Search results for "Secretin"

showing 10 items of 49 documents

Effects on α- and β-cell function of sequentially adding empagliflozin and linagliptin to therapy in people with type 2 diabetes previously receiving…

2017

Aims The aim of the study was to investigate the effect of sequential treatment escalation with empagliflozin and linagliptin on laboratory markers of alpha- and beta cell function in patients with type 2 diabetes mellitus (T2DM) insufficiently controlled on metformin monotherapy. Methods Forty-four patients with T2DM received 25 mg empagliflozin for a duration of one month in an open-label fashion (treatment period (TP 1). Thereafter, patients were randomised to a double-blind add-on therapy with linagliptin 5 mg or placebo (TP 2) for one additional month. Alpha- and beta cell function were assessed with a standardised liquid meal test (LMT) and an intravenous (iv.) glucose challenge. Effi…

Blood GlucoseMalemedicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatment030209 endocrinology & metabolismLinagliptinType 2 diabetes030204 cardiovascular system & hematologyLinagliptinGlucagon03 medical and health sciences0302 clinical medicineEndocrinologyDouble-Blind MethodGlucosidesInternal medicineInsulin-Secreting CellsInternal MedicinemedicineEmpagliflozinHumansHypoglycemic AgentsInsulinBenzhydryl CompoundsProinsulinAgedbusiness.industryInsulinMiddle Agedmedicine.diseaseGlucagonPostprandial PeriodMetforminEndocrinologyPostprandialTreatment OutcomeDiabetes Mellitus Type 2Glucagon-Secreting CellsGlucose Clamp TechniqueFemalebusinessmedicine.drugProinsulinDiabetes, obesitymetabolism
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Predictors of microvascular complications in type 1 diabetic patients at onset: The role of metabolic memory

2011

Background Several epidemiological studies showed a close association between metabolic control and microvascular complications in type 1 Diabetes Mellitus (T1DM). The aim of our longitudinal observational study was to evaluate the predictive role of the main clinical and biochemical parameters in determining microvascular complications. Methods 376 T1DM patients, hospitalized in our division from 1991 to 2005 (mean follow-up = 10.93 ± 4.26 years) were studied. Stepwise Cox regression analysis was used to identify the influence of residual ß-cell function, ß-cell autoimmunity, HbA1c levels and other clinical and laboratory parameters in the development of microalbuminuria and retinopathy. R…

Malemedicine.medical_specialtyAdolescentendocrine system diseasesSettore MED/13 - EndocrinologiaNephropathyYoung AdultPredictive Value of TestsInsulin-Secreting CellsInternal medicineDiabetes mellitusInternal MedicinemedicineAlbuminuriaHumansDiabetic NephropathiesLongitudinal StudiesAge of OnsetChildAutoantibodiesProportional Hazards ModelsGlycated HemoglobinInpatientsType 1 diabetesDiabetic Retinopathybusiness.industryMicrocirculationMicroangiopathyType 1 diabetes microvascular Complicationsnutritional and metabolic diseasesmedicine.diseaseSurgeryDiabetes Mellitus Type 1Metabolic control analysisPredictive value of testsFemaleMicroalbuminuriaAge of onsetbusinessDiabetic AngiopathiesFollow-Up StudiesEuropean Journal of Internal Medicine
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In vivo reprogramming for tissue repair.

2015

Berninger and colleagues define milestones for in vivo reprogramming and discuss recent developments in reprogramming into pancreatic b-cells and neurons. Vital organs such as the pancreas and the brain lack the capacity for effective regeneration. To overcome this limitation, an emerging strategy consists of converting resident tissue-specific cells into the cell types that are lost due to disease by a process called in vivo lineage reprogramming. Here we discuss recent breakthroughs in regenerating pancreatic β-cells and neurons from various cell types, and highlight fundamental challenges that need to be overcome for the translation of in vivo lineage reprogramming into therapy.

