Search results for "Stearoyl-CoA"

showing 10 items of 22 documents

Effects of streptozotocin and dietary fructose on delta-6 desaturation in spontaneously hypertensive rat liver.

2004

We have investigated the effects of hypertension associated with diabetes mellitus on polyunsaturated fatty acid biosynthesis. For this purpose, two rat models for these pathologies have been established: a type 1 diabetic hypertensive model obtained by streptozotocin injection to spontaneously hypertensive rat (SHR), followed or not by insulin treatment (experiment 1); a type 2 diabetic hypertensive model by feeding SHR with a fructose enriched diet (experiment 2). Liver gene expression of delta-6 desaturase (D6D), microsomal D6D activities and fatty acid composition of total lipids were estimated. In experiment 1, an increase of linoleic acid (18:2 n-6) level was observed in the streptozo…

Malemedicine.medical_specialtymedicine.medical_treatmentType 2 diabetesFructoseBiochemistryGene Expression Regulation EnzymologicStreptozocinchemistry.chemical_compoundSpontaneously hypertensive ratInternal medicineDiabetes mellitusMicrosomesRats Inbred SHRmedicineDietary CarbohydratesAnimalsHumansInsulinInsulinFatty AcidsFructoseGeneral Medicinemedicine.diseaseStreptozotocinDietRatsDisease Models AnimalEndocrinologyDiabetes Mellitus Type 1chemistryDiabetes Mellitus Type 2LiverLipogenesisHypertensionFatty Acids UnsaturatedMetabolic syndromeStearoyl-CoA Desaturasemedicine.drugBiochimie
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Chronic exposure of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces an obesogenic effect in C57BL/6J mice fed a high fat diet

2017

IF 3.582; International audience; Contaminant involvement in the pathophysiology of obesity is widely recognized. It has been shown that low dose and chronic exposure to endocrine disruptor compounds (EDCs) potentiated diet- induced obesity. High and acute exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), a persistent organic pollutant (POP) and an EDC with anti-estrogenic property, causes wasting syndrome . However at lower doses, the TCDD metabolic effects remain poorly understood. We investigated the obesogenic effect during chronic exposure of TCDD at 1μg/kg body weight (bw)/week in adult C57BL/6J mice fed with a high fat diet (HFD) and exposed from 10 to 42 weeks old to TCDD or e…

Blood GlucoseLeptinMale0301 basic medicineTCDDPolychlorinated DibenzodioxinsTime FactorsAdipose tissue010501 environmental sciencesToxicology01 natural sciencesBasic Helix-Loop-Helix Transcription FactorsInsulinAdiposity2. Zero hunger[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism3. Good healthLiverEndocrine disruptorReceptors AndrogenCytokinesEnvironmental PollutantsFemaleInflammation Mediatorsmedicine.symptomStearoyl-CoA Desaturasemedicine.medical_specialtyLipolysisInflammationchronic exposureIntra-Abdominal FatDiet High-FatRisk Assessment03 medical and health sciencesSex FactorsobesogenInternal medicinemedicineAnimalsEndocrine systemObesityRNA MessengerWasting SyndromeTriglycerides0105 earth and related environmental sciencesbusiness.industrymedicine.diseaseObesityMice Inbred C57BL030104 developmental biologyEndocrinologyReceptors Aryl HydrocarbonInsulin ResistancebusinessBiomarkersObesogenDrug metabolism
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INHIBITION OF FATTY ACID DESATURASES INDrosophila melanogasterLARVAE BLOCKS FEEDING AND DEVELOPMENTAL PROGRESSION

2016

International audience; Fatty acid desaturases are metabolic setscrews. To study their systemic impact on growth in Drosophila melanogaster, we inhibited fatty acid desaturases using the inhibitor CAY10566. As expected, the amount of desaturated lipids is reduced in larvae fed with CAY10566. These animals cease feeding soon after hatching, and their growth is strongly attenuated. A starvation program is not launched, but the expression of distinct metabolic genes is activated, possibly to mobilize storage material. Without attaining the normal size, inhibitor-fed larvae molt to the next stage indicating that the steroid hormone ecdysone triggers molting correctly. Nevertheless, after moltin…

Fatty Acid Desaturases0301 basic medicinePhysiologymedicine.medical_treatmentMoltingBiochemistrychemistry.chemical_compound0302 clinical medicinehomeostasisDrosophila Proteins2. Zero hungerchemistry.chemical_classificationsex-pheromonesGene Expression Regulation DevelopmentalGeneral Medicineinsulin-like peptidesAmino acidDrosophila melanogastersynthaseBiochemistryLarvaDrosophila melanogasterMoultingEcdysoneEcdysoneinsulinanimal structuresgrowthamino-acidsBiologylipids03 medical and health sciencesdesat1medicineAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biologydevelopment[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biologystearoyl-coa desaturase-1fungiFatty acidFeeding Behaviorbiology.organism_classificationgene-expressionSteroid hormone030104 developmental biologyEnzymechemistryInsect SciencecellsStearoyl-CoA desaturase-1030217 neurology & neurosurgeryArchives of Insect Biochemistry and Physiology
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Regulation of yeast fatty acid desaturase in response to iron deficiency

