Search results for "T cell"

showing 10 items of 2228 documents

Stimulation of human T cells by microbial 'superantigens'.

1991

The enterotoxins and the TSST of S. aureus, the erythrogenic toxins A and C of S. pyogenes and a still uncharacterized exoprotein of M. arthritidis belong to a family of exotoxins that have in common a potent mitogenic activity for T lymphocytes of several species. These proteins stimulate CD4+ and C8+ T cells, as well as a fraction of gamma delta TCR-bearing T cells by cross-linking variable parts of the T cell antigen receptor with MHC class II molecules on accessory or target cells. They are functionally bivalent molecules having distinct interaction sites for variable parts of the TCR and for nonpolymorphic parts of the MHC class II molecule. For alpha beta TCR-bearing T cells the V bet…

Antigens BacterialT cellReceptors Antigen T-Cell alpha-betaT-LymphocytesImmunologyCD1CD28ExotoxinsStreptamerMHC restrictionBiologyIn Vitro TechniquesLymphocyte ActivationMicrobiologyInterleukin 21Enterotoxinsmedicine.anatomical_structuremedicineCytotoxic T cellHumansMitogensAntigen-presenting cellImmunologic research
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Requirements for the growth of TH1 lymphocyte clones.

1990

Besides the signal generated in a T lymphocyte after triggering the T cell receptor (TcR), most lymphocytes need a "second signal" to become fully activated. The necessity and nature of the "second signal" differs between different types of T cells. At the level of CD4-positive T helper lymphocytes interleukin 1 (IL 1) serves as "second signal" for those of the TH2 subtype (IL4, 5, 6 producer) but not for those of the TH1 subtype (IL 2, IFN-gamma producer). This correlates with the absence of the IL 1 receptor at the surface of TH1 clones. We report herein the further purification of T cell stimulating factor (TSF), a soluble mediator involved in the proliferation of TH1 lymphocytes. A prep…

Antigens Differentiation T-LymphocyteCD3 Complexmedicine.medical_treatmentT cellLymphocyteImmunologyReceptors Antigen T-CellAntigen-Presenting CellsBiologyLymphocyte ActivationMicemedicineImmunology and AllergyAnimalsAntigen-presenting cellInterleukin 4Mice Inbred BALB CCell growthMacrophage Colony-Stimulating FactorMacrophagesT-cell receptorAntibodies MonoclonalReceptors Interleukin-2T lymphocyteT-Lymphocytes Helper-InducerMolecular biologyCytokinemedicine.anatomical_structureImmunologyInterleukin-1European journal of immunology
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Depletion of alloreactive T cells via CD69: implications on antiviral, antileukemic and immunoregulatory T lymphocytes

2005

Selective depletion of alloreactive T cells from stem-cell allografts should abrogate graft-versus-host disease while preserving beneficial T cell specificities to facilitate engraftment and immune reconstitution. We therefore explored a refined immunomagnetic separation strategy to effectively deplete alloreactive donor lymphocytes expressing the activation antigen CD69 upon stimulation, and examined the retainment of antiviral, antileukemic, and immunoregulatory T cells. In addition to the CD69high T cell fraction, our studies retrieved two T cell subsets based on residual CD69 expression. Whereas, truly CD69(neg) cells were devoid of detectable alloresponses to original stimulators, CD69…

Antigens Differentiation T-LymphocyteCD4-Positive T-LymphocytesEpstein-Barr Virus InfectionsHerpesvirus 4 HumanT cellCytomegalovirusGraft vs Host DiseaseCell Cycle Proteinschemical and pharmacologic phenomenaStreptamerBiologyLymphocyte ActivationLymphocyte DepletionCell LineInterleukin 21Antigens CDmedicineHumansTransplantation HomologousCytotoxic T cellLectins C-TypeIL-2 receptorAntigen-presenting cellTransplantationHematopoietic Stem Cell TransplantationNuclear ProteinsForkhead Transcription FactorsReceptors Interleukin-2hemic and immune systemsHematologyT lymphocyteNatural killer T cellDNA-Binding Proteinsmedicine.anatomical_structureCytomegalovirus InfectionsImmunologyRNA Splicing FactorsCarrier ProteinsImmunologic MemoryBone Marrow Transplantation
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Postnatal development of functional T cell subsets in the mouse: a frequency analysis of mitogen reactive precursors of proliferating, of cytotoxic a…

1985

In order to study the postnatal development of functional T cell subsets in the mouse, a mitogen-driven limiting dilution culture system was used for a precursor frequency analysis of proliferating, of cytolytic and of IL 2-producing T cells, respectively, present in spleen and thymus of mice from neonatal to adult age. In adult mice, the majority (up to 100%) of splenic T cells was capable to respond to Concanavalin A. In contrast, an up to tenfold lower frequency of mitogen-reactive precursors was found within positively selected Thy-1+ spleen cells of neonatal mice. Within this fraction of Con A reactive neonatal T cells, there was an apparent imbalance in the CTLp/PTLp ratio within the …

