Search results for "THD"

showing 10 items of 129 documents

Different brain oxidative and neuroinflammation status in rats during prolonged abstinence depending on their ethanol relapse-like drinking behavior:…

2022

Rationale: Accumulating evidence suggests that chronic alcohol consumption is associated with excessive oxidative damage and neuroinflammatory processes and these events have been associated to early alcohol withdrawal. In the present research we wonder if brain oxidative stress and neuroinflammation remains altered during prolonged withdrawal situations and whether these alterations can be correlated with relapse behavior in alcohol consumption. The effects of alcohol reintroduction were also evaluated Methods: We have used a model based on the alcohol deprivation effect (ADE) within a cohort of wild-type male Wistar rats. Two subpopulations were identified according to the alcohol relapse…

MalePharmacologyAlcohol Efectes fisiològicsAlcohol DrinkingEthanolEstrès oxidatiuPrefrontal CortexToxicologyRatsSubstance Withdrawal SyndromeAlcoholismOxidative StressPsychiatry and Mental healthAlcohol relapseAlcohol deprivation effectNeuroinflammationRecurrenceOxidative stressNeuroinflammatory DiseasesAnimalsPharmacology (medical)Rats WistarOxidation-ReductionCraving
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Effects of desipramine and alprazolam in the forced swim test in rats after long-lasting termination of chronic exposure to picrotoxin and pentylenet…

1993

Abstract Rats were treated for 5 weeks with three subconvulsant doses of picrotoxin (PTX) and pentylenetetrazol (PTZ) per week to induce a persistent reduction of the GABA A receptor function which results in chemical kindling. Fifteen days after termination of this treatment schedule, the effect of desipramine (DMI) and alpraxolam (ALP) on immobility time in the forced swim test (FST) was evaluated. Chronic PTX and PTZ did not alter the immobility time. Acute PTX and PTZ reduced the immobility of rats chronically treated with vehicle but not of those exposed chronically to PTX and PTZ. Chronic PTX did not influence the anti-immobility effect of DMI, but blocked that of ALP. Chronic PTZ mar…

MalePharmacologyMotor ActivityChlordiazepoxidechemistry.chemical_compoundDesipraminemedicineAnimalsPicrotoxinPharmacology (medical)GABA-A Receptor AntagonistsPentylenetetrazolBiological PsychiatrySwimmingPharmacologyAlprazolamGABAA receptorKindlingbusiness.industryDesipramineChlordiazepoxideRatsSubstance Withdrawal SyndromePsychiatry and Mental healthNeurologyAlprazolamchemistryPentylenetetrazoleNeurology (clinical)businesshuman activitiesPsychomotor Performancemedicine.drugBehavioural despair testPicrotoxinEuropean neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology
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Adolescent but not adult ethanol binge drinking modulates cocaine withdrawal symptoms in mice.

2016

Background Ethanol (EtOH) binge drinking is an increasingly common behavior among teenagers that induces long-lasting neurobehavioral alterations in adulthood. An early history of EtOH abuse during adolescence is highly correlated with cocaine addiction in adulthood. Abstinence of cocaine abuse can cause psychiatric symptoms, such as anxiety, psychosis, depression, and cognitive impairments. This study assessed the consequences of adolescent exposure to EtOH on the behavioral alterations promoted by cocaine withdrawal in adulthood. Methods We pretreated juvenile (34-47 days old) or adult (68-81 days old) mice with EtOH (1.25 g/kg) following a binge-drinking pattern. Then, after a three-week…

MalePhysiologylcsh:MedicineAdolescentsOpen fieldMice0302 clinical medicineCocaineMedicine and Health Scienceslcsh:SciencePrepulse inhibitionmedia_commonMammalsMultidisciplinaryAlcohol ConsumptionAnimal BehaviorDepressionAge FactorsSubstance Withdrawal SyndromeChemistryBehavioral PharmacologyPhysical SciencesVertebratesResearch Articlemedicine.medical_specialtyElevated plus mazeAlcohol Drinkingmedia_common.quotation_subjectBinge drinkingRodents03 medical and health sciencesAlkaloidsInternal medicineRecreational Drug Usemental disordersMental Health and PsychiatrymedicineAnimalsAdultsNutritionPharmacologyBehaviorbusiness.industryMood DisordersBiological LocomotionAddictionlcsh:RChemical CompoundsOrganismsBiology and Life SciencesAbstinenceTail suspension test030227 psychiatryDietEndocrinologyAnxiogenicAge GroupsAmniotesPeople and Placeslcsh:QPopulation GroupingsbusinessZoology030217 neurology & neurosurgeryPloS one
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The impact of chronic daily nicotine exposure and its overnight withdrawal on the structure of anxiety-related behaviors in rats: Role of the lateral…

2021

Tobacco smoking is a serious health problem worldwide and a leading cause of mortality. Nicotine, the addictive component of tobacco, affects a range of emotional responses, including anxiety-related behaviors. Although perceived by smokers to be anxiolytic, evidence suggests that smoking increases anxiety and that mood fluctuates with nicotine intake. Thus, nicotine addiction may depend on easing the psychobiological distress caused by its abuse. The lateral habenula (LHb) has been implicated as a neural substrate for acute nicotine-induced anxiety, but its role in anxiety-like behaviors associated with chronic nicotine exposure has not been explored. Here, we assessed the effect of chroni…

