Search results for "Sweat"

showing 10 items of 63 documents

Tissue kallikrein and kininogen in human sweat glands and psoriatic skin

1991

The cellular localization of immunoreactive tissue kallikrein and kininogen was studied in normal and psoriatic human skin. Immunoreactivity to both enzyme and substrate was observed in secretory granules of the dark cells in the secretory fundus (acinus) of the sweat glands. Double immunostaining revealed a segmental distribution of the two antigens. Each acinar section contained either tissue kallikrein or kininogen. However, there appeared to be a junctional zone in which both were present, but in separate dark cells. Immunoreactivity for both antigens was also observed in close apposition to the luminal microvilli of the duct cells. No specific immunostaining was seen in sebaceous gland…

Kininogenmedicine.medical_specialtyPathologyStaining and LabelingKininogensTissue kallikreinMyoepithelial cellHuman skinDermatologyKallikreinBiologyKininImmunohistochemistrySweat GlandsEndocrinologymedicine.anatomical_structureSweat glandInternal medicinemedicineHumansPsoriasisKallikreinsCellular localizationSkincirculatory and respiratory physiologyBritish Journal of Dermatology
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Axonal pathology of the skin in infantile neuroaxonal dystrophy.

1987

Ultrastructural studies on the skin of two patients affected by infantile neuroaxonal dystrophy (INAD) were performed to evaluate its diagnostic value and to discuss the etiology of INAD. While the majority of terminal axons around intradermal glands were dystophic consisting of tubulomembranous and tubulovesicular profiles sometimes accompanied by synaptic vesicles, there were only few dystophic axons inside intradermal nerve bundles. These observations suggest that the primary lesion of INAD is located in terminal and presynaptic axons. Therefore, terminal axons have to be investigated when a diagnostic skin biopsy is performed in INAD.

MalePathologymedicine.medical_specialtyAxonal pathologySynaptic vesiclePathology and Forensic MedicineInfantile neuroaxonal dystrophyCellular and Molecular NeurosciencemedicineHumansAxonNeuroaxonal dystrophySkinmedicine.diagnostic_testbusiness.industryLeukodystrophyInfantAnatomyPrimary lesionmedicine.diseaseAxonsSweat Glandsmedicine.anatomical_structurenervous systemChild PreschoolSkin biopsyFemaleNeurology (clinical)Nervous System DiseasesbusinessActa neuropathologica
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Ultrastructural Pathology of Eccrine Sweat Gland Epithelial Cells in Globoid Cell Leukodystrophy

1993

Three of four children were recognized by deficient β-galactocerebrosidase activities as having globoid cell leukodystrophy inclusions in sweat gland epithelial cells, similar in ultrastructure to those seen in Schwann cells. This observation in globoid cell leukodystrophy emphasizes the need to include sweat gland epithelial cells in examinations of skin in globoid cell leukodystrophy, as well as in any neurometabolic disorder. ( J Child Neurol 1993;8:171-174).

MalePathologymedicine.medical_specialtyBiopsyCellEccrine GlandsBiologyEpitheliumInclusion bodiesUltrastructural Pathology03 medical and health sciences0302 clinical medicine030225 pediatricsSweat glandmedicineHumansEccrine sweat glandChildSkinInclusion Bodiesintegumentary systemLeukodystrophyInfantLipid Metabolismmedicine.diseaseEpitheliumLeukodystrophy Globoid CellMicroscopy Electronmedicine.anatomical_structureChild PreschoolVacuolesPediatrics Perinatology and Child HealthUltrastructureFemaleNeurology (clinical)030217 neurology & neurosurgeryJournal of Child Neurology
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Infant developmental profile of Crisponi syndrome due to compound heterozygosity for CRLF1 deletion.

2020

Crisponi syndrome/CISS1, is an autosomal recessive ciliary neurotrophic factor receptor (CNTFR)-related genodermatosis caused in 95% of cases by mutations in CRLF1 on chromosome 19p13. The CNTFR pathway is important for CNS development. Crisponi syndrome/ CISS1 can be suspected in the presence of the following clinical triad: camptodactyly with fisted hands, intermittent hyperthermia and muscular contractions with feeding difficulties.

MalePathologymedicine.medical_specialtyCrisponi syndromeCompound heterozygosityPathology and Forensic MedicineCamptodactylyDeath SuddenPeriodic feverMedicineHumansHyperhidrosisReceptors CytokineGenetics (clinical)Sequence DeletionDevelopmental profiledevelopmental delay thin corpus callosum clinical profilebusiness.industryInfant NewbornFaciesInfantCold-induced sweating syndromeGeneral MedicineThin corpus callosumPediatrics Perinatology and Child HealthTrismusAnatomymedicine.symptomDevelopmental DelayCold-induced sweating syndrome CamptodactylyThin corpus callosum Periodic feverbusinessHand Deformities CongenitalClinical dysmorphology
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Enzyme replacement therapy with agalsidase alfa in children with Fabry disease.

2006

Aim: To assess the effects of enzyme replacement therapy (ERT) in children with Fabry disease. Methods: Safety and efficacy of ERT with agalsidase alfa, 0.2 mg/kg infused over 40 minutes every 2 weeks for 23 weeks, were studied in a multicentre open-label trial in nine boys and four girls. Median age at the start of the study was 11.0 years (range 3.5–18 years). Results: Fifty-four adverse events were reported in 11 patients. No serious adverse events related to ERT were reported. Twelve of the 54 adverse events were considered possibly or probably related to ERT. Infusion reactions (8 mild, 3 moderate) occurred in four boys, in seven infusions. One boy developed IgG antibodies, although he…

MalePediatricsmedicine.medical_specialtyAdolescentGlobotriaosylceramideSweatingchemistry.chemical_compoundQuality of lifemedicineHumansBrief Pain InventoryAdverse effectChildPain Measurementbusiness.industryTrihexosylceramidesGeneral MedicineEnzyme replacement therapymedicine.diseaseFabry diseaseRecombinant ProteinsSurgeryClinical trialIsoenzymesTreatment OutcomeEl NiñochemistryChild Preschoolalpha-GalactosidasePediatrics Perinatology and Child HealthFabry DiseaseFemalebusinessActa paediatrica (Oslo, Norway : 1992)
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Chemosensory anxiety signals prime defensive behavior in prepubertal girls

2017

indexation en cours; Chemosensory anxiety signals effectively prime motor responses related to withdrawal behavior, such as the startle reflex, in adult humans. As the reproductive status strongly affects the response to social chemosignals, the current study examined whether chemosensory anxiety signals would augment the startle response in prepubertal children as it does in adults. Using cotton pads, axillary sweat was collected from 28 men while waiting for an important oral examination (anxiety condition), and during ergometer training (sport control condition). Using a constant-flow olfactometer, sweat samples and pure cotton samples (cotton control) were presented to 10 prepubertal gi…

MaleReflex Startlepuberty[SDV.OT]Life Sciences [q-bio]/Other [q-bio.OT]Startle responsemedicine.medical_specialtyAdolescentEmotionsSweatingExperimental and Cognitive PsychologyElectromyographyAnxietystartle responseAudiologyStimulus (physiology)Alarm signalFear-potentiated startle050105 experimental psychologyDevelopmental psychologysex hormoneSWEAT03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineMoro reflex[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]medicineHumans0501 psychology and cognitive sciences[ SDV.OT ] Life Sciences [q-bio]/Other [q-bio.OT]ChildPsychiatric Status Rating Scalesmedicine.diagnostic_test[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]05 social sciencesStimulation ChemicalAcoustic Stimulationchemosensory communication[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]AxillaOdorantsAnxietyFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]medicine.symptomPsychologychemosensory anxiety030217 neurology & neurosurgeryPhysiology & Behavior
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Botulinum Toxin Type B Blocks Sudomotor Function Effectively: A 6 Month Follow Up

2003

This study analyzes the suppression of sweat gland activity by botulinum toxin type B. We injected botulinum toxin type B (between 2 and 1000 mouse units subcutaneously) in the lateral side of both lower legs in 15 healthy volunteers. Sweat tests were carried out before botulinum toxin type B injections, and at 3 wk, 3 mo, and 6 mo. We studied focal anhidrosis by iodine–starch staining and by capacitance hygrometry after carbachol iontophoresis, according to the quantitative sudomotor axon reflex test (QSART). Iodine starch staining indicated that a threshold dose of 8 mouse units botulinum toxin type B leads to anhidrotic skin spots (>4 cm2) after 3 wk. Duration of anhidrosis was prolonged…

Malemedicine.medical_specialtyCarbacholBotulinum ToxinsSweatingDermatologyBiochemistrySWEATSweat glandInternal medicinemedicineHumansHyperhidrosisAnhidrosisBotulinum Toxins Type AMolecular BiologyHypohidrosisLegIontophoresisStaining and Labelingbusiness.industryHyperhidrosisautonomic nervous systemStarchCell BiologySweat GlandsSudomotorEndocrinologymedicine.anatomical_structureAxon reflexFemalemedicine.symptombusinessiodine starch stainingbotulinum toxin Bmedicine.drugFollow-Up StudiesIodineJournal of Investigative Dermatology
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Nerve injury in fractures of the condylar neck.

1977

Summary Although fractures of the condylar neck belong to the most frequent injuries of the jaws, and various nerves are found in close proximity to the temporomandibular joints, only little mention is made in the literature of neurological complications arising from accidents. We have therefore carried out a prospective study on nerve injuries in 237 fractures of the articular process in the period 1971–1975. We found two cases of post-traumatic Frey's syndrome, whereby the syndrome developed from a post-traumatic auriculotemporal neuralgia. We also found loss of function of auriculotemporal, buccal and facial nerves, and loss of taste sensation of the tongue in another case, caused by dam…

Malemedicine.medical_specialtyChordaFacial ParalysisSweating GustatoryAuriculotemporal nerveCondylestomatognathic systemTongueMandibular FracturesmedicineHumansProspective Studiesmedicine.cranial_nerveFacial Nerve Injuriesbiologybusiness.industryMandibular CondyleGeneral MedicineNerve injurymedicine.diseasebiology.organism_classificationFacial nerveSurgerymedicine.anatomical_structureNeuralgiaSurgeryFemaleBuccal nerveChorda Tympani Nervemedicine.symptombusinessJournal of maxillofacial surgery
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PANI-Based Wearable Electrochemical Sensor for pH Sweat Monitoring

2021

Nowadays, we are assisting in the exceptional growth in research relating to the development of wearable devices for sweat analysis. Sweat is a biofluid that contains useful health information and allows a non-invasive, continuous and comfortable collection. For this reason, it is an excellent biofluid for the detection of different analytes. In this work, electrochemical sensors based on polyaniline thin films deposited on the flexible substrate polyethylene terephthalate coated with indium tin oxide were studied. Polyaniline thin films were abstained by the potentiostatic deposition technique, applying a potential of +2 V vs. SCE for 90 s. To improve the sensor performance, the electronic…

Materials scienceelectrochemical sensor02 engineering and technologySubstrate (electronics)QD415-436wearable sensor010402 general chemistry01 natural sciencespH meterBiochemistryreduced graphene oxidepolyanilineAnalytical ChemistryContact anglechemistry.chemical_compoundPolyanilineSettore ING-IND/17 - Impianti Industriali MeccanicipH sensorPhysical and Theoretical ChemistryThin filmSettore ING-IND/34 - Bioingegneria Industriale021001 nanoscience & nanotechnology0104 chemical sciencesElectrochemical gas sensorIndium tin oxidesweatSettore ING-IND/23 - Chimica Fisica ApplicataChemical engineeringchemistryElectrode0210 nano-technologyChemosensors
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A mild juvenile variant of type IV glycogenosis.

1992

The mild juvenile form of type IV glycogenosis, confirmed by a profound deficiency of the brancher enzyme in tissue specimens is reported from three Turkish male siblings who, foremost, suffered from chronic progressive myopathy. Muscle fibers contained polyglucosan inclusions of typical fine structure, i.e. a mixture of granular and filamentous glycogen. They reacted strongly for myophosphorylase, but were resistant to diastase. These inclusions were ubiquitinated and reacted with antibody KM-279 which previously has been shown to bind to Lafora bodies, corpora amylacea and polyglucosan material in hepatic and cardiac cells of type IV glycogenosis as well as polyglucosan body myopathy with…

Muscle tissueMalemedicine.medical_specialtyBiologychemistry.chemical_compoundGlycogen Storage Disease Type IVDevelopmental NeuroscienceInternal medicineSweat glandmedicineHumansGlycogen storage disease type IVMyopathyChildGlycogenStaining and LabelingHistocytochemistryMusclesInfantGeneral Medicinemedicine.diseaseEnzyme assaySweat Glandsmedicine.anatomical_structureEndocrinologychemistryMyophosphorylasePediatrics Perinatology and Child Healthbiology.proteinNeurology (clinical)medicine.symptomCorpora amylaceaBraindevelopment
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