologyta1181570 Life sciences; biology590 Animals (Zoology)Parasitology
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Structure–Activity Relationship of Plesiomonas shigelloides Lipid A to the Production of TNF-α, IL-1β, and IL-6 by Human and Murine Macrophages

2017

Plesiomonas shigelloides is a Gram-negative bacterium that is associated with diarrheal disease in humans. Lipopolysaccharide (LPS) is the main surface antigen and virulence factor of this bacterium. The lipid A (LA) moiety of LPS is the main region recognized by target cells of immune system. Here, we evaluated the biological activities of P. shigelloides LA for their abilities to induce the productions of proinflammatory cytokines (TNF-α, IL-1β, and IL-6) by human and murine macrophages [THP-1 macrophages and immortalized murine bone marrow-derived macrophages (iBMDM)]. Four native P. shigelloides LA preparations differing in their phosphoethanolamine (PEtn) substitution, length, number, …

0301 basic medicinelcsh:Immunologic diseases. AllergyReceptor complexLipopolysaccharideImmunologymedicine.disease_causeVirulence factorProinflammatory cytokineLipid A03 medical and health scienceschemistry.chemical_compoundmedicineImmunology and AllergyEscherichia colilipid AOriginal ResearchbiologyChemistrylipopolysaccharidebiology.organism_classificationBMDMMolecular biologyIn vitro030104 developmental biologyPlesiomonas shigelloidesproinflammatory cytokinesPlesiomonasTHP-1lcsh:RC581-607Frontiers in Immunology
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Functional insights into the infective larval stage of Anisakis simplex s.s., Anisakis pegreffii and their hybrids based on gene expression patterns

2018

[Background]: Anisakis simplex sensu stricto and Anisakis pegreffii are sibling species of nematodes parasitic on marine mammals. Zoonotic human infection with third stage infective larvae causes anisakiasis, a debilitating and potentially fatal disease. These 2 species show evidence of hybridisation in geographical areas where they are sympatric. How the species and their hybrids differ is still poorly understood. [Results]: Third stage larvae of Anisakis simplex s.s., Anisakis pegreffii and hybrids were sampled from Merluccius merluccius (Teleosti) hosts captured in waters of the FAO 27 geographical area. Specimens of each species and hybrids were distinguished with a diagnostic genetic m…

0301 basic medicinelcsh:QH426-470Virulence Factorslcsh:BiotechnologyAnisakis simplexBreedingBiologyAnisakisTranscriptomeFish Diseases03 medical and health scienceslcsh:TP248.13-248.65parasitic diseasesGeneticsAnimalsAlleleGeneGeneticsSequence Analysis RNAGene Expression ProfilingAnisakis simplexMolecular Sequence AnnotationHelminth Proteins030108 mycology & parasitologyAllergensbiology.organism_classificationA. PegreffiiAnisakisGene expression profilingGadiformeslcsh:Genetics030104 developmental biologyGene Expression RegulationSympatric speciationGenetic markerLarvaGene expressionEnergy MetabolismTranscriptomeResearch ArticleBiotechnologyBMC Genomics
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MARTX Toxin in the Zoonotic Serovar of Vibrio vulnificus Triggers an Early Cytokine Storm in Mice

2017

Vibrio vulnificus biotype 2-serovar E is a zoonotic clonal complex that can cause death by sepsis in humans and fish. Unlike other biotypes, Bt2 produces a unique type of MARTXVv (Multifunctional-Autoprocessive-Repeats-in-Toxin; RtxA13), which is encoded by a gene duplicated in the pVvBt2 plasmid and chromosome II. In this work, we analyzed the activity of this toxin and its role in human sepsis by performing in vitro, ex vivo, and in vivo assays. First, we demonstrated that the ACD domain, present exclusively in this toxin variant, effectively has an actin-cross-linking activity. Second, we determined that the whole toxin caused death of human endotheliocytes and monocytes by lysis and apo…

