Search results for "SIGNALLING"

showing 10 items of 249 documents

Exercise acts as a drug; the pharmacological benefits of exercise

2012

The beneficial effects of regular exercise for the promotion of health and cure of diseases have been clearly shown. In this review, we would like to postulate the idea that exercise can be considered as a drug. Exercise causes a myriad of beneficial effects for health, including the promotion of health and lifespan, and these are reviewed in the first section of this paper. Then we deal with the dosing of exercise. As with many drugs, dosing is extremely important to get the beneficial effects of exercise. To this end, the organism adapts to exercise. We review the molecular signalling pathways involved in these adaptations because understanding them is of great importance to be able to pr…

PharmacologyDrugeducation.field_of_studymedicine.medical_specialtybusiness.industrymedia_common.quotation_subjectPopulationPsychoactive drugAlternative medicinePromotion (rank)medicinePhysical therapyDosingExercise physiologyeducationbusinessSignalling pathwaysmedicine.drugmedia_commonBritish Journal of Pharmacology
researchProduct

The cholinergic 'pitfall': acetylcholine, a universal cell molecule in biological systems, including humans.

1999

1. Acetylcholine (ACh) represents one of the most exemplary neurotransmitters. In addition to its presence in neuronal tissue, there is increasing experimental evidence that ACh is widely expressed in pro- and eukaryotic non-neuronal cells. Thus, ACh has been detected in bacteria, algae, protozoa, tubellariae and primitive plants, suggesting an extremely early appearance of ACh in the evolutionary process. 2. In humans, ACh and/or the synthesizing enzyme, choline acetyltransferase, has been demonstrated in epithelial cells (airways, alimentary tract, urogenital tract, epidermis), mesothelial (pleura, pericardium) and endothelial and muscle cells. In addition, immune cells express the non-ne…

PharmacologyNeuronsPhysiologyCellular differentiationBiologyCholine acetyltransferaseAcetylcholineCell biologyEvolution MolecularParacrine signallingNicotinic agonistBiochemistryPhysiology (medical)Muscarinic acetylcholine receptormedicineCholinergicAnimalsCholinesterasesHumansAcetylcholinemedicine.drugCalcium signalingClinical and experimental pharmacologyphysiology
researchProduct

Endocannabinoid signals in the control of emotion.

2008

The appropriate control of emotional responses evoked by environmental stimuli is an important innate mechanism for ensuring quality of life and even for survival. Inappropriate responses and decreased abilities to adjust to changed environmental situations can lead to psychiatric disorders, such as posttraumatic stress disorders, phobia and depression. Endocannabinoid signalling has emerged as one of the regulatory systems of the brain supporting appropriate emotional responses. As various components of the endocannabinoid system have become therapeutic targets, understanding the endocannabinoids’ mechanism of action is an important research topic for a rationalized drug design and optimal…

PharmacologyPosttraumatic stress disordersNeuronal PlasticityMechanism (biology)Mental DisordersEmotionsEndocannabinoid systemDevelopmental psychologyImportant researchQuality of life (healthcare)SignallingDrug DiscoveryCannabinoid Receptor ModulatorsSynapsesAnimalsHumansControl (linguistics)PsychologyReceptors CannabinoidNeuroscienceEndocannabinoidsSignal TransductionCurrent opinion in pharmacology
researchProduct

Down-regulation of microglial cyclo-oxygenase-2 and inducible nitric oxide synthase expression by lipocortin 1

1999

Activated microglial cells are believed to play an active role in most brain pathologies, during which they can contribute to host defence and repair but also to the establishment of tissue damage. These actions are largely mediated by microglial secretory products, among which are prostaglandins (PGs) and nitric oxide (NO). The anti-inflammatory protein, lipocortin 1 (LC1) was reported to have neuroprotective action and to be induced by glucocorticoids in several brain structures, with a preferential expression in microglia. In this paper we tested whether the neuroprotective effect of LC1 could be explained by an inhibitory effect on microglial activation. We have previously shown that ba…

Pharmacologymedicine.medical_specialtymedicine.diagnostic_testbiologyMicrogliaNeuroprotectionNitric oxideCell biologyNitric oxide synthasechemistry.chemical_compoundmedicine.anatomical_structureEndocrinologychemistryWestern blotInternal medicinemedicinebiology.proteinNeurogliaAutocrine signallingAnnexin A1British Journal of Pharmacology
researchProduct

Adenosine negatively regulates duodenal motility in mice: role of A1 and A2A receptors

2011

BACKGROUND AND PURPOSE Adenosine is considered to be an important modulator of intestinal motility. This study was undertaken to investigate the role of adenosine in the modulation of contractility in the mouse duodenum and to characterize the adenosine receptor subtypes involved. EXPERIMENTAL APPROACH RT-PCR was used to investigate the expression of mRNA encoding for A1, A2A, A2B and A3 receptors. Contractile activity was examined in vitro as changes in isometric tension. KEY RESULTS In mouse duodenum, all four classes of adenosine receptors were expressed, with the A2B receptor subtype being confined to the mucosal layer. Adenosine caused relaxation of mouse longitudinal duodenal muscle; …

Pharmacologymedicine.medical_specialtymedicine.drug_classPurinergic signallingBiologyAdenosine A3 receptorReceptor antagonistAdenosineAdenosine receptorAdenosine A1 receptorEndocrinologyInternal medicinemedicineReceptorAdenosine A2B receptormedicine.drugBritish Journal of Pharmacology
researchProduct

Nitric oxide: a new player in plant signalling and defence responses.

