Search results for "SULFATE"

showing 10 items of 657 documents

The effects of three absorption-modifying critical excipients on the in vivo intestinal absorption of six model compounds in rats and dogs.

2018

Pharmaceutical excipients that may affect gastrointestinal (GI) drug absorption are called critical pharmaceutical excipients, or absorption-modifying excipients (AMEs) if they act by altering the integrity of the intestinal epithelial cell membrane. Some of these excipients increase intestinal permeability, and subsequently the absorption and bioavailability of the drug. This could have implications for both the assessment of bioequivalence and the efficacy of the absorption-enhancing drug delivery system. The absorption-enhancing effects of AMEs with different mechanisms (chitosan, sodium caprate, sodium dodecyl sulfate (SDS)) have previously been evaluated in the rat single-pass intestin…

MalePharmaceutical ScienceExcipientBiological Availability02 engineering and technologyBioequivalencePharmacology030226 pharmacology & pharmacyIntestinal absorptionPermeabilityExcipients03 medical and health sciences0302 clinical medicineDogsIn vivomedicineAnimalsPharmaceutical sciencesIntestinal MucosaChitosanIntestinal permeabilityChemistrySodium Dodecyl Sulfate021001 nanoscience & nanotechnologymedicine.diseaseBioavailabilityRatsIntestinesIntestinal AbsorptionPharmaceutical PreparationsDrug delivery0210 nano-technologyDecanoic Acidsmedicine.drugInternational journal of pharmaceutics
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Different mechanisms of the inhibition of the transient outward current in rat ventricular myocytes.

1994

The mechanism of drug-induced inhibition of the transient outward current, Ito, has been investigated in rat ventricular myocytes using the whole cell patch clamp technique. Ito was activated by 300 ms depolarizing voltage clamp steps in 10 mV increments from −50 mV up to +40 mV. At +40 mV, Ito peaked after about 3 ms, and the time course of inactivation was appropriately described by two time constants, τfast = 17 ms and τslow = 203 ms. Verapamil, quinidine sulfate and nifedipine preferentially depressed Ito at the end of the 300 ms depolarizing voltage clamp step; the inactivation of Ito was accelerated by all drugs, whereas peak Ito was less affected. The time course of drug action at +4…

MalePotassium ChannelsVoltage clampHeart VentriclesPharmacologydigestive systemMembrane PotentialsRats Sprague-Dawleychemistry.chemical_compoundQuinidine SulfateNifedipinemedicineAnimalsVentricular FunctionPatch clampCells CulturedPharmacologyMembrane potentialCardiac transient outward potassium currentMyocardiumHeartGeneral MedicineTetraethylammonium chlorideRatsElectrophysiologychemistryBiophysicsVerapamilmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Neurosteroid modulation of the presynaptic NMDA receptors regulating hippocampal noradrenaline release in normal rats and those exposed prenatally to…

2003

Abstract Prenatal exposure to diazepam (DZ), a positive allosteric modulator of the γ-aminobutyric acidA (GABAA) receptor complex, exerts profound effects that become more evident during puberty and in many cases are sex-specific, suggesting that such exposure interferes with the activity of steroid hormones. Apart from their well known effects on the genome, the reduced metabolites of many steroid hormones also interact directly with membrane receptors, including those for N-methyl- d -aspartate (NMDA). In this study, we compared the effects of several neurosteroids on NMDA receptors from normal rats and those exposed in utero to DZ (1.25 mg/kg per day) from the 14th through the 20th day o…

MalePregnenolone sulfatemedicine.medical_specialtyReceptor complexNeuroactive steroidAllosteric modulatorGlycinePharmacologyHippocampusReceptors N-Methyl-D-AspartateNorepinephrineCellular and Molecular Neurosciencechemistry.chemical_compoundPregnancyInternal medicineNeurosteroidmedicinepregnenolone sulphateAnimalsRats WistarReceptorDiazepamGABAA receptorHippocampal synaptosomesCell BiologyRatsEndocrinologyNMDA/GLY-mediated [3H]NA releasechemistryPregnenolonePrenatal Exposure Delayed EffectsSettore BIO/14 - FarmacologiaNMDA receptorFemalePregnenolone sulfateSynaptosomesHormoneNeurochemistry International
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Perlecan controls neurogenesis in the developing telencephalon.