2017

Unsaturated fatty acids (UFA) are essential components of phospholipids that greatly contribute to the biophysical properties of cellular membranes. Biosynthesis of UFAs relies on a conserved family of iron-dependent fatty acid desaturases, whose representative in the model yeast Saccharomyces cerevisiae is Ole1. OLE1 expression is tightly regulated to adapt UFA biosynthesis and lipid bilayer properties to changes in temperature, and in UFA or oxygen availability. Despite iron deficiency being the most extended nutritional disorder worldwide, very little is known about the mechanisms and the biological relevance of fatty acid desaturases regulation in response to iron starvation. In this re…

0301 basic medicineSaccharomyces cerevisiae ProteinsMga2Ole1Saccharomyces cerevisiaeSaccharomyces cerevisiaeGene Expression Regulation Enzymologic03 medical and health scienceschemistry.chemical_compoundBiosynthesisValosin Containing ProteinGene Expression Regulation FungalFatty acidsHypoxiaMolecular BiologyTranscription factorEndosomal Sorting Complexes Required for Transport030102 biochemistry & molecular biologybiologyChemistryIron deficiencyEndoplasmic reticulumMembrane ProteinsUbiquitin-Protein Ligase ComplexesIron DeficienciesCell Biologybiology.organism_classificationYeastYeastUbiquitin ligase030104 developmental biologyFatty acid desaturaseBiochemistryProteasomebiology.proteinStearoyl-CoA DesaturaseTranscription FactorsColdBiochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
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Stearoyl-CoA desaturase 1 coding sequences abd antisense RNA affect lipid secretion in transfected chicken LMH hepatoma cells

2000

Hepatic stearoyl CoA desaturase (SCD) activity in chickens from a fat line is higher than that of chickens from a lean line and correlates with plasma triacylglycerol concentrations. Furthermore, in these lines, the hepatic SCD1 mRNA level is positively correlated with the adipose tissue weight. To analyze the contribution of the SCD1 gene in the regulation of adiposity in the early stages of triacylglycerol secretion, SCD1 coding sequence and antisense RNA expression vectors were transfected in LMH cells. After selection, these cells were analyzed with regard to SCD1 expression and lipid secretion. The amounts of secreted triacylglycerols and phospholipids were shown to be higher in LMH ce…

animal structures[SDV]Life Sciences [q-bio]BiophysicsGene ExpressionAdipose tissueBiologyTransfectionBiochemistry03 medical and health sciencesLiver Neoplasms ExperimentalGene expressionTumor Cells CulturedAnimalsRNA AntisenseRNA MessengerRNA NeoplasmMolecular BiologyComputingMilieux_MISCELLANEOUSDNA Primers030304 developmental biology0303 health sciencesExpression vectorBase Sequence030302 biochemistry & molecular biologynutritional and metabolic diseasesRNATransfectionLipid MetabolismMolecular biologyRecombinant ProteinsAntisense RNAIsoenzymesStearoyl-CoA DesaturaseLiverembryonic structureslipids (amino acids peptides and proteins)Stearoyl-CoA desaturase-1ChickensStearoyl-CoA Desaturase
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Inhibition of stearoyl-CoA desaturase 1 expression induces CHOP-dependent cell death in human cancer cells.

2010

Background Cancer cells present a sustained de novo fatty acid synthesis with an increase of saturated and monounsaturated fatty acid (MUFA) production. This change in fatty acid metabolism is associated with overexpression of stearoyl-CoA desaturase 1 (Scd1), which catalyses the transformation of saturated fatty acids into monounsaturated fatty acids (e.g., oleic acid). Several reports demonstrated that inhibition of Scd1 led to the blocking of proliferation and induction of apoptosis in cancer cells. Nevertheless, mechanisms of cell death activation remain to be better understood. Principal Findings In this study, we demonstrated that Scd1 extinction by siRNA triggered abolition of de nov…

