Search results for "Smooth Muscle"

showing 10 items of 156 documents

Effects of Dehydroepiandrosterone Sulfate on Cellular Calcium Responsiveness and Vascular Contractility

1995

Abstract Dehydroepiandrosterone sulfate (DHEAS) is an endogenous steroid having a wide variety of biological effects, but its physiological role remains undefined. Since an age-related decline of DHEAS corresponds to the progressive onset of atherosclerosis, cardiovascular diseases, and overall mortality, we investigated a possible protective role of DHEAS in vascular disease by studying the effects of this hormone (10 −7 to 10 −5 mol/L) on cytosolic free calcium and contractility in different in vitro vascular tissue preparations. DHEAS produced a significant, dose-dependent relaxation of isolated helical strips of rat tail artery precontracted with KCl (60 mmol/L) (89.7±18.7%, P <.01)…

MaleVasopressinmedicine.medical_specialtyVascular smooth muscleArgininechemistry.chemical_elementIn Vitro TechniquesBiologyCalciumMuscle Smooth VascularCalcium in biologyRats Sprague-DawleyContractilitychemistry.chemical_compoundCytosolDehydroepiandrosterone sulfateInternal medicineInternal MedicinemedicineAnimalsCells CulturedVascular tissueAnalysis of VarianceDehydroepiandrosterone SulfateDehydroepiandrosteroneRatsEndocrinologychemistryCalciumMuscle ContractionHypertension
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Interplay between PACAP and NO in mouse ileum

2003

Abstract We investigated the possibility that pituitary adenylate cyclase activating peptide (PACAP) has a role in the control of contractility in the mouse ileum. PACAP-(1-27) produced tetrodotoxin (TTX)-insensitive, concentration-dependent reduction of the amplitude of the spontaneous contractions of longitudinal muscle up to their complete disappearance. This effect was inhibited by PACAP-(6-38), PACAP receptor antagonist, and by apamin, blocker of small-conductance Ca2+-activated K+-channels. Nω-nitro- l -arginine methyl ester (L-NAME), nitric oxide (NO) synthase inhibitor, reduced the PACAP-inhibitory response, and the joint application of apamin plus L-NAME produced additive effects. …

Maleendocrine systemmedicine.medical_specialtyMuscle RelaxationMouse ileumStimulationIn Vitro TechniquesApaminSettore BIO/09 - FisiologiaContractilityMiceCellular and Molecular Neurosciencechemistry.chemical_compoundSmooth muscleIleumInternal medicinemedicineAnimalsMyocyteNitric Oxide DonorsPharmacologyDose-Response Relationship DrugNeuropeptidesNitric oxideSmooth muscle contractionMice Inbred C57BLPituitary adenylate cyclase-activating peptideEndocrinologyApaminchemistryTetrodotoxinPituitary Adenylate Cyclase-Activating PolypeptideSodium nitroprussidePACAP (pituitary adenylate cyclase activating peptide)hormones hormone substitutes and hormone antagonistsmedicine.drugNeuropharmacology
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Gastric relaxation induced by apigenin and quercetin: Analysis of the mechanism of action

2009

Abstract Aims Recently, flavonoids have been shown to cause murine gastric relaxation. In the present study we examined the mechanism of action underlying gastric relaxation induced by apigenin and quercetin in isolated mouse stomach. Main methods The mechanical activity from the whole stomach was detected as changes in the endoluminal pressure and the response to increasing concentrations of both flavonoids were tested before and after different pharmacological treatments. Key findings Apigenin and quercetin-induced a concentration-dependent gastric relaxation, apigenin being more potent than quercetin. The responses were unaffected by 2′5′dideoxyadenosine, an inhibitor of adenylate cyclas…

Malemedicine.medical_specialtyCarbacholNifedipineMuscle Relaxationchemistry.chemical_elementCalcium antagonistIn Vitro TechniquesMuscarinic AgonistsCalciumPharmacologySettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular BiologyPotassium ChlorideMicechemistry.chemical_compoundSmooth muscleInternal medicineCyclic AMPmedicineAnimalsheterocyclic compoundsApigeninGeneral Pharmacology Toxicology and PharmaceuticsCyclic GMPCyclic nucleotide phosphodiesteraseChemistryRyanodine receptorStomachMuscle SmoothGeneral MedicineCalcium Channel BlockersMice Inbred C57BLEndocrinologyMechanism of actionGastric toneApigeninFlavonoidCalciumCarbacholQuercetinmedicine.symptomQuercetinIntracellularMuscle Contractionmedicine.drugLife Sciences
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Spontaneous electromechanical activity in the rat duodenumin vitro