MaleSerotoninNicotinemedicine.drug_classmedia_common.quotation_subjectmedicine.medical_treatmentPhysiologyAnxietyT-pattern analysisSettore BIO/09 - FisiologiaAnxiolyticNicotineRats Sprague-Dawley03 medical and health sciences0302 clinical medicineBehavior; Rats; Serotonin; T-pattern analysis; Withdrawal symptomsmedicineAnimalsBiological Psychiatrymedia_commonPharmacologyNeuronsBehaviorHabenulaWithdrawal symptombusiness.industryAddictionT-pattern analysimedicine.disease030227 psychiatryRatsSubstance Withdrawal SyndromeNicotine withdrawalMoodAnxiogenicExploratory BehaviorRatSmoking cessationAnxietyWithdrawal symptomsmedicine.symptombusinessmedicine.drugProgress in neuro-psychopharmacologybiological psychiatry
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Adolescent Exposure to the Synthetic Cannabinoid WIN 55212-2 Modifies Cocaine Withdrawal Symptoms in Adult Mice

2017

Chronic cannabinoid consumption is an increasingly common behavior among teenagers and has been shown to cause long-lasting neurobehavioral alterations. Besides, it has been demonstrated that cocaine addiction in adulthood is highly correlated with cannabis abuse during adolescence. Cocaine consumption and subsequent abstinence from it can cause psychiatric symptoms, such as psychosis, cognitive impairment, anxiety, and depression. The aim of the present research was to study the consequences of adolescent exposure to cannabis on the psychiatric-like effects promoted by cocaine withdrawal in adult mice. We pre-treated juvenile mice with the cannabinoid CB1 receptor agonist WIN 55212-2 (WIN)…

MalecannabisCannabinoid receptormedicine.medical_treatment0302 clinical medicineCocaineDopamine Uptake InhibitorsReceptor Cannabinoid CB1Spectroscopymedia_commoncocaine withdrawalBehavior Animalbiologyadolescence; cannabis; WIN 55212-2; cocaine withdrawal; miceGeneral MedicineSubstance Withdrawal SyndromeComputer Science ApplicationsHindlimb SuspensionElevated plus mazePsychosismedicine.medical_specialtymiceMorpholinesmedia_common.quotation_subjectNaphthalenesArticleCatalysisInorganic ChemistryCocaine-Related Disorders03 medical and health sciencesMemoryInternal medicinemedicineAnimalsWIN 55212-2Physical and Theoretical ChemistryPsychiatryMolecular BiologyCannabinoid Receptor Agonistsbusiness.industryAddictionOrganic ChemistryAbstinencemedicine.diseasebiology.organism_classificationBenzoxazines030227 psychiatryEndocrinologyAnxiogenicExploratory BehavioradolescenceCannabisCannabinoidbusiness030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
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Germline copy number variation in theYTHDC2gene: does it have a role in finding a novel potential molecular target involved in pancreatic adenocarcin…

2014

Abstract: Objective: The vast majority of pancreatic cancers occurs sporadically. The discovery of frequent variations in germline gene copy number can significantly influence the expression levels of genes that predispose to pancreatic adenocarcinoma. We prospectively investigated whether patients with sporadic pancreatic adenocarcinoma share specific gene copy number variations (CNVs) in their germline DNA. Patients and methods: DNA samples were analyzed from peripheral leukocytes from 72 patients with a diagnosis of sporadic pancreatic adenocarcinoma and from 60 controls using Affymetrix 500K array set. Multiplex ligation-dependent probe amplification (MLPA) assay was performed using a s…

Malecopy number variations germline alteration pancreatic cancer susceptibility YTHDC2 geneDNA Copy Number VariationsSettore MED/06 - Oncologia MedicaClinical BiochemistryAdenocarcinomaBiologyGermlinePancreatic cancerDrug DiscoverymedicineHumansGenetic Predisposition to DiseaseMultiplexProspective StudiesMultiplex ligation-dependent probe amplificationCopy-number variationAlleleGeneGerm-Line MutationAgedAdenosine TriphosphatasesAged 80 and overPharmacologyPharmacology. TherapyDNA HelicasesMiddle Agedmedicine.diseaseMolecular biologyPancreatic NeoplasmsCase-Control StudiesMolecular MedicineAdenocarcinomaFemaleMultiplex Polymerase Chain ReactionRNA HelicasesExpert Opinion on Therapeutic Targets
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Cannabidiol prevents several of the behavioral alterations related to cocaine addiction in mice.