Cell SurvivalEukaryotic Initiation Factor-2lcsh:MedicineApoptosisCHOPBiologyCell Biology/Cell SignalingCell Linechemistry.chemical_compoundCell Line TumorNeoplasmsHumansRNA Small Interferinglcsh:ScienceEndoplasmic Reticulum Chaperone BiPFatty acid synthesisHeat-Shock ProteinsCell ProliferationTranscription Factor CHOPMultidisciplinaryFatty acid metabolismCell DeathCell growthFatty Acidslcsh:RCell Biology/Cellular Death and Stress ResponsesMolecular biologyCell biologychemistryOncologyApoptosisCancer celllipids (amino acids peptides and proteins)lcsh:QStearoyl-CoA desaturase-1Stearoyl-CoA DesaturaseTranscription Factor CHOPResearch ArticleOleic AcidPLoS ONE
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SCD5-induced oleic acid production reduces melanoma malignancy by intracellular retention of SPARC and cathepsin B

2014

A proper balance between saturated and unsaturated fatty acids (FAs) is required for maintaining cell homeostasis. The increased demand of FAs to assemble the plasma membranes of continuously dividing cancer cells might unbalance this ratio and critically affect tumour outgrowth. We unveiled the role of the stearoyl-CoA desaturase SCD5 in converting saturated FAs into mono-unsaturated FAs during melanoma progression. SCD5 is down-regulated in advanced melanoma and its restored expression significantly reduced melanoma malignancy, both in vitro and in vivo, through a mechanism governing the secretion of extracellular matrix proteins, such as secreted protein acidic and rich in cysteine (SPAR…

cathepsin B2734Intracellular SpaceDown-RegulationCell LineMelanocyteCell Line TumormelanomaHumansintracellular acidityOsteonectinNeoplasticTumorMedicine (all)Fatty AcidsSPARCHydrogen-Ion ConcentrationGene Expression Regulation NeoplasticSCD5Gene Expression Regulationoleic acidDisease ProgressionMelanocytesFatty AcidStearoyl-CoA Desaturasecathepsin B; intracellular acidity; melanoma; oleic acid; SCD5; SPARC; Cathepsin B; Cell Line Tumor; Disease Progression; Down-Regulation; Fatty Acids; Humans; Hydrogen-Ion Concentration; Intracellular Space; Melanocytes; Melanoma; Oleic Acid; Osteonectin; Stearoyl-CoA Desaturase; Gene Expression Regulation Neoplastic; 2734; Medicine (all)Human
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Modulation of the hepatic fatty acid pool in peroxisomal 3-ketoacyl-CoA thiolase B-null mice exposed to the selective PPARalpha agonist Wy14,643

2009

10 pages; International audience; The peroxisomal 3-ketoacyl-CoA thiolase B (Thb) gene was previously identified as a direct target gene of PPARalpha, a nuclear hormone receptor activated by hypolipidemic fibrate drugs. To better understand the role of ThB in hepatic lipid metabolism in mice, Sv129 wild-type and Thb null mice were fed or not the selective PPARalpha agonist Wy14,643 (Wy). Here, it is shown that in contrast to some other mouse models deficient for peroxisomal enzymes, the hepatic PPARalpha signaling cascade in Thb null mice was normal under regular conditions. It is of interest that the hypotriglyceridemic action of Wy was reduced in Thb null mice underlining the conclusion t…

MESH : RNA MessengerMESH: Microsomes LiverMESH : PyrimidinesMono-unsaturated fatty acids n-7 and n-9MESH : Hepatocytes[SDV.BBM.BM] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyMESH: Mice KnockoutPPARαBiochemistryMESH: Acetyl-CoA C-AcetyltransferaseStearoyl-CoA desaturase-1MESH: HepatocytesMicechemistry.chemical_compoundMESH : Lipid MetabolismWy14MESH: AnimalsPeroxisomal 3-ketoacyl-CoA thiolase BAcetyl-CoA C-AcetyltransferaseMESH: PPAR alphaMESH : Fatty AcidsMESH: Lipid MetabolismMice Knockoutchemistry.chemical_classificationThiolaseFatty Acids643General MedicinePeroxisomeMESH : Stearoyl-CoA DesaturaseMESH: Fatty AcidsMESH : Microsomes LiverMESH : Acetyl-CoA C-AcetyltransferaseMicrosomes LiverMono-unsaturated fatty acids n-7 and n-9; Peroxisomal 3-ketoacyl-CoA thiolase B; PPARα; Stearoyl-CoA desaturase-1; Wy14643lipids (amino acids peptides and proteins)Stearoyl-CoA DesaturasePolyunsaturated fatty acidmedicine.medical_specialtyMESH : PPAR alphaMESH : Mice Inbred C57BL[ SDV.BBM.BM ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyBiologyMESH: Mice Inbred C57BLInternal medicineMESH : MicePeroxisomesmedicineAnimalsHumansPPAR alphaRNA MessengerMESH: MiceMESH: RNA MessengerSCP2MESH: HumansMESH : HumansFatty acid[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyStearoyl-CoALipid MetabolismMESH: PeroxisomesSterol regulatory element-binding proteinMice Inbred C57BLPyrimidinesEndocrinologychemistryMESH: PyrimidinesMESH: Stearoyl-CoA DesaturaseHepatocytesMESH : Mice KnockoutMESH : AnimalsStearoyl-CoA desaturase-1MESH : PeroxisomesBiochimie
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Heterogeneity of the Stearoyl-CoA desaturase-1 (SCD1) Gene and Metabolic Risk Factors in the EPIC-Potsdam Study