0301 basic medicinelcsh:QR1-502MicrobiologiaVibrio vulnificusmedicine.disease_causeMonocyteslcsh:MicrobiologysepsisVibrio vulnificusCells CulturedOriginal ResearchMice Inbred BALB CCell DeathVirulencebiologyqPCR arrayInfectious DiseasesHost-Pathogen InteractionsBacteris patògensCytokinesV. vulnificusFemaleMicrobiology (medical)Virulence FactorsBacterial ToxinsImmunologyVirulenceSerogroupMicrobiologyMicrobiology03 medical and health sciencesImmune systemMARTXIn vivomedicineAnimalsHumansVibrioToxinEndothelial Cellsbiology.organism_classificationmedicine.diseaseVirologyVibrioinfectionDisease Models Animal030104 developmental biologyInfecciógene expressionCytokine stormEx vivoFrontiers in Cellular and Infection Microbiology
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A new class of phenylhydrazinylidene derivatives as inhibitors of Staphylococcus aureus biofilm formation

2016

In the struggle against the emergence of the antibiotic resistance, new molecules targeting biofilm formation could be useful as adjuvant of conventional antibiotics. This study focused on a new class of 2-phenylhydrazinylidene derivatives as antivirulence agents. The compound 12e showed interesting activities against biofilm formation of all tested Staphylococcus aureus strains with IC50 ranging from 1.7 to 43 µM; compounds 12f and 13a resulted strong inhibitors of S. aureus ATCC 6538 and ATCC 29213 biofilm formation with IC50 of 0.9 and 0.8 µM, respectively. A preliminary study on the mechanism of action was carried on evaluating the inhibition of sortase A transpeptidase. Compound 12e re…

0301 basic medicinemedicine.drug_class030106 microbiologyAntibioticsBacterial adhesionAntibiofilm agentSettore BIO/19 - Microbiologia Generalemedicine.disease_causeMicrobiologyAntivirulence agent03 medical and health sciencesAntibiotic resistanceIn vivomedicineGeneral Pharmacology Toxicology and PharmaceuticsbiologyChemistrySortase AOrganic ChemistryBiofilmPhenylhydrazinylidene derivativebiochemical phenomena metabolism and nutritionbiology.organism_classificationSettore CHIM/08 - Chimica FarmaceuticaGalleria mellonellaSettore AGR/11 - Entomologia Generale E Applicata030104 developmental biologyMechanism of actionBiochemistryStaphylococcus aureusPharmacology Toxicology and Pharmaceutics (all)Sortase Amedicine.symptomMedicinal Chemistry Research
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Minor environmental concentrations of antibiotics can modify bacterial virulence in co-infection with a non-targeted parasite.

2018

Leakage of medical residues into the environment can significantly impact natural communities. For example, antibiotic contamination from agriculture and aquaculture can directly influence targeted pathogens, but also other non-targeted taxa of commensals and parasites that regularly co-occur and co-infect the same host. Consequently, antibiotics could significantly alter interspecific interactions and epidemiology of the co-infecting parasite community. We studied how minor environmental concentrations of antibiotic affects the co-infection of two parasites, the bacterium Flavobacterium columnare and the fluke Diplostomum pseudospathaceum , in their fish host. We found that antibiotic in …

0301 basic medicinemedicine.drug_classAntibioticsVirulenceOxytetracyclineAquacultureTrematode InfectionsinfektiotFlavobacteriumantibioticsMicrobiologybakteerit03 medical and health sciencesFish DiseasesAntibiotic resistanceco-infectionAquacultureFlavobacteriaceae InfectionsloisetDrug Resistance BacterialmedicineWater Pollution ChemicalParasite hostingAnimalsbacteriabiologyVirulencebusiness.industryCoinfectionimumadotta1183antibiootitvirulenssiCommensalismbiology.organism_classificationAgricultural and Biological Sciences (miscellaneous)Anti-Bacterial Agents030104 developmental biology13. Climate actionympäristövaikutuksetOncorhynchus mykissFlavobacterium columnareparasiteta1181TrematodaGeneral Agricultural and Biological SciencesbusinessPathogen BiologyBacteriaBiology letters
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