Search results for "Sarcomere"

showing 10 items of 25 documents

Considerations for an In Vitro, Cell-Based Testing Platform for Detection of Drug-Induced Inotropic Effects in Early Drug Development. Part 2: Design…

2019

Contractility of the myocardium engines the pumping function of the heart and is enabled by the collective contractile activity of its muscle cells: cardiomyocytes. The effects of drugs on the contractility of human cardiomyocytes in vitro can provide mechanistic insight that can support the prediction of clinical cardiac drug effects early in drug development. Cardiomyocytes differentiated from human-induced pluripotent stem cells have high potential for overcoming the current limitations of contractility assays because they attach easily to extracellular materials and last long in culture, while having human- and patient-specific properties. Under these conditions, contractility measureme…

0301 basic medicinePharmacologyInotropeCell typelcsh:RM1-950cellular alignmentBiologymicroenvironmentco-cultureSarcomereCell biologyContractility03 medical and health scienceslcsh:Therapeutics. Pharmacology030104 developmental biology0302 clinical medicineDrug development030220 oncology & carcinogenesisMyocytePharmacology (medical)sarcomereelectrical stimulationInduced pluripotent stem cellFunction (biology)Frontiers in Pharmacology
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Protein aggregation in congenital myopathies.

2011

Protein aggregation in congenital myopathies may be encountered among different myofibrillar myopathies such as granulofilamentous myopathy, cytoplasmic body myopathy, or spheroid body myopathy, which are designated as αB crystallinopathy, desminopathy, and myotilinopathy, respectively, according to the respective mutant proteins. Caps in cap disease and reducing bodies in reducing body myopathy were disclosed to contain numerous proteins. The multitude of diverse proteins aggregating within muscle fibers suggests impaired extralysosomal degradation of proteins, a disturbance of catabolism. The lack of different proteins accruing, but the mutant ones at an early age of affected patients in …

AdultCatabolismMutantInfantProtein aggregationBiologySarcomereActinsDesminBiochemistryChild PreschoolPediatrics Perinatology and Child HealthMyosinMutationmedicineHumansMutant ProteinsNeurology (clinical)medicine.symptomMyopathyMyofibrilChildActinMyopathies Structural CongenitalSeminars in pediatric neurology
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Medial gastrocnemius muscle behavior during human running and walking.

2007

Utilization of elastic energy in the tendinous tissues (TT) of the human skeletal muscle may be task dependent. The present study was designed to investigate this problem by comparing the fascicle-TT interaction of the medial gastrocnemius muscle (MG) during ground contact of running and walking. Seven subjects ran and walked with a natural cadence. Ankle and knee joint angular data were recorded by electrogoniometers for estimating the entire MG muscle-tendon unit (MTU) length, together with the ground reaction forces. The MG fascicle length was measured by using the high-speed ultrasound image scanning during movements. The results showed that in running, after the rapid early fascicle st…

AdultMaleMaterials scienceMuscle RelaxationMuscle Fibers SkeletalBiophysicsMedial gastrocnemiusWalkingKnee JointSarcomereRunningmedicineHumansOrthopedics and Sports MedicineGround reaction forceMuscle SkeletalUltrasonographyLegElectromyographyRehabilitationSkeletal muscleAnatomyFasciclemedicine.anatomical_structureAnkleCadenceMuscle ContractionGaitposture
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Store and recoil of elastic energy in slow and fast types of human skeletal muscles.

1982

Stretch-shortening cycle refers to the mechanical condition in which store and recoil of elastic energy occur in the skeletal muscle. This leads to a greater work output when compared to a simple shortening contraction. The subjects performed vertical jumps with and without preliminary counter-movement and with small and large knee angular displacement. The results indicated that those subjects who had more fast twitch (FT) fibers benefited more from the stretching phase performed with high speed and short angular displacement. The amounts of elastic energy stored in this phase were 30 and 26 N X kgBW-1, respectively, for FT and slow twitch (ST) type subjects. The recoil of elastic energy w…