1990

Isolated rat duodenum shows spontaneous mechanical and electrical activities. Mechanical activity consists in changes both in endoluminal pressure and in isometric tension. Electrical activity is characterized by slow waves with superimposed bursts. This spontaneous activity is tetrodotoxin (TTX) resistant and therefore it is myogenic in origin. Indeed, TTX pretreatment, even in the presence of atropine and guanethidine, caused an increase in amplitude and in frequency of the electrical and mechanical activities. This finding indicates the presence of tonically active inhibitory intramural non adrenergic, non cholinergic (NANC) nerves. Duodenal longitudinal strips showed a spontaneous mecha…

Malemedicine.medical_specialtyDuodenumPhysiologyAdrenergicIsometric exerciseIn Vitro TechniquesInhibitory postsynaptic potentialBiochemistrychemistry.chemical_compoundInternal medicinemedicineAnimalsGuanethidinemusculoskeletal neural and ocular physiologySmooth muscle layerMuscle SmoothBiomechanical PhenomenaRatsElectrophysiologyElectrophysiologyEndocrinologychemistryTetrodotoxinCholinergicGastrointestinal Motilitymedicine.drugArchives Internationales de Physiologie et de Biochimie
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β-Adrenoceptors differentially regulate vascular tone and angiogenesis of rat aorta via ERK1/2 and p38

2014

β-Adrenoceptors (β-ARs) modulate ERK1/2 and p38 in different cells, but little is known about the contribution of these signaling pathways to the function of β-ARs in vascular tissue. Immunoblotting analysis of rat aortic rings, primary endothelial (ECs) and smooth muscle cells (SMCs) isolated from aorta showed that β-AR stimulation with isoprenaline activated p38 in aortic rings and in both cultured cell types, whereas it had a dual effect on ERK1/2 phosphorylation, decreasing it in ECs while increasing it in SMCs. These effects were reversed by propranolol, which by itself increased p-ERK1/2 in ECs. Isoprenaline β-AR mediated vasodilation of aortic rings was potentiated by the ERK1/2 inhi…

Malemedicine.medical_specialtyEndotheliumPhysiologyAngiogenesisVasodilator AgentsAdrenergic beta-AntagonistsMyocytes Smooth MuscleNeovascularization PhysiologicAorta ThoracicStimulationVasodilationFibroblast growth factorp38 Mitogen-Activated Protein KinasesMuscle Smooth VascularInternal medicineIsoprenalinemedicine.arteryReceptors Adrenergic betamedicineAnimalsHumansRats WistarMitogen-Activated Protein Kinase 1PharmacologyMatrigelAortaMitogen-Activated Protein Kinase 3business.industryAdrenergic beta-AgonistsPropranololRatsVasodilationHEK293 Cellsmedicine.anatomical_structureEndocrinologycardiovascular systemMolecular MedicineEndothelium Vascularbusinessmedicine.drugVascular Pharmacology
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Muscarinic inhibition of acetylcholine release from a novel in vitro preparation of the guinea-pig trachea

1990

An isolated preparation of the guinea-pig trachea is described which allows the simultaneous measurement of acetylcholine release and smooth muscle contraction. Incubation of the epithelium-free preparation with [3H]choline resulted in the formation of [3H]acetylcholine. Electrical stimulation caused the release of [3H]acetylcholine and a contractile response. Tetrodotoxin and omission of calcium from the medium abolished both the evoked release and contractions. The muscarinic agonists oxotremorine, carbachol and pilocarpine concentration-dependently inhibited the electrically evoked acetylcholine release and contracted the tracheal smooth muscle. Pre- and postsynaptic EC50 values for a gi…

Malemedicine.medical_specialtyGuinea Pigschemistry.chemical_elementStimulationTetrodotoxinIn Vitro TechniquesCalciumEpitheliumCholineGuinea pigchemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineAnimalsPharmacologyOxotremorineMuscle SmoothGeneral MedicineSmooth muscle contractionReceptors MuscarinicAcetylcholineElectric StimulationTracheaEndocrinologychemistryTetrodotoxinBiophysicsCalciummedicine.symptomAcetylcholinemedicine.drugMuscle contractionNaunyn-Schmiedeberg's Archives of Pharmacology
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Inhibitory effects of indicaxanthin on mouse ileal contractility: analysis of the mechanism of action.

2011

Recently, we have showed that indicaxanthin, the yellow betalain pigment abundant in the fruit of Opuntia ficus indica, has remarkable spasmolytic effects on the intestinal contractility in vitro. Thus, the purpose of the present study was to investigate the mechanism of action underlying the observed response. We used organ bath technique to record the mechanical activity of the mouse ileum longitudinal muscle and ELISA to measure the levels of cAMP. Indicaxanthin induced inhibitory effects on spontaneous mechanical activity, which were unaffected by indomethacin, a non-selective inhibitor of cycloxygenase; 1H-[1,2,4]oxadiazolo[4,3-a]quinoxalin-1-one, a selective inhibitor of nitric oxide-…

Malemedicine.medical_specialtyIBMXPyridinesIndicaxanthinBiologyIn Vitro TechniquesContractilitySettore BIO/09 - FisiologiaAdenylyl cyclaseContractilitychemistry.chemical_compoundMiceSmooth muscleCactus pear fruitIleumSettore BIO/10 - BiochimicaInternal medicinemedicineCyclic AMPAnimalsEnzyme InhibitorsPharmacologyForskolinPhosphodiesteraseMuscle SmoothBetaxanthinsBiomechanical PhenomenaMice Inbred C57BLEndocrinologychemistryPhosphodiesterasesCarbacholZaprinastSoluble guanylyl cyclaseIndicaxanthinMuscle ContractionSignal TransductionEuropean journal of pharmacology
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Muscarinic receptors, leukotriene B4 production and neutrophilic inflammation in COPD patients.