2006

This article is available from: http://www.biomedcentral.com/1471-213X/7/29

MaleTelencephalonLaminaOrganogenesisApoptosisCell CountNeocortexPerlecanExencephalyBiologyBasement MembraneMiceFetal Organ MaturityInterneuronsPregnancymedicineAnimalsHedgehog Proteinslcsh:QH301-705.5Embryonic Stem CellsCell ProliferationBasement membraneNeuronsCerebrumNeurogenesisAnatomymedicine.diseaseImmunohistochemistryCell biologyNeuroepithelial cellmedicine.anatomical_structurelcsh:Biology (General)embryonic structuresbiology.proteinMicrocephalyBasal laminaFemaleFibroblast Growth Factor 2Heparan Sulfate ProteoglycansDevelopmental BiologyResearch ArticleBMC developmental biology
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Mutations in the PDS Gene in German Families with Pendred’s Syndrome: V138F Is a Founder Mutation

2003

Pendred's syndrome, an autosomal-recessive condition characterized by congenital sensorineural hearing loss and goiter, is caused by mutations in the PDS gene. Located on chromosome 7q22-q31, it encodes a chloride-iodide transporter expressed in the thyroid, inner ear, and kidney. We investigated the PDS gene of six affected individuals from four unrelated families with Pendred's syndrome by direct sequencing. PDS mutations were identified in homozygous or compound heterozygous state in all six cases. A homozygous missense mutation leading to the amino acid substitution S133T was detected in a family of Turkish origin. The mutations found in the other affected individuals, who originate fro…

MaleThreoninemedicine.medical_specialtyAdolescentTurkeyHearing Loss SensorineuralEndocrinology Diabetes and MetabolismClinical BiochemistryMutation MissenseBiologyCompound heterozygositymedicine.disease_causeBiochemistryGenetic determinismEndocrinologyHypothyroidismGermanyInternal medicineSerinemedicineHumansMissense mutationAlleleChildPendred syndromeGeneticsMutationBase SequenceBiochemistry (medical)HaplotypeInfant NewbornMembrane Transport Proteinsfood and beveragesSyndromemedicine.diseaseFounder EffectPedigreeEndocrinologyAmino Acid SubstitutionHaplotypesSulfate TransportersChild PreschoolMicrosatelliteFemaleCarrier ProteinsThe Journal of Clinical Endocrinology & Metabolism
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Augmentation of tibial plateau fractures with an injectable bone substitute: CERAMENT™. Three year follow-up from a prospective study

2015

Background: Reduction of tibial plateau fractures and maintain a level of well aligned congruent joint is key to a satisfactory clinical outcome and is important for the return to pre-trauma level of activity. Stable internal fixation support early mobility and weight bearing. The augmentation with bone graft substitute is often required to support the fixation to mantain reduction. For these reasons there has been development of novel bone graft substitutes for trauma applications and in particular synthetic materials based on calcium phosphates and/or apatite combined with calcium sulfates. Injectable bone substitutes can optimize the filling of irregular bone defects. The purpose of this…

