Search results for "Simendan"

showing 10 items of 11 documents

Levosimendan prevents doxorubicin-induced cardiotoxicity in time- and dose-dependent manner: implications for inotropy.

2019

Abstract Aims Levosimendan (LEVO) a clinically-used inodilator, exerts multifaceted cardioprotective effects. Case-studies indicate protection against doxorubicin (DXR)-induced cardiotoxicity, but this effect remains obscure. We investigated the effect and mechanism of different regimens of levosimendan on sub-chronic and chronic doxorubicin cardiotoxicity. Methods and results Based on preliminary in vivo experiments, rats serving as a sub-chronic model of doxorubicin-cardiotoxicity and were divided into: Control (N/S-0.9%), DXR (18 mg/kg-cumulative), DXR+LEVO (LEVO, 24 μg/kg-cumulative), and DXR+LEVO (acute) (LEVO, 24 μg/kg-bolus) for 14 days. Protein kinase-B (Akt), endothelial nitric oxi…

0301 basic medicineMaleMice 129 StrainTime FactorsHeart DiseasesNitric Oxide Synthase Type IIIPhysiology030204 cardiovascular system & hematologyPharmacology03 medical and health sciences0302 clinical medicineEnosPhysiology (medical)medicineCyclic AMPCyclic GMP-Dependent Protein KinasesAnimalsDoxorubicinMyocytes CardiacCalcium SignalingRats WistarProtein kinase BCyclic GMPCells CulturedSimendanCardioprotectionMice KnockoutCardiotoxicityAntibiotics AntineoplasticbiologyDose-Response Relationship DrugChemistryCalcium-Binding ProteinsMammary Neoplasms ExperimentalCardiovascular AgentsLevosimendanbiology.organism_classificationCyclic AMP-Dependent Protein KinasesMyocardial ContractionCardiotoxicityPhospholambanMice Inbred C57BL030104 developmental biologyDoxorubicinMilrinoneFemaleCardiology and Cardiovascular MedicineProto-Oncogene Proteins c-aktmedicine.drugCardiovascular research
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Levosimendan protects human hepatocytes from ischemia-reperfusion injury.

2017

Background Ischemia-reperfusion injury (IRI) is a major challenge in liver transplantation. The mitochondrial pathway plays a pivotal role in hepatic IRI. Levosimendan, a calcium channel sensitizer, was shown to attenuate apoptosis after IRI in animal livers. The aim of this study was to investigate the effect of levosimendan on apoptosis in human hepatocytes. Methods Primary human hepatocytes were either exposed to hypoxia or cultured under normoxic conditions. After the hypoxic phase, reoxygenation was implemented and cells were treated with different concentrations of levosimendan (10ng/ml, 100ng/ml, 1000ng/ml). The overall metabolic activity of the cells was measured using 3-(4,5-dimeth…

0301 basic medicineNecrosisCritical Care and Emergency Medicinelcsh:MedicineApoptosis030204 cardiovascular system & hematologyBiochemistry0302 clinical medicineAnimal CellsMedicine and Health SciencesEnzyme assaysColorimetric assayslcsh:ScienceBioassays and physiological analysisCells CulturedEnergy-Producing Organellesbcl-2-Associated X ProteinMultidisciplinaryMTT assaybiologyCell DeathMitochondriaPyridazinesLiverCell ProcessesReperfusion Injurymedicine.symptomCellular TypesAnatomyCellular Structures and Organellesmedicine.drugResearch Articlemedicine.medical_specialtyCell PhysiologyIschemiaCardiologySurgical and Invasive Medical ProceduresBioenergetics03 medical and health sciencesDigestive System ProceduresBcl-2-associated X proteinInternal medicinemedicineHumansMTT assayddc:610SimendanHeart FailureTransplantationbusiness.industrylcsh:RHydrazonesBiology and Life SciencesLevosimendanCell BiologyOrgan TransplantationHypoxia (medical)medicine.diseaseLiver TransplantationCell MetabolismResearch and analysis methods030104 developmental biologyEndocrinologyApoptosisReperfusionBiochemical analysisbiology.proteinHepatocyteslcsh:QbusinessReperfusion injuryPloS one
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Repetitive use of LEvosimendan in Ambulatory Heart Failure patients (LEIA-HF) - The rationale and study design

2022

Abstract Purpose Clinical practice forces the necessity to conduct a clinical trial concerning the group of outpatients with chronically advanced heart failure in III or IV NYHA functional class, frequently requiring hospitalizations due to HF exacerbation, and often left without any additional therapeutic option. The current trial aims to determine the efficacy and safety of repeated levosimendan infusions in the group of severe outpatients with reduced ejection fraction (HFrEF). Material and methods LEIA-HF (LEvosimendan In Ambulatory Heart Failure Patients) is a multicentre, randomized, double-blind, placebo-controlled, phase 4 clinical trial to determine whether the repetitive use of le…

Heart Failuremedicine.medical_specialtyCardiotonic AgentsEjection fractionExacerbationbusiness.industryStroke VolumeGeneral MedicineLevosimendanPlacebomedicine.diseaseClinical trialTreatment OutcomeDouble-Blind MethodHeart failureEmergency medicineAmbulatoryClinical endpointmedicineHumansbusinessSimendanmedicine.drugAdvances in Medical Sciences
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Old and New Drugs for Treatment of Advanced Heart Failure.

