Search results for "Synaptic Transmission"

showing 8 items of 178 documents

Toxicity effects of the organic UV-filter 4-Methylbenzylidene camphor in zebrafish embryos

2019

Abstract Ultraviolet (UV) filters are widely used in personal care products and due to their lipophilicity these chemicals tend to bioaccumulate in the aquatic biota. 4-Methylbenzylidene camphor (4-MBC) is one of the most used UV-filters, and it is commonly detected in freshwater fish tissues. This substance is suspected to be an endocrine disruptor due to its interaction with Hypothalamus-Pituitary-Gonadal (HPG) and HP-Thyroid (HPT)-axis. The main objective of this study was to evaluate the effects of 4-MBC on apical endpoints, biochemical markers and on genes involved in endocrine pathways in Danio rerio. Zebrafish embryos were exposed to 4-MBC (0.083–0.77 mg/l) from 0 to 96 h post-fertil…

medicine.medical_specialtyEmbryo Nonmammaliananimal structuresEnvironmental EngineeringHealth Toxicology and Mutagenesis0208 environmental biotechnologyDanioEndocrine System02 engineering and technologyEndocrine Disruptors010501 environmental sciencesmedicine.disease_causeSynaptic Transmission01 natural scienceschemistry.chemical_compoundInternal medicinemedicineAnimalsEnvironmental ChemistryYolk sacZebrafishZebrafishGlutathione Transferase0105 earth and related environmental sciencesbiologyEmbryogenesisPublic Health Environmental and Occupational HealthGeneral MedicineGeneral Chemistrybiology.organism_classificationPollutionAcetylcholinesteraseCamphor020801 environmental engineeringOxidative Stressmedicine.anatomical_structureEndocrinologychemistryEndocrine disruptorEnzyme InductionToxicityAcetylcholinesteraseFiltrationOxidative stressChemosphere
researchProduct

Effects of ovariectomy and steroid replacement on GABAA receptor binding in female rat brain.

1991

Abstract The specific binding of tritiated muscimol to γ-aminobutyric acid (GABA) receptor sites was studied in distinct brain areas of female rats during different endocrine states. In diestrous rats with intact ovaries the highest receptor densities were found in the cortex (10.24 pmol/mg protein) and the lowest concentrations in the mediobasal hypothalamus (3.29 pmol/mg protein). Four weeks after removal of the ovaries, the number of binding sites was enhanced up to 2.4-fold in all brain areas investigated: the preoptic brain area, mediobasal hypothalamus, corticomedial amygdala, and cerebral cortex. The affinity of the binding sites remained unchanged. Substitution of estradiol and prog…

medicine.medical_specialtyEndocrinology Diabetes and MetabolismOvariectomyClinical BiochemistryBiologyIn Vitro TechniquesTritiumBiochemistrySynaptic Transmissionchemistry.chemical_compoundEndocrinologyInternal medicineCortex (anatomy)medicineAnimalsReceptorMolecular BiologyProgesteroneEstradiolGABAA receptorMuscimolBrainRats Inbred StrainsCell BiologyLuteinizing HormoneReceptors GABA-ARatsPreoptic areamedicine.anatomical_structureEndocrinologynervous systemMuscimolchemistryCerebral cortexMolecular MedicineGABAergicFemaleHormoneThe Journal of steroid biochemistry and molecular biology
researchProduct

gamma-Aminobutyric acid and cholinergic transmission in the guinea-pig ileum.

1983

1. The effects of GABA on release of acetylcholine and on contractility of the smooth muscle were studied in the myenteric plexus-longitudinal muscle preparation of the guinea pig. Acetylcholine was determined as radiolabelled transmitter from strips preloaded with 3H-choline. 2. GABA (1–300 μM) caused an increase in resting tension of smooth muscle as well as an increase in release of acetylcholine that was considerably reduced by tetrodotoxin. The facilitation by GABA of acetylcholine release exhibited a marked tachyphylaxis. The increase in muscle tension was clearly related to the increase in acetylcholine release. Muscimol (0.1–10 μM) also enhanced the release of acetylcholine. The eff…

medicine.medical_specialtyGuinea PigsIn Vitro TechniquesBicucullineTritiumSynaptic Transmissiongamma-Aminobutyric acidchemistry.chemical_compoundIleumInternal medicineMuscle tensionMuscarinic acetylcholine receptormedicineAnimalsPicrotoxingamma-Aminobutyric AcidPharmacologyMuscimolMuscle SmoothGeneral MedicineBicucullineAcetylcholineElectric StimulationEndocrinologynervous systemchemistryMuscimolCholinergicAcetylcholinemedicine.drugPicrotoxinMuscle ContractionNaunyn-Schmiedeberg's archives of pharmacology
researchProduct

Glucose plus choline improve passive avoidance behaviour and increase hippocampal acetylcholine release in mice.

2001

The present study tests the effects of glucose and choline, the biosynthetic precursors of acetylcholine, on passive avoidance behaviour and hippocampal acetylcholine release measured by microdialysis in awake mice. Glucose (10 and 30mg/kg) or choline chloride (6-60mg/kg), given by i.p. injection immediately after training, dose-dependently enhanced retention in an inhibitory avoidance task. Combinations of low doses of glucose (10mg/kg) and choline chloride (20mg/kg) which alone were submaximally effective significantly increased retention latencies in a synergistic manner, an effect which was sensitive to atropine (0.5mg/kg). This beneficial effect vanished when higher doses of glucose or…

medicine.medical_specialtyMicrodialysisMicrodialysisHippocampal formationHippocampusSynaptic TransmissionCholinechemistry.chemical_compoundMiceMemoryInternal medicinemedicineAvoidance LearningCholineAnimalsNeurotransmitterNootropic AgentsMice Inbred BALB CGeneral NeuroscienceAcetylcholineAtropineEndocrinologyGlucosechemistryExploratory BehaviorCholinergicFemaleAcetylcholinemedicine.drugCholine chlorideNeuroscience
researchProduct

Study and modulation of human cortical excitability with transcranial magnetic stimulation.