Antigens Differentiation T-LymphocyteCytotoxicity ImmunologicInterleukin 2T-LymphocytesCellular differentiationT cellImmunologySpleenThymus GlandLymphocyte ActivationAndrologyMice03 medical and health sciences0302 clinical medicineAntigenmedicineAnimalsAntigens LyImmunology and AllergyCytotoxic T cell030304 developmental biologyMice Inbred BALB C0303 health sciencesbiologyAge FactorsAntibodies MonoclonalCell DifferentiationHematologyCytolysismedicine.anatomical_structureAnimals NewbornConcanavalin AAntigens SurfaceImmunologyMice Inbred CBAbiology.proteinInterleukin-2Thy-1 AntigensSpleenT-Lymphocytes Cytotoxic030215 immunologymedicine.drugImmunobiology
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Identification of epitopes of Mycobacterium tuberculosis 16-kDa protein recognized by human leukocyte antigen-A*0201 CD8(+) T lymphocytes.

2002

CD8(+) T cells could make an important contribution to protection against tuberculosis (TB), but the antigenic determinants recognized in the context of major histocompatibility complex class I molecules remain ill defined. Our aim was to identify nonamer peptides derived from the acr/16-kDa antigen. Two immunogenic peptides (p21-29 and p120-128) were identified by their ability to elicit cytotoxic CD8(+) T cells from juvenile patients recovering from TB. Epitope-specific recognition was demonstrated by the lysis of both Mycobacterium tuberculosis-infected and peptide-pulsed macrophages, the release of cytotoxic granules, and interferon-gamma and tumor necrosis factor-alpha production. CD8(…

Antigens Differentiation T-LymphocyteCytotoxicity ImmunologicMalePore Forming Cytotoxic ProteinsT cellEpitopes T-LymphocyteHuman leukocyte antigenCD8-Positive T-LymphocytesMajor histocompatibility complexEpitopeInterferon-gammaImmune systemAntigenBacterial ProteinsHLA-A2 AntigenmedicineImmunology and AllergyCytotoxic T cellHumansChildTuberculosis PulmonaryMembrane GlycoproteinsbiologyHLA-A AntigensPerforinTumor Necrosis Factor-alphaMacrophagesMycobacterium tuberculosisFlow CytometryPeptide FragmentsMolecular WeightInfectious Diseasesmedicine.anatomical_structureImmunologybiology.proteinFemaleCD8The Journal of infectious diseases
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Clonal analysis of human T cell activation by the Mycoplasma arthritidis mitogen (MAS)

1988

Mycoplasma arthritidis produces an as yet undefined soluble molecule (MAS) that has a potent mitogenic effect on T cells of several species. We have used cloned human cytotoxic and proliferative T lymphocytes to dissect the molecular mechanism of T cell activation by this mitogen. Reactivity to MAS is clonally expressed among T cell receptor (TcR) alpha/beta chain-expressing T cell clones of CD4+ or CD8+ phenotype, as well as CD4-8- TcR alpha/beta chain-negative T lymphocyte clones expressing the CD3-associated TcR gamma chain. MAS is able to induce cytotoxicity and/or proliferation in these T cell clones. For triggering of these T cells, regardless of their phenotype of specificity, the pr…

Antigens Differentiation T-LymphocyteCytotoxicity ImmunologicT-LymphocytesT cellCD3ImmunologyReceptors Antigen T-CellStreptamerIn Vitro TechniquesBiologyLymphocyte ActivationAntigen-Antibody ReactionsInterleukin 21MycoplasmaSpecies SpecificitymedicineHumansImmunology and AllergyCytotoxic T cellAntigen-presenting cellAntigens BacterialHLA-D AntigensfungiNatural killer T cellVirologyMolecular biologyClone Cellsmedicine.anatomical_structurebiology.proteinCD8European Journal of Immunology
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Enzymatic Activity of CD26 (Dipeptidylpeptidase IV) is not Required for Its Signalling Function in T Cells

1993

Abstract CD26 is a proteolytic enzyme (dipeptidylpeptidase IV) expressed on the T cell surface that defines an alternative activation signal for human T lymphocytes. Crosslinking of CD26 via monoclonal antibodies triggers proliferation and cytotoxicity in preactivated T cells. In this study, we used highly specific competitive and irreversible inhibitors of dipeptidylpeptidase IV to study the role of the enzymatic activity in activation of CD26- transfected T cells as well as of CD26-expressing normal human T cell clones. These inhibitors at concentrations that blocked up to 95% of the enzymatic activity, did not specifically inhibit T cell activation neither via TCR/CD3 nor via CD26 itself…