AdultMaleMaterials scienceWork outputAdolescentPhysiologyIsometric exerciseSports MedicineSarcomereModels BiologicalStretch shortening cycleRecoilCrossBridgeIsometric ContractionmedicineHumansMusclesElastic energyMechanicsAnatomyElastic TissueFemalemedicine.symptomEnergy MetabolismMuscle contractionMuscle ContractionActa physiologica Scandinavica
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Reducing Body Myopathy with Cytoplasmic Bodies and Rigid Spine Syndrome: A Mixed Congenital Myopathy

2001

At the age of five years a male child started to develop a progressive rigid spine, torsion scoliosis, and flexion contractures of his elbows, knees, hips, and ankles owing to severe proximal and distal muscle weakness. He had three muscle biopsies from three different muscles at ages 7, 11, and 14 years, respectively. Myopathologically, these muscle tissues contained numerous inclusions which, at the ultrastructural level, turned out to be reducing bodies and cytoplasmic bodies, often in close spatial proximity. Similar histological inclusions, although not further identified by histochemistry and electron microscopy, were seen in his maternal grandmother's biopsied muscle tissue who had d…

AdultMaleMuscle tissuePathologymedicine.medical_specialtyWeaknessScoliosisSpinal Muscular Atrophies of ChildhoodSarcomereMyositis Inclusion BodymedicineHumansGenetic Predisposition to DiseaseMuscle SkeletalMyopathyMyositisAgedInclusion Bodiesbusiness.industrySyndromeGeneral MedicineAnatomymedicine.diseasePenetrancePedigreemedicine.anatomical_structureChild PreschoolPediatrics Perinatology and Child HealthDisease ProgressionLordosisFemaleDesminNeurology (clinical)medicine.symptombusinessMyopathies Structural CongenitalNeuropediatrics
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Acute change of titin at mid-sarcomere remains despite 8 wk of plyometric training

2014

Purpose: The purpose of this study was to investigate skeletal muscle changes induced by an acute bout of plyometric exercise (PlyEx) both before and after PlyEx training, to understand if titin is affected differently after PlyEx training. Methods: Healthy untrained individuals (N=11) completed the 1stPlyEx (10x10 squat-jumps, 1min rest). Thereafter, 6 subjects completed 8 weeks of PlyEx, while 5 controls abstained from any jumping activity. Seven days after the last training session all subjects completed the 2ndPlyEx. Blood samples were collected before, 6 hours and 1, 2, 3 and 4 days after each acute bout of PlyEx, and muscle biopsies 4 days before and 3 days after each acute bout of Pl…

AdultMaleSarcomeresmedicine.medical_specialtyPhysiologyZ-disk streaming acute bouts calpain eccentric exerciseMuscle ProteinsPlyometric ExerciseSarcomerePhysiology (medical)HumansMedicinePlyometricsConnectinTissue DistributionMuscle SkeletalExercisebiologySettore BIO/16 - Anatomia UmanaCalpainbusiness.industrySkeletal musclemusculoskeletal systemAdaptation Physiologicalmedicine.anatomical_structurebiology.proteinPhysical therapyFemaleTitinPlyometric trainingAutolysisbusinessMuscle ContractionJournal of Applied Physiology
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The effect of fatigue on store and re-use of elastic energy in slow and fast types of human skeletal muscle

1986

Stretch-shortening exercises are characterized by enhancement of performance when compared to the work output performed in shortening conditions. There is evidence that fast subjects are unable to re-use great amounts of elastic energy during stretch-shortening cycles performed with slow stretching speed and large stretching length. In the present study, 14 subjects possessing different fibre types in m. vastus lateralis performed vertical jumps with and without preliminary countermovement and with large angular displacement and slow stretching speed The jumping tests were executed before and immediately after fatigue induced by short intense exercises (60 s of continuous rebound jumping). …

AdultMaleWork outputMaterials scienceMuscle fatiguePhysiologyPhysical ExertionElastic energySkeletal muscleAnatomyElastic Tissuemedicine.disease_causeSarcomereStretch shortening cycleBiomechanical PhenomenaJumpingmedicine.anatomical_structuremedicineHumansmedicine.symptomMechanoreceptorsMuscle ContractionMuscle contractionBiomedical engineeringActa Physiologica Scandinavica
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Ultrastructural pathology in emetine-induced myopathy

