Search results for "Smooth"

showing 10 items of 710 documents

Results of the smooth-muscle fold-over double-plasty (SMFD)

1986

Since 1980, when we developed substitute for the internal anal sphicter, the smooth-muscle fold-over double-plasty (SMFD-plasty), we have operated on seven children using this method, usually in combination with the pull-through procedure, and on five children after one or more previous operations. Since 1983, 11 cases with previous operations have undergone the modified “door-wing” SMFD-plasty. The results are excellent if the operation is used in primary combination with the pull-through procedure or if the “door-wing” modification is combined with reconstruction of the pelvic floor. It is also necessary that all cases undergo a biofeedback conditioning program.

medicine.medical_specialtyPelvic floorAnorectal diseaseFold (higher-order function)business.industryGeneral MedicineAnusSurgerymedicine.anatomical_structureSmooth musclePediatrics Perinatology and Child HealthBiofeedback conditioningmedicineSurgeryAnal sphincterbusinessPediatric Surgery International
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Pentobarbital-sensitive EDHF comediates ACh-induced arteriolar dilation in the hamster microcirculation

1999

It is unclear to what extent the endothelium-derived hyperpolarizing factor (EDHF) contributes to the control of microcirculatory blood flow in vivo. We analyzed, by intravital microscopy in hamster muscles, the potential role of EDHF along the vascular tree under stimulated (ACh) or basal conditions. Experiments were performed in conscious as well as anesthetized (pentobarbital, urethan) animals. Additionally, cellular effects of the potential EDHF were studied in isolated small arteries. In pentobarbital-anesthetized animals, treatment with N ω-nitro-l-arginine (l-NNA; 30 μmol/l) and indomethacin (3 μmol/l) reduced the dilation in response to 10 μmol/l ACh from 60 ± 6 to 20 ± 4%. This ni…

medicine.medical_specialtyPentobarbitalEndothelium-derived hyperpolarizing factorPotassium ChannelsCharybdotoxinPhysiologyVasodilator AgentsIndomethacinHamsterVasodilationNitroarginineMuscle Smooth VascularMicrocirculationGlibenclamideBiological FactorsCytochrome P-450 Enzyme SystemArterioleCricetinaePhysiology (medical)Internal medicinemedicine.arterymedicineAnimalsCyclooxygenase InhibitorsMuscle SkeletalPentobarbitalSkinMesocricetusChemistryMicrocirculationPenicillamineAcetylcholineArteriolesEndocrinologyAnesthesiaFatty Acids UnsaturatedPotassiumEndothelium VascularCardiology and Cardiovascular MedicineIntravital microscopyAdjuvants Anesthesiamedicine.drugAmerican Journal of Physiology-Heart and Circulatory Physiology
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Selective inhibition of calcium entry induced by benzylisoquinolines in rat smooth muscle.

1992

Abstract The mechanism of relaxant activity of six benzylisoquinolines was examined in order to determine the minimal structural requirements that enable these compounds to have either a non-specific action like papaverine or an inhibitory activity on calcium entry via potential-operated channels. All the alkaloids tested totally or partially relaxed KCl-depolarized rat uterus and inhibited oxytocin-induced rhythmic contractions. Only glaucine and laudanosine inhibited K+-induced uterine contractions more than oxytocin-induced uterine contractions. In Ca+-free medium, sustained contractions induced by oxytocin or vanadate were relaxed by the alkaloids tested except for glaucine and laudanos…

medicine.medical_specialtyPharmaceutical Sciencechemistry.chemical_elementPharmacologyCalciumIn Vitro TechniquesOxytocinCalcium in biologyUterine contractionLaudanosinechemistry.chemical_compoundUterine ContractionAlkaloidsInternal medicinePapaverinemedicineAnimalsBenzylisoquinolinesPharmacologyPapaverineEstradiolChemistryMuscle SmoothRats Inbred StrainsCalcium Channel BlockersIsoquinolinesGlaucineRatsEndocrinologyPotassiumCalciumFemalemedicine.symptomMuscle contractionmedicine.drugMuscle ContractionThe Journal of pharmacy and pharmacology
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Effects of dopamine in isolated rat colon strips.

