Search results for "T-box"

showing 10 items of 43 documents

Expression pattern of the Brachyury and Tbx2 homologues from the sponge Suberites domuncula.

2005

Background information. T-box transcription factors are a large family of transcriptional regulators involved in many aspects of embryonic development. In a previous report, we described the isolation and genomic characterization of two T-box genes from the siliceous sponge Suberites domuncula: a Brachyury homologue, Sd-Bra, and a Tbx2 homologue, Sd-Tbx2. Elucidation of the genomic structure of Sd-Bra allowed us to demonstrate the existence of two different isoforms, resulting from alternative splicing. Moreover, we demonstrated that the shorter isoform exists in two different glycosylation states. Results. In the present study, we demonstrate a differential subcellular localization of the …

Gene isoformFetal ProteinsBrachyuryCytoplasmGlycosylationBlotting WesternAnimalsProtein IsoformsGeneTranscription factorCells CulturedGeneticsRegulation of gene expressionCell NucleusbiologyAlternative splicingCell BiologyGeneral Medicinebiology.organism_classificationImmunohistochemistryPoriferaSuberites domunculaAlternative SplicingGene Expression RegulationT-Box Domain ProteinsFunction (biology)Biology of the cell
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A key pathogenic role for the STAT1/T-bet signaling pathway in T-cell-mediated liver inflammation.

2003

TH1 cytokines have been suggested to contribute to the pathogenesis of T-cell-mediated liver injury and inflammation. However, the molecular signaling pathways involved in such injury are still poorly understood. In the present study, we investigated the role of the STAT1/T-bet signaling pathway in a murine model of T-cell-mediated liver inflammation induced by the application of concanavalin A (Con A) using newly created STAT1 transgenic mice as well as STAT1- and T-bet-deficient mice. Liver injury induced by Con A was associated with an increase of both pSTAT1 and T-bet levels in the liver. Furthermore, functional studies suggested a pathogenic role for STAT1 in Con A-induced liver injury…

Genetically modified mouseT cellTransgeneT-LymphocytesInflammationMice TransgenicBiologyHepatitisInterferon-gammaMicemedicineConcanavalin AAnimalsInterferon gammaLiver injuryHepatologymedicine.diseasePhosphoproteinsDNA-Binding ProteinsMice Inbred C57BLIRF1medicine.anatomical_structureSTAT1 Transcription FactorLiverImmunologyTrans-ActivatorsSignal transductionmedicine.symptomT-Box Domain Proteinsmedicine.drugInterferon Regulatory Factor-1Signal TransductionTranscription FactorsHepatology (Baltimore, Md.)
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optomotor-blind suppresses instability at the A/P compartment boundary of the Drosophila wing.

2008

Formation and function of the A/P compartment boundary of the Drosophila wing have been studied intensely. The boundary prevents mingling of A and P cells, is characterized by an expression discontinuity of several genes like engrailed, Cubitus interruptus, hedgehog and decapentaplegic and is essential for patterning the wing. Compared with segmental or compartmental boundaries in several other systems which generally manifest as folds or clefts, the wing A/P boundary is morphologically inconspicuous in both the larval and adult stage. We show here that the Drosophila wing A/P boundary, too, is susceptible to fold and cleft formation and that these processes are suppressed by the T-box tran…

GeneticsEmbryologyanimal structuresWingDecapentaplegicMorphogenesisGene Expression Regulation DevelopmentalNerve Tissue ProteinsBiologyMicrotubulesengrailedCell biologyAdherens junctionCompartment (development)AnimalsDrosophila ProteinsWings AnimalDrosophilaEnhancerT-Box Domain ProteinsHedgehogDevelopmental BiologyBody PatterningSequence DeletionMechanisms of development
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Spatial discontinuity of Optomotor-blind expression in the Drosophila wing imaginal disc disrupts epithelial architecture and promotes cell sorting

2010

Abstract Background Decapentaplegic (Dpp) is one of the best characterized morphogens, required for dorso-ventral patterning of the Drosophila embryo and for anterior-posterior (A/P) patterning of the wing imaginal disc. In the larval wing pouch, the Dpp target gene optomotor-blind (omb) is generally assumed to be expressed in a step function above a certain threshold of Dpp signaling activity. Results We show that the transcription factor Omb forms, in fact, a symmetrical gradient on both sides of the A/P compartment boundary. Disruptions of the Omb gradient lead to a re-organization of the epithelial cytoskeleton and to a retraction of cells toward the basal membrane suggesting that the O…

GeneticsWinganimal structuresbiologyDecapentaplegicMorphogenesisNerve Tissue ProteinsCell sortingbiology.organism_classificationCell biologyImaginal discDrosophila melanogasterlcsh:Biology (General)Research articleAnimalsDrosophila ProteinsWings AnimalCompartment (development)Drosophila melanogasterT-Box Domain Proteinslcsh:QH301-705.5Drosophila ProteinSignal TransductionDevelopmental BiologyBMC Developmental Biology
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The Dorsocross T-box transcription factors promote tissue morphogenesis in the Drosophila wing imaginal disc.

