Search results for "SORBENT"

showing 10 items of 635 documents

In vitro secretion of specific antimitochondrial antibodies in primary biliary cirrhosis

1992

Antimitochondrial antibodies are present in the serum of virtually all patients with primary biliary cirrhosis. They have a well-defined antigen reactivity that is diagnostic for the disease. The role of these autoantibodies in the disease process remains to be defined. In this study we show that antimitochondrial antibodies can be produced in vitro by peripheral blood lymphocytes, that the cells producing antimitochondrial antibodies are present in the peripheral blood in a high frequency and seem to be maximally activated. Stimulation with pokeweed mitogen did not augment the in vitro production of antimitochondrial antibodies in patients nor did it induce the production of these antibodi…

Pathologymedicine.medical_specialtyLymphocyteImmunoblottingRadioimmunoassayEnzyme-Linked Immunosorbent AssayPrimary biliary cirrhosisAntigenAntibody SpecificitymedicineHumansCells CulturedAutoantibodiesHepatologybiologyLiver Cirrhosis Biliarybusiness.industryPokeweed mitogenAutoantibodyRadioimmunoassaymedicine.diseaseMitochondriamedicine.anatomical_structurePolyclonal antibodiesImmunologybiology.proteinAntibodybusinessJournal of Hepatology
researchProduct

Targeting of multiple myeloma-related angiogenesis by miR-199a-5p mimics: in vitro and in vivo anti-tumor activity

2014

// Lavinia Raimondi 1 , Nicola Amodio 1 , Maria Teresa Di Martino 1 , Emanuela Altomare 1 , Marzia Leotta 1 , Daniele Caracciolo 1 , Annamaria Gulla 1 , Antonino Neri 2 , Simona Taverna 3 , Patrizia D’Aquila 4 , Riccardo Alessandro 3 , Antonio Giordano 5 , Pierosandro Tagliaferri 1 and Pierfrancesco Tassone 1,5 . 1 Department of Experimental and Clinical Medicine, Magna Graecia University and Medical Oncology Unit, T. Campanella Cancer Center, Salvatore Venuta University Campus, Catanzaro, Italy 2 Department of Medical Sciences University of Milan, Hematology1, IRCCS Policlinico Foundation, Milan, Italy 3 Department of Pathology and Forensic and Medical Biotechnology, Section of Biology and…

Pathologymedicine.medical_specialtyStromal cellAngiogenesisMultiple Myeloma; microRNA AngiogenesisBlotting WesternEnzyme-Linked Immunosorbent AssayMice SCIDIn Vitro TechniquesBiologyReal-Time Polymerase Chain ReactionTransfectionMicemiR-199-5pCell MovementMice Inbred NODSettore BIO/13 - Biologia ApplicataCell Line TumorCell AdhesionmedicineAnimalsHumansHypoxiaCell adhesionProtein kinase BCell ProliferationPlasma cell leukemiaNeovascularization PathologicmicroRNA AngiogenesisMicroRNATransfectionPlasma cell leukemiamedicine.diseaseXenograft Model Antitumor AssaysMolecular medicineCell HypoxiaMicroRNAsmedicine.anatomical_structureOncologyAngiogenesis; Hypoxia; Microenviroment; MicroRNA; miR-199-5p; MiRNA; Multiple myeloma; Plasma cell leukemiaCancer researchFemaleAngiogenesisBone marrowMicroenviromentMiRNAMultiple MyelomaResearch Paper
researchProduct

Spatial and temporal heterogeneity of ventilator-associated lung injury after surfactant depletion.

2008

Volutrauma and atelectrauma have been proposed as mechanisms of ventilator-associated lung injury, but few studies have compared their relative importance in mediating lung injury. The objective of our study was to compare the injury produced by stretch (volutrauma) vs. cyclical recruitment (atelectrauma) after surfactant depletion. In saline-lavaged rabbits, we used high tidal volume, low respiratory rate, and low positive end-expiratory pressure to produce stretch injury in nondependent lung regions and cyclical recruitment in dependent lung regions. Tidal changes in shunt fraction were assessed by measuring arterial Po2 oscillations. After ventilating for times ranging from 0 to 6 h, lu…

Pathologymedicine.medical_specialtyVentilator-associated lung injuryPhysiologyNitric Oxide Synthase Type IIInflammationEnzyme-Linked Immunosorbent AssayPulmonary EdemaRespiratory physiologyLung injuryPhysiology (medical)medicineAnimalsInterleukin 8LungChemokine CCL2PeroxidaseLungVentilators Mechanicalbusiness.industryRespiratory diseaseInterleukin-8Pulmonary SurfactantsLung Injuryrespiratory systemmedicine.diseasePulmonary edemarespiratory tract diseasesOxygenmedicine.anatomical_structureNeutrophil InfiltrationCalibrationRespiratory MechanicsCytokinesFluid TherapyFemaleRabbitsmedicine.symptomBlood Gas AnalysisChemokinesbusinessJournal of applied physiology (Bethesda, Md. : 1985)
researchProduct

