Search results for "Synovial sarcoma"

showing 7 items of 17 documents

p16INK4A (CDKN2A) gene deletion is a frequent genetic event in synovial sarcomas.

2006

We assessed the frequency of genomic deletion of p16 INK4A (CDKN2A) in synovial sarcomas (SSs) and its possible association with immunoexpression of p16 and cyclin D1 and the Ki-67 proliferation index using dualcolor fluorescence in situ hybridization (FISH) on tissue microarray sections of 41 histologically and molecularly confirmed SSs. A heterozygous p16 INK4A gene deletion was identified in 28 (74%) of 38 cases, with 25 (89%) of them showing abnormal p16 protein expression (20 negative and 5 heterogeneous). Of 25 cases, 19 (76%) exhibiting increased cyclin D1 expression also demonstrated heterozygous p16 INK4A deletion. No significant association was observed between p16 INK4A deletion …

HeterozygoteProliferation indexTumor suppressor geneSoft Tissue NeoplasmsBiologySarcoma SynovialCyclin D1CDKN2ACyclin DCyclinsmedicineBiomarkers TumorHumansCDKN2A Gene DeletionCyclin-Dependent Kinase Inhibitor p16In Situ Hybridization FluorescenceCell Nucleusmedicine.diagnostic_testGeneral Medicinemedicine.diseaseImmunohistochemistrySynovial sarcomaKi-67 AntigenTumor progressionTissue Array AnalysisCancer researchGene DeletionFluorescence in situ hybridizationAmerican journal of clinical pathology
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PML down-regulation in soft tissue sarcomas

2010

To date, little is known concerning the promyelocytic leukemia gene (PML) status in tumors of different origin, and its expression has never been evaluated in soft tissue sarcoma. The aim of the present study is focused on the identification of differences in terms of PML protein expression between different types of soft tissue sarcoma and the corresponding normal surrounding tissue. PML protein expression has been assessed by immunohistochemistry in six different histologic types of soft tissue sarcoma (synovial sarcoma, myofibroblastic sarcoma, angiosarcoma, liposarcoma, pleomorphic sarcoma, and leiomyosarcoma) and in the corresponding normal surrounding tissue. PML resulted significantl…

LeiomyosarcomaPathologymedicine.medical_specialtyPhysiologysoft tissue tumorSettore MED/06 - Oncologia MedicaClinical BiochemistryDown-RegulationLiposarcomaPromyelocytic Leukemia ProteinPleomorphic LiposarcomaPML sarcomasPromyelocytic leukemia proteinmedicineHumanssarcomasneoplasmsMyxoid liposarcomaPMLbiologybusiness.industrySoft tissue sarcomaTumor Suppressor ProteinsNuclear ProteinsSarcomaCell BiologyAnatomymedicine.diseaseImmunohistochemistrySynovial sarcomabody regionsbiology.proteinSarcomabusinessTranscription Factors
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Treatment of soft tissue sarcoma in childhood and adolescence: A report of the german cooperative soft tissue sarcoma study

1992

Background In the first German soft tissue sarcoma (STS) study, CWS-81, 344 patients younger than 19 years of age who had previously untreated soft tissue sarcoma were studied. For this analysis, there were 218 patients with chemosensitive STS (Group A: rhabdomyosarcoma [RMS], synovial sarcoma, extraosseous Ewing sarcoma, leiomyosarcoma, undifferentiated sarcoma, and malignant peripheral neuroectodermal tumor) who could be studied for a minimum potential follow-up time of 6 years. Methods A staging system based on the postoperative extent of the disease was used. The chemotherapy for Stage I-III disease consisted of vincristine, dactinomycin, cyclophosphamide, and doxorubicin (VACA). Patien…

MaleReoperationLeiomyosarcomaCancer Researchmedicine.medical_specialtyVincristineAdolescentmedicine.medical_treatmentSoft Tissue NeoplasmsGastroenterologyInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansChildRhabdomyosarcomaCyclophosphamideSurvival rateNeoplasm StagingChemotherapybusiness.industrySoft tissue sarcomaSarcomaPrognosismedicine.diseaseCombined Modality TherapySurvival AnalysisSynovial sarcomaSurgeryOncologyChemotherapy AdjuvantDoxorubicinVincristineChild PreschoolDactinomycinRegression AnalysisFemaleSarcomabusinessFollow-Up Studiesmedicine.drugCancer
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Immunoreactivity using anti-ERG monoclonal antibodies in sarcomas is influenced by clone selection.