Cell typeLineage (genetic)Cell- and Tissue-Based TherapyAcinar CellsBiologyIn vivoInsulin-Secreting CellsmedicineHumansRegenerationCell LineagePancreasNeuronsBrain DiseasesRegeneration (biology)BrainPancreatic DiseasesTranslation (biology)Cell DifferentiationCell BiologyTissue repairCellular ReprogrammingCell biologymedicine.anatomical_structurePancreasReprogrammingNeurogliaNature cell biology
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Heterogeneity of circulating CD8 T-cells specific to islet, neo-antigen and virus in patients with type 1 diabetes mellitus

2018

Auto-reactive CD8 T-cells play an important role in the destruction of pancreatic β-cells resulting in type 1 diabetes (T1D). However, the phenotype of these auto-reactive cytolytic CD8 T-cells has not yet been extensively described. We used high-dimensional mass cytometry to phenotype autoantigen- (pre-proinsulin), neoantigen- (insulin-DRIP) and virus- (cytomegalovirus) reactive CD8 T-cells in peripheral blood mononuclear cells (PBMCs) of T1D patients. A panel of 33 monoclonal antibodies was designed to further characterise these cells at the single-cell level. HLA-A2 class I tetramers were used for the detection of antigen-specific CD8 T-cells. Using a novel Hierarchical Stochastic Neighb…

Male0301 basic medicinelcsh:MedicineCD8-Positive T-LymphocytesBiochemistryAutoantigensEndocrinologyInsulin-Secreting CellsCellular typesCytotoxic T celllcsh:ScienceStainingMultidisciplinaryImmune cellsCell StainingPhenotypePhenotypesData AcquisitionPhenotypeWhite blood cellsFemaleSingle-Cell AnalysisResearch ArticleAdultCell biologyBlood cellsComputer and Information SciencesEndocrine Disordersmedicine.drug_classImmunologyT cellsCytotoxic T cellsBiologyResearch and Analysis MethodsMonoclonal antibodyPeripheral blood mononuclear cellVirus03 medical and health sciencesHLA-A2 AntigenGeneticsDiabetes MellitusmedicineHumansMass cytometryMedicine and health sciencesType 1 diabetesBiology and life scienceslcsh:Rmedicine.diseaseDiabetes Mellitus Type 1030104 developmental biologyAnimal cellsSpecimen Preparation and TreatmentMetabolic DisordersImmunologyLeukocytes Mononuclearlcsh:QCytometryBiomarkersCD8
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Increased Gastrin and Calcitonin Secretion after Oral Calcium or Peptones Administration in Patients with Hypercalciuria: A Clue to an Alteration in …

2005

The calcium-sensing receptor (CaSR) has been detected in human antral gastrin-secreting cells, where, upon calcium and/or amino acid allosteric activation, it stimulates gastrin secretion. Patients with absorptive hypercalciuria (AH) display an enhanced gastric acid output; therefore, we evaluated the secretion of gastrin in subjects with AH ( 30 subjects vs. 30 healthy female controls, all postmenopausal) after oral calcium administration ( 1 g calcium gluconate) and, on a separate occasion, after peptone loading test ( protein hydrolyzed, 10 g). Gastrin and monomeric calcitonin responses were higher in AH after both oral calcium administration ( P < 0.01) and peptone loading ( P< 0.01). B…

Calcitoninmedicine.medical_specialtyEndocrinology Diabetes and MetabolismClinical BiochemistryThyroid GlandAdministration Oralchemistry.chemical_element.CalciumBiochemistryKidney CalculiEndocrinologyOral administrationCalcium Metabolism DisordersInternal medicineGastrinsmedicineHumansGastrin-Secreting CellsHypercalciuriaAgedGastrinBiochemistry (medical)Middle AgedCalcitonin secretionmedicine.diseaseCalcium GluconateEndocrinologychemistryGastrointestinal hormoneParathyroid HormoneCalcitoninPeptonesFemaleCalcium-sensing receptorReceptors Calcium-Sensinghormones hormone substitutes and hormone antagonistsThe Journal of Clinical Endocrinology &amp; Metabolism
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Early Lung Function Abnormalities in Acromegaly.