2004

There is increasing evidence that nitric oxide (NO), which was first identified as a unique diffusible molecular messenger in animals, plays important roles in diverse (patho)physiological processes in plants. NO functions include the modulation of hormonal, wounding and defence responses, as well as the regulation of cell death. Enzymes that catalyse NO synthesis and signalling cascades that mediate NO effects have recently been discovered, providing a better understanding of the mechanisms by which NO influences plant responses to various stimuli. Additionally, growing evidence suggests that NO signalling interacts with the salicylic acid and jasmonic acid signalling pathways.

Programmed cell deathCell DeathJasmonic acidPlant ScienceBiologyPlantsNitric OxideModels BiologicalNitric oxideCell biologychemistry.chemical_compoundSignallingchemistryBiochemistrySignal transductionSignalling pathwaysSignalling cascadesSalicylic acidSignal TransductionCurrent opinion in plant biology
researchProduct

Heat shock proteins: essential proteins for apoptosis regulation

2008

Abstract Many different external and intrinsic apoptotic stimuli induce the accumulation in the cells of a set of proteins known as stress or heat shock proteins (HSPs). HSPs are conserved proteins present in both prokaryotes and eukaryotes. These proteins play an essential role as molecular chaperones by assisting the correct folding of nascent and stress-accumulated misfolded proteins, and by preventing their aggregation. HSPs have a protective function, that is they allow the cells to survive to otherwise lethal conditions. Various mechanisms have been proposed to account for the cytoprotective functions of HSPs. Several of these proteins have demonstrated to directly interact with compo…

Programmed cell deathCell signalingReviewsMitochondrionBiologyModels BiologicallysosomesLysosomeHeat shock proteindeath receptorsmedicineAnimalsHumansemerging chemotherapeutic treatmentsHeat-Shock ProteinsCell Deathhaematopoietic malignanciesapoptosiscell signallingCell BiologyMitochondriaNeoplasm ProteinsCell biologymedicine.anatomical_structurecaspasesHematologic Neoplasmsheat shock proteinsMolecular MedicineProtein foldingHSP60Signal transductionMolecular ChaperonesSignal TransductionJournal of Cellular and Molecular Medicine
researchProduct

Signalling Three-Way Intersections: Is Redundancy Better Than Only Mandatory or Prohibitory Signs?

2021

This work was supported by the Spanish Government, Ministry of Economy and Competitiveness (PGC2018-095868-B-I00).

Prohibitory signThree-way intersectionsApplied psychologyprohibitory signMandatory signSign (semiotics)Experimental researchmandatory signBF1-990Mental modelsSignallingmental modelsThree waythree-way intersectionsRedundancy (engineering)Psychologyredundant informationRedundant informationPsychologyGeneral PsychologyOriginal Research
researchProduct

Different protein turnover of interleukin-6-type cytokine signalling components.

1999

Interleukin (IL)-6 and IL-6-type cytokines signal through the gp130/Jak/STAT signal transduction pathway. The key components involved are the signal transducing receptor subunit gp130, the Janus kinases Jak1, Jak2 and Tyk2, STAT1 and STAT3 of the family of signal transducers and activators of transcription, the protein tyrosine phosphatase SHP2 and the suppressors of cytokine signalling SOCS1, SOCS2 and SOCS3. Whereas considerable information has been accumulated concerning the time-course of activation for the individual signalling molecules, data on the availability of the proteins involved in IL-6-type cytokine signal transduction are scarce. Nevertheless, availability of these molecules…

Protein Tyrosine Phosphatase Non-Receptor Type 11Protein tyrosine phosphataseBiologyBiochemistrySuppressor of cytokine signallingAntigens CDCytokine Receptor gp130Membrane GlycoproteinsSuppressor of cytokine signaling 1Interleukin-6Protein Tyrosine Phosphatase Non-Receptor Type 6Intracellular Signaling Peptides and ProteinsJAK-STAT signaling pathwaySignal transducing adaptor proteinSTAT2 Transcription FactorProtein-Tyrosine KinasesGlycoprotein 130Recombinant ProteinsCell biologyDNA-Binding ProteinsSTAT1 Transcription FactorBiochemistryTrans-ActivatorsCytokinesSignal transductionProtein Tyrosine PhosphatasesJanus kinaseHalf-LifeSignal TransductionEuropean journal of biochemistry
researchProduct

Epithelial to mesenchymal transition is increased in patients with COPD and induced by cigarette smoke

2013

Background Cigarette smoking contributes to lung remodelling in chronic obstructive pulmonary disease (COPD). As part of remodelling, peribronchiolar fibrosis is observed in the small airways of patients with COPD and contributes to airway obstruction. Epithelial to mesenchymal transition (EMT) appears to be involved in the formation of peribronchiolar fibrosis. This study examines the EMT process in human bronchial epithelial cells (HBECs) from non-smokers, smokers and patients with COPD as well as the in vitro effect of cigarette smoke extract (CSE) on EMT. Methods HBECs from non-smokers (n=5), smokers (n=12) and patients with COPD (n=15) were collected to measure the mesenchymal markers …

Pulmonary and Respiratory MedicineCOPDPathologymedicine.medical_specialtyLungbiologybusiness.industryMesenchymal stem cellVimentinrespiratory systemmedicine.diseaserespiratory tract diseasesCytokeratinmedicine.anatomical_structureFibrosismedicinebiology.proteinEpithelial–mesenchymal transitionAutocrine signallingbusinessThorax
researchProduct