MaleTime FactorsPercutaneousKnee Jointmedicine.medical_treatmentDentistryFracture Fixation InternalFracture FixationTibial plateau fracture Surgical treatment Bone graft Ceramic injectable biphasic bone substitute Clinical and radiographic outcomeFracture fixationTibial plateau fractureOrthopedics and Sports MedicineProspective StudiesTomographyFracture HealingSurgical treatmentBone TransplantationMiddle AgedCombined Modality TherapyBiomechanical PhenomenaX-Ray ComputedDrug CombinationsTreatment Outcomemedicine.anatomical_structureSettore MED/03FemaleResearch ArticleAdultmedicine.medical_specialtyBone healingCalcium SulfateInjectionsRheumatologyClinical and radiographic outcomeTibial plateau fracturemedicineBone graftHumansInternal fixationTibiaTibiabusiness.industryRecovery of Functionmedicine.diseaseInternalSurgeryTibial FracturesDurapatiteCeramic injectable biphasic bone substituteIrregular boneBone SubstitutesOrthopedic surgeryAdult; Biomechanical Phenomena; Bone Substitutes; Bone Transplantation; Calcium Sulfate; Combined Modality Therapy; Drug Combinations; Durapatite; Female; Follow-Up Studies; Fracture Fixation Internal; Fracture Healing; Humans; Injections; Knee Joint; Male; Middle Aged; Prospective Studies; Recovery of Function; Tibia; Tibial Fractures; Time Factors; Tomography X-Ray Computed; Treatment OutcomeTomography X-Ray ComputedbusinessFollow-Up StudiesBMC Musculoskeletal Disorders
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Toxicological investigations in a fatal and non-fatal accident due to hydrogen sulphide (H2S)poisoning

2019

Hydrogen sulphide (H2S) is one of the most toxic natural gas and represents a not rare cause of fatal events in workplaces. We report here a serious accidental poisoning by hydrogen sulphide inhalation involving six sailors. Three of them died while the other three survived and were transported to the emergency room. No greenish discolouration of the body, that could be a feature of these type of deaths, was observed at autopsy. Given that blood and/or urine H2S detection does not allow to discriminate if it is related to inhalation or to putrefactive processes, the determination of thiosulphate, H2S main metabolite, is decisive. The succession of fatal events reported here can be rebuilt b…

MaleTime FactorsPoison controlAutopsyFatal accidentBrain EdemaUrine01 natural scienceschemistry.chemical_compound0302 clinical medicineThiosulfateHydrogen sulphideMedicineHydrogen SulfideThiosulfateAir PollutantsOccupational accidentInhalationFatal poisoningMiddle AgedPulmonary edemaFatal poisoning; Hydrogen sulphide; Non-fatal poisoning; Occupational accident; Thiosulphate; Administration Inhalation; Adult; Air Pollutants; Brain Edema; Emphysema; Humans; Hydrogen Sulfide; Hyperemia; Italy; Male; Middle Aged; Military Personnel; Pulmonary Edema; Thiosulfates; Time Factors; Accidents OccupationalOccupationalMilitary PersonnelInhalationItalyAir PollutantAnesthesiaAdministrationNon-fatal poisoningHumanAdultTime FactorThiosulphateThiosulfatesHyperemiaPulmonary EdemaHydrogen sulphidePathology and Forensic Medicine03 medical and health sciencesAdministration InhalationHumansAccidents Occupational030216 legal & forensic medicineEmphysemabusiness.industry010401 analytical chemistrymedicine.disease0104 chemical scienceschemistryAccidentsbusinessLaw
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Wheat Consumption Aggravates Colitis in Mice via Amylase Trypsin Inhibitor–mediated Dysbiosis

2020

Background & Aims Wheat has become the world's major staple and its consumption correlates with prevalence of noncommunicable disorders such as inflammatory bowel diseases. Amylase trypsin inhibitors (ATIs), a component of wheat, activate the intestine's innate immune response via toll-like receptor 4 (TLR4). We investigated the effects of wheat and ATIs on severity of colitis and fecal microbiota in mice. Methods C57BL/6 wild-type and Tlr4–/– mice were fed wheat- or ATI-containing diets or a wheat-free (control) diet and then given dextran sodium sulfate to induce colitis; we also studied Il10–/– mice, which develop spontaneous colitis. Changes in fecal bacteria were assessed by taxa-speci…