2005

The aim of the present work is to investigate the effects of dopamine on isolated rat colon strips, and whether dopamine receptors are involved in these effects. Experiments on spontaneous motility and under potassium contraction were performed with dopamine and isoprenaline, both in the absence and presence of antagonists (distal colon strips, isotonic recording, Tyrode solution, 31 °C, 1 g of resting tension). At higher concentration (10–4mol/L), dopamine abolished spontaneous motility of the rat colon and this effect was not modified by antagonists. In isolated rat colon strips that were depolarized with potassium, dopamine produced concentration-dependent relaxation, without significant…

medicine.medical_specialtyPhysiologyColonDopamineBiologyIn Vitro TechniquesReceptors Dopaminechemistry.chemical_compoundDopaminePhysiology (medical)IsoprenalineInternal medicinemedicinePrazosinAnimalsRats WistarNeurotransmitterPharmacologyDose-Response Relationship DrugMuscle SmoothGeneral MedicineYohimbineRatsEndocrinologychemistryDopamine receptorDopamine AgonistsCatecholamineDopamine AntagonistsSulpirideGastrointestinal Motilitymedicine.drugMuscle ContractionCanadian journal of physiology and pharmacology
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Substance P inactivation by transglutaminase in vitro.

1992

Gamma(glutamyl5)spermine derivative of substance P (Spm-SP) was synthesized in vitro in the presence of purified guinea pig liver transglutaminase and Ca2+. The spermine adduct of the neuropeptide was purified by HPLC on a reversed-phase column and characterized by fast atom bombardment mass spectrometry. The biological activities of Spm-SP were tested by assaying, in comparison with substance P, its ability to induce both the contractions of smooth muscle in vitro and the edema formation in vivo. Spm-SP was shown not to elicit contractile responses in the isolated rat stomach strip and duodenum and not to antagonize the spasmogenic effect evoked by the native neuropeptide. Furthermore, Spm…

medicine.medical_specialtyPhysiologyGuinea PigsMolecular Sequence DataHistamine AntagonistsNeuropeptideSpermineSubstance PSubstance PPharmacologyBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundEndocrinologyIn vivoInternal medicinemedicineAnimalsEdemaAmino Acid SequenceReceptorPeptide modificationTransglutaminasesChemistryExtremitiesMuscle SmoothBiological activityIn vitroEndocrinologyLiverHistamineMuscle Contraction
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Influence of Vascular Load on Plasma Endothelin-1, Cytokines and Catecholamine Levels in Essential Hypertensives

1998

In vitro studies demonstrated a relationship between ET-1 and basic Fibroblast Growth Factor (bFGF), and of bFGF with Platelet Derived Growth Factor (PDGF). The present study was carried out to investigate in vivo the behaviour after vascular stress of circulating ET-1, bFGF and PDGF, and catecholamines, and their relationship. In 12 healthy normotensives (NTs) and 15 essential hypertensives (Ehs) venous blood samples to determine circulating ET-1, bFGF and PDGF, and catecholamine (EPI and NE) levels were drawn before and at the third minute of a handgrip test. Blood pressures (BP) and heart rate were automatically recorded before starting, and at 1, 2, and 3 minutes during the test. The NT…

medicine.medical_specialtyPlatelet-derived growth factorBasic fibroblast growth factorMuscle Smooth Vascularchemistry.chemical_compoundCatecholaminesStress PhysiologicalInternal medicineBlood plasmaHeart rateInternal MedicinemedicineHumansPlatelet-Derived Growth FactorEndothelin-1Hand Strengthbiologybusiness.industryHypertrophyGeneral MedicineVenous bloodEndothelin 1EndocrinologychemistryVasoconstrictionHypertensioncardiovascular systembiology.proteinCatecholamineFibroblast Growth Factor 2Endothelium VascularCardiology and Cardiovascular MedicinebusinessPlatelet-derived growth factor receptorMuscle Contractionmedicine.drugBlood Pressure
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Endocannabinoid anandamide mediates hypoxic pulmonary vasoconstriction

2013

Endocannabinoids are important regulators of organ homeostasis. Although their role in systemic vasculature has been extensively studied, their impact on pulmonary vessels remains less clear. Herein, we show that the endocannabinoid anandamide (AEA) is a key mediator of hypoxic pulmonary vasoconstriction (HPV) via fatty acid amide hydrolase (FAAH)-dependent metabolites. This is underscored by the prominent vasoconstrictive effect of AEA on pulmonary arteries and strongly reduced HPV in FAAH(-/-) mice and wild-type mice upon pharmacological treatment with FAAH inhibitor URB597. In addition, mass spectrometry measurements revealed a clear increase of AEA and the FAAH-dependent metabolite arac…

medicine.medical_specialtyPolyunsaturated Alkamidesmedicine.medical_treatmentHypertension PulmonaryBlotting WesternMyocytes Smooth MuscleArachidonic AcidsBiologyAmidohydrolaseschemistry.chemical_compoundMiceFatty acid amide hydrolaseInternal medicineHypoxic pulmonary vasoconstrictionmedicineAnimalsHypoxiaLungDNA PrimersMice KnockoutAnalysis of VarianceMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionAnandamideHypoxia (medical)URB597Biological Sciencesmedicine.diseaseEndocannabinoid systemPulmonary hypertensionImmunohistochemistryEndocrinologynervous systemchemistryVasoconstrictionBenzamideslipids (amino acids peptides and proteins)CannabinoidCarbamatesmedicine.symptompsychological phenomena and processesChromatography LiquidEndocannabinoidsSignal Transduction
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Perivascular nerve fiber α-synuclein regulates contractility of mouse aorta: A link to autonomic dysfunction in Parkinson's disease

