Search results for "STRESS"

showing 10 items of 6278 documents

On the severity of aortic stenosis in ascending aortic aneurysm: A computational tool to examine ventricular-arterial interaction and aortic wall str…

2020

Abstract An ascending thoracic aortic aneurysm (ATAA) is a life-threatening cardiovascular consequence of vessel dilatation that portends adverse events and death. From a clinical perspective, ATAA should not be treated as an isolated disease, and surgery is often carried out in the presence of AS, aortic insufficiency or a calcified valve leaflet. Aortic stenosis (AS) is common in ATAAs and leads to both vessel rigidity and left ventricular (LV) impairment. In this study, lumped-parameter modeling and computational analysis were used to assess the change in the wall shear stress (WSS) and intramural wall stress of patient-specific ATAA models with different degrees of AS (i.e., mild to sev…

Aortic valvemedicine.medical_specialtyHemodynamicsThoracic aortic aneurysmImaging dataAortic aneurysmInternal medicinemedicineGeneral Materials ScienceIn patientFinite-element analysisCivil and Structural EngineeringShear stressbusiness.industryAortic stenosisMechanical EngineeringAscending thoracic aortic aneurysmCondensed Matter Physicsmedicine.diseaseAortic wallStenosismedicine.anatomical_structureMechanics of Materialscardiovascular systemCardiologyCFDbusinessMechanics Research Communications
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Constitutive modeling of ascending thoracic aortic aneurysms using microstructural parameters.

2016

Ascending thoracic aortic aneurysm (ATAA) has been associated with diminished biomechanical strength and disruption in the collagen fiber microarchitecture. Additionally, the congenital bicuspid aortic valve (BAV) leads to a distinct extracellular matrix structure that may be related to ATAA development at an earlier age than degenerative aneurysms arising in patients with the morphological normal tricuspid aortic valve (TAV). The purpose of this study was to model the fiber-reinforced mechanical response of ATAA specimens from patients with either BAV or TAV. This was achieved by combining image-analysis derived parameters of collagen fiber dispersion and alignment with tensile testing dat…

Aortic valvemedicine.medical_specialtyMaterials scienceBicuspid aortic valve0206 medical engineeringFinite Element AnalysisBiomedical EngineeringBiophysics02 engineering and technology030204 cardiovascular system & hematologyThoracic aortic aneurysmModels BiologicalArticleExtracellular matrixSettore ING-IND/14 - Progettazione Meccanica E Costruzione Di Macchine03 medical and health sciencesAortic aneurysmFinite Element Analysi0302 clinical medicineBicuspid aortic valveFinite elementmedicine.arteryInternal medicineTensile StrengthAscending aortamedicineHumansAortaMechanical PhenomenaAortaAortic aneurysmAortic Aneurysm ThoracicSettore ING-IND/34 - Bioingegneria IndustrialeBiomechanical strengthExtracellular matrixAnatomymedicine.disease020601 biomedical engineeringBiomechanical Phenomenamedicine.anatomical_structureBiophysicCardiologycardiovascular systemCollagenStress MechanicalAortic failureHumanMedical engineeringphysics
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Statistical shape analysis of ascending thoracic aortic aneurysm: Correlation between shape and biomechanical descriptors

2020

An ascending thoracic aortic aneurysm (ATAA) is a heterogeneous disease showing different patterns of aortic dilatation and valve morphologies, each with distinct clinical course. This study aimed to explore the aortic morphology and the associations between shape and function in a population of ATAA, while further assessing novel risk models of aortic surgery not based on aortic size. Shape variability of n = 106 patients with ATAA and different valve morphologies (i.e., bicuspid versus tricuspid aortic valve) was estimated by statistical shape analysis (SSA) to compute a mean aortic shape and its deformation. Once the computational atlas was built, principal component analysis (PCA) allow…