2020

Background: Advanced heart failure (HF) is a progressive disease with high mortality and limited medical therapeutic options. Long-term mechanical circulatory support and heart transplantation remain goldstandard treatments for these patients; however, access to these therapies is limited by the advanced age and multiple comorbidities of affected patients, as well as by the limited number of organs available. Methods: Traditional and new drugs available for the treatment of advanced HF have been researched. Results: To date, the cornerstone for the treatment of patients with advanced HF remains water restriction, intravenous loop diuretic therapy and inotropic support. However, many patien…

Inotropemedicine.medical_specialtymedicine.drug_classmedicine.medical_treatment030204 cardiovascular system & hematology03 medical and health sciences0302 clinical medicineFurosemideDrug DiscoverymedicineHumansDecompensation030212 general & internal medicine: Dobutamine dopamine nitroglycerine sodium nitroprusside vaptans ivabradineIntensive care medicineDiureticsSimendanPharmacologyHeart transplantationHeart Failurebusiness.industryLevosimendanLoop diureticmedicine.diseasePrognosisOmecamtiv mecarbilIstaroximeHeart failurebusinessmedicine.drugCurrent pharmaceutical design
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Effects of levosimendan on hemodynamics, local cerebral blood flow, neuronal injury, and neuroinflammation after asphyctic cardiac arrest in rats.

2014

Despite advances in cardiac arrest treatment, high mortality and morbidity rates after successful cardiopulmonary resuscitation are still a major clinical relevant problem. The post cardiac arrest syndrome subsumes myocardial dysfunction, impaired microcirculation, systemic inflammatory response, and neurological impairment. The calcium-sensitizer levosimendan was able to improve myocardial function and initial resuscitation success after experimental cardiac arrest/cardiopulmonary resuscitation. We hypothesized that levosimendan exerts beneficial effects on cerebral blood flow, neuronal injury, neurological outcome, and inflammation 24 hours after experimental cardiac arrest/cardiopulmonar…

Malemedicine.medical_specialtyCardiotonic AgentsHemodynamicsGene ExpressionEnzyme-Linked Immunosorbent AssayCritical Care and Intensive Care MedicineHippocampusRats Sprague-DawleyInternal medicineMedicineAnimalsNeuroinflammationSimendanCerebral CortexNeuronsAnalysis of Variancebusiness.industryInterleukin-6Reverse Transcriptase Polymerase Chain ReactionHemodynamicsHydrazonesLevosimendanCardiopulmonary ResuscitationHeart ArrestRatsSprague dawleyPyridazinesCerebral blood flowAnesthesiaCerebrovascular CirculationCardiologybusinessmedicine.drugCritical care medicine
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Milrinone is preferred to levosimendan for mesenteric perfusion in hypoxia-reoxygenated newborn piglets treated with dopamine

2012

INTRODUCTION There is little information regarding the comparative hemodynamic effects of adding milrinone or levosimendan to dopamine infusion in hypoxia-reoxygenated (H-R) newborns. RESULTS Severely hypoxic piglets had cardiogenic shock with depressed cardiac index (CI) and mean arterial pressure (MAP). The hemodynamics deteriorated gradually after initial recovery upon reoxygenation. Heart rate and CI improved with milrinone (D+M) and levosimendan (D+L) administration (P < 0.05 vs. control). Both regimens improved carotid arterial flow and carotid vascular resistance; D+M additionally improved superior mesentric arterial flow (all P < 0.05 vs. control). No effect was found on renal arter…

Mean arterial pressureCardiotonic AgentsSwineDopamineVasodilator AgentsHemodynamicsMyocardial ReperfusionMyocardial Reperfusion Injury030204 cardiovascular system & hematologyArticle03 medical and health sciences0302 clinical medicine030225 pediatricsMedicineAnimalsHypoxiaSimendanbusiness.industryCardiogenic shockHemodynamicsHydrazonesLevosimendanHypoxia (medical)medicine.disease3. Good healthMesenteric ArteriesPyridazinesOxidative Stressmedicine.anatomical_structureAnimals NewbornAnesthesiaPediatrics Perinatology and Child HealthModels AnimalVascular resistanceMilrinoneDrug Therapy Combinationmedicine.symptombusinessPerfusionmedicine.drugMilrinonePediatric research
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P3488Mechanistic insight on the cardioprotective effect of levosimendan against doxorubicin induced cardiomyopathy: Pivotal role of PKA signaling