2012

Background: Stearoyl-CoA desaturase-1 (SCD1) is an enzyme involved in lipid metabolism. In mice and humans its activity has been associated with traits of the metabolic syndrome, but also with the prevention of saturated fatty acids accumulation and subsequent inflammation, whereas for liver fat content inconsistent results have been reported. Thus, variants of the gene encoding SCD1 (SCD1) could potentially modify metabolic risk factors, but few human studies have addressed this question. Methods: In a sample of 2157 middle-aged men and women randomly drawn from the Potsdam cohort of the European Prospective Investigation into Cancer and Nutrition, we investigated the impact of 7 SCD1 tagg…

MaleEpidemiologyPopulationlcsh:Medicine610Single-nucleotide polymorphismBiologyPolymorphism Single NucleotideBiochemistryCohort StudiesGenetic HeterogeneityMiceRisk FactorsGermanyNeoplasmsGenotypemedicineGeneticsAnimalsHumansGenetic Predisposition to Diseaselcsh:ScienceeducationBiologyGenetic Association StudiesCardiovascular Disease EpidemiologyAgedGeneticseducation.field_of_studyMultidisciplinaryGenetic heterogeneitylcsh:RHaplotypeHuman GeneticsMiddle Agedmedicine.diseaseEuropean Prospective Investigation into Cancer and NutritionEnzymesMinor allele frequencyHaplotypesGenetic EpidemiologyGenetic PolymorphismMedicinelcsh:QFemaleMetabolic syndromeStearoyl-CoA DesaturasePopulation GeneticsResearch Article
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Mechanisms involved in lipid accumulation and apoptosis induced by 1-nitropyrene in Hepa1c1c7 cells

2011

International audience; 1-Nitropyrene (1-NP) is a nitro-polycyclic aromatic hydrocarbon (nitro-PAH) present in diesel exhaust and bound to particular matter in urban air. We show that 1-NP and the referent PAH benzo(a)pyrene (BP) induce apoptosis and a lipid accumulation dependent on cytochrome P450 1A1-metabolites in mouse hepatoma cells, whereas 1-amino-pyrene had no effect. The caspase inhibitor, N-benzyloxycarbonyl-Val-Ala-Asp(O-Me) fluoromethyl ketone (Z-VAD-fmk), inhibits 1-NP-induced apoptosis, but failed to alter 1-NP-triggered lipid accumulation determined by Nile red staining. We further show that cholesterol and fatty acid contents are modified after nitro-PAH exposure and that 1…

endoplasmic-reticulum stressMESH: PyrenesHepatoma cellsliver-cellsactivated protein-kinaseApoptosisAMP-Activated Protein KinasesToxicologyMESH: Liver Neoplasms ExperimentalMicechemistry.chemical_compoundMESH: CholesterolLiver Neoplasms ExperimentalMESH: AnimalsMESH: AMP-Activated Protein KinasesStearoyl-CoA desaturase 1CaspaseMESH: Lipid Metabolismchemistry.chemical_classificationhuman macrophages0303 health sciencesPyrenesbiology8-tetrachlorodibenzo-p-dioxin tcdd030302 biochemistry & molecular biologyGeneral Medicineinhibition[SDV.BBM.BC]Life Sciences [q-bio]/Biochemistry Molecular Biology/Biomolecules [q-bio.BM]CholesterolBiochemistry[SDV.TOX]Life Sciences [q-bio]/ToxicologyCaspaseslipids (amino acids peptides and proteins)stearoyl-coaStearoyl-CoA DesaturaseMESH: Cell Line Tumor[SDV.BC]Life Sciences [q-bio]/Cellular Biology03 medical and health sciencesMESH: Benzo(a)pyreneCell Line Tumor1-NitropyreneBenzo(a)pyreneAnimalsFatty acidsProtein kinase AMESH: Mice030304 developmental biologyaromatic-hydrocarbonsMESH: CaspasesCholesterolMESH: Apoptosisc-srcFatty acidAMPKCytochrome P450Lipid MetabolismMolecular biologychemistryApoptosisMESH: Stearoyl-CoA Desaturasebiology.proteinStearoyl-CoA desaturase-1desaturaseToxicology Letters
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