1998

Transcranial magnetic stimulation (TMS) can be applied in different paradigms to obtain a measure of various aspects of cortical excitability. These different TMS paradigms provide information about different neurotransmitter systems, enhance our understanding about the pathophysiology of neuropsychiatric conditions, and in the future may be helpful as a guide for pharmacological interventions. In addition, repetitive TMS (rTMS) modulates cortical excitability beyond the duration of the rTMS trains themselves. Depending on rTMS parameters, a lasting inhibition or facilitation of cortical excitability can be induced. These effects can be demonstrated neurophysiologically or by combining rTMS…

medicine.medical_specialtyNeurologyPhysiologyNerve netmedicine.medical_treatmentNeural ConductionNeural Inhibitionbehavioral disciplines and activitiesSynaptic TransmissionElectromagnetic FieldsNeuroimagingPhysiology (medical)Physical Stimulationmental disordersNeural PathwaysmedicineHumansEvoked PotentialsNeurorehabilitationCerebral CortexDepressive DisorderMovement Disordersbusiness.industrymusculoskeletal neural and ocular physiologyNeural InhibitionTranscranial magnetic stimulationmedicine.anatomical_structurenervous systemNeurologyCerebral cortexNeurology (clinical)Nerve NetbusinessNeurosciencepsychological phenomena and processesPhysical StimulationJournal of clinical neurophysiology : official publication of the American Electroencephalographic Society
researchProduct

IRS-2 deficiency impairs NMDA receptor-dependent long-term potentiation

2011

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License.-- et al.

medicine.medical_specialtyPatch-Clamp TechniquesCognitive Neurosciencemedicine.medical_treatmentBlotting WesterneducationHippocampusComputingMilieux_LEGALASPECTSOFCOMPUTINGNeurotransmissionBiologyHippocampusReceptors N-Methyl-D-AspartateSynaptic TransmissionMiceCellular and Molecular NeuroscienceInternal medicinemedicineAnimalsImmunoprecipitationlong-term potentiationMice Knockoutsynaptic plasticitydiabetesInsulinDiabetesLong-term potentiationArticlesNMDA receptorIRS2insulin receptor signalingSynaptic fatigueEndocrinologynervous systemSynaptic plasticityInsulin Receptor Substrate ProteinsNMDA receptorFemaleNeuroscienceCerebral Cortex
researchProduct

Acetylcholine overflow during infusion of a high potassium-low sodium solution into the perfused chicken heart in the absence and presence of physost…

1977

1. The effect of infusion of a modified Tyrode's solution (“high K+-low Na+ solution”) into the isolated chicken heart on the content of acetylcholine in the tissue and the overflow of acetylcholine were compared to those evoked by vagal stimulation. 2. The release of acetylcholine was measured over 15-min periods of either stimulation of the vagus nerves (40 V, 1 ms) at 20 Hz or of infusion of the high K+-low Na+ solution (108 mM K+, 44 mM Na+). 3. Both stimuli caused a maximum overflow of acetylcholine in the first few minutes whether or not 10−6 M physostigmine was present. The overflow was maintained during the vagal stimulation at a constant rate of at least 35% the initial rate, where…

medicine.medical_specialtyPhysostigmineSodiumPhysostigminechemistry.chemical_elementStimulationSynaptic Transmissionchemistry.chemical_compoundInternal medicinemedicineCholineAnimalsPharmacologyMyocardiumPotassium lowSodiumGeneral MedicineAcetylcholinePerfusionSolutionsEndocrinologychemistrycardiovascular systemPotassiumSteady state (chemistry)PerfusionChickensAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
researchProduct

Modulatory action of acetylcholine on cerebrovascular sympathetic neurotransmission

1991

1. Acetylcholine (10 micrograms/min) diminished the electrically-induced cerebral blood flow reductions. Atropine (1-2 mg) partially blocked this inhibitory effect. 2. Exogenously administered noradrenaline (1-10 micrograms) and tyramine (50-500 micrograms) reduced cerebral blood flow but this effect was unchanged by acetylcholine infusion. 3. Acetylcholine inhibited the nonadrenergic component of the electrically-induced contraction at a concentration greater than or equal to 10(-6) M and potentiated the adrenergic component at a concentration greater than or equal to 10(5) M. Atropine 10(-7) M) inhibited both of these effects. In addition, acetylcholine (10(-4) M) enhanced the electricall…

medicine.medical_specialtySympathetic Nervous SystemContraction (grammar)Cerebral arteriesTyramineAdrenergicTetrodotoxinIn Vitro TechniquesSynaptic TransmissionMuscle Smooth VascularNorepinephrinechemistry.chemical_compoundIsometric ContractionInternal medicineMuscarinic acetylcholine receptormedicineAnimalsPharmacologyChemistryGoatsMuscarinic acetylcholine receptor M3Cerebral ArteriesTyramineAcetylcholineElectric StimulationAtropineEndocrinologyCerebrovascular CirculationFemaleAcetylcholinemedicine.drugGeneral Pharmacology: The Vascular System
researchProduct