1988

Progressive myopathy developed in two women who consumed ipecac syrup containing emetine hydrochloride to induce vomiting as part of their anorexia nervosa. Muscle biopsy specimens were characterized by severe disruption of the sarcomeres. The ultrastructural spectrum extended from "Z-band streaming" to the formation of cytoplasmic bodies and also comprised abnormalities of the sarcotubular system, thus suggesting that muscle weakness may be related to both sarcomeric and sarcotubular lesions in this self-inflicted myopathy. It is tempting to suggest that muscle weakness may be correlated with or based on the pathology in sarcomeres and the sarcotubular system. As the myopathy is clinically…

AdultSarcomeresPathologymedicine.medical_specialtyAnorexia NervosaSubstance-Related DisordersEmetineEmetine HydrochlorideEmetineBiologySarcomereUltrastructural PathologyPathology and Forensic MedicineCellular and Molecular NeuroscienceMuscular DiseasesmedicineHumansMyopathyMuscle biopsymedicine.diagnostic_testMusclesMuscle weaknessMitochondriaSarcoplasmic ReticulumAnorexia nervosa (differential diagnoses)FemaleNeurology (clinical)medicine.symptommedicine.drugActa Neuropathologica
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Fetal akinesia caused by a novel actin filament aggregate myopathy skeletal muscle actin gene (ACTA1) mutation.

2010

We report a female newborn, diagnosed with fetal akinesia in utero, who died one hour after birth. Post-mortem muscle biopsy demonstrated actin-filament myopathy based on immunolabelling for sarcomeric actin, and large areas of filaments, without rod formation, ultrastructurally. Analysis of DNA extracted from the muscle disclosed a novel de novo heterozygous c.44G>A, GGC>GAC, 'p.Gly15Asp' mutation in the ACTA1 gene. Analysis of the location of the mutated amino-acid in the actin molecule suggests the mutation most likely causes abnormal nucleotide binding, and consequent pathological actin polymerization. This case emphasizes the association of fetal akinesia with actin-filament myopathy.

AdultSarcomeresmacromolecular substancesBiologymedicine.disease_causeSarcomereNemaline myopathyPregnancymedicineHumansMyopathyMuscle SkeletalGenetics (clinical)ActinMutationMuscle biopsymedicine.diagnostic_testMicrofilament ProteinsInfant NewbornSkeletal muscleDNANeuromuscular DiseasesActin cytoskeletonmedicine.diseaseMolecular biologyActin CytoskeletonFetal Diseasesmedicine.anatomical_structureNeurologyBiochemistryPediatrics Perinatology and Child HealthMutationFemaleNeurology (clinical)medicine.symptomNeuromuscular disorders : NMD
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Deleting Full Length Titin Versus the Titin M-Band Region Leads to Differential Mechanosignaling and Cardiac Phenotypes

2019

Background: Titin is a giant elastic protein that spans the half-sarcomere from Z-disk to M-band. It acts as a molecular spring and mechanosensor and has been linked to striated muscle disease. The pathways that govern titin-dependent cardiac growth and contribute to disease are diverse and difficult to dissect. Methods: To study titin deficiency versus dysfunction, the authors generated and compared striated muscle specific knockouts (KOs) with progressive postnatal loss of the complete titin protein by removing exon 2 (E2-KO) or an M-band truncation that eliminates proper sarcomeric integration, but retains all other functional domains (M-band exon 1/2 [M1/2]-KO). The authors evaluated c…

Cardiomyopathy DilatedMaleSarcomeresanimal structuresVentricular Dysfunction Rightmacromolecular substances030204 cardiovascular system & hematologyMechanotransduction CellularVentricular Function LeftArticleMuscle hypertrophyVentricular Dysfunction Left03 medical and health sciences0302 clinical medicinePhysiology (medical)AnimalsMedicineMyocytes CardiacMuscle Skeletal030304 developmental biologyMice Knockout0303 health sciencesbiologybusiness.industryMolecular springmusculoskeletal systemPhenotypeCell biologyMuscular AtrophyPhenotypeMuscle diseasecardiovascular systemVentricular Function Rightbiology.proteinTitinCardiology and Cardiovascular MedicinebusinessProtein KinasesGene DeletionDifferential (mathematics)Circulation
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