Search results for "Signal Transduction"

showing 10 items of 2020 documents

Prostaglandin E2 activates the ciliary beat frequency of cultured human nasal mucosa via the second messenger cyclic adenosine monophosphate.

2001

Prostaglandins influence the ciliary beat frequency (CBF) of ciliated nasal epithelial cells and a stimulatory effect has been described for prostaglandin E2 (PGE2). Until now, it is not known whether PGE2 has direct ciliostimulatory properties or acts through a second messenger. In this study we investigated whether cyclic adenosine monophosphate (cAMP) is implicated in the signal transduction pathway of PGE2-induced activation of CBF. Ciliated cells of the nasal mucosa were cultured for up to 5 days whereafter the culture medium was removed and the cells were incubated with different concentrations of test solutions. The ciliated cells were recorded under a phase-contrast microscope and v…

AdultMalemedicine.medical_specialtyStimulationMucous membrane of noseBiologyIn Vitro TechniquesSecond Messenger SystemsDinoprostonechemistry.chemical_compoundInternal medicinemedicineCyclic AMPHumansCyclic adenosine monophosphateCiliaProstaglandin E2Cells CulturedAgedDose-Response Relationship DrugColforsinEpithelial CellsGeneral MedicineMiddle AgedEpitheliumCell biologyNasal Mucosamedicine.anatomical_structureEndocrinologyOtorhinolaryngologychemistryCell cultureSecond messenger systemFemaleSignal transductionmedicine.drugSignal TransductionEuropean archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery
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VEGF-D expression correlates with colorectal cancer aggressiveness and is downregulated by cetuximab

2008

AIM: To gain mechanistic insights into the role played by epidermal growth factor receptor (EGFR) in the regulation of vascular endothelial growth factors (VEGFs) in colorectal cancer (CRC). METHODS: The impact of high-level expression of the growth factor receptors EGFR and VEGF receptor (VEGFR)3 and the VEGFR3 ligands VEGF-C and VEGF-D on disease progression and prognosis in human CRC was investigated in 108 patients using immunohistochemistry. Furthermore, the expression of the lymphangiogenic factors in response to the modulation of EGFR signalling by the EGFR-targeted monoclonal antibody cetuximab was investigated at the mRNA and protein level in human SW480 and SW620 CRC cell lines an…

AdultPathologymedicine.medical_specialtyColorectal cancerTransplantation HeterologousVascular Endothelial Growth Factor CVascular Endothelial Growth Factor DCetuximabAntineoplastic AgentsMice SCIDAntibodies Monoclonal HumanizedMiceGrowth factor receptorMice Inbred NODCell Line TumorMedicineAnimalsHumansEpidermal growth factor receptorneoplasmsAgedColorectal CancerAged 80 and overCetuximabbiologyintegumentary systembusiness.industryGastroenterologyVascular Endothelial Growth Factor DAntibodies MonoclonalGeneral MedicineMiddle Agedmedicine.diseaseVascular Endothelial Growth Factor Receptor-3digestive system diseasesLymphangiogenesisErbB ReceptorsVascular endothelial growth factor CCancer researchbiology.proteinImmunohistochemistryFemalebusinessColorectal NeoplasmsNeoplasm Transplantationmedicine.drugSignal Transduction
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Genes involved in immune response/inflammation, IGF1/insulin pathway and response to oxidative stress play a major role in the genetics of human long…

2005

In this paper, we review data of recent literature on the distribution in centenarians of candidate germ-line polymorphisms that likely affect the individual chance to reach the extreme limit of human life. On the basis of previous observations on the immunology, endocrinology and cellular biology of centenarians we focused on genes that regulate immune responses and inflammation (IL-6, IL-1 cluster, IL-10), genes involved in the insulin/IGF-I signalling pathway and genes that counteract oxidative stress (PON1). On the whole, data indicate that polymorphisms of these genes likely contribute to human longevity, in accord with observations emerging from a variety of animal models, and suggest…