MaleTrypsin inhibitorPlant Proteins DietarySeverity of Illness IndexInflammatory bowel diseaseMicrobiologyFecesMicemedicineAnimalsHumansAmylaseColitisTriticumFecesMice KnockoutHepatologybiologyDextran SulfateGastroenterologyfood and beveragesFecal Microbiota TransplantationColitisInflammatory Bowel Diseasesmedicine.diseaseAnimal FeedImmunity InnateGastrointestinal MicrobiomeToll-Like Receptor 4TransplantationDisease Models Animalbiology.proteinTLR4DysbiosisTrypsin InhibitorsDysbiosisSignal TransductionGastroenterology
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Perlecan-Induced Suppression of Smooth Muscle Cell Proliferation Is Mediated Through Increased Activity of the Tumor Suppressor PTEN

2004

We were interested in the elucidation of the interaction between the heparan sulfate proteoglycan, perlecan, and PTEN in the regulation of vascular smooth muscle cell (SMC) growth. We verified serum-stimulated DNA synthesis, and Akt and FAK phosphorylation were significantly reduced in SMCs overexpressing wild-type PTEN. Our previous studies showed perlecan is a potent inhibitor of serum-stimulated SMC growth. We report in the present study, compared with SMCs plated on fibronectin, serum-stimulated SMCs plated on perlecan exhibited increased PTEN activity, decreased FAK and Akt activities, and high levels of p27, consistent with SMC growth arrest. Adenoviral-mediated overexpression of cons…

MaleVascular smooth musclePhysiology:CIENCIAS MÉDICAS ::Farmacodinámica [UNESCO]Aorta ThoracicBasement MembraneCulture Media Serum-FreeMuscle Smooth VascularRats Sprague-DawleyMicePhosphorylationCells CulturedGlycosaminoglycansbiologyProtein-Tyrosine KinasesCell cycle:CIENCIAS MÉDICAS [UNESCO]musculoskeletal systemUNESCO::CIENCIAS MÉDICAS ::FarmacodinámicaUNESCO::CIENCIAS MÉDICAScardiovascular systemPhosphorylationSmooth muscle cell proliferationCardiology and Cardiovascular MedicineCell DivisionDNA ReplicationBasement membraneRecombinant Fusion ProteinsPerlecanProtein Serine-Threonine KinasesVascular injurySmooth muscle cell proliferation ; Restenosis ; Vascular injury ; Vascular development ; Basement membraneCatheterizationProto-Oncogene ProteinsAnimalsPTENProtein kinase BRestenosisCell growthVascular developmentOligonucleotides AntisenseFibronectinsRatsFibronectinFocal Adhesion Kinase 1Focal Adhesion Protein-Tyrosine Kinasesbiology.proteinCancer researchHeparitin SulfateCarotid Artery InjuriesProtein Processing Post-TranslationalProto-Oncogene Proteins c-aktHeparan Sulfate ProteoglycansCirculation Research
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Effects of Dehydroepiandrosterone Sulfate on Cellular Calcium Responsiveness and Vascular Contractility

1995

Abstract Dehydroepiandrosterone sulfate (DHEAS) is an endogenous steroid having a wide variety of biological effects, but its physiological role remains undefined. Since an age-related decline of DHEAS corresponds to the progressive onset of atherosclerosis, cardiovascular diseases, and overall mortality, we investigated a possible protective role of DHEAS in vascular disease by studying the effects of this hormone (10 −7 to 10 −5 mol/L) on cytosolic free calcium and contractility in different in vitro vascular tissue preparations. DHEAS produced a significant, dose-dependent relaxation of isolated helical strips of rat tail artery precontracted with KCl (60 mmol/L) (89.7±18.7%, P <.01)…

MaleVasopressinmedicine.medical_specialtyVascular smooth muscleArgininechemistry.chemical_elementIn Vitro TechniquesBiologyCalciumMuscle Smooth VascularCalcium in biologyRats Sprague-DawleyContractilitychemistry.chemical_compoundCytosolDehydroepiandrosterone sulfateInternal medicineInternal MedicinemedicineAnimalsCells CulturedVascular tissueAnalysis of VarianceDehydroepiandrosterone SulfateDehydroepiandrosteroneRatsEndocrinologychemistryCalciumMuscle ContractionHypertension
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