2021

Abstract Cocaine dependence is a highly prevalent disease in modern society and lacks an effective treatment. Cannabidiol (CBD), a major non-psychoactive constituent of Cannabis sativa, has been shown to be a promising tool in the management of some neuropsychiatric disorders, including cocaine abuse. However, its therapeutic effects on the behavioral outcomes related to cocaine addiction remain unclear. The present research evaluates the effects of CBD (30, 60 and 120 mg/kg; injected intraperitoneally) on the acquisition, expression, extinction and reinstatement of cocaine (10 mg/kg)-induced conditioned place preference (CPP; Study 1); cocaine (25 mg/kg)-induced locomotor stimulation (Stud…

Malemedia_common.quotation_subjectConditioning ClassicalPharmacologyHyperkinesisCocaine dependenceExtinction Psychological03 medical and health sciencesCocaine-Related DisordersMice0302 clinical medicineCocainemedicineAnimalsCannabidiolBiological Psychiatrymedia_commonPharmacologyDose-Response Relationship Drugbusiness.industryAddictionTherapeutic effectExtinction (psychology)Abstinencemedicine.diseaseConditioned place preferenceTail suspension test030227 psychiatrySubstance Withdrawal SyndromeMice Inbred C57BLbusinessCannabidiolInjections IntraperitonealLocomotionmedicine.drugProgress in neuro-psychopharmacologybiological psychiatry
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The Hikikomori Phenomenon of Social Withdrawal: An Emerging Condition Involving Youth's Mental Health and Social Participation.

2020

The objective of this editorial is to raise awareness on the burden and risks faced by adolescents developing this emerging form of social withdrawal. The hikikomori phenomenon is part of the group of new morbidities causing children and adolescents to limit their activity owing to a chronic health condition with attendant psychological problems. We further emphasize the importance of including new morbidities involving mental health and social participation in formal pediatric training, to enable new generations of pediatricians to identify and properly manage these disorders.

Malemedicine.medical_specialtySocial psychology (sociology)Adolescentmedia_common.quotation_subjectMEDLINEhikikomori phenomenonShameSocial epidemiologyShameGlobal HealthYoung AdultSettore MED/38 - Pediatria Generale E SpecialisticamedicineGlobal healthHumansPsychiatrymedia_commonbusiness.industryPhobia SocialSocial engagementmedicine.diseaseMental healthHikikomoriSocial IsolationPediatrics Perinatology and Child HealthFemalebusinessHikikomori Phenomenon Social Withdrawal Youth Mental HealthThe Journal of pediatrics
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Delayed effects of ciprofibrate on rat liver peroxisomal properties and proto-oncogene expression.

1995

Peroxisome proliferators (PPs) are non-genotoxic carcinogens in rodents. Their reversible effects on rat liver have been studied with ciprofibrate and fenofibrate. We found that with the hypolipemic drug fenofibrate a pause of 28 days is sufficient for a return to normal status, whereas with the highly potent PP ciprofibrate, the stimulation of ACO mRNA levels remains after its withdrawal. We investigated the effects of the renewal of the treatment with PPs on other peroxisomal parameters and proto-oncogene expression using Wistar rats. Interestingly, c-myc expression was enhanced even upon drug withdrawal, and was more stimulated by the second exposure to ciprofibrate, while c-fos expressi…

Malemedicine.medical_specialtyTime FactorseducationStimulationMitochondria LiverBiologyBiochemistryMicrobodiesDrug withdrawalClofibric AcidFenofibrateInternal medicineProto-Oncogene ProteinsGene expressionmedicineAnimalsRats WistarCarcinogenPharmacologyFenofibrateOncogeneFibric AcidsPeroxisomemedicine.diseaseRatsEndocrinologyLiverMicrosomes LiverCiprofibrateAcyl-CoA OxidaseOxidoreductasesmedicine.drugBiochemical pharmacology
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Episodic Social Stress-Escalated Cocaine Self-Administration: Role of Phasic and Tonic Corticotropin Releasing Factor in the Anterior and Posterior V…

2016

Intermittent social defeat stress escalates later cocaine self-administration. Reward and stress both activate ventral tegmental area (VTA) dopamine neurons, increasing downstream extracellular dopamine concentration in the medial prefrontal cortex and nucleus accumbens. The stress neuropeptide corticotropin releasing factor (CRF) and its receptors (CRF-R1, CRF-R2) are located in the VTA and influence dopaminergic activity. These experiments explore how CRF release and the activation of its receptors within the VTA both during and after stress influence later cocaine self-administration in rats.In vivomicrodialysis of CRF in the VTA demonstrated that CRF is phasically released in the poster…

Malemedicine.medical_specialtyendocrine systemCorticotropin-Releasing HormoneMicrodialysisDrug-Seeking BehaviorNeuropeptideSelf AdministrationNucleus accumbensSocial EnvironmentReceptors Corticotropin-Releasing HormoneSocial defeat03 medical and health sciencesCorticotropin-releasing hormoneCocaine-Related Disorders0302 clinical medicineDopamineInternal medicinemental disordersmedicineAnimalsRats Long-EvansSocial stressGeneral Neurosciencemusculoskeletal neural and ocular physiologyDopaminergicVentral Tegmental AreaArticles030227 psychiatryRatsSubstance Withdrawal SyndromeVentral tegmental areamedicine.anatomical_structureEndocrinologynervous systemPsychologyNeuroscience030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonistsStress Psychologicalmedicine.drug
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