Search results for "TLR4"

showing 10 items of 96 documents

TLR2, TLR4 and Dectin-1 signalling in hematopoietic stem and progenitor cells determines the antifungal phenotype of the macrophages they produce

2016

TLRs represent an attractive target for the stimulation of myeloid cell production by HSPCs. We have previously demonstrated that HSPCs use TLR2 to sense Candida albicans in vivo and induce the production of macrophages. In this work, we used an in vitro model of HSPCs differentiation to investigate the functional consequences for macrophages of exposure of HSPCs to various PAMPs and C. albicans cells. Mouse HSPCs (Lin(-) cells) were cultured with M-CSF to induce macrophage differentiation, in the presence or absence of the following PRR agonists: Pam3CSK4 (TLR2 ligand), LPS (TLR4 ligand), depleted zymosan (which only activates Dectin-1), or C. albicans yeasts (which activate several PRRs, …

Lipopolysaccharides0301 basic medicineMacrophage colony-stimulating factorCellular differentiationImmunologyBiologyMicrobiologyMicrobiologyProinflammatory cytokineLipopeptidesMice03 medical and health sciences0302 clinical medicineCandida albicansAnimalsLectins C-TypeProgenitor cellCandida albicansInnate immune systemMacrophage Colony-Stimulating FactorMacrophagesZymosanCell DifferentiationHematopoietic Stem Cellsbiology.organism_classificationToll-Like Receptor 2Cell biologyMice Inbred C57BLToll-Like Receptor 4TLR2030104 developmental biologyInfectious DiseasesTLR4Female030215 immunologyMicrobes and Infection
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Neuroendocrine Modulation of IL-27 in Macrophages

2017

Abstract Heterodimeric IL-27 (p28/EBV-induced gene 3) is an important member of the IL-6/IL-12 cytokine family. IL-27 is predominantly synthesized by mononuclear phagocytes and exerts immunoregulatory functional activities on lymphocytic and nonlymphocytic cells during infection, autoimmunity or neoplasms. There is a great body of evidence on the bidirectional interplay between the autonomic nervous system and immune responses during inflammatory disorders, but so far IL-27 has not been defined as a part of these multifaceted neuroendocrine networks. In this study, we describe the role of catecholamines (as mediators of the sympathetic nervous system) related to IL-27 production in primary …

Lipopolysaccharides0301 basic medicinemedicine.medical_specialtySympathetic nervous systemSympathetic Nervous SystemEpinephrinemedicine.medical_treatmentImmunologyInflammationMiceNorepinephrine03 medical and health sciences0302 clinical medicineFormoterol FumarateInternal medicineThiadiazolesmedicineAnimalsImmunology and AllergyAlbuterolCells CulturedAnthracenesInflammationSulfonamidesbiologyInterleukinsMacrophagesZymosanTLR7Macrophage ActivationShock SepticInterleukin-10Receptors AdrenergicToll-Like Receptor 3Mice Inbred C57BLTLR2Interleukin 10Poly I-C030104 developmental biologyCytokineEndocrinologymedicine.anatomical_structureIntegrin alpha Mbiology.proteinTLR4medicine.symptomSignal Transduction030215 immunologyThe Journal of Immunology
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Adaptive suppression of the ATF4–CHOP branch of the unfolded protein response by toll-like receptor signalling

2009

The endoplasmic reticulum (ER) unfolded protein response (UPR) restores equilibrium to the ER, but prolonged expression of the UPR effector CHOP (GADD153) is cytotoxic. We found that CHOP expression induced by ER stress was suppressed by prior engagement of toll-like receptor (TLR) 3 or 4 through a TRIF-dependent pathway. TLR engagement did not suppress phosphorylation of PERK or eIF-2alpha, which are upstream of CHOP, but phospho-eIF-2alpha failed to promote translation of the CHOP activator ATF4. In mice subjected to systemic ER stress, pretreatment with low dose lipopolysaccharide (LPS), a TLR4 ligand, suppressed CHOP expression and apoptosis in splenic macrophages, renal tubule cells an…

LipopolysaccharidesBiologyCHOPEndoplasmic ReticulumArticleMice03 medical and health sciences0302 clinical medicineStress Physiologicalhemic and lymphatic diseasesAnimalsHumansCells Cultured030304 developmental biologyMice Knockout0303 health sciencesToll-like receptorEndoplasmic reticulumToll-Like ReceptorsATF4Cell BiologyActivating Transcription Factor 4Cell biologyMice Inbred C57BLAdaptor Proteins Vesicular TransportTRIF030220 oncology & carcinogenesisUnfolded Protein ResponseUnfolded protein responseTLR4biological phenomena cell phenomena and immunitySignal transductionTranscription Factor CHOPSignal TransductionNature Cell Biology
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Reduced expression of TLR4 is associated with the metastatic status of human colorectal cancer.

2007

Signaling mediating colorectal cancer (CRC) progression is incompletely understood. Previously, we identified lipopolysaccharide (LPS), an endotoxin of ubiquitously existing colonic bacteria, as a pivotal stimulus increasing the metastatic potential of human CRC. Since the ubiquitous colonic bacteria release large amounts of LPS this observation could be of enormous relevance for the progression of CRC. In this study we present data contributing to the elucidation of its mode of action. Since both receptors CD14 and TLR4 act as LPS mediators, we determined their expression in various CRC cell lines and in 115 non-metastatic, lymphogenous-metastatic and haematogenous-metastatic CRC specimens…

LipopolysaccharidesColorectal cancerCellLipopolysaccharide ReceptorsBiologyDownregulation and upregulationCell Line TumorGeneticsmedicineHumansNeoplasm MetastasisReceptorFluorescent Antibody Technique IndirectNeoplasm StagingRetrospective StudiesOncogeneCarcinomaGeneral MedicineCell cyclemedicine.diseaseMolecular medicineImmunohistochemistrydigestive system diseasesGene Expression Regulation NeoplasticToll-Like Receptor 4medicine.anatomical_structureLymphatic MetastasisImmunologyTLR4Cancer researchlipids (amino acids peptides and proteins)Caco-2 CellsColorectal NeoplasmsInternational journal of molecular medicine
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Interaction of TLR2 and TLR4 ligands with the N-terminal domain of Gp96 amplifies innate and adaptive immune responses.

2006

Activation of dendritic cells by ligands for Toll-like receptors (TLR) is a crucial event in the initiation of innate and adaptive immune responses. Several classes of TLR ligands have been identified that interact with distinct members of the TLR-family. TLR4 ligands include lipopolysaccharide derived from different Gram-negative bacteria and viral proteins. Recent reports have demonstrated the TLR-mediated activation of dendritic cells by heat shock proteins (HSPs). However, doubts were raised as to what extent this effect was due to lipopolysaccharide contaminations of the HSP preparations. We re-examined this phenomenon using Gp96 or its N-terminal domain, nominally endotoxin-free (0.5 …

LipopolysaccharidesLipopolysaccharideBiologyCD8-Positive T-LymphocytesBiochemistrychemistry.chemical_compoundMiceImmune systemDogsHeat shock proteinAnimalsHumansReceptorMolecular BiologyInflammationMice Inbred BALB CInnate immune systemMembrane GlycoproteinsCCL18Cell BiologyToll-Like Receptor 2Cell biologyEndotoxinsMice Inbred C57BLToll-Like Receptor 4TLR2BiochemistrychemistryTLR4The Journal of biological chemistry
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Oxidative stress and innate immunity responses in cigarette smoke stimulated nasal epithelial cells

2013

Cigarette smoke extracts (CSE) may play a significant role in diseases of the upper airway including chronic rhinosinusitis. Even short term exposure of cigarette smoke has adverse effects on mitochondrial functions and redox homeostasis in tissues which may progress to further complications associated with chronic smoking. Cigarette smoke alters toll-like receptor 4 (TLR4) expression and activation in bronchial epithelial cells. Carbocysteine is an anti-oxidant and mucolytic agent. The effects of carbocysteine on CSE induced oxidative stress and on associated innate immune and inflammatory responses in nasal epithelial cells are largely unknown. The present study was aimed to assess in CSE…

LipopolysaccharidesNecrosisNeutrophilsPhalloidineCARB CSE Cigarette smoke LPS Nasal epithelial cells ROS Reactive oxygen species TLR4 carbocysteine cigarette smoke extracts lipolysaccharide reactive oxygen species toll like receptor 4Fluorescent Antibody TechniqueApoptosisMucous membrane of noseCell SeparationBiologyToxicologymedicine.disease_causeCell LineNecrosisSmokeTobaccomedicineHumansExpectorantschemistry.chemical_classificationReactive oxygen speciesInnate immune systemCarbocysteineEpithelial CellsCarbocysteineTobacco ProductsGeneral MedicineActinsImmunity InnateToll-Like Receptor 4Nasal MucosaOxidative StresschemistryApoptosisImmunologyTLR4medicine.symptomReactive Oxygen SpeciesOxidative stressToxicology in Vitro
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A role for Toll-like receptor mediated signals in neutrophils in the pathogenesis of the anti-phospholipid syndrome.