2014

The aim of the present study was to explore ERG immunoreactivity in a series of sarcomas, GIST and malignant rhabdoid tumor (MRT), considering the not fully elucidated specificity and sensitivity of this antibody. Paraffin-embedded tissue microarrays from those tumors were stained with anti-ERG against the C-terminus [(EPR3864(2)] and N-terminus (Clone 9FY). EPR3864(2) was positive in almost all angiosarcomas, and MRT.GIST were positive in a large proportion of cases (38.4%), and more than half the synovial sarcomas (52.7%) revealed EPR3864(2) staining. Several chondrosarcomas, osteosarcomas, rhabdomyosarcoma and Ewing's sarcoma family of tumors (ESFT) presented EPR3864(2) expression in a l…

Pathologymedicine.medical_specialtyGastrointestinal Stromal TumorsClone (cell biology)BiologySensitivity and SpecificityPathology and Forensic MedicineFusion geneTranscriptional Regulator ERGmedicineHumansRhabdomyosarcomaRhabdoid TumorRetrospective StudiesTissue microarrayBrain NeoplasmsSarcomasEwing's sarcomaAntibodies MonoclonalEwing's sarcomaSarcomaCell Biologymedicine.diseaseImmunohistochemistrySynovial sarcomaKidney NeoplasmsERGTrans-ActivatorsImmunohistochemistrySarcomaPathology, research and practice
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FU-3 monoclonal antibody: a specific marker for malignant fibrous histiocytoma? An analysis of 32 malignant soft tissue and bone sarcomas.

1994

An immunohistochemical study on frozen sections was carried out on 51 malignant tumours of soft tissue and bone using the FU-3 monoclonal antibody. This antibody is claimed to be specific for malignant fibrous histiocytoma (MFH) and liposarcoma and for normal and tumour cells located in perivascular fields. The results show a lack of specificity in MFH staining: several malignant tumours such as synovial sarcoma, fibrosarcoma, rhabdomyosarcoma, osteogenic sarcoma, and including an anaplastic malignant melanoma, presented positive staining somewhat similar to that found in MFH. The value of this antibody in the differential diagnosis of MFH is doubtful. It might be useful to recognize a comm…

Pathologymedicine.medical_specialtyHistiocytoma Benign Fibrousbusiness.industrySoft tissueAntibodies MonoclonalBone NeoplasmsSarcomaCell BiologyGeneral MedicineBone SarcomaLiposarcomamedicine.diseaseImmunohistochemistrySynovial sarcomaPathology and Forensic MedicinemedicineImmunohistochemistryHumansSarcomaFibrosarcomabusinessRhabdomyosarcomaMolecular BiologyVirchows Archiv : an international journal of pathology
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Pulmonary metastasectomy in soft tissue sarcomas: a systematic review

2021

Background Soft tissue sarcoma (STS) tend to metastasis to the lungs. Pulmonary metastasectomy seems to be a common practice always when plausible. The objective of this article was to review systematically the results of a literature search on pulmonary metastasectomy for STSs published in the last ten years and to offer a brief overview about the current practice as well. Methods Eight retrospective studies published in the period 2010-2020, which included patients with pulmonary metastases and metastasectomy were selected. Indication for surgery, survival rate and factors influencing survival were the primary outcomes, while further interesting findings in the studies were also collected…

Pulmonary and Respiratory MedicineLeiomyosarcomamedicine.medical_specialtybusiness.industrySoft tissue sarcomaOriginal Article on Pulmonary MetastasesSoft tissueRetrospective cohort studymedicine.diseaseSynovial sarcomaMetastasisMedicineRadiologyMetastasectomybusinessSurvival rateJournal of Thoracic Disease
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Mutational analysis of E-cadherin, β-catenin and APC genes in synovial sarcomas

2010

medicine.medical_specialtyMutationPathologyHistologyCadherinCell adhesion moleculeCancerAnatomical pathologyGeneral MedicineBiologymedicine.disease_causemedicine.diseaseSynovial sarcomaPathology and Forensic MedicineCateninmedicineCancer researchGeneHistopathology
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