Search results for "TOXICITY"

showing 10 items of 2261 documents

Cytotoxicity and antimitotic activity of Rhinella schneideri and Rhinella marina venoms.

2019

Abstract Ethnopharmacological relevance Rhinella schneideri and Rhinella marina are toad venoms distributed in different parts of the world, including Brazil, Columbia and amazon. Venoms extracted from different species have many clinical applications such as antimicrobial cardiotonics and treatment of cancer. Aim of the study; In this study, we aim to investigate the effect of venoms extracted from R. schneideri and R. marina on cancer cells and verify possible mechanism of action. Material and method Cytotoxicity analyses was performed using the resazurin reduction assay, where different concentrations of venoms were tested against sensitive CCRF-CEM and P-gp overexpressing ADR/CEM5000 le…

Programmed cell deathCell SurvivalAntimitotic AgentsLethal Dose 5003 medical and health scienceschemistry.chemical_compound0302 clinical medicineTubulinRhinella schneideriCell Line TumorDrug DiscoveryAnimalