Search results for "Stellate cell"

showing 10 items of 106 documents

Paracrine Activation of Hepatic CB1 Receptors by Stellate Cell-Derived Endocannabinoids Mediates Alcoholic Fatty Liver

2008

SummaryAlcohol-induced fatty liver, a major cause of morbidity, has been attributed to enhanced hepatic lipogenesis and decreased fat clearance of unknown mechanism. Here we report that the steatosis induced in mice by a low-fat, liquid ethanol diet is attenuated by concurrent blockade of cannabinoid CB1 receptors. Global or hepatocyte-specific CB1 knockout mice are resistant to ethanol-induced steatosis and increases in lipogenic gene expression and have increased carnitine palmitoyltransferase 1 activity, which, unlike in controls, is not reduced by ethanol treatment. Ethanol feeding increases the hepatic expression of CB1 receptors and upregulates the endocannabinoid 2-arachidonoylglycer…

Malemedicine.medical_specialtyPhysiologyHUMDISEASEArachidonic AcidsGlyceridesMiceCarnitine palmitoyltransferase 1PiperidinesReceptor Cannabinoid CB1Internal medicineCannabinoid Receptor ModulatorsParacrine CommunicationmedicineAnimalsReceptorDiet Fat-RestrictedMolecular BiologyCells CulturedMice KnockoutCarnitine O-PalmitoyltransferaseEthanolChemistryLipogenesisFatty AcidsFatty liverCell Biologymedicine.diseaseEndocannabinoid systemCoculture TechniquesUp-RegulationMice Inbred C57BLDisease Models AnimalLipoprotein LipaseEndocrinologyLiverLipogenesisHepatocytesHepatic stellate cellPyrazoleslipids (amino acids peptides and proteins)Alcoholic fatty liverFatty Acid SynthasesRimonabantSteatosisSterol Regulatory Element Binding Protein 1Oxidation-ReductionEndocannabinoidsFatty Liver AlcoholicCell Metabolism
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Hepatic progenitors for liver disease: current position

2010

Alice Conigliaro1, David A Brenner2, Tatiana Kisseleva21University “La Sapienza”, Dipartimento di Biotecnologie Cellulari ed Ematologia Policlinico Umberto I, V Clinica Medica, Rome, Italy; 2Department of Medicine, University of California, San Diego, La Jolla, CA, USAAbstract: Liver regeneration restores the original functionality of hepatocytes and cholangiocytes in response to injury. It is regulated on several levels, with different cellular populations contributing to this process, eg, hepatocytes, liver precursor cells, intrahepatic stem cells. In response to injury, mature hepatocytes have the capability to proliferate and give rise to new hepatocytes and cholangi…

Pathologymedicine.medical_specialtyoval cellsLiver cytologyMedicine (miscellaneous)cholangiocytes; hepatic progenitor; hepatocytes; intrahepatic stem cells; liver disease; liver precursor cells; oval cellsReviewBiologyhepatocytemedicinecholangiocytesProgenitor cellliver precursor cellQH573-671Mesenchymal stem cellintrahepatic stem cellCell Biologyhepatic progenitorliver precursor cellsintrahepatic stem cellsLiver regenerationCell biologyHaematopoiesisAmniotic epithelial cellsHepatic stellate cellhepatocytesStem cellCytologyliver diseasecholangiocyteStem Cells and Cloning: Advances and Applications
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Hepatocyte cell lines: their use, scope and limitations in drug metabolism studies.

2006

Gaining knowledge on the metabolism of a drug, the enzymes involved and its inhibition or induction potential is a necessary step in pharmaceutical development of new compounds. Primary human hepatocytes are considered a cellular model of reference, as they express the majority of drug-metabolising enzymes, respond to enzyme inducers and are capable of generating in vitro a metabolic profile similar to what is found in vivo. However, hepatocytes show phenotypic instability and have a restricted accessibility. Different alternatives have been explored in the past recent years to overcome the limitations of primary hepatocytes. These include immortalisation of adult or fetal human hepatic cel…

PharmacologyCell fusionCell Culture TechniquesDrug Evaluation PreclinicalReproducibility of ResultsGeneral MedicineBiologyToxicologyCell biologyCell LineXenobioticsmedicine.anatomical_structureBiochemistryDownregulation and upregulationCell cultureHepatocytemedicineHepatic stellate cellHepatocytesAnimalsHumansProgenitor cellCellular modelDrug metabolismCell Line TransformedExpert opinion on drug metabolismtoxicology
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Mitochondrial (dys)function - a factor underlying the variability of efavirenz-induced hepatotoxicity?

