Search results for "Smut"

showing 10 items of 493 documents

Perspectives for CNO neutrino detection in Borexino

2018

International audience; Borexino measured with unprecedented accuracy the fluxes of solar neutrinos emitted at all the steps of the pp fusion chain. Still missing is the measurement of the flux of neutrinos produced in the CNO cycle. A positive measurement of the CNO neutrino flux is of fundamental importance for understanding the evolution of stars and addressing the unresolved controversy on the solar abundances. The measurement of the CNO neutrino flux in Borexino is challenging because of the low intensity of this component (CNO cycle accounts for about 1% of the energy emitted by Sun), the lack of prominent spectral features and the presence of background sources. The main background c…

CNO cycleexperimental methodsneutrino: solarPhysics::Instrumentation and DetectorsSolar neutrinoAstrophysics::High Energy Astrophysical Phenomenascintillation counter: liquidSolar neutrinosbismuth: admixtureAstrophysics[PHYS.NEXP]Physics [physics]/Nuclear Experiment [nucl-ex]7. Clean energySolar neutrinoCNO-cycleneutrino: fluxAstrophysics::Solar and Stellar Astrophysics[PHYS.PHYS.PHYS-INS-DET]Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det]Stellar evolutionBorexinoliquid scintillatorAstrophysics::Galaxy AstrophysicsPhysicsEnergy distributiondetectorbackgroundbismuth: nuclideCNO cycleNeutrino detector13. Climate actionBorexinoExperimental methodsNeutrino
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Cathodic reduction of CO2 to formic acid: Effect of the nature of the cathode for pressurized systems

2023

Electrochemical conversion of CO2 into formic acid (FA) in an aqueous electrolyte is considered a promising strategy to valorise waste-CO2. Some studies, mainly performed using Sn cathodes, have shown that the performance of the process can be strongly improved using pressurized systems. On the other hand, other studies, usually carried out in non-pressurized systems, have indicated that the nature of the cathode can strongly affect the process. Hence, in this work, we have investigated the coupled effect of nature of the cathode and CO2 pressure (PCO2 ) on the electrochemical conversion of CO2 to FA. Four electrodes (Sn, Sn/C-NP, Bi, Bi/C-NP) have been used as model cathodes. The results o…

CO2 reductionFormic acidPressureTinBismuthElectrochemistryCO2 reductionTinProcess Chemistry and TechnologyPressureElectrochemistryChemical Engineering (miscellaneous)Formic acidSettore ING-IND/27 - Chimica Industriale E TecnologicaBismuthPollutionWaste Management and DisposalJournal of Environmental Chemical Engineering
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Natural polyphenols facilitate elimination of HT-29 colorectal cancer xenografts by chemoradiotherapy: a Bcl-2- and superoxide dismutase 2-dependent …

2008

AbstractColorectal cancer is one of the most common malignancies worldwide. The treatment of advanced colorectal cancer with chemotherapy and radiation has two major problems: development of tumor resistance to therapy and nonspecific toxicity towards normal tissues. Different plant-derived polyphenols show anticancer properties and are pharmacologically safe. In vitro growth of human HT-29 colorectal cancer cells is inhibited (∼56%) by bioavailable concentrations of trans-pterostilbene (trans-3,5-dimethoxy-4′-hydroxystilbene; t-PTER) and quercetin (3,3′,4′,5,6-pentahydroxyflavone; QUER), two structurally related and naturally occurring small polyphenols. I.v. administration of t-PTER and Q…

Cancer ResearchAntioxidantColorectal cancerSp1 Transcription Factormedicine.medical_treatmentDown-RegulationMice NudeAntineoplastic AgentsBiologyAntioxidantsSuperoxide dismutaseMicePhenolsIn vivoGene expressionmedicineAnimalsHumansCell ProliferationFlavonoidsChemotherapySuperoxide DismutaseGene Expression ProfilingNF-kappa BPolyphenolsmedicine.diseaseChemotherapy regimenXenograft Model Antitumor AssaysOxaliplatinUp-RegulationOncologyBiochemistryProto-Oncogene Proteins c-bcl-2Drug Resistance NeoplasmCancer researchbiology.proteinFemaleColorectal NeoplasmsHT29 Cellsmedicine.drugMolecular cancer therapeutics
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Antioxidant defenses in a B16 melanoma line resistant to doxorubicin: an in vivo study.

