Search results for "Sud"

showing 10 items of 571 documents

Quantitative and qualitative analysis of DNA extracted from postmortem muscle tissues

1990

DNA extracted from 33 postmortem muscle specimens was analyzed using MZ 1.3, a hypervariable minisatellite probe, as well as locus-specific minisatellite probes (g3, MS1 and MS43). After storage at -25 degrees C for 10 months, DNA from all the samples was partially (approximately 21% of total DNA) degraded even when autopsy was performed 1 day postmortem. However, more than 90% of DNA samples up to at least 3 days postmortem were suitable to obtain good restriction fragment length polymorphism (RFLP) patterns. When small strips of specimen were stored for 8 days at room temperature in moist chambers, approximately 42% of total DNA was degraded. Only 30% of these DNA samples still showed goo…

AdultMaleTime FactorsAdolescentBiologyDNA SatellitePostmortem ChangesPathology and Forensic Medicinechemistry.chemical_compoundDeath SuddenQualitative analysisHumansChildAgedMultiple TraumaHybridization probeMusclesDNAMiddle AgedDNA extractionMolecular biologyMolecular WeightMinisatellitechemistryDNA profilingAccidents AviationPostmortem ChangesFemaleRestriction fragment length polymorphismAnatomyBurnsDNA ProbesDNAPolymorphism Restriction Fragment LengthZeitschrift f�r Rechtsmedizin
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Prospective risk stratification of sudden cardiac death in Marfan's syndrome.

2013

Marfan syndrome (MFS) is a variable, autosomal-dominant disorder of the connective tissue. In MFS serious ventricular arrhythmias and sudden cardiac death (SCD) can occur. The aim of this prospective study was to reveal underlying risk factors and to prospectively investigate the association between MFS and SCD in a long-term follow-up.77 patients with MFS were included. At baseline serum N-terminal pro-brain natriuretic peptide (NT-proBNP), transthoracic echocardiogram, 12-lead resting ECG, signal-averaged ECG (SAECG) and a 24-h Holter ECG with time- and frequency domain analyses were performed. The primary composite endpoint was defined as SCD, ventricular tachycardia (VT), ventricular fi…

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyCardiomyopathyVentricular tachycardiaRisk AssessmentSudden cardiac deathMarfan SyndromeYoung AdultInternal medicineClinical endpointMedicineHumanscardiovascular diseasesProspective StudiesUltrasonographybusiness.industryHazard ratioMiddle Agedmedicine.diseaseSignal-averaged electrocardiogramDeath Sudden CardiacVentricular fibrillationCardiologyFemaleTransthoracic echocardiogramCardiology and Cardiovascular MedicinebusinessFollow-Up StudiesInternational journal of cardiology
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Traumatic lesion of the extracranial vertebral artery--a note-worthy potentially lethal injury.

1994

The autopsy findings from routine neuropathological investigations of the cervical spine after any history of trauma emphasized the vulnerability of the extracranial vertebral arteries. In 21 cases with trauma to the head and neck, normal autopsy procedures did not succeed in revealing an obvious cause of death. Traumatic lesions of the spinal cord such as contusion or neurorrhexis were seen in 10 cases. In 15 cases we observed different degrees and stages of traumatic lesions of the extracranial vertebral arteries. Sudden death due to acute brain stem ischemia might be considered as an explanation in some of these cases. Six case reports with traumatic vertebral artery (VA) lesions after s…

AdultMalemedicine.medical_specialtyAdolescentVertebral arteryPoison controlAutopsyHemorrhageWounds NonpenetratingSudden deathPathology and Forensic MedicineBrain IschemiaDeath SuddenFatal Outcomemedicine.arteryCause of DeathmedicineCraniocerebral TraumaHumansVertebrobasilar insufficiencySpinal Cord InjuriesVertebral ArteryAgedAged 80 and overbusiness.industryThrombosisMiddle AgedSpinal cordmedicine.diseaseThrombosisSurgerymedicine.anatomical_structureBlunt traumaCerebrovascular CirculationCervical VertebraeFemaleAutopsybusinessBrain StemInternational journal of legal medicine
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Botulinum toxin A (Botox) and sweating-dose efficacy and comparison to other BoNT preparations.