2012

The anti-phospholipid syndrome (APS) is characterized by recurrent thrombosis and occurrence of anti-phospholipid antibodies (aPL). aPL are necessary, but not sufficient for the clinical manifestations of APS. Growing evidence suggests a role of innate immune cells, in particular polymorphonuclear neutrophils (PMN) and Toll-like receptors (TLR) to be additionally involved. aPL activate endothelial cells and monocytes through a TLR4-dependent signalling pathway. Whether this is also relevant for PMN in a similar way is currently not known. To address this issue, we used purified PMN from healthy donors and stimulated them in the presence or absence of human monoclonal aPL and the TLR4 agonis…

LipopolysaccharidesNeutrophilsImmunology610 MedizinImmunoglobulinslcsh:MedicineInflammationApoptosisImmunopathologyBiologyNeutrophil ActivationAutoimmune DiseasesPhagocytosisimmune system diseases610 Medical sciencesmedicineHumansInterleukin 8L-SelectinReceptorlcsh:ScienceBiologyImmune ResponseneoplasmsRespiratory BurstInflammationToll-like receptorMultidisciplinaryInnate immune systemCD11b AntigenCoagulation DisordersEffectorInterleukin-8lcsh:RImmunityHematologyAntiphospholipid SyndromeFlow CytometryInnate ImmunityRespiratory burstToll-Like Receptor 4ImmunologyTLR4MedicineClinical Immunologylcsh:Qmedicine.symptomResearch ArticleSignal TransductionPLoS ONE
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Nanoscale distribution of TLR4 on primary human macrophages stimulated with LPS and ATI

2019

Toll-like receptor 4 (TLR4) plays a crucial role in the recognition of invading pathogens. Upon activation by lipopolysaccharides (LPS), TLR4 is recruited into specific membrane domains and dimerizes. In addition to LPS, TLR4 can be stimulated by wheat amylase-trypsin inhibitors (ATI). ATI are proteins associated with gluten containing grains, whose ingestion promotes intestinal and extraintestinal inflammation. However, the effect of ATI vs. LPS on the membrane distribution of TLR4 at the nanoscale has not been analyzed. In this study, we investigated the effect of LPS and ATI stimulation on the membrane distribution of TLR4 in primary human macrophages using single molecule localization m…

LipopolysaccharidesSingle molecule localizationStimulationInflammation02 engineering and technology010402 general chemistry01 natural sciencesmedicineHumansDistribution (pharmacology)General Materials ScienceReceptorCells CulturedChemistryMacrophagesCell Membrane021001 nanoscience & nanotechnology0104 chemical sciencesCell biologyToll-Like Receptor 4MembraneMicroscopy FluorescenceTLR4lipids (amino acids peptides and proteins)Receptor clusteringmedicine.symptomTrypsin Inhibitors0210 nano-technologyNanoscale
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Release of IL-12 by dendritic cells activated by TLR ligation is dependent on MyD88 signaling, whereas TRIF signaling is indispensable for TLR synerg…

2010

Abstract Synergistic activation of dendritic cells by combinations of TLR ligands requires both MyD88- and TRIF-dependent signaling. Recently, it has been shown that certain combinations of TLR ligands act in synergy to induce the release of IL-12 by DCs. In this study, we sought to define the critical parameters underlying TLR synergy. Our data show that TLR ligands act synergistically if MyD88- and TRIF-dependent ligands are combined. TLR4 uses both of these adaptor molecules, thus activation via TLR4 proved to be a synergistic event on its own. TLR synergy did not affect all aspects of DC activation but enhanced primarily the release of certain cytokines, particularly IL-12, whereas the …

LipopolysaccharidesT cellImmunologyBiologyLymphocyte ActivationInterferon-gammaMicemedicineImmunology and AllergyAnimalsCD40 AntigensAutocrine signallingMice Inbred BALB CToll-Like ReceptorsSignal transducing adaptor proteinCell PolarityCell BiologyDendritic CellsInterleukin-12Cell biologyMice Inbred C57BLAdaptor Proteins Vesicular Transportmedicine.anatomical_structurePoly I-CTRIFImmunologyMyeloid Differentiation Factor 88TLR4Interleukin 12Myeloid Differentiation Factor 88Signal transductionSignal TransductionJournal of leukocyte biology
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Phenolic Extract from Sonchus oleraceus L. Protects Diabetes-related Liver Injury in Rats through TLR4/NF-κB Signaling Pathway

2019

Sonchus oleraceus Linn. is of great interest in scientific platform because it exhibits significant biological activities. This study investigated the liver protective effects and mechanism of phenolic extract from S. oleraceus (SOL) on Streptozotocin (STZ)-induced diabetic rats. SOL significantly increased both superoxide dismutase activity and glutathione level, while causing a reduction of malondialdehyde level in the liver. Moreover, SOL ameliorates STZ-induced liver function and pathological damages. Diabetic rats fed with S. oleraceus daily for 6 weeks showed significantly decreased levels of tumor necrosis factor alpha and interleukin-1β in the liver. Sonchus oleraceus decreased the …

Liver injurybiologylcsh:TP368-456ChemistryAnti inflammationPharmacologybiology.organism_classificationmedicine.diseaseSonchus oleraceusin vivoSonchus oleraceuslcsh:Food processing and manufactureIn vivolcsh:RA1190-1270Diabetes mellitusAnti-inflammationTlr4 nf κbmedicineSignal transductionNF-κB signaling pathwayliver injurylcsh:Toxicology. PoisonseFood
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