Antigens Differentiation T-LymphocyteDipeptidyl Peptidase 4T-LymphocytesT cellCD3ImmunologyBiologyLymphocyte ActivationCell LineMiceTumor Cells CulturedmedicineAnimalsHumansImmunology and AllergyCytotoxic T cellDipeptidyl-Peptidases and Tripeptidyl-PeptidasesT-cell receptorProteolytic enzymesHematologyTransfectionT lymphocyteCytotoxicity Tests ImmunologicCell biologymedicine.anatomical_structureBiochemistrybiology.proteinInterleukin-2Clone (B-cell biology)Signal TransductionImmunobiology
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Function of Dipeptidyl Peptidase IV (CD26, Tp103) in Transfected Human T Cells

1993

CD26 (Tp103) is a proteolytic enzyme (dipeptidyl peptidase IV) expressed on the T cell surface that defines an alternative activation signal for human T lymphocytes. It is absent from or present in only low amounts on resting T cells but it is expressed strongly after activation. Crosslinking of CD26/Tp103 via the monoclonal antibody CB.1 triggers functional activities in preactivated T cells. To study the molecular requirements for T cell activation via CD26 we transfected a cDNA encoding CD26 into several CD26-negative cells. In Jurkat T cell leukemia cells that normally do not express the CD26 antigen, the transfected CD26 molecule is functional because the monoclonal antibody CB.1 induc…

Antigens Differentiation T-LymphocyteDipeptidyl Peptidase 4T-LymphocytesT cellZAP70ImmunologyT-cell receptorReceptors Antigen T-CellBiologyTransfectionNatural killer T cellMolecular biologyJurkat cellsRecombinant ProteinsCell LineMicemedicine.anatomical_structureAntigenTumor Cells CulturedmedicineAnimalsHumansCytotoxic T cellIL-2 receptorDipeptidyl-Peptidases and Tripeptidyl-PeptidasesCellular Immunology
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Involvement of soluble mediator(s) different from interleukin (IL)1 in the antigen-induced IL 2 receptor expression and proliferation of L3T4+ (CD4+)…

1988

Proliferation of T lymphocytes (T cells) requires the interaction of interleukin 2 (IL 2) with the high affinity form of the IL 2 receptor (IL 2R). IL 2 production as well as IL 2R expression are generally induced simultaneously in T cells by the recognition of specific antigen displayed on the surface of syngeneic antigen-presenting cells. The experiments described herein show that the expression of IL 2R has different requirements than the production of IL 2 (and other lymphokines). Stimulation of antigen-specific L3T4+ T cell lines with antigen-pulsed spleen cells (SC) treated with ultraviolet (UV) light results in efficient IL 2 production but only minimal proliferation due to reduced I…

Antigens Differentiation T-LymphocyteInterleukin 2Ultraviolet RaysT-LymphocytesT cellImmunologyAntigen-Presenting CellsBiologyLymphocyte ActivationMiceInterleukin 21medicineAnimalsImmunology and AllergyCytotoxic T cellIL-2 receptorAntigensReceptors ImmunologicInterleukin 3Mice Inbred BALB CMice Inbred C3HLymphokineReceptors Interleukin-2Molecular biologymedicine.anatomical_structureGene Expression RegulationImmunologyInterleukin 12Interleukin-2Cell DivisionSpleenmedicine.drugEuropean Journal of Immunology
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Peritoneal Cavity is a Route for Gut-Derived Microbial Signals to Promote Autoimmunity in Non-Obese Diabetic Mice

2015

Macrophages play a crucial role in innate immune reactions, and peritoneal macrophages (PMs) guard the sterility of this compartment mainly against microbial threat from the gut. Type 1 diabetes (T1D) is an autoimmune disease in which gut microbiota and gut immune system appear to contribute to disease pathogenesis. We have recently reported elevated free radical production and increased permeability of gut epithelium in non-obese diabetic (NOD) mice. Impaired barrier function could lead to bacterial leakage to the peritoneal cavity. To explore the consequences of impaired gut barrier function on extra-intestinal immune regulation, we characterized peritoneal lavage cells from young newly w…

Antigens Differentiation T-LymphocyteLipopolysaccharidesmedicine.medical_specialtymiceT-LymphocytesT cellBlotting WesternImmunologyWeaningNodBiologyta3111Peritoneal cavityImmune systemSpecies SpecificityAntigens CDMice Inbred NODInternal medicinediabeticmedicineAnimalsLectins C-TypeIntestinal Mucosamicrobial signalsCells CulturedNOD miceMice Inbred BALB CInnate immune systemTumor Necrosis Factor-alphanon-obeseMicrobiotaautoimmunityta1182ta3141General MedicineFlow CytometryGut EpitheliumIntestinesMice Inbred C57BLInterleukin-1 Receptor-Associated KinasesEndocrinologymedicine.anatomical_structureperitoneal cavityImmunologyMacrophages PeritonealTumor necrosis factor alphaInjections IntraperitonealSignal TransductionScandinavian Journal of Immunology
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