2004

Abstract Background Botulinum toxin type A (BoNT/A) is 20–50 times more effective than Botulinum toxin type B (BoNT/B) concerning the treatment of muscular hypercontractions [Sloop, R.R., Cole, B.A., Escutin, R.O., 1997. Human response to botulinum toxin injection: type B compared with type A. Neurology 49, 189–194]. Botulinum toxins block motor nerves as well as autonomic fibres [Rand, M.J., Whaler, B.C., 1965. Impairment of sympathetic transmission by botulinum toxin. Nature 206, 588–591]. Objective Purpose of this study was to analyse the dose dependent reduction of sweating using the BoNT/A preparation Botox® and to compare the results with our earlier results analysing Dysport® [Braune…

AdultMalemedicine.medical_specialtyBotulinum ToxinsTime FactorsInjections SubcutaneousSweatingBotulinum toxin aCellular and Molecular NeuroscienceMedicineHumansBotulinum toxin type BAnhidrosisBotulinum Toxins Type AHypohidrosisDose-Response Relationship DrugEndocrine and Autonomic Systemsbusiness.industryStarchBotulinum toxinSurgerySudomotorDose–response relationshipThreshold doseNeuromuscular AgentsAnesthesiaFemaleNeurology (clinical)medicine.symptombusinessBotulinum toxin typemedicine.drugFollow-Up StudiesAutonomic neuroscience : basicclinical
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Long-Term Enoximone Therapy in Unstable Chronic Heart Failure

1989

Long-term safety and efficacy of oral enoximone were evaluated in 32 patients with unstable chronic heart failure despite digitalis, diuretics, and vasodilator therapy. Oral enoximone, 75-150 mg t.i.d. was given for an average of 32 weeks. At baseline, 21 patients were in NYHA functional class IV, 10 patients in class III, and 1 patient in class II. Within 12 weeks, 14 of 20 patients surviving for more than 26 weeks had improved by at least one functional class. Hemodynamic data showed an 18% increase of cardiac index and a 34% decrease of diastolic pulmonary artery pressure. Echocardiographic recordings revealed an increase of fractional shortening from 13.9 +/- 7 to 15.6 +/- 5% after 12 w…

AdultMalemedicine.medical_specialtyCardiotonic AgentsHeart diseaseCardiac indexDiastoleSudden deathElectrocardiographyInternal medicinemedicine.arterymedicineHumansEnoximoneEnoximoneSurvival rateAgedAged 80 and overHeart FailurePharmacologybusiness.industryHemodynamicsImidazolesMiddle Agedmedicine.diseaseSurgeryHeart failureChronic DiseasePulmonary arteryCardiologyFemalebusinessCardiology and Cardiovascular Medicinemedicine.drugJournal of Cardiovascular Pharmacology
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Clinical characteristics and determinants of the phenotype in TMEM43 arrhythmogenic right ventricular cardiomyopathy type 5.

2020

Arrhythmogenic right ventricular cardiomyopathy type V (ARVC-5) is the most aggressive heterozygous form of ARVC. It is predominantly caused by a fully penetrant mutation (p.S358L) in the nondesmosomal gene TMEM43-endemic to Newfoundland, Canada. To date, all familial cases reported worldwide share a common ancestral haplotype. It is unknown whether the p.S358L mutation by itself causes ARVC-5 or whether the disease is influenced by genetic or environmental factors. The purpose of this study was to examine the phenotype, clinical course, and the impact of exercise on patients with p.S358L ARVC-5 without the Newfoundland genetic background. We studied 62 affected individuals and 73 noncarrie…

AdultMalemedicine.medical_specialtyDNA Mutational AnalysisMutation MissenseDisease030204 cardiovascular system & hematologyVentricular Function LeftRight ventricular cardiomyopathySudden cardiac deathElectrocardiography03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicinemedicineGeneticsHumans030212 general & internal medicineExerciseArrhythmogenic Right Ventricular DysplasiaEjection fractionTMEM43business.industryIncidence (epidemiology)HaplotypeMembrane ProteinsStroke VolumeDNAmedicine.diseasePhenotypePedigree3. Good healthPhenotypeMutation (genetic algorithm)CardiologyFemaleCardiology and Cardiovascular MedicinebusinessArrhythmogenic right ventricular cardiomyopathyArrhythmia
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Chemically and electrically induced sweating and flare reaction

2004

Both thin afferent (nociceptors) and efferent (sympathetic sudomotor) nerve fibers can be activated electrically and chemically, resulting in neurogenic erythema and sweating. These reactions have been used before to assess the impairment of sympathetic and nociceptor fibers in humans. In this study, electrically induced sweating and erythema were assessed simultaneously in the foot dorsum and thigh, and were compared to chemically induced activation. Reproducible intensity-response relations (stimulation intensities 0-30 mA, 1 Hz) were obtained from 32 subjects. The steepest increase of the sweat response was induced at lower intensities as compared to that of the erythema (18.3 mA vs. 25.…