2015

Background and Purpose The non-nucleoside analogue reverse transcriptase inhibitor efavirenz is associated with hepatic toxicity and metabolic disturbances. Although the mechanisms involved are not clear, recent evidence has pinpointed a specific mitochondrial action of efavirenz accompanied by the induction of an endoplasmic reticulum (ER) stress/unfolded protein response in human hepatic cells. The aim of this study was to further investigate the involvement of this organelle by evaluating efavirenz's effects in cells lacking functional mitochondria (rho°) and comparing them with those of the typical mitotoxic agent rotenone, a standard complex I inhibitor, and the ER stress inducer thaps…

PharmacologyThapsigarginEfavirenzReverse-transcriptase inhibitorEndoplasmic reticulumRotenoneBiologyMitochondrionPharmacologychemistry.chemical_compoundchemistryUnfolded protein responseHepatic stellate cellmedicinemedicine.drugBritish Journal of Pharmacology
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Prevention of 7-ketocholesterol-induced side effects by natural compounds

2018

Cholesterol oxidation products, also named oxysterols, can be formed either by cholesterol auto-oxidation, enzymatically or both. Among these oxysterols, 7-ketocholesterol (7KC) is mainly formed during radical attacks that take place on the carbon 7 of cholesterol. As increased levels of 7KC have been found in the tissues, plasma and/or cerebrospinal fluid of patients with major diseases, especially age-related diseases (cardiovascular diseases, eye diseases, neurodegenerative diseases), some cancers, and chronic inflammatory diseases, it is suspected that 7KC, could contribute to their development. Since 7KC, provided by the diet or endogenously formed, is not or little efficiently metabol…

Programmed cell deathOxysterol030309 nutrition & dieteticsTocopherolsInflammationPharmacologymedicine.disease_causeAntioxidantsIndustrial and Manufacturing Engineering03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologymedicineHumansNoncommunicable DiseasesKetocholesterolsInflammation0303 health sciencesCholesterolFatty AcidsPolyphenols04 agricultural and veterinary sciencesGeneral Medicine040401 food scienceCytoprotectionOxidative StressMetabolic pathwaychemistryHepatic stellate cellmedicine.symptomOxidation-ReductionOxidative stressFood ScienceCritical Reviews in Food Science and Nutrition
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Nitric oxide synthesis is related to astroglial cell cycle in the mouse retina via p53 gene regulation

2009

Purpose To analyze the influence of p53 gene in retinal astrocytes morphology and function. Methods Adult mice of the strain C57BL/6 were distributed into two groups: 1) wild type TP53, as the controls (cp53; n=20) and 2) mice with an extra copy of p53 (sp53; n=22). Both eyeballs were enucleated, after death by ether narcosis, to obtain the retinas. Right retinas were frozen (-85oC) until processing to assess nitric oxide (NO) synthesis. Left retinas were obtained and proccesed to perform immunohistochemical localization of the glial fibrillary acidic protein (GFAP). Data are expressed as the mean ± SD. Statistical analysis was carried out using Student’s t-test. Confidence interval was tak…

Regulation of gene expressionRetinaeducation.field_of_studyGlial fibrillary acidic proteinPopulationWild typeRetinalGeneral MedicineBiologyMolecular biologyNitric oxideOphthalmologychemistry.chemical_compoundmedicine.anatomical_structurechemistryBiochemistrybiology.proteinHepatic stellate cellmedicineeducationActa Ophthalmologica
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Mboat7 down-regulation by hyper-insulinemia induces fat accumulation in hepatocytes.