AdultSenescenceAgingCandidate geneGenotypemedia_common.quotation_subjectLongevityBiologyModels BiologicalGenomeImmune systemHumansInsulinInsulin-Like Growth Factor IGeneAgedmedia_commonAged 80 and overInflammationGeneticsPolymorphism GeneticAryldialkylphosphataseInterleukin-6Age FactorsImmunityLongevityHedgehog signaling pathwayInterleukin-10Oxidative StressMultigene FamilyFunction (biology)Interleukin-1Signal TransductionDevelopmental BiologyMechanisms of Ageing and Development
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C4-dicarboxylate carriers and sensors in bacteria

2002

AbstractBacteria contain secondary carriers for the uptake, exchange or efflux of C4-dicarboxylates. In aerobic bacteria, dicarboxylate transport (Dct)A carriers catalyze uptake of C4-dicarboxylates in a H+- or Na+-C4-dicarboxylate symport. Carriers of the dicarboxylate uptake (Dcu)AB family are used for electroneutral fumarate:succinate antiport which is required in anaerobic fumarate respiration. The DcuC carriers apparently function in succinate efflux during fermentation. The tripartite ATP-independent periplasmic (TRAP) transporter carriers are secondary uptake carriers requiring a periplasmic solute binding protein. For heterologous exchange of C4-dicarboxylates with other carboxylic …

Aerobic bacteriaAntiporterSuccinic AcidBiophysicsOrganic Anion TransportersReceptors Cell Surfacemedicine.disease_causeBiochemistryFumarate (succinate) sensorTwo-component systemBacterial ProteinsFumaratesEscherichia colimedicineAmino Acid SequenceEscherichia coliDicarboxylate uptake SHistidine protein kinasePhylogenyHistidineDicarboxylic Acid TransportersDicarboxylate transport BbiologyEscherichia coli ProteinsBiological TransportPeriplasmic spaceCell Biologybiology.organism_classificationTwo-component regulatory systemBacteria AerobicModels ChemicalBiochemistryAntiportFumarate/succinate transportEffluxDicarboxylate uptake carrierProtein KinasesDicarboxylate transport A carrierBacteriaSignal TransductionBiochimica et Biophysica Acta (BBA) - Bioenergetics
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Aging Negatively Affects Estrogens-Mediated Effects on Nitric Oxide Bioavailability by Shifting ERα/ERβ Balance in Female Mice

2011

AIMS: Aging is among the major causes for the lack of cardiovascular protection by estrogen (E2) during postmenopause. Our study aims to determine the mechanisms whereby aging changes E2 effects on nitric oxide (NO) production in a mouse model of accelerated senescence (SAM). METHODS AND RESULTS: Although we found no differences on NO production in females SAM prone (SAMP, aged) compared to SAM resistant (SAMR, young), by either DAF-2 fluorescence or plasmatic nitrite/nitrate (NO2/NO3), in both cases, E2 treatment increased NO production in SAMR but had no effect in SAMP. Those results are in agreement with changes of eNOS protein and gene expression. E2 up-regulated eNOS expression in SAMR…

AgingAnatomy and Physiologylcsh:MedicineEstrogen receptorFluorescent Antibody TechniqueCardiovascularCardiovascular SystemBiochemistrychemistry.chemical_compoundMiceEndocrinologyEnosMolecular Cell BiologyMembrane Receptor Signalinglcsh:ScienceReceptorMultidisciplinarybiologySuperoxideNeurochemistryHormone Receptor SignalingReceptors EstrogenDNA methylationCirculatory PhysiologyMedicineFemaleNeurochemicalsResearch ArticleSignal TransductionSenescencemedicine.medical_specialtymedicine.drug_classBlotting WesternEndocrine SystemNitric OxideReal-Time Polymerase Chain ReactionCardiovascular PharmacologyNitric oxideInternal medicinemedicineCardiovascular Diseases in WomenAnimalsBiologyEndocrine Physiologylcsh:RNADPH OxidasesEstrogensDNA Methylationbiology.organism_classificationHormonesEndocrinologychemistryEstrogenWomen's Healthlcsh:QNeurosciencePLoS ONE
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Calorie Restriction in Mammals and Simple Model Organisms