2019

Abstract Background Levosimendan (LEVO) an inodilator indicated for the treatment of heart failure exerts multifaceted cardioprotective effects. Case-studies indicate protection against doxorubicin (DXR)-induced cardiotoxicity, but this effect remains elusive. We have previously shown that LEVO exerts cardioprotection against DXR-induced cardiomyopathy in a rat in vivo model, in a PKA/PKG-dependent manner. Purpose We sought to elucidate the mechanism of LEVO's induced cardioprotection and clarify the contribution of PKG and PKA pathways converging onto phospholamban (PLN). Methods As previously observed, LEVO at a dose of 24μg/kg protects against DXR cardiotoxicity, with protein kinase B (A…

business.industryPka signalingMedicineLevosimendanPharmacologyCardiology and Cardiovascular MedicinebusinessDoxorubicin induced cardiomyopathymedicine.drugEuropean Heart Journal
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Lewosimendan - cenny gracz w leczeniu ciężkiej prawokomorowej niewydolności krążenia

2019

66-letni otyły mężczyzna, obciążony cukrzycą typu 2 i nadciśnieniem tętniczym, przewlekłą chorobą nerek w okresie 4, utrwalonym migotaniem przedsionków z bradykardią, przyjęty został na oddział kardiologii z powodu objawów ciężkiej prawokomorowej niewydolności krążenia. W leczeniu zastosowano standardową terapię, w tym złożone leczenie moczopędne. Z uwagi na jej nieskuteczność rozpoznano oporność na diuretyki i włączono do leczenia lewosimendan, uzyskując obfitą diurezę, znaczną redukcję wagi ciała i istotną poprawę wydolności krążenia, co pozwoliło na dalszą diagnostykę i adekwatne, przyczynowe leczenie zabiegowe. W artykule zarysowano ponadto aktualny stan wiedzy na temat miejsca lewosime…

levosimendanresistance to diureticsoporność na diuretykiright-sided heart failureprawokomorowa niewydolność krążenialewosimendanWiadomości Lekarskie
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Levosimendan in the treatment of patients with acute cardiac conditions: an expert opinion of the Association of Intensive Cardiac Care of the Polish…

2020

Levosimendan is a new inodilator which involves 3 main mechanisms: increases the calcium sensitivity of cardiomyocytes, acts as a vasodilator due to the opening of potassium channels, and has a cardioprotective effect. Levosimendan is mainly used in the treatment of acute decompensated heart failure (class IIb recommendation according to the European Society of Cardiology guidelines). However, numerous clinical trials indicate the validity of repeated infusions of levosimendan in patients with stable heart failure as a bridge therapy to heart transplantation, and in patients with accompanying right ventricular heart failure and pulmonary hypertension. Due to the complex mechanism of action,…

medicine.medical_specialtyCardiotonic AgentsAcute decompensated heart failureinotropic agentsacute heart failuremedicine.medical_treatment030204 cardiovascular system & hematologylevosimendan03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansinodilatorAdverse effectExpert TestimonySimendanHeart FailureHeart transplantationbusiness.industryHydrazonesLevosimendanmedicine.diseasePulmonary hypertensionCardiac surgeryPyridazinesClinical trialHeart failurechronic advanced systolic heart failureCardiologyPolandCardiology and Cardiovascular Medicinebusinessmedicine.drugKardiologia Polska
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Predictors of diuresis response to levosimendan administration in patients with acute heart failure

2021

Abstract Funding Acknowledgements Type of funding sources: None. Levosimendan, a calcium sensitizer and potassium channel-opener, is appreciated  for its effects on systemic and pulmonary hemodynamic and for the relief of symptoms in acute heart failure (AHF). Positive effects of levosimendan on renal function have been also described. The aim of the present analysis was to assess the predictors of the diuresis response to levosimendan administration in high risk acute heart failure patients. Methods We analysed 34 consecutive patients admitted with high risk AHF to one centre and treated in intensive cardiac care unit. Levosimendan was administered on top of other treatment as a 24-hour in…

medicine.medical_specialtyEjection fractionLungbusiness.industryFurosemideDiuresisRenal functionHemodynamicsGeneral MedicineLevosimendan16.4.1 - PharmacotherapyCritical Care and Intensive Care Medicinemedicine.diseasemedicine.anatomical_structureInternal medicineHeart failuremedicineCardiologyCardiology and Cardiovascular Medicinebusinessmedicine.drug
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