Search results for "Streptococcus pyogenes"

showing 8 items of 18 documents

Major histocompatibility complex class II binding site for streptococcal pyrogenic (erythrogenic) toxin A.

1994

Streptococcal pyrogenic exotoxin A (SPEA) is an important pathogenicity factor of group A streptococci. It is a member of the family of „superantigens” produced by Staphylococcus aureus and Streptococcus pyogenes and its T lymphocyte stimulating activity is involved into the pathogenesis of certain diseases caused by pyogenic streptococci. In this study we have produced and characterized recombinant SPEA molecules in Escherichia coli. These molecules are indistinguishable from natural SPEA in both T cell stimulatory and HLA class II binding activities. Human class II molecules are more efficient than mouse class II molecules in presenting SPEA to T cells. In binding tests to major histocomp…

Microbiology (medical)Recombinant Fusion ProteinsT-LymphocytesImmunologyAntigen presentationErythrogenic toxinBacterial ToxinsMolecular Sequence DataExotoxinsEnterotoxinmedicine.disease_causeMajor histocompatibility complexLymphocyte ActivationMicrobiologyCell LineMajor Histocompatibility ComplexEnterotoxinsMicestomatognathic systemBacterial ProteinsmedicineEscherichia coliImmunology and AllergyAnimalsHumansCells CulturedMice Inbred BALB CBinding SitesSuperantigensbiologyBase SequencePyrogensToxic shock syndromeMembrane ProteinsStreptococcusGeneral MedicineGene Expression Regulation BacterialHLA-DR Antigensmedicine.diseasebiology.organism_classificationSpeaStreptococcus pyogenesbiology.proteinExotoxinMedical microbiology and immunology
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Identification of a plasminogen-binding motif in PAM, a bacterial surface protein.

1995

Surface-associated plasmin(ogen) may contribute to the invasive properties of various cells. Analysis of plasmin(ogen)-binding surface proteins is therefore of interest. The N-terminal variable regions of M-like (ML) proteins from five different group A streptococcal serotypes (33, 41, 52, 53 and 56) exhibiting the plasminogen-binding phenotype were cloned and expressed in Escherichia coli. The recombinant proteins all bound plasminogen with high affinity. The binding involved the kringle domains of plasminogen and was blocked by a lysine analogue, 6-aminohexanoic acid, indicating that lysine residues in the M-like proteins participate in the interaction. Sequence analysis revealed that the…

PlasminStreptococcus pyogenesMolecular Sequence DataPlasma protein bindingBiologyMicrobiologyKringle domainBacterial ProteinsKringlesmedicineEscherichia coliAmino Acid SequenceBinding siteCloning MolecularMolecular BiologyPeptide sequenceBinding SitesBase SequenceLysinePlasminogenFusion proteinMolecular biologyRecombinant ProteinsPhenotypeBiochemistryCarrier ProteinsPlasminogen activatorSequence AlignmentBinding domainmedicine.drugProtein BindingMolecular microbiology
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TRIC: an automated alignment strategy for reproducible protein quantification in targeted proteomics

2016

Nature Methods, 13 (9)

Pluripotent Stem CellsProteomics0301 basic medicineAnalyteStreptococcus pyogenesSoftware toolQuantitative proteomicsProteomic analysisComputational biologyBiologyProteome informaticsProteomicsBioinformaticsBiochemistryArticleMass Spectrometry03 medical and health sciencesSequence Analysis ProteinProtein methodsHumansProtein PrecursorsHuman Induced Pluripotent Stem CellsMolecular BiologyElectronic Data ProcessingReproducibility of ResultsCell BiologyMass spectrometricTargeted proteomics030104 developmental biologyProteolysissense organsPeptidesSequence AlignmentAlgorithmsSoftwareBiotechnologyNature Methods
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EDEMA EMORRAGICO ACUTO DELL'INFANZIA: UN CASE REPORT

2019

Un lattante di 6 mesi maschio giunge alla nostra osservazione per comparsa di lesioni maculo-papulari alle guance estesesi, in poche ore, ai quattro arti ed evolventi in manifestazioni purpuriche su cute edematosa senza segno della fovea. All’EO all’ingresso si evidenziavano inoltre: angioedema dei padiglioni auricolari, aspetto a coccarda delle lesioni aventi diametro max di 2 cm con coinvolgimento anche dello scroto, cute del tronco e mucose integre, assenza di segni neurologici. Due giorni prima del ricovero e della comparsa delle manifestazioni cutanee il piccolo aveva presentato febbre (T max 39,5°C) e faringite trattata con amoxi-clavulanato. Esami al ricovero: PCR mossa (2,3 mg/dl; V…