2014

BACKGROUND: Acromegaly is an insidious disorder caused by a pituitary growth hormone (GH)-secreting adenoma resulting in high circulating levels of GH and insulin-like growth factor I (IGF-I). Respiratory disorders are common complications in acromegaly, and can severely impact on quality of life, eventually affecting mortality. OBJECTIVES: The present study aimed to explore structural and functional lung alterations of acromegalic subjects. METHODS: We enrolled 10 consecutive patients (M/F: 5/5) affected by acromegaly. In all patients, magnetic resonance imaging (MRI) revealed the presence of pituitary tumor. All patients underwent clinical, lung functional, biological, and radiological as…

AdultLung DiseasesMalePulmonary and Respiratory MedicinePathologymedicine.medical_specialtyTime FactorsAdenomaVital CapacitySettore MED/10 - Malattie Dell'Apparato RespiratorioGastroenterologyspirometry dyspnea acromegalySettore MED/13 - EndocrinologiaPredictive Value of TestsDLCOForced Expiratory VolumeDiffusing capacityInternal medicineAcromegalymedicineHumansLung volumesRespiratory systemLungAgedLungbusiness.industryPituitary tumorsMiddle Agedrespiratory systemmedicine.diseaseMagnetic Resonance Imagingrespiratory tract diseasesRadiographyEarly Diagnosismedicine.anatomical_structureCase-Control StudiesPulmonary Diffusing CapacityFemaleGrowth Hormone-Secreting Pituitary AdenomaLung Volume Measurementsbusiness
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Molecular aspects of pancreatic β-cell dysfunction: Oxidative stress, microRNA, and long noncoding RNA.

2018

Metabolic syndrome is known as a frequent precursor of type 2 diabetes mellitus (T2D). This disease could affect 8% of the people worldwide. Given that pancreatic β-cell dysfunction and loss have central roles in the initiation and progression of the disease, the understanding of cellular and molecular pathways associated with pancreatic β-cell dysfunction can provide more information about the underlying pathways involved in T2D. Multiple lines evidence indicated that oxidative stress, microRNA, and long noncoding RNA play significant roles in various steps of diseases. Oxidative stress is one of the important factors involved in T2D pathogenesis. This could affect the function and surviva…

0301 basic medicinePhysiologyClinical BiochemistryCellDiseaseBiologymedicine.disease_causePathogenesis03 medical and health sciences0302 clinical medicineInsulin-Secreting CellsGene expressionmicroRNAmedicineHumansEpigeneticsPancreasCell BiologyLong non-coding RNACell biologyMicroRNAsOxidative Stress030104 developmental biologymedicine.anatomical_structureDiabetes Mellitus Type 2030220 oncology & carcinogenesisRNA Long NoncodingOxidative stressSignal TransductionJournal of cellular physiology
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Recovery of Endogenous β-Cell Function in Nonhuman Primates After Chemical Diabetes Induction and Islet Transplantation

2008

OBJECTIVE—To describe the ability of nonhuman primate endocrine pancreata to reestablish endogenous insulin production after chemical β-cell destruction. RESEARCH DESIGN AND METHODS—Eleven monkeys (Macaca fascicularis) were rendered diabetic with streptozotocin. Eight diabetic monkeys received intraportal porcine islet transplantation. RESULTS—Two monkeys transplanted after 75 days of type 1 diabetes showed recovery of endogenous C-peptide production a few weeks after transplantation, concomitant with graft failure. Histological analysis of the pancreas of these monkeys showed insulin-positive cells, single or in small aggregates, scattered in the pancreas and adjacent to ducts. Interesting…