2012

The Drosophila wing imaginal disc is subdivided into notum, hinge and blade territories during the third larval instar by formation of several deep apical folds. The molecular mechanisms of these subdivisions and the subsequent initiation of morphogenic processes during metamorphosis are poorly understood. Here, we demonstrate that the Dorsocross (Doc) T-box genes promote the progression of epithelial folds that not only separate the hinge and blade regions of the wing disc but also contribute to metamorphic development by changing cell shapes and bending the wing disc. We found that Doc expression was restricted by two inhibitors, Vestigial and Homothorax, leading to two narrow Doc stripes…

Integrinsanimal structuresTime FactorsMorphogenesisBiologyMicrotubulesExtracellular matrixMicrotubuleMorphogenesisAnimalsDrosophila ProteinsWings AnimalTransgenesMolecular BiologyAllelesWingAnatomyNotumCell biologyExtracellular MatrixImaginal discT-boxDrosophila melanogasterMutationMatrix Metalloproteinase 2RNA InterferenceDrosophila ProteinDevelopmental BiologyProtein BindingSignal TransductionTranscription FactorsDevelopment (Cambridge, England)
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Regulation of T cells in asthma: implications for genetic manipulation

2004

PURPOSE OF THE REVIEW Allergic asthma is a disease characterized by airway hyperresponsiveness, inflammation and remodeling. In the past few decades it has become clear that the pathogenesis and development of this disease is controlled by cytokines released by CD4 T helper type 2 lymphocytes that develop under the influence of natural killer lymphocytes. At birth, T cell priming exhibits a T helper type 2 bias and the development of the T helper phenotype is determined in the first year of life by environmental exposure to virus or bacterial substances or environmental allergens in genetically predisposed individuals. Decreased exposure to infection in early childhood has thus been linked …

LipopolysaccharidesT-LymphocytesT cellImmunologyPriming (immunology)Receptors Cell SurfaceInflammationBiologyType 2 immune responseImmune systemAntigenHygiene hypothesismedicineHumansImmunology and AllergyGeneticsMembrane GlycoproteinsToll-Like ReceptorsT-Lymphocytes Helper-InducerEnvironmental exposureAsthmamedicine.anatomical_structureImmunologyCytokinesmedicine.symptomT-Box Domain ProteinsTranscription FactorsCurrent Opinion in Allergy and Clinical Immunology
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Diagnostic strategy in segmentation defect of the vertebrae: a retrospective study of 73 patients

2018

BackgroundSegmentation defects of the vertebrae (SDV) are non-specific features found in various syndromes. The molecular bases of SDV are not fully elucidated due to the wide range of phenotypes and classification issues. The genes involved are in the Notch signalling pathway, which is a key system in somitogenesis. Here we report on mutations identified in a diagnosis cohort of SDV. We focused on spondylocostal dysostosis (SCD) and the phenotype of these patients in order to establish a diagnostic strategy when confronted with SDV.Patients and methodsWe used DNA samples from a cohort of 73 patients and performed targeted sequencing of the five known SCD-causing genes (DLL3,MESP2,LFNG,HES7…

Male0301 basic medicineOncologymedicine.medical_specialtyCandidate geneAdolescent030105 genetics & heredityspondylocostal dysostosisdiagnostic strategysegmentation defect of the vertebraewhole exome sequencingLFNG03 medical and health sciencesgene panelInternal medicineExome SequencingBasic Helix-Loop-Helix Transcription FactorsGeneticsmedicineHumansFLNBChildGenetics (clinical)Exome sequencingBone Diseases Developmentalbusiness.industryIntracellular Signaling Peptides and ProteinsGlycosyltransferasesInfantMembrane ProteinsRetrospective cohort studymedicine.diseasePhenotypeSpineSpondylocostal dysostosisPedigreePhenotype[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsChild PreschoolMutationCohortFemaleT-Box Domain Proteinsbusiness
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The T-box transcription factor eomesodermin controls CD8 T cell activity and lymph node metastasis in human colorectal cancer.