C1q as a novel player in angiogenesis with therapeutic implication in wound healing

2014

We have previously shown that C1q is expressed on endothelial cells (ECs) of newly formed decidual tissue. Here we demonstrate that C1q is deposited in wound-healing skin in the absence of C4 and C3 and that C1q mRNA is locally expressed as revealed by real-time PCR and in situ hybridization. C1q was found to induce permeability of the EC monolayer, to stimulate EC proliferation and migration, and to promote tube formation and sprouting of new vessels in a rat aortic ring assay. Using a murine model of wound healing we observed that vessel formation was defective in C1qa(-/-) mice and was restored to normal after local application of C1q. The mean vessel density of wound-healing tissue and …

Pathologymedicine.medical_specialtycomplement C1qAngiogenesisImmunoblottingNeovascularization Physiologicchemical and pharmacologic phenomenaEnzyme-Linked Immunosorbent AssayIn situ hybridizationBiologyReal-Time Polymerase Chain ReactionangiogenesisMiceVasculogenesiscomplement; vasculogenesis; animal modelsimmune system diseasesmedicineangiogenesis; complement C1q; wound-healing; endothelial cellsHuman Umbilical Vein Endothelial CellsAnimalsHumanscomplementRats WistarIn Situ HybridizationCell ProliferationDNA PrimersTube formationMice KnockoutWound HealingMultidisciplinaryCell growthComplement C1qEndothelial CellsangiogenesivasculogenesiBiological Scienceswound-healingImmunohistochemistryanimal modelsendothelial cellsRatsMice Inbred C57BLReal-time polymerase chain reactionImmunohistochemistryWound healing
researchProduct

Determination of Human Granulocyte Elastase by the Immunoactivation Method on the Hitachi® 717 Automated Analyser

1991

This paper describes a fully mechanized homogeneous immunoassay using the immunoactivation method for the rapid and specific determination of human granulocyte elastase (EC 3.4.21.37) in plasma. The method uses anti-elastase antibody fragments from sheep, conjugated to horseradish peroxidase. These enzyme-antibody conjugates bind to the elastase-alpha 1-proteinase inhibitor complex present in plasma. A separate sample blank with non-specific sheep antibody fragments conjugated to horseradish peroxidase corrects for errors introduced by the sample matrix. Measurements were performed with the clinical chemistry analyser Hitachi 717. A single determination can be performed in 10 min, requiring…

Pathologymedicine.medical_specialtyeducationClinical BiochemistryEnzyme-Linked Immunosorbent AssayGranulocyteHorseradish peroxidaseReference ValuesBlood plasmamedicineHumansAutomated analyserAutoanalysisChromatographyPancreatic Elastasebiologymedicine.diagnostic_testBiochemistry (medical)ElastaseGeneral Medicinemedicine.anatomical_structureImmunoassaybiology.proteinLeukocyte ElastaseQuantitative analysis (chemistry)ConjugateClinical Chemistry and Laboratory Medicine
researchProduct

A monoclonal Ro-antibody and the serum of a Ro-positive patient with subacute cutaneous lupus erythematosus (SCLE) react with basal layers of human e…

1988

Skin lesions, especially at areas exposed to sunlight, prove to be a major form of manifestation of diseases related to Ro-antibodies and neonatal-, 'ANA-negative-', and cutaneous types of lupus erythe- matosus. A monoclonal Ro-antibody established by our group reacts with a 60 kD polypeptide in extracts from human spleen, whereas in extracts from human epidermis the monoclonal Ro-antibody and a purified Ro-antibody from a monospecific serum of a patient with subacute cutaneous lupus erythematosus reacted with a 60 kD and a 48 kD protein. Performing immunofluorescence microscopy on HEp2-cells both antibodies showed a nuclear speckled staining pattern and a reaction with cytokeratin filament…

Pathologymedicine.medical_specialtymedicine.drug_classClinical BiochemistryBlotting WesternFluorescent Antibody TechniqueEnzyme-Linked Immunosorbent AssayMonoclonal antibodyImmunofluorescenceBiochemistrySubacute cutaneous lupus erythematosusmedicineLupus Erythematosus CutaneousHumansskin and connective tissue diseasesSystemic lupus erythematosusbiologyEpidermis (botany)medicine.diagnostic_testAntibodies MonoclonalGeneral Medicinemedicine.diseaseAntibodies AntinuclearMonoclonalbiology.proteinAntibodyEpidermisAnti-SSA/Ro autoantibodiesEuropean journal of clinical investigation
researchProduct

Acute Morphological and Toxicological Effects in a Human Bronchial Coculture Model after Sulfur Mustard Exposure

2009

International audience; Sulfur mustard (SM) is a strong alkylating agent. Inhalation of SM causes acute lung injury accompanied by severe disruption of the airway barrier. In our study, we tested the acute effects after mustard exposure in an in vitro coculture bronchial model of the proximal barrier. To achieve this, we seeded normal human bronchial epithelial explant-outgrowth cells (HBEC) together with lung fibroblasts as a bilayer on filter plates and exposed the bronchial model after 31 days of differentiation to various concentrations of SM (30, 100, 300, and 500mM). The HBEC formed confluent layers, expressing functional tight junctions as measured by transepithelial electrical resis…