2020

Background: Naturally occurring variation in Membrane-bound O-acyltransferase domain-containing 7 (MBOAT7), encoding for an enzyme involved in phosphatidylinositol acyl-chain remodelling, has been associated with fatty liver and hepatic disorders. Here, we examined the relationship between hepatic Mboat7 down-regulation and fat accumulation. Methods: Hepatic MBOAT7 expression was surveyed in 119 obese individuals and in experimental models. MBOAT7 was acutely silenced by antisense oligonucleotides in C57Bl/6 mice, and by CRISPR/Cas9 in HepG2 hepatocytes. Findings: In obese individuals, hepatic MBOAT7 mRNA decreased from normal liver to steatohepatitis, independently of diabetes, inflammatio…

Research paperTGFβ Transforming Growth Factor BetaIntracellular SpaceCRISPR Clustered Regularly Interspaced Short Palindromic RepeatshHEPS Human HepatocytesMice0302 clinical medicineLPIAT1DAG Diacylglyceroli.p. Intraperitonealmedia_commonFatty AcidsGeneral Medicine3. Good health030220 oncology & carcinogenesisHOMA-IR homeostasis Model Assessment of Insulin ResistanceMPO morpholinolcsh:Medicine (General)medicine.medical_specialtyPE Phosphatidyl-EthanolamineNashGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesTNFα tumor Necrosis Factor AlphaLDL Low Density LipoproteinsHyperinsulinismNAFLDSD Standard Dietmedia_common.cataloged_instanceHumansCPT1 Carnitine Palmitoyltransferase IPhosphatidylinositolGene SilencingEuropean unionVLDL Very Low Density Lipoproteinlcsh:RhHSC Human Hepatic Stellate Cellsmedicine.diseaseLipid MetabolismOA Oleic AcidCI Confidence IntervalMboat7 Membrane bound O-acyltransferase domain containing 7MCD methionine choline deficient diet030104 developmental biologyEndocrinologychemistryCDP Cytidine-DiphosphateFOXO1 Forkhead Box protein O1NAFLD nonalcoholic fatty liver diseaseSteatohepatitisBMI Body Mass IndexCL CardiolipinAcyltransferases0301 basic medicineAlcoholic liver diseaseCXCL10 C-X-C Motif Chemokine 10lcsh:Medicinechemistry.chemical_compoundNon-alcoholic Fatty Liver DiseaseIFG Impaired Fasting GlucoseAPOB Apolipoprotein BNonalcoholic fatty liver diseasePIP Phosphatidyl-Inositol-PhosphateSteatohepatitisqRT-PCR quantitative Real Time Polymerase Chain ReactionMice Knockoutlcsh:R5-920ORO Oil Red O StainingPI PhosphatidylinositolFatty liverTM6SF2 Transmembrane 6 Superfamily Member 2PhospholipidTAG TriglyceridesNASH Nonalcoholic SteatohepatitisLipogenesisLPA Lyso-Phosphatidic AcidPhosphatidylinositolSignal TransductionPS Phosphatidyl-SerinePA Palmitic AcidALD alcoholic liver diseasePC Phosphatidylcholinei.v. IntravenousFATP1 Fatty Acid Transport Protein 1Models BiologicalInternal medicinemedicineAnimalsNonalcoholic fatty liver diseasePPARα Peroxisome Proliferator-Activated Receptor alphaObesityG3P Glyceraldehyde-3-PhosphateSREBP1c Sterol Regulatory Element-Binding Protein 1HDL High Density Lipoproteinsbusiness.industryPI3K Phosphatidylinositol 3 KinaseMembrane ProteinsNHEJ Non-Homologues End JoiningPNPLA3 Patatin-like Phospholipase Domain-containing-3MTTP Microsomal Triglyceride Transfer ProteinLPIAT1 Lysophosphatidylinositol Acyltransferase 1TMC4 Transmembrane Channel-Like 4Disease Models AnimalGene Expression RegulationHepatocytesFOXA2 Forkhead Box A2mTOR mammalian target of RapamycinSteatosisInsulin ResistancebusinessPG Phosphatidyl-GlycerolFABP1 Fatty Acid-Binding Protein 1 FAS Fatty Acid SynthaseT2DM Type 2 Diabetes MellitusEBioMedicine
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Cultured Ito cells of rat liver express the alpha 2-macroglobulin gene.