2014

Calorie restriction (CR), which usually refers to a 20–40% reduction in calorie intake, can effectively prolong lifespan preventing most age-associated diseases in several species. However, recent data from both human and nonhumans point to the ratio of macronutrients rather than the caloric intake as a major regulator of both lifespan and health-span. In addition, specific components of the diet have recently been identified as regulators of some age-associated intracellular signaling pathways in simple model systems. The comprehension of the mechanisms underpinning these findings is crucial since it may increase the beneficial effects of calorie restriction making it accessible to a broad…

AgingCalorie restrictionPopulationved/biology.organism_classification_rank.speciesRegulatorlcsh:MedicineReview ArticleBiologylongevity ageing calorie restriction dietBioinformaticsGeneral Biochemistry Genetics and Molecular BiologyIntracellular signaling pathwaysSettore BIO/13 - Biologia ApplicataYeastsAnimalsHumansCaenorhabditis eleganseducationModel organismBeneficial effectsCaloric RestrictionMammalseducation.field_of_studyGeneral Immunology and Microbiologyved/biologylcsh:RGeneral MedicineCaloric intakeDietCalorie intakeDrosophila melanogasterBiochemistryEnergy IntakeSignal Transduction
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Anti-Aging Effects of GDF11 on Skin

2020

International audience; Human skin is composed of three layers: the epidermis, the dermis, and the hypodermis. The epidermis has four major cell layers made up of keratinocytes in varying stages of progressive differentiation. Skin aging is a multi-factorial process that affects every phase of its biology and function. The expression profiles of inflammation-related genes analyzed in resident immune cells demonstrated that these cells have a strong ability to regenerate adult skin stem cells and to produce endogenous substances such as growth differentiation factor 11 (GDF11). GDF11 appears to be the key to progenitor proliferation and/or differentiation. The preservation of youthful phenot…

AgingHuman skinReviewSkin Aginglcsh:Chemistry0302 clinical medicineSkin Physiological Phenomenalcsh:QH301-705.5SpectroscopySkin0303 health sciencesintegumentary systemGeneral Medicine3. Good healthComputer Science ApplicationsCell biologyGrowth Differentiation Factorsmedicine.anatomical_structureBone Morphogenetic ProteinsIntercellular Signaling Peptides and ProteinsDisease SusceptibilityStem cellSignal TransductionBiologyCatalysisInorganic Chemistry03 medical and health sciencesImmune systemDermisgrowth factorsmedicineAnimalsHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyPhysical and Theoretical Chemistryskin agingMolecular Biology030304 developmental biologyWound HealingdiseaseEpidermis (botany)Regeneration (biology)Organic Chemistrylcsh:Biology (General)lcsh:QD1-999Gene Expression RegulationregenerationGDF11[SDV.MHEP.DERM]Life Sciences [q-bio]/Human health and pathology/Dermatology030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
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Hedgehog signaling and primary cilia are required for the formation of adult neural stem cells.

2008

Neural stem cells that continue to produce neurons are retained in the adult hippocampal dentate gyrus. The mechanisms by which embryonic neural progenitors expand and transform into postnatal neural stem cells, an essential process for the continual production of neurons throughout life, remain unknown. We found that radial astrocytes, the postnatal progenitors in the dentate gyrus, failed to develop after embryonic ablation of ciliary genes or Smoothened (Smo), an essential component for Sonic hedgehog (Shh) signaling. Postnatal dentate neurogenesis failed in these mutant mice, and the dentate gyrus became severely hypotrophic. In contrast, expression of a constitutively active Smo (SmoM2…

