Search results for "SCAR"

showing 10 items of 914 documents

The variation of acetylcholine release from myenteric neurones with stimulation frequency and train length. Role of presynaptic muscarine receptors.

1983

1. The effects of scopolamine on the release of 3H-acetylcholine (ACh) from the guinea-pig myenteric plexus were studied at different stimulation frequencies (0.03–10 Hz) and train lengths (1–180 pulses). Release of 3H-ACh was measured in the absence of cholinesterase inhibitors as the outflow of tritium from myenteric plexus-longitudinal muscle preparations preloaded with 3H-choline. 2. In control experiments the volley output of 3H-ACh declined with increasing train length and increasing stimulation frequency. Stimulation by one pulse produced the highest volley output. 3. Scopolamine facilitated the evoked output of 3H-ACh via blockade of presynaptic muscarine receptors. A significant in…

PharmacologyMuscarinePulse (signal processing)Guinea PigsScopolamineMyenteric PlexusStimulationGeneral MedicineInhibitory postsynaptic potentialReceptors MuscarinicAcetylcholineElectric StimulationFeedbackchemistry.chemical_compoundchemistryNegative feedbackMuscarinic acetylcholine receptormedicineAnimalsNeuroscienceAcetylcholineMyenteric plexusmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
researchProduct

Acetylcholine release at motor endplates and autonomic neuroeffector junctions: a comparison.

1996

Acetylcholine released at motor endplates and at autonomic neuroeffector junctions binds to nicotinic and muscarinic receptors to affect the activity of the corresponding target cells. Additionally, nicotonic and muscarinic receptors modulate various intracellular regulatory pathways (second messengers, gene expression) and mediate trophic effects. To maintain homeostasis of the individual cell and of the whole organism the release of acetylcholine has to be strictly controlled within both nervous systems. The basic events of synthesis, storage, and release are comparable at motoneurones and autonomic neurones, but mechanisms regulating transmitter release appear to differ. The motor endpla…

PharmacologyMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2BiologyMotor EndplateReceptors MuscarinicAcetylcholineNeuroeffector junctionNicotinic agonistMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptorMuscarinic acetylcholine receptor M4medicineNeuroeffector JunctionAnimalsNeuroscienceAcetylcholinemedicine.drugPharmacological research
researchProduct

The cholinergic 'pitfall': acetylcholine, a universal cell molecule in biological systems, including humans.

1999

1. Acetylcholine (ACh) represents one of the most exemplary neurotransmitters. In addition to its presence in neuronal tissue, there is increasing experimental evidence that ACh is widely expressed in pro- and eukaryotic non-neuronal cells. Thus, ACh has been detected in bacteria, algae, protozoa, tubellariae and primitive plants, suggesting an extremely early appearance of ACh in the evolutionary process. 2. In humans, ACh and/or the synthesizing enzyme, choline acetyltransferase, has been demonstrated in epithelial cells (airways, alimentary tract, urogenital tract, epidermis), mesothelial (pleura, pericardium) and endothelial and muscle cells. In addition, immune cells express the non-ne…

PharmacologyNeuronsPhysiologyCellular differentiationBiologyCholine acetyltransferaseAcetylcholineCell biologyEvolution MolecularParacrine signallingNicotinic agonistBiochemistryPhysiology (medical)Muscarinic acetylcholine receptormedicineCholinergicAnimalsCholinesterasesHumansAcetylcholinemedicine.drugCalcium signalingClinical and experimental pharmacologyphysiology
researchProduct

Acetylcholine beyond neurons: the non-neuronal cholinergic system in humans

2008

Animal life is controlled by neurons and in this setting cholinergic neurons play an important role. Cholinergic neurons release ACh, which via nicotinic and muscarinic receptors (n- and mAChRs) mediate chemical neurotransmission, a highly integrative process. Thus, the organism responds to external and internal stimuli to maintain and optimize survival and mood. Blockade of cholinergic neurotransmission is followed by immediate death. However, cholinergic communication has been established from the beginning of life in primitive organisms such as bacteria, algae, protozoa, sponge and primitive plants and fungi, irrespective of neurons. Tubocurarine- and atropine-sensitive effects are obser…