endocrine systemmedicine.medical_specialtySwineEndocrinology Diabetes and Metabolismmedicine.medical_treatmentIslets of Langerhans TransplantationBiologyStreptozocinDiabetes Mellitus ExperimentalInsulin-Secreting CellsInternal medicineDiabetes mellitusInternal MedicinemedicineAnimalsInsulinProinsulingeographyType 1 diabetesgeography.geographical_feature_categoryInsulinHaplorhiniStreptozotocinmedicine.diseaseIsletTransplantationmedicine.anatomical_structureEndocrinologyIslet StudiesPancreasmedicine.drugDiabetes
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A Good Breath of Oxygen for Beta-Like Cells Obtained From Porcine Exocrine Pancreatic Tissue

2011

Ischemia is the most important factor that affects organ survival during harvesting. The two-layer method (TLM) is one of several cold storage solutions that seeks to preserve organs and cells avoiding in vivo and in vitro ischemia. We compared the retrieval of beta-like elements from exocrine pancreatic cells using TLM versus University of Wisconsin (UW) solutions. For this purpose pancreata laparoscopically harvested from 20 female pigs were preserved in UW solution or TLM before digestion. The resulting exocrine cells were divided into 2 groups: the first was cultured in a designed medium to allow differentiation into beta-like cells and the second was cryopreserved before the differenti…

AdenosineTime FactorsCell SurvivalSwineAllopurinolCellular differentiationOrgan Preservation Solutionsbeta-like-cells porcine esocrine pancreatic tisuueCold storageCell SeparationCryopreservationAndrologyPancreatectomyRaffinoseIn vivoSettore BIO/13 - Biologia ApplicataInsulin-Secreting CellsmedicineAnimalsInsulinViaspanCells CulturedCryopreservationFluorocarbonsTransplantationbusiness.industryCell DifferentiationAnatomyGlutathionePancreas ExocrineIn vitroCulture MediaOxygenTransplantationSettore MED/18 - Chirurgia GeneraleGlucosemedicine.anatomical_structureTissue and Organ HarvestingFemaleLaparoscopySurgeryPancreasbusiness
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Adding insulin glargine vs. NPH insulin to metformin results in a more efficient postprandial β-cell protection in individuals with type 2 diabetes

2010

AIM Postprandial release of intact proinsulin (IP) is an independent marker for beta-cell dysfunction in patients with type 2 diabetes. This open-label, parallel-group, two-arm, pilot study compared the beta-cell protective effect of adding insulin glargine (GLA) vs. NPH insulin to ongoing metformin. MATERIAL AND METHODS Overall, 28 insulin-naive type 2 diabetes subjects (mean +/- SD age, 61.5 +/- 6.7 years; diabetes duration, 9.8 +/- 6.5 years; HbA1c, 7.1 +/- 0.5%; BMI, 30.7 +/- 4.3 kg/m(2)) treated with metformin and sulfonylurea were randomized to add once-daily GLA or NPH at bedtime. At baseline and after 3 months, subjects received a standardized breakfast, lunch and dinner, with pre- …

Malemedicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentInsulin IsophaneInsulin GlarginePilot ProjectsNPH insulinType 2 diabetesNPH insulinDrug Administration ScheduleEndocrinologyInsulin-Secreting CellsInternal medicineDiabetes mellitusInternal MedicinemedicineHumansHypoglycemic AgentsInsulinintact proinsulinGlycated Hemoglobinbusiness.industryInsulin glargineInsulindigestive oral and skin physiologynutritional and metabolic diseasesFastingOriginal ArticlesMiddle AgedPostprandial Periodmedicine.diseaseMetforminMetforminInsulin Long-ActingEndocrinologyPostprandialDiabetes Mellitus Type 2beta cell stressDrug Therapy CombinationFemalebusinessmedicine.drugBlood samplingDiabetes, Obesity and Metabolism
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