AdultMalemedicine.medical_specialtyErythemaEfferentSweatingStimulationFunctional LateralityCellular and Molecular NeuroscienceSex FactorsInternal medicineReflexLaser-Doppler FlowmetrymedicineHumansAgedSkinAnalysis of VarianceNeurogenic inflammationintegumentary systemFootEndocrine and Autonomic SystemsChemistryAge FactorsReproducibility of ResultsDose-Response Relationship RadiationMiddle AgedAcetylcholineElectric StimulationStimulation ChemicalSudomotorAutonomic nervous systemEndocrinologyThighErythemaAnesthesiaNociceptorFemaleNeurology (clinical)medicine.symptomAcetylcholinemedicine.drugAutonomic Neuroscience
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Prevalence of myxomatous mitral valve prolapse in patients with lymphocytic thyroiditis

1995

Abstract In conclusion, given the cardiac (mitral regurgitation, endocarditis, thromboembolic complications, arrhythmic sudden death) and neurologic (cerebral embolic event) complications of the pathologic forms of MVP, 6,8 physicians should look carefully for myxomatous involvement of the mitral valve and prolapse in patients with autoimmune thyroid diseases. Patients should be monitored and prophylactic antibiotic treatment recommended when appropriate.

AdultMalemedicine.medical_specialtyGraves' diseaseSudden deathInternal medicineMitral valvemedicineHumansEndocarditisAgedAutoimmune diseaseMitral regurgitationMitral Valve Prolapsebusiness.industryThyroidThyroiditis AutoimmuneMiddle Agedmedicine.diseaseSurgerymedicine.anatomical_structureEchocardiographycardiovascular systemCardiologyFemaleCardiology and Cardiovascular MedicinebusinessLymphocytic ThyroiditisThe American Journal of Cardiology
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Prognostic significance of repetitive ventricular response in chronic coronary artery disease.

1985

A prospective study was conducted in 267 patients with angiographically defined coronary artery disease without documented ventricular tachycardia to determine the prognostic significance of repetitive ventricular response (RVR) after programmed electrical stimulation (PES). The patients were classified inducible if RVR with 3 or more echo beats (RVR greater than or equal to 3) could be induced. 89 patients without previous myocardial infarction (MI), 61 survivors of MI occurring between 6 weeks and 3 months before and 117 patients who had survived longer than 3 months after MI were studied. A standardized stimulation protocol with single (S1S2) and double (S1S2S3) extrastimuli during ventr…

AdultMalemedicine.medical_specialtyHeart VentriclesMyocardial InfarctionCoronary DiseaseVentricular tachycardiaCoronary AngiographySudden deathCoronary artery diseaseDeath SuddenInternal medicineTachycardiaMedicineHumansIn patientMyocardial infarctionProspective cohort studyCycle lengthAgedbusiness.industryIncidence (epidemiology)Cardiac Pacing ArtificialMiddle Agedmedicine.diseasePrognosisSurgeryCardiologyFemaleCardiology and Cardiovascular MedicinebusinessFollow-Up StudiesEuropean heart journal
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Botulinum Toxin A reduces neurogenic flare but has almost no effect on pain and hyperalgesia in human skin.

2003

Botulinum toxin A (BoNT/A) has been used therapeutically to treat muscular hypercontractions and sudomotor hyperactivity. There is increasing evidence that BoNT/A might also have analgesic properties, in particular in headache. In the present investigation we tested the often cited hypothesis that BoNT/A-induced analgesia can be attributed to inhibition of neuropeptide release from nociceptive nerve fibers. In 15 healthy volunteers BoNT/A (5, 10, 20 mouse units BOTOX) or saline (contralateral side) was injected intracutaneously on the volar forearm. On day zero, the day of injection, no further tests were performed. We repeatedly elicited pain, mechanical hyperalgesia and neurogenic flare b…

AdultMalemedicine.medical_specialtyNeurologyAnalgesicNeuropeptidePainStimulationNerve FibersPsychophysicsMedicineHumansBotulinum Toxins Type APain MeasurementSkinHypohidrosisNeurogenic inflammationbusiness.industryNociceptorsAxonsElectric StimulationSudomotorNociceptionNeurologyHyperalgesiaAnesthesiaHyperalgesiaFemaleNeurology (clinical)medicine.symptomNeurogenic InflammationbusinessJournal of neurology
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