1991

A B16 melanoma line was repeatedly transplanted subcutaneously in C57BL/6 mice. On day 4 after every transplant, the animals were treated with doxorubicin (DXR), 10 mg/kg i.p. The aim of the work was to develop an in-vivo model of resistance to the antiblastic in order to analyze some possible mechanistic aspects of the process in the course of time. After 16 transplants and treatments the melanoma completely lost its sensitivity to the antiproliferative effects of maximal tolerated doses of DXR and showed over-expression of P-glycoprotein. Compared to the parental line, the in vitro resistance index was 4.6. After 27 transplants and treatments the melanoma did not increase its in vitro res…

Cancer ResearchAntioxidantmedicine.medical_treatmentGlutathione reductaseDrug ResistanceMelanoma ExperimentalPharmacologySuperoxide dismutasechemistry.chemical_compoundMiceIn vivoTumor Cells CulturedMedicineAnimalsPharmacology (medical)DoxorubicinATP Binding Cassette Transporter Subfamily B Member 1Pharmacologychemistry.chemical_classificationMembrane Glycoproteinsbiologybusiness.industryMelanomaGlutathione peroxidaseGlutathionemedicine.diseaseGlutathioneImmunohistochemistryMice Inbred C57BLOncologychemistryDoxorubicinVincristinebiology.proteinFemalebusinessNeoplasm Transplantationmedicine.drugAnti-cancer drugs
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Role of glutathione in the induction of apoptosis and c-fos and c-jun mRNAs by oxidative stress in tumor cells.

2003

We have used two tumor cell clones (B9 and G2), derived from the methylcholanthrene-induced murine fibrosarcoma GR9 and normal BALB/c3T3 fibroblasts, to study the ability of t-BOOH derived reactive oxygen radicals to induce oxidative stress, apoptosis and c-fos and c-jun mRNA transcription. These clones differ in terms of their major histocompatibility complex (MHC) (H-2) class I genes expression, their tumor induction and metastatic potential and their reduced glutathione (GSH) levels. Incubation of both cell clones in the presence of t-BOOH results in the increase of 8-oxo-2'-deoxyguanosine (8-oxo-dG) and malondialdehyde and the decrease of GSH. The xenobiotic also induces the transcripti…

Cancer ResearchBALB 3T3 CellsTranscription GeneticProto-Oncogene Proteins c-junFibrosarcomaCellApoptosisBiologymedicine.disease_causeAntioxidantsSuperoxide dismutasechemistry.chemical_compoundMicetert-ButylhydroperoxideCell CloneMalondialdehydemedicineTumor Cells CulturedAnimalsRNA MessengerDNA Primerschemistry.chemical_classificationReactive oxygen speciesReverse Transcriptase Polymerase Chain Reactionc-junHistocompatibility Antigens Class IDeoxyguanosineGlutathioneFibroblastsMolecular biologyGlutathioneOxidative Stressmedicine.anatomical_structureOncologychemistryGene Expression RegulationApoptosis8-Hydroxy-2'-Deoxyguanosinebiology.proteinReactive Oxygen SpeciesProto-Oncogene Proteins c-fosOxidative stressCancer letters
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Spontaneous mutagenesis in Csb(m/m)Ogg1⁻(/)⁻ mice is attenuated by dietary resveratrol.

2010

Oxidative DNA modifications such as 7,8-dihydro-8-oxoguanine (8-oxoG) are generated endogenously in apparently all living cells. The defect of the repair of 8-oxoG in Csb m/m Ogg1 ―/― mice results in elevated basal levels of these lesions and increased frequencies of spontaneous mutations, which initiate tumorigenesis in the liver if cell proliferation is stimulated. Here, we describe that the phytoalexin resveratrol, applied either for 7 days per gavage (100 mg/kg body wt) or for 3―9 months in the diet (0.04% ad libitum), reduces the endogenous oxidative DNA base damage in the livers of the Csb m/m Ogg1 ―/― mice by 20―30% (P < 0.01). A small but consistent effect is also observed in the wi…

Cancer ResearchDNA damageSOD1SOD2Gene ExpressionMice TransgenicBiologyResveratrolSuperoxide dismutasechemistry.chemical_compoundMiceStilbenesAnimalschemistry.chemical_classificationReverse Transcriptase Polymerase Chain ReactionGlutathione peroxidaseMutagenesisAntimutagenic AgentsGeneral MedicineMolecular biologyDietOxidative StressCell killingchemistryBiochemistryLiverMutagenesisResveratrolbiology.proteinDNA DamageCarcinogenesis
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Production of superoxide by human malignant melanoma cells.

1998

Metastasis is a complicated multi-step process involving interactions between tumour cells, the extracellular matrix and the vessel walls. Experimental observations suggest that leucocyte migration and function could be a suitable model in order to understand tumour cell dissemination. In the present report we show and quantify the production of free radicals by human malignant melanoma cells (St-ml12) by means of a spectrophotometrical method, using an enzyme immunoassay reader. Endothelial cells and activated polymorphonuclear leucocytes were used as controls. Melanoma cells without stimulants produced large amounts of superoxide anion at an increasing rate in relation to time, which coul…

Cancer ResearchbiologySuperoxideMelanomaRadicalCellDermatologyHydrogen Peroxidemedicine.diseaseMolecular biologyMalignant transformationExtracellular matrixSuperoxide dismutaseImmunoenzyme Techniqueschemistry.chemical_compoundmedicine.anatomical_structureOncologychemistrySuperoxidesmedicinebiology.proteinTumor Cells CulturedHumansHydrogen peroxideMelanomaMelanoma research
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The oxygen radicals involved in the toxicity induced by parthenolide in MDA-MB-231 cells