Aortic valveprincipal component analysis0206 medical engineeringPopulationshape analysislcsh:MedicineMedicine (miscellaneous)Principal component analysi02 engineering and technology030204 cardiovascular system & hematologyThoracic aortic aneurysmTortuosityArticleCorrelation03 medical and health sciences0302 clinical medicinemedicine.arterymedicineShear stress: straineducationeducation.field_of_studyAortaShape analysibusiness.industryStatistical shape analysislcsh:RComputational modelingAnatomymedicine.disease020601 biomedical engineeringmedicine.anatomical_structurecardiovascular systembusinessShape analysis (digital geometry)
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mHealth for the Monitoring of Brace Compliance and Wellbeing in Adolescents with Idiopathic Scoliosis: Study Protocol for a Feasibility Study

2021

Escoliosis idiopática del adolescente; Aplicación móvil; Eficacia del tratamiento Escoliosi idiopàtica de l'adolescent; Aplicació mòbil; Eficàcia del tractament Adolescent idiopathic scoliosis; Mobile application; Treatment effectiveness Attempts to optimize monitoring of brace adherence prescribed to adolescents with idiopathic scoliosis (IS) have generally relied on sensors. Sensors, however, are intrusive and do not allow the assessment of psychological and physical consequences of brace use that might underlie poor adherence. Mobile applications have emerged as alternatives to monitor brace compliance. However, the feasibility and utility of these app-based systems to assess key psychol…

Aparells ortopèdicsFerides i lesions TractamentHealth Toxicology and Mutagenesis:Health Services Administration::Patient Care Management::Delivery of Health Care::Telemedicine [HEALTH CARE]Adolescents0302 clinical medicineMedicine030212 general & internal medicinemHealthEscoliosiRbrace adherenceTelemedicinaInformation and Communication TechnologiesTelemedicineTest (assessment)DistressScoliosistreatment effectiveness:Musculoskeletal Diseases::Bone Diseases::Spinal Diseases::Spinal Curvatures::Scoliosis [DISEASES]adolescent idiopathic scoliosisMedicinemusculoskeletal diseasesmedicine.medical_specialtyAdolescentTelefonia mòbilArticle:administración de los servicios de salud::gestión de la atención al paciente::prestación sanitaria::telemedicina [ATENCIÓN DE SALUD]03 medical and health sciencesQuality of life (healthcare)HumansComunicació i tecnologiaProtocol (science)adolescent idiopathic scoliosis; Information and Communication Technologies; mobile application; brace adherence; treatment effectivenessBracesbusiness.industry:enfermedades musculoesqueléticas::enfermedades óseas::enfermedades de la columna vertebral::desviaciones de la columna vertebral::escoliosis [ENFERMEDADES]:equipos y suministros::equipo quirúrgico::equipos ortopédicos::aparatos ortésicos::tirantes [TÉCNICAS Y EQUIPOS ANALÍTICOS DIAGNÓSTICOS Y TERAPÉUTICOS]Public Health Environmental and Occupational HealthUsabilitymobile applicationBraceMood:Equipment and Supplies::Surgical Equipment::Orthopedic Equipment::Orthotic Devices::Braces [ANALYTICAL DIAGNOSTIC AND THERAPEUTIC TECHNIQUES AND EQUIPMENT]Physical therapyQuality of LifeFeasibility StudiesPatient Compliancebusiness030217 neurology & neurosurgeryInternational Journal of Environmental Research and Public Health
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Reductive stress in young healthy individuals at risk of Alzheimer disease.

2013

Oxidative stress is a hallmark of Alzheimer disease (AD) but this has not been studied in young healthy persons at risk of the disease. Carrying an Apo e4 allele is the major genetic risk factor for AD. We have observed that lymphocytes from young, healthy persons carrying at least one Apo e4 allele suffer from reductive rather than oxidative stress, i.e., lower oxidized glutathione and P-p38 levels and higher expression of enzymes involved in antioxidant defense, such as glutamylcysteinyl ligase and glutathione peroxidase. In contrast, in the full-blown disease, the situation is reversed and oxidative stress occurs, probably because of the exhaustion of the antioxidant mechanisms just ment…