Settore MED/38 - Pediatria Generale E SpecialisticaEDEMA EMORRAGICO ACUTO Streptococcus pyogenes
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Molecular Co-operation between Protein PAM and Streptokinase for Plasmin Acquisition by Streptococcus pyogenes

1998

Bacterial surface-associated plasmin formation is believed to contribute to invasion, although the underlying molecular mechanisms are poorly understood. To define the components necessary for plasmin generation on group A streptococci we used strain AP53 which exposes an M-like protein ("PAM") that contains a plasminogen-binding sequence with two 13-amino acid residues long tandem repeats (a1 and a2). Utilizing an Escherichia coli-streptococcal shuttle vector, we replaced a 29-residue long sequence segment of Arp4, an M-like protein that does not bind plasminogen, with a single (a1) or the combined a1a2 repeats of PAM. When expressed in E. coli, the purified chimeric Arp/PAM proteins both …

Streptococcus pyogenesPlasminRecombinant Fusion Proteinsmedicine.medical_treatmentStreptokinasemedicine.disease_causeBiochemistryMicrobiologyBacterial Proteinsstomatognathic systemShuttle vectorTandem repeatEscherichiaparasitic diseasesmedicineStreptokinaseFibrinolysinMolecular BiologyGeneAntigens BacterialProteasebiologyPlasminogenCell Biologybiology.organism_classificationBiochemistryStreptococcus pyogenesTransformation BacterialCarrier ProteinsBacterial Outer Membrane Proteinsmedicine.drugJournal of Biological Chemistry
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Mutations affecting MHC class II binding of the superantigen streptococcal erythrogenic toxin A.

1993

Streptococcal pyrogenic exotoxin A (SPEA) is an important pathogenicity factor of group A streptococci. It is a member of the family of 'superantigens' produced by Staphylococcus aureus and Streptococcus pyogenes, and its T lymphocyte stimulating activity is involved in the pathogenesis of certain diseases caused by pyogenic streptococci. In this study we have generated nine mutant SPEA molecules by substituting amino acids in the regions of homology between different streptococcal and staphylococcal superantigens. An additional mutant was created by deletion of the 10 N-terminal amino acids. The mutants were expressed as fusion proteins. Several mutations led to a loss of function due to a…

Streptococcus pyogenesT-LymphocytesImmunologyMutantMolecular Sequence DataExotoxinsBiologymedicine.disease_causeLymphocyte ActivationMicrobiologyMiceStructure-Activity Relationshipstomatognathic systemBacterial ProteinsSuperantigenmedicineImmunology and AllergyAnimalsHumansAmino Acid SequencePeptide sequencechemistry.chemical_classificationMice Inbred BALB CSuperantigensBase SequenceHistocompatibility Antigens Class IIMembrane ProteinsGeneral Medicinebiology.organism_classificationFusion proteinAmino acidchemistrySpeaStreptococcus pyogenesMutationExotoxinInternational immunology
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Absorption of kininogen from human plasma by Streptococcus pyogenes is followed by the release of bradykinin.

1997

H-kininogen (high-molecular-mass kininogen, HK) is the precursor of the vasoactive peptide hormone bradykinin (BK). Previous work has demonstrated that HK binds to Streptococcus pyogenesthrough M-proteins, fibrous surface proteins and important virulence factors of these bacteria. Here we find that M-protein-expressing bacteria absorb HK from human plasma. The HK bound to the bacteria was found to be cleaved, and analysis of the degradation pattern suggested that the cleavage of HK at the bacterial surface is associated with the release of BK. Moreover, addition of activated plasma prekallikrein to bacteria preincubated with human plasma, resulted in BK release. This mechanism, by which a p…

chemistry.chemical_classificationKininogenbiologyKininogensStreptococcus pyogenesBradykininVirulencePeptideCell BiologyPlasma protein bindingbiology.organism_classificationmedicine.disease_causeBradykininBiochemistryMicrobiologyProinflammatory cytokinechemistry.chemical_compoundchemistryStreptococcus pyogenesmedicineHumansMolecular BiologyBacteriaProtein BindingResearch Article
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PIN30: HOSPITALIZATION EXPENDITURE OF STREPTOCOCCUS PYOGENES-ASSOCIATED CELLULITIS IN THE UNITED STATES

2003

medicine.medical_specialtybusiness.industryInternal medicineCellulitisHealth PolicyStreptococcus pyogenesmedicinePublic Health Environmental and Occupational Healthmedicine.disease_causebusinessmedicine.diseaseValue in Health
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