1987

Ito cells were isolated from rat liver and kept in culture for up to 13 days. The capability of the Ito cells to synthesize a2-macroglobulin was analyzed at different times after isolation and by pulse-chase experiments. Newly synthesized a2-macroglobulin was determined by immunoprecipitation followed by sodium dodecyl sulfate/ polyacrylamide gel electrophoresis and fluorography. a2-Macroglobulin synthesis was hardly detectable in Ito cells and their media 3 days after plating. However, 5 - 11 days after the isolation of the cells, increasing amounts of a2-macroglobulin were synthesized. The results of pulse-chase experiments performed on day 7 showed that radioactively labeled a2-macroglob…

Time FactorsBiologydigestive systemBiochemistrychemistry.chemical_compoundfluids and secretionsAnimalsalpha-MacroglobulinsNorthern blotRNA MessengerSodium dodecyl sulfatePancreatic elastasePolyacrylamide gel electrophoresisCells CulturedImmunoassayDNALipid MetabolismMolecular biologyMacroglobulinRatsSecretory proteinPerisinusoidal spaceBiochemistrychemistryGene Expression RegulationLiverHepatic stellate cellElectrophoresis Polyacrylamide GelFemalecirculatory and respiratory physiologyEuropean journal of biochemistry
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Differential expression of collagen types I, III, and IV by fat-storing (Ito) cells in vitro

1992

It has been observed that Ito cells in vitro undergo phenotypical changes ("activation") similar to those noted in vivo during the development of liver fibrosis. Because conflicting data have been published on the amount and different types of collagens synthesized by Ito cells in vitro, collagen biosynthesis was studied at different "activation" stages on both the protein and RNA levels. Immunoprecipitation of endogenously labeled collagen showed that freshly isolated ("resting") Ito cells synthesize mainly collagen type IV. Collagen type I was hardly detectable in the earlier stage of primary culture, but it clearly increased starting 5 days after isolation. Compared with the basal rates …

Transcription GeneticMolecular Sequence DataCell SeparationBiologychemistry.chemical_compoundBiosynthesisIn vivomedicineAnimalsCells CulturedBasement membraneBase SequenceHepatologyGastroenterologyRNARats Inbred StrainsImmunohistochemistryPrecipitin TestsMolecular biologyIn vitroRatsmedicine.anatomical_structureAdipose TissueBiochemistrychemistryCell cultureHepatic stellate cellRNAImmunohistochemistryElectrophoresis Polyacrylamide GelFemaleCollagenGastroenterology
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Alpha-(13′-hydroxy)-6-hydroxychroman, the main product of alpha-tocopherol metabolism in human hepatocytes, regulates CYP4F2 and PPAR-γ expression

2017

The enzymatic metabolism of vitamin E in liver cells generates long chain metabolites (LCMs) with proposed regulatory activity on inflammatory and atherogenic genes. In this study the LCM formation kinetics was characterized in HepG2 and HepaRG human hepatic cells, supplemented with RRR-α-tocopherol (α-TOH). α-13’OH was the main product of α-TOH metabolism, while α-13’COOH metabolite and the short chain metabolite α-CEHC, were detected only in traces, thus demonstrating the poor efficiency of vitamin E catabolism in these cells. However, this metabolism was significantly simulated when the hepatic cells were challenged with (lipo)toxic agents, such as ethanol or palmitate. Under such condit…

chemistry.chemical_classificationCatabolismMetaboliteVitamin Emedicine.medical_treatmentPeroxisome proliferator-activated receptorBiologyBiochemistrychemistry.chemical_compoundchemistryBiochemistryNuclear receptorPhysiology (medical)medicineHepatic stellate cellalpha-TocopherolDrug metabolismFree Radical Biology and Medicine
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