2005

Background:  Acetylcholine (ACh) plays an important role in smooth muscle contraction and in the development of airway narrowing; preliminary evidences led us to hypothesize that ACh might also play a role in the development of airways inflammation in chronic obstructive pulmonary disease (COPD). Methods:  We evaluated the concentrations of leukotriene B4 (LTB4) in induced sputum, and the expression of Ach M1, M2, and M3 receptors in sputum cells (SC) obtained from 16 patients with COPD, 11 smokers, and 14 control subjects. The SC were also treated with ACh and the production of LTB4 assessed in the presence or absence of a muscarinic antagonist (oxitropium). In blood monocytes, we evaluate…

Malemedicine.medical_specialtyLeukotriene B4NeutrophilsImmunologyProtozoan ProteinsLeukotriene B4chemistry.chemical_compoundPulmonary Disease Chronic ObstructiveInternal medicineMuscarinic acetylcholine receptorImmunology and AllergyMedicineHumansReceptorCells CulturedAgedFlavonoidsCOPDMitogen-Activated Protein Kinase 3business.industryCalcium-Binding ProteinsSputumMuscarinic acetylcholine receptor M3Muscarinic antagonistSmooth muscle contractionMiddle Agedmedicine.diseaseImmunohistochemistryReceptors MuscarinicAcetylcholinerespiratory tract diseasesChemotaxis LeukocyteEndocrinologychemistryLeukocytes MononuclearFemalebusinessAcetylcholinemedicine.drugAllergy
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Voltage-Dependent Effects of Barnidipine in Rat Vascular Smooth Muscle

2003

The effects of the dihydropyridine nifedipine and its more lipophilic congener, barnidipine, were investigated in smooth muscle preparations from the rat in resting and depolarizing conditions. Both drugs relaxed precontracted aortic rings more potently in depolarizing conditions, barnidipine being more potent than nifedipine. Currents through Ca 2+ channels in rat vascular smooth muscle cells (A7r5) and in isolated rat cardiomyocytes were reduced more potently by both drugs at a holding potential of-40 mV than at -80 mV. However, barnidipine and nifedipine were more effective in reducing the current in A7r5 cells than in cardiomyocytes. The IC 50 obtained in aortic rings and in A7r5 cells …

Malemedicine.medical_specialtyPatch-Clamp TechniquesVascular smooth muscleBarnidipineNifedipinechemistry.chemical_elementPharmacologyCalciumMuscle Smooth VascularRats Sprague-DawleyNifedipineInternal medicinemedicineAnimalsMyocyteCells CulturedPharmacologyChemistryDihydropyridineDepolarizationCalcium Channel BlockersRatsEndocrinologyMechanism of actioncardiovascular systemFemalemedicine.symptomCardiology and Cardiovascular Medicinemedicine.drugJournal of Cardiovascular Pharmacology
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Bradykinin Contracts Rat Urinary Bladder Largely Independently of Phospholipase C

2014

Several receptor systems in the bladder causing detrusor smooth muscle contraction stimulate phospholipase C (PLC). PLC inhibition abolishes bladder contraction via P2Y6 but not that via M3 muscarinic receptors, indicating a receptor-dependent role of PLC. Therefore, we explored the role of PLC in rat bladder contraction by bradykinin. The PLC inhibitor U 73,122 [1-(6-[([17β]-3-methoxyestra-1,3,5[10]-trien-17-yl)-amino]hexyl)-1H-pyrrole-2,5-dione] did not affect the bradykinin response to a significantly greater degree than its inactive analog U 73,343 [10 μM each; 1-(6-[-([17β]-3-methoxyestra-1,3,5[10]-trien-17-yl)-amino]hexyl)-2,5-pyrrolidinedione], whereas the phospholipase D inhibitor b…

Malemedicine.medical_specialtyUrinary BladderMedizinBradykininPharmacologyBradykininCyclooxygenase pathwaychemistry.chemical_compoundOrgan Culture TechniquesInternal medicinemedicineAnimalsCalphostinRats WistarBradykinin receptorPharmacologyDose-Response Relationship DrugPhospholipase CMuscle SmoothSmooth muscle contractionRatsChelerythrineEndocrinologychemistryRho kinase inhibitorType C PhospholipasesMolecular MedicineMuscle ContractionJournal of Pharmacology and Experimental Therapeutics
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