AgingKinesinsHippocampal formationHippocampusReceptors G-Protein-CoupledMiceMice Neurologic MutantsAnimalsHedgehog ProteinsCiliaSonic hedgehogCells CulturedCell ProliferationMice KnockoutbiologyGeneral NeuroscienceDentate gyrusStem CellsNeurogenesisCell DifferentiationSmoothened ReceptorNeural stem cellHedgehog signaling pathwaySmoothened Receptornervous systemAstrocytesDentate Gyrusbiology.proteinSmoothenedNeuroscienceSignal TransductionNature neuroscience
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YAP/TAZ activity in stromal cells prevents ageing by controlling cGAS-STING

2022

Ageing is intimately connected to the induction of cell senescence(1,2), but why this is so remains poorly understood. A key challenge isthe identification of pathways that normally suppress senescence, are lost during ageing and are functionally relevant to oppose ageing(3). Here we connected the structural and functional decline of ageing tissues to attenuated function of the master effectors of cellular mechanosignalling YAP and TAZ. YAP/TAZ activity declines during physiological ageing in stromal cells, and mimicking such decline through genetic inactivation of YAP/TAZ in these cells leads to accelerated ageing. Conversely, sustaining YAP function rejuvenates old cells and opposes the e…

AgingMechanotransductionActin-Related Protein 2; Cellular Senescence; Extracellular Matrix; Healthy Aging; Immunity Innate; Lamin Type B; Mechanotransduction Cellular; Nuclear Envelope; Signal Transduction; Aging; Membrane Proteins; Nucleotidyltransferases; Stromal Cells; Transcriptional Coactivator with PDZ-Binding Motif Proteins; YAP-Signaling ProteinsNuclear EnvelopeSettore MED/08 - Anatomia PatologicaYAP TAZ ageing C-GAS STINGMechanotransduction CellularArticleHealthy AgingInnateCellular SenescenceAdaptor Proteins Signal TransducingMultidisciplinaryLamin Type BImmunityMembrane ProteinsYAP-Signaling ProteinsPhosphoproteinsNucleotidyltransferasesImmunity InnateExtracellular MatrixTranscriptional Coactivator with PDZ-Binding Motif ProteinsActin-Related Protein 2CellularStromal CellsSignal Transduction
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Tif1γ regulates the TGF-β1 receptor and promotes physiological aging of hematopoietic stem cells.

2014

The hematopoietic system declines with age. Myeloid-biased differentiation and increased incidence of myeloid malignancies feature aging of hematopoietic stem cells (HSCs), but the mechanisms involved remain uncertain. Here, we report that 4-mo-old mice deleted for transcription intermediary factor 1γ (Tif1γ) in HSCs developed an accelerated aging phenotype. To reinforce this result, we also show that Tif1γ is down-regulated in HSCs during aging in 20-mo-old wild-type mice. We established that Tif1γ controls TGF-β1 receptor (Tgfbr1) turnover. Compared with young HSCs, Tif1γ(-/-) and old HSCs are more sensitive to TGF-β signaling. Importantly, we identified two populations of HSCs specifical…

AgingMyeloidReceptor Transforming Growth Factor-beta Type IReceptors Cell SurfaceCell SeparationBiologyProtein Serine-Threonine KinasesTransforming Growth Factor beta1MiceSignaling Lymphocytic Activation Molecule Family Member 1Antigens CDmedicineAnimalsMyeloid CellsRNA MessengerPolyubiquitinTranscription factorCellular SenescenceRegulation of gene expressionMultidisciplinaryUbiquitinationhemic and immune systemsBiological SciencesHematopoietic Stem CellsCell biologyHematopoiesisHaematopoiesismedicine.anatomical_structurePhysiological AgingPhenotypeGene Expression RegulationSignal transductionStem cellCell agingReceptors Transforming Growth Factor betaSignal TransductionTranscription FactorsProceedings of the National Academy of Sciences of the United States of America
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