Apolipoprotein EAdultMaleAntioxidantGenotypemedicine.medical_treatmentApolipoprotein E4DiseaseBiologymedicine.disease_causeBiochemistryAntioxidantschemistry.chemical_compoundAlzheimer DiseaseRisk FactorsPhysiology (medical)medicineHumansAlleleAlleleschemistry.chemical_classificationGlutathione PeroxidaseGlutathione peroxidaseGlutathioneMiddle Agedmedicine.diseaseGlutathioneOxidative StresschemistryImmunologyFemaleLipid PeroxidationAlzheimer's diseaseOxidative stressBiomarkersFree radical biologymedicine
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Amyloid-β toxicity and tau hyperphosphorylation are linked via RCAN1 in Alzheimer's disease.

2011

Amyloid-β peptide (Aβ) toxicity and tau hyperphosphorylation are hallmarks of Alzheimer’s disease (AD). How their molecular relationships may affect the etiology, progression, and severity of the disease, however, has not been elucidated. We now report that incubation of foetal rat cortical neurons with Aβ up-regulates expression of the Regulator of Calcineurin gene RCAN1, and this is mediated by Aβ-induced oxidative stress. Calcineurin (PPP3CA) is a serine-threonine phosphatase that dephosphorylates tau. RCAN1 proteins inhibit this phosphatase activity of calcineurin. Increased expression of RCAN1 also causes up-regulation of glycogen synthase kinase-3beta (GSK3β), a tau kinase. Thus, incr…

Apolipoprotein EAdultMuscle Proteinstau ProteinsBiologymedicine.disease_causeTransfectionArticleDephosphorylationGlycogen Synthase Kinase 3GSK-3Alzheimer DiseasemedicineAnimalsHumansLymphocytesPhosphorylationRNA Small InterferingGSK3BCells CulturedChromatography High Pressure LiquidRegulation of gene expressionCerebral CortexNeuronsAmyloid beta-PeptidesGlycogen Synthase Kinase 3 betaGeneral NeuroscienceCalcineurinIntracellular Signaling Peptides and ProteinsGeneral MedicineMiddle Agedmedicine.diseaseEmbryo MammalianMolecular biologyGlutathionePeptide FragmentsCell biologyRatsCalcineurinDNA-Binding ProteinsPsychiatry and Mental healthClinical PsychologyOxidative StressGene Expression RegulationFemaleGeriatrics and GerontologyAlzheimer's diseaseOxidative stressJournal of Alzheimer's disease : JAD
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Inflammation, Cytokines, Immune Response, Apolipoprotein E, Cholesterol, and Oxidative Stress in Alzheimer Disease: Therapeutic Implications

2010

Alzheimer disease (AD) is a heterogeneous and progressive neurodegenerative disease, which in Western society mainly accounts for senile dementia. Today many countries have rising aging populations and are facing an increased prevalence of age-related diseases, such as AD, with increasing health-care costs. Understanding the pathophysiology process of AD plays a prominent role in new strategies for extending the health of the elderly population. Considering the future epidemic of AD, prevention and treatment are important goals of ongoing research. However, a better understanding of AD pathophysiology must be accomplished to make this objective feasible. In this paper, we review some hot to…

Apolipoprotein EAgingAlzheimer Disease Inflammationmedicine.medical_treatmentInflammationDiseasemedicine.disease_causeImmune System PhenomenaImmune systemApolipoproteins EAlzheimer DiseasemedicineAnimalsHumansInflammationSettore MED/04 - Patologia Generalebusiness.industryImmunotherapymedicine.diseaseDietOxidative StressCytokineCholesterolImmunologyCytokinesImmunotherapyGeriatrics and Gerontologymedicine.symptomAlzheimer's diseasebusinessOxidative stress
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The relationship between cortisol and cognitive function in healthy older people: The moderating role of Apolipoprotein E polymorphism.