2014

It has been shown that the sesquiterpene lactone parthenolide lowers the viability of MDA-MB-231 breast cancer cells, in correlation with oxidative stress. The present report examined the different radical species produced during parthenolide treatment and their possible role in the toxicity caused by the drug. Time course experiments showed that in the first phase of treatment (0-8 h), and in particular in the first 3 h, parthenolide induced dichlorofluorescein (DCF) signal in a large percentage of cells, while dihydroethidium (DHE) signal was not stimulated. Since the effect on DCF signal was suppressed by apocynin and diphenyleneiodonium (DPI), two inhibitors of NADPH oxidase (NOX), we s…

Cancer Researchparthenolide oxygen radicals NADPH oxidase breast cancer cellsCell SurvivalBreast NeoplasmsSuperoxide dismutasechemistry.chemical_compoundSuperoxide Dismutase-1DichlorofluoresceinSuperoxidesCell Line TumorSettore BIO/10 - BiochimicaHumansParthenolidechemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologySuperoxideSuperoxide DismutaseAnti-Inflammatory Agents Non-SteroidalNADPH OxidasesGeneral MedicineHydrogen PeroxideMolecular biologyMitochondriaOncologychemistryApocyninbiology.proteinFemaleSesquiterpenesPeroxynitrite
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WIN 55,212-2, agonist of cannabinoid receptors, prevents amyloid β1-42 effects on astrocytes in primary culture

2015

Alzheimer's disease (AD), a neurodegenerative illness involving synaptic dysfunction with extracellular accumulation of Aβ1-42 toxic peptide, glial activation, inflammatory response and oxidative stress, can lead to neuronal death. Endogenous cannabinoid system is implicated in physiological and physiopathological events in central nervous system (CNS), and changes in this system are related to many human diseases, including AD. However, studies on the effects of cannabinoids on astrocytes functions are scarce. In primary cultured astrocytes we studied cellular viability using MTT assay. Inflammatory and oxidative stress mediators were determined by ELISA and Western-blot techniques both in…

Cannabinoid receptormedicine.medical_treatmentInterleukin-1betaNitric Oxide Synthase Type IIlcsh:Medicinemedicine.disease_causeReceptors CannabinoidWIN 55212-2Receptorlcsh:ScienceCerebral CortexMultidisciplinaryCalcium Channel BlockersSistema nerviós Malaltiesmedicine.symptomSignal transductionResearch ArticleSignal Transductionmedicine.drugmedicine.medical_specialtyCell SurvivalMorpholinesPrimary Cell CultureInflammationNaphthalenesBiologyNeurologiaFetusInternal medicinemedicineAnimalsViability assayCannabinoid Receptor AgonistsAmyloid beta-PeptidesSuperoxide DismutaseTumor Necrosis Factor-alphalcsh:RTranscription Factor RelAPeptide FragmentsBenzoxazinesRatsPPAR gammaOxidative StressEndocrinologyGene Expression RegulationCyclooxygenase 2Astrocyteslcsh:QFisiologia humanaCannabinoidOxidative stress
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Heterodimer formation of wild-type and amyotrophic lateral sclerosis-causing mutant Cu/Zn-superoxide dismutase induces toxicity independent of protei…

2008

Recent studies provide evidence that wild-type Cu/Zn-superoxide dismutase (SOD1(hWT)) might be an important factor in mutant SOD1-mediated amyotrophic lateral sclerosis (ALS). In order to investigate its functional role in the pathogenesis of ALS, we designed fusion proteins of two SOD1 monomers linked by a polypeptide. We demonstrated that wild-type-like mutants, but not SOD1(G85R) homodimers, as well as mutant heterodimers including SOD1(G85R)-SOD1(hWT) display dismutase activity. Mutant homodimers showed an increased aggregation compared with the corresponding heterodimers in cell cultures and transgenic Caenorhabditis elegans, although SOD1(G85R) heterodimers are more toxic in functiona…

Cell SurvivalRecombinant Fusion Proteinsanimal diseasesSOD1MutantProtein aggregationAnimals Genetically ModifiedProtein CarbonylationSuperoxide dismutaseMicechemistry.chemical_compoundSuperoxide Dismutase-1Cell Line TumorGeneticsAnimalsHumansAmino Acid SequenceCaenorhabditis elegansMolecular BiologyGenetics (clinical)Motor NeuronsbiologySuperoxide DismutaseSuperoxideAmyotrophic Lateral SclerosisWild typenutritional and metabolic diseasesHydrogen PeroxideGeneral MedicineFusion proteinProtein Structure Tertiarynervous system diseasesCell biologyAmino Acid Substitutionnervous systemchemistryBiochemistrybiology.proteinDismutaseDimerizationHuman Molecular Genetics
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