Pathologymedicine.medical_specialtysulfur mustard[SDV]Life Sciences [q-bio]ApoptosisBronchiEnzyme-Linked Immunosorbent Assay[SDV.BC]Life Sciences [q-bio]/Cellular BiologyBiologyLung injuryToxicologyCell LinelungProinflammatory cytokinechemistry.chemical_compoundIn vivoMustard GasmedicineHumansChemical Warfare AgentsInterleukin 8Tight junctionInterleukinSulfur mustardprimary bronchial cellsMolecular biologyCoculture TechniqueschemistryApoptosis[SDV.TOX]Life Sciences [q-bio]/ToxicologyMicroscopy Electron ScanningbarriercocultureToxicological Sciences
researchProduct

Transient suppression of atopy in early childhood is associated with high vaccination coverage.

2003

Objective. To analyze prevalences of allergic sensitization and atopic disease in relation to vaccination coverage. Methods. A German atopy risk-enhanced birth cohort of 1314 neonates who were born in 1990 in 5 German cities was studied. A total of 943 children participated in the follow-up visit at 5 years of age. Atopic symptoms and diagnoses (derived from structured interviews), total serum immunoglobulin E, and specific immunoglobulin E against 9 common allergens (CAP Radio-Allergo-Sorbent Test Fluoro-Enzyme Immunoassay) were evaluated. Children were grouped into dose percentiles according to cumulative doses of any vaccine given up to 5 years of age (<10%, 0–11 doses; 10%–50%, 1…

Pediatricsmedicine.medical_specialtyPercentileDose-Response Relationship ImmunologicImmunoglobulin EDermatitis AtopicAtopyRadioallergosorbent TestHypersensitivityMedicineHumansEarly childhoodSensitizationAsthmaVaccinesbiologybusiness.industryImmunization ProgramsVaccinationInfant NewbornInfantAtopic dermatitisImmunoglobulin Emedicine.diseaseAsthmamedicine.anatomical_structureVaccination coverageChild PreschoolPediatrics Perinatology and Child Healthbiology.proteinbusinessFollow-Up StudiesPediatrics
researchProduct

Peptide microarrays enable rapid mimotope optimization for pharmacokinetic analysis of the novel therapeutic antibody IMAB362.

2014

As membrane proteins play an important role in a variety of life-threatening diseases, the development of therapeutic monoclonal antibodies against membrane proteins is of significant interest. Among many other requirements, the process of antibody drug development requires a set of tailor-made assays for the characterization of the antibodies and for monitoring their activity. Designing assays to characterize antibodies directed to membrane proteins is challenging, because the natural targets are often not available in a format that is compatible with a biochemical assay setup. Thus, alternatives that mimic the targeted membrane proteins are needed. In this study, we developed optimal pept…

Phage displaymedicine.drug_classProtein Array AnalysisEnzyme-Linked Immunosorbent AssayComputational biologyBiologyMonoclonal antibodyApplied Microbiology and BiotechnologyStructure-Activity RelationshipPeptide LibrarymedicineAnimalsIMAB362Mice Inbred BALB CMimotopeAssayAntibodies MonoclonalGeneral MedicineMolecular biologyMembrane proteinDrug developmentMolecular MedicineFemaleDNA microarrayPeptidesProtein BindingBiotechnology journal
researchProduct

Expression of membrane C1q in human monocyte-derived macrophages is developmentally regulated and enhanced by interferon-γ

2001

The present study investigated when during "in vitro" maturation macrophages (MPhi) express membrane C1q (mC1q), and whether cell activation affects expression and function of mC1q. Although C1q mRNA was repeatedly detected in freshly isolated monocytes using reverse transcriptase-polymerase chain reaction, C1q protein was observed only in developing MPhi from day 1 to 4 on using immunodetection and flow cytometry. However, the quantity of mC1q and other MPhi membrane proteins differed strikingly in cells from different donors. We report here for the first time that CD14(+) and CD14(-) mC1q-bearing MPhi can develop, and that interferon-gamma increases mC1q display at the cell surface, and m…

PhagocytosisCD14CellLipopolysaccharide ReceptorsBiophysicsMonocyte/macrophageComplementEnzyme-Linked Immunosorbent AssayBiologyLymphocyte ActivationBiochemistryFlow cytometryInterferon-gammaPhagocytosisStructural BiologyGeneticsmedicineHumansMolecular BiologyCells CulturedC1qMessenger RNAmedicine.diagnostic_testComplement C1qMacrophagesCell DifferentiationCell BiologyFlow CytometryPrecipitin TestsMolecular biologyIn vitromedicine.anatomical_structureGene Expression RegulationMembrane proteinDifferentiationCell activationFEBS Letters
researchProduct