2010

Parkinson's disease and other neurodegenerative disorders associated to changes in alpha-synuclein often result in autonomic dysfunction, most of the time accompanied by abundant expression of this synaptic protein in peripheral autonomic neurons. Given that expression of alpha-synuclein in vascular elements has been previously reported, the present study was undertaken to determine whether alpha-synuclein directly participates in the regulation of vascular responsiveness. We detected by immunohistochemistry perivascular nerve fibers containing alpha-synuclein in the aorta of mice while aortic endothelial cells and muscular fibers themselves did not exhibit detectable levels of this protein…

medicine.medical_specialtyPresynaptic TerminalsAorta ThoracicVasodilationBiologyMuscle Smooth VascularMiceCellular and Molecular Neurosciencechemistry.chemical_compoundSympathetic Fibers PostganglionicDopaminemedicine.arteryInternal medicinemedicineAnimalsNeurotransmitterMice KnockoutAortaEndothelial CellsParkinson DiseaseCell Biologynervous system diseasesMice Inbred C57BLEndocrinologyAutonomic Nervous System Diseasesnervous systemchemistryVasoconstrictionKnockout mousealpha-SynucleinCatecholaminemedicine.symptomVasoconstrictionAcetylcholineMuscle Contractionmedicine.drugNeurochemistry International
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Can you blame cold feet on Epac (and Rap1A)? Focus on “Cyclic AMP-Rap1A signaling activates RhoA to induce α2C-adrenoceptor translocation to the cell…

2012

Intracellular signaling by the second messenger cyclic AMP (cAMP) activates the Ras-related small GTPase Rap1 through the guanine exchange factor Epac. This activation leads to effector protein interactions, activation, and biological responses in the vasculature, including vasorelaxation. In vascular smooth muscle cells derived from human dermal arterioles (microVSM), Rap1 selectively regulates expression of G protein-coupled α2C-adrenoceptors (α2C-ARs) through JNK-c-jun nuclear signaling. The α2C-ARs are generally retained in the trans-Golgi compartment and mobilize to the cell surface and elicit vasoconstriction in response to cellular stress. The present study used human microVSM to exa…

medicine.medical_specialtyRHOAPhysiologyMyocytes Smooth MuscleCellChromosomal translocationSmooth muscleReceptors Adrenergic alpha-2Internal medicineCyclic AMPmedicineAnimalsHumansReceptorbiologyrap1 GTP-Binding ProteinsArticlesCell Biologyα2c adrenoceptorCell biologyProtein Transportmedicine.anatomical_structureEndocrinologybiology.proteinmedicine.symptomrhoA GTP-Binding ProteinVasoconstrictionAmerican Journal of Physiology-Cell Physiology
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Kinin receptors in human vascular tissue: their role in atheromatous disease

1997

Using samples of many human blood vessels, obtained at autopsy and specific antibodies directed to peptide sequences of the kinin B1 and B2 receptors, we demonstrate the localisation of these receptors within the human vascular system using standard immunolabelling techniques. In large elastic arteries and veins, kinin receptors are present only in the endothelial cells whereas in all muscular arteries and arterioles, these receptors are present in both the endothelial and smooth muscle cells. The identification of kinin receptors in human blood vessels confirms that kinins may modulate both vascular permeability and contractility. The incidental finding at histology, of patchy atheromatous…

medicine.medical_specialtyReceptor Bradykinin B2EndotheliumArteriosclerosisMolecular Sequence DataImmunocytochemistryEnzyme-Linked Immunosorbent AssayVascular permeabilityBiologyReceptor Bradykinin B1Muscle Smooth VascularVeinsCapillary PermeabilityContractilityAntibody SpecificityInternal medicinemedicineHumansVasoconstrictor AgentsAmino Acid SequenceReceptorVascular tissuePharmacologyStaining and LabelingReceptors BradykininArteriesKininImmunohistochemistryMolecular WeightArteriolesmedicine.anatomical_structureEndocrinologycardiovascular systemImmunohistochemistryKallikreinsAutopsyEndothelium VascularTissue KallikreinsMuscle ContractionImmunopharmacology
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