2007

An efficient cytolytic T cell function is essential for immune mediated rejection of colorectal cancer. However, the molecular mechanisms driving T cell mediated cancer rejection are still poorly understood. Here, we assessed the relevance of the T-box transcription factor eomesodermin in colorectal cancer. METHODS/ RESULTS: By analysing tissue probes from 88 different colorectal tumours, a significant (p0.02) inverse correlation between eomesodermin expression in colorectal cancers and the presence of lymph node metastases could be shown, whereas no such correlation was noted for the master transcription factor of regulatory T cells, FoxP3 and CD8 alpha expression. To evaluate whether this…

MaleT cellEomesoderminEnzyme-Linked Immunosorbent AssayCD8-Positive T-LymphocytesTransforming Growth Factor betamedicineCytotoxic T cellHumansTranscription factorColorectal Cancerbiologybusiness.industryReverse Transcriptase Polymerase Chain ReactionGastroenterologyCancerT lymphocytemedicine.diseasemedicine.anatomical_structurePerforinLymphatic MetastasisImmunologybiology.proteinFemaleInterleukin-4businessColorectal NeoplasmsT-Box Domain ProteinsCD8Gut
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Partial Sleep Restriction Activates Immune Response-Related Gene Expression Pathways: Experimental and Epidemiological Studies in Humans

2013

Epidemiological studies have shown that short or insufficient sleep is associated with increased risk for metabolic diseases and mortality. To elucidate mechanisms behind this connection, we aimed to identify genes and pathways affected by experimentally induced, partial sleep restriction and to verify their connection to insufficient sleep at population level. The experimental design simulated sleep restriction during a working week: sleep of healthy men (N = 9) was restricted to 4 h/night for five nights. The control subjects (N = 4) spent 8 h/night in bed. Leukocyte RNA expression was analyzed at baseline, after sleep restriction, and after recovery using whole genome microarrays complem…

MaleTBX21NF-KAPPA-Blcsh:MedicineNK cellsBioinformaticskokeellinen tutkimusReceptors G-Protein-Coupled0302 clinical medicineCARDIOMETABOLIC RISKLeukocytesta319geeniekspressiolcsh:Scienceta515Sleep restrictionRegulation of gene expression0303 health sciencesMultidisciplinaryNATURAL-KILLERNF-kappa Bta3142Sleep in non-human animalsC-REACTIVE PROTEIN3. Good healthMACROPHAGE APOPTOSISINSUFFICIENT SLEEPSTAT1 Transcription FactorCARDIOVASCULAR-DISEASEimmuunivasteProteoglycansmedicine.symptomResearch ArticleAdulteducationENDOPLASMIC-RETICULUMMETABOLIC CONSEQUENCESSyntaxin 16Biologyepidemiologinen tutkimusuni (lepotila)03 medical and health sciencesImmune systemmedicineHumans030304 developmental biologyTOLL-LIKE RECEPTORSB cellsuniMicroarray analysis techniquesGene Expression Profilingsytokiinitlcsh:RMicroarray AnalysisGene expression profilingSleep deprivationGene Expression RegulationImmunologyRNASleep Deprivationlcsh:Q3111 BiomedicineT-Box Domain ProteinsReceptors Transforming Growth Factor beta030217 neurology & neurosurgery
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T-bet as a possible therapeutic target in autoimmune disease

2002

The prominent role of pro-inflammatory cytokines produced by T helper-1 (T(H1)) cells in regulating autoimmune responses in vitro and in vivo has been demonstrated. Recent observations of T cell polarisation by regulatory transcription factors--especially T-bet (T-box expressed in T cells)--raise the question of their influence in controlling autoimmune diseases. Here, the authors summarise recent observations of the role of T-bet in controlling chronic inflammatory and autoimmune diseases and discuss the implications of these findings for future therapeutic approaches.

Mice Inbred MRL lprTranscription GeneticTransgeneT cellCellular differentiationClinical BiochemistryMice TransgenicLymphocyte ActivationAutoimmune DiseasesInterferon-gammaMiceTh2 CellsCrohn DiseaseDrug DiscoverymedicineAnimalsLupus Erythematosus SystemicIL-2 receptorIntestinal MucosaMice KnockoutPharmacologyAutoimmune diseaseLupus erythematosusbusiness.industryZAP70Cell DifferentiationTh1 CellsColitisInflammatory Bowel Diseasesmedicine.diseaseCeliac DiseaseDisease Models Animalmedicine.anatomical_structureCTLA-4ImmunologyCytokinesMolecular MedicineT-Box Domain ProteinsbusinessTranscription FactorsExpert Opinion on Therapeutic Targets
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