2018

The Apolipoprotein E4 (ApoE-epsilon 4) allele has been suggested as the main risk factor for late onset Alzheimer's disease (AD), whereas the ApoE-epsilon 2 allele has been proposed as a protective factor. These proposals have increased the interest in the effect of the ApoE genotype in healthy people. Additionally, high cortisol levels have been related to negative effects on cognition. However, few studies have investigated the relationship between cognitive performance and cortisol, taking into account the different ApoE alleles. For this reason, the aim of this study was to evaluate different cognitive domains (declarative and working memory, attention, and executive function) and their…

Apolipoprotein EMaleSALIVARY CORTISOLHydrocortisonePituitary-Adrenal SystemCortisolBehavioral NeuroscienceExecutive FunctionPOSTTRAUMATIC-STRESS-DISORDER0302 clinical medicineCognitionGenotypeSOCIOECONOMIC-STATUSAttentionPOPULATIONeducation.field_of_study05 social sciencesNeuropsychologyCognitionMiddle AgedALZHEIMERS-DISEASElipids (amino acids peptides and proteins)FemaleApolipoprotein Emedicine.medical_specialtyHypothalamo-Hypophyseal SystemSEX-DIFFERENCESCognitive NeurosciencePopulationExperimental and Cognitive PsychologyPolymorphism Single Nucleotide050105 experimental psychology03 medical and health sciencesApolipoproteins EMemoryAWAKENING RESPONSEInternal medicinemedicineHumans0501 psychology and cognitive sciencesEffects of sleep deprivation on cognitive performanceAlleleeducationAgedELDERLYWorking memorybusiness.industryMEMORY PERFORMANCEE GENOTYPEBODY-MASS INDEXEndocrinologyOlder peoplebusinessNeuroscience030217 neurology & neurosurgeryNeurobiology of learning and memory
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Deficiency of glutathione peroxidase-1 accelerates the progression of atherosclerosis in apolipoprotein E-deficient mice.

2007

Background— We have recently demonstrated that activity of red blood cell glutathione peroxidase-1 is inversely associated with the risk of cardiovascular events in patients with coronary artery disease. The present study analyzed the effect of glutathione peroxidase-1 deficiency on atherogenesis in the apolipoprotein E-deficient mouse. Methods and Results— Female apolipoprotein E-deficient mice with and without glutathione peroxidase-1 deficiency were placed on a Western-type diet for another 6, 12, or 24 weeks. After 24 weeks on Western-type diet, double-knockout mice (GPx-1 −/− ApoE −/− ) developed significantly more atherosclerosis than control apolipoprotein E-deficient mice. Moreover…

Apolipoprotein Emedicine.medical_specialtyGPX1AntioxidantApolipoprotein Bmedicine.medical_treatmentLipoproteinsApoptosisBlood Pressuremedicine.disease_causeNitric OxideMitochondria HeartMonocyteschemistry.chemical_compoundMiceApolipoproteins EGlutathione Peroxidase GPX1SuperoxidesInternal medicinePeroxynitrous AcidmedicineAnimalsAortaCell Proliferationchemistry.chemical_classificationMice KnockoutReactive oxygen speciesGlutathione PeroxidaseMembranesbiologyGlutathione peroxidaseGlutathioneAtherosclerosisEndocrinologyPhenotypechemistryImmunologybiology.proteinDisease ProgressionFemaleCardiology and Cardiovascular MedicineReactive Oxygen SpeciesOxidation-ReductionOxidative stressArteriosclerosis, thrombosis, and vascular biology
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From stress proteins to apoptosis and autophagy in sea urchin embryos

2019

Marine invertebrates inhabit a key position as intermediate consumers in the pelagic as well as in the benthonic food chains, making them suitable model systems for ecotoxicological studies. Among benthonic organisms, echinoderms represent a simple, though significant, model system to test how specific stress can simultaneously provoke dangerous effects on growth and vitality of organisms. Sea urchins provide an attractive and exceptional model for investigating environmental pollution. Most studies investigating the effects of Cd stress were conducted on Paracentrotus lividus, one of the most important marine invertebrates used as bioindicator of metal/heavy metal pollution and an importan…

ApoptosiSettore BIO/06 - Anatomia Comparata E CitologiaSea Urchin Embryos HSPS AutophagyCadmium Stress
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