PharmacologyNicotinic agonistMuscarinic acetylcholine receptormedicineCholinergicBiologySignal transductionCholinergic neuronReceptorNeuroscienceAcetylcholineAcetylcholine receptormedicine.drugBritish Journal of Pharmacology
researchProduct

Volatile components of aerial parts of Centaurea nigrescens an C. stenolepis growing wild in the Balkans

2010

The volatile constituents of the aerial parts of Centaurea nigrescens Willd, collected in Romania and of two samples of C. stenolepis A. Kerner from Bulgaria and Romania were obtained by hydrodistillation and analyzed. The main components of C. stenolepis were caryophyllene oxide (6.9-15.6%), hexahydrofarnesyl acetone (6.5-4.4%), heptacosane (6.0-4.9%) and p-vinyl guiacol (4.3-5.0%). The main components of C. nigrescens were caryophyllene oxide (9.9%), β-eudesmol (9.5%), spathulenol (7.6%), heptacosane (6.1%) and p-vinyl guiacol (5.5%). The chemotaxonomic significance with respect to their co-location in Sections Lepteranthus and Nigrescentes, respectively, is discussed.

PharmacologybiologyCentaurea stenolepisCentaurea nigrescens Centaurea stenolepis Asteraceae volatile componentscaryophyllene oxide hexahydrofarnesyl acetone beta-eudesmol spathulenol p-vinyl guaiacolCentaureaPlant ScienceGeneral MedicineSettore CHIM/06 - Chimica OrganicaAsteraceaePlant Components Aerialbiology.organism_classificationSpathulenolComplementary and alternative medicineCaryophyllene oxideDrug DiscoveryBotanyCentaurea nigrescensOils VolatilePlant OilsSettore BIO/15 - Biologia FarmaceuticaStenolepisBeta-eudesmol
researchProduct

Differential effects of anandamide on acetylcholine release in the guinea-pig ileum mediated via vanilloid and non-CB1 cannabinoid receptors

2001

The effects of anandamide on [3H]-acetylcholine release and muscle contraction were studied on the myenteric plexus-longitudinal muscle preparation of the guinea-pig ileum preincubated with [3H]-choline. Anandamide increased both basal [3H]-acetylcholine release (pEC50 6.3) and muscle tone (pEC50 6.3). The concentration-response curves for anandamide were shifted to the right by 1 μM capsazepine (pKB 7.5 and 7.6), and by the combined blockade of NK1 and NK3 tachykinin receptors with the antagonists CP99994 plus SR142801 (each 0.1 μM). The CB1 and CB2 receptor antagonists, SR141716A (1 μM) and SR144528 (30 nM), did not modify the facilitatory effects of anandamide. Anandamide inhibited the e…

Pharmacologymedicine.medical_specialtyCannabinoid receptormedicine.medical_treatmentTRPV1AnandamideMuscarinic agonistchemistry.chemical_compoundEndocrinologychemistryInternal medicinemedicineCannabinoidCapsazepineTachykinin receptorAcetylcholinemedicine.drugBritish Journal of Pharmacology
researchProduct

The Non-neuronal Cholinergic System

2001

Acetylcholine, one of the most exemplary neurotransmitters, has been detected in bacteria, algae, protozoa, tubellariae and primitive plants, suggesting an extremely early appearance in the evolutionary process and a wide expression in non-neuronal cells. In plants (Urtica dioica), acetylcholine is involved in the regulation of water resorption and photosynthesis. In humans, acetylcholine and/or the synthesizing enzyme, choline acetyltransferase, have been demonstrated in epithelial (airways, alimentary tract, urogenital tract, epidermis), mesothelial (pleura, pericardium), endothelial, muscle and immune cells (granulocytes, lymphocytes, macrophages, mast cells). The widespread expression o…

Pharmacologymedicine.medical_specialtyInflammationBiologyCholine acetyltransferaseCell biologyNicotinic agonistEndocrinologyInternal medicineMuscarinic acetylcholine receptormedicinebiology.proteinSignal transductionmedicine.symptomReceptorAcetylcholinemedicine.drugCholinesteraseJapanese Journal of Pharmacology
researchProduct

Adrenoceptor-mediated changes of excitation and contraction in isolated heart muscle preparations.

1989

The inotropic effects of sympathetic stimulation on the heart are mainly ascribed to the activation of beta-adrenoceptors. However, several findings suggest that alpha-adrenoceptors also may help mediate the inotropic response to catecholamines under certain conditions. The onset of the positive inotropic effect mediated by beta-adrenoceptors occurs within seconds and is associated with a faster rate of relaxation. Both beta 1- and beta 2-adrenoceptors are stimulatorily coupled to the enzyme adenylate cyclase, thereby leading to the generation of cyclic AMP. Cyclic AMP increases the slow inward calcium current and enhances the uptake of calcium into the sarcoplasmic reticulum. GTP-binding p…

Pharmacologymedicine.medical_specialtyMuscarineAdrenergic receptorAdenylate kinaseStimulationHeartIn Vitro TechniquesAdenosine receptorCyclaseMyocardial ContractionReceptors AdrenergicBeta-1 adrenergic receptorchemistry.chemical_compoundEndocrinologychemistryInternal medicinemedicineAnimalsHumansmedicine.symptomCardiology and Cardiovascular MedicineMuscle contractionJournal of cardiovascular pharmacology
researchProduct

Non-neuronal acetylcholine, a locally acting molecule, widely distributed in biological systems: expression and function in humans.

1998

Acetylcholine acts as a neurotransmitter in the central and peripheral nervous systems in humans. However, recent experiments demonstrate a widespread expression of the cholinergic system in non-neuronal cells in humans. The synthesizing enzyme choline acetyltransferase, the signalling molecule acetylcholine, and the respective receptors (nicotinic or muscarinic) are expressed in epithelial cells (human airways, alimentary tract, epidermis). Acetylcholine is also found in mesothelial, endothelial, glial, and circulating blood cells (platelets, mononuclear cells), as well as in alveolar macrophages. The existence of non-neuronal acetylcholine explains the widespread expression of muscarinic …

Pharmacologymedicine.medical_specialtyMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2BiologyAcetylcholineCell biologyCholine O-AcetyltransferaseCircadian RhythmEndocrinologyNicotinic agonistInternal medicineMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4CholinergicHumansPharmacology (medical)Acetylcholinemedicine.drugPharmacologytherapeutics
researchProduct

A role for Rho in receptor- and G protein-stimulated phospholipase C Reduction in phosphatidylinositol 4,5-bisphosphate by Clostridium difficile toxi…

1996

Receptors coupled to heterotrimeric guanine nucleotide-binding proteins (G proteins) activate phosphatidylinositol 4,5-bisphosphate (PtdIns(4,5)P2)-hydrolyzing phospholipase C (PLC) enzymes by activated alpha of free beta gamma subunits of the relevant G proteins. To study whether low molecular weight G proteins of the Rho family are involved in receptor signaling to PLC, we examined the effect of Clostridium difficile toxin B, which glucosylates and thereby inactivates Rho proteins, on the regulation of PLC activity in human embryonic kidney (HEK) cells stably expressing the m3 muscarinic acetylcholine receptor (mAChR) subtype. Toxin B treatment of HEK cells did not affect basal PLC activi…

Phosphatidylinositol 45-DiphosphateBotulinum ToxinsG proteinBacterial ToxinsClostridium difficile toxin AClostridium difficile toxin BBiologychemistry.chemical_compoundBacterial ProteinsGTP-Binding ProteinsHeterotrimeric G proteinHumansPhosphatidylinositolCells CulturedADP Ribose TransferasesPharmacologyPhospholipase CHEK 293 cellsGeneral MedicineReceptors MuscarinicMolecular biologyCell biologychemistryPhosphatidylinositol 45-bisphosphateType C PhospholipasesrhoA GTP-Binding ProteinNaunyn-Schmiedeberg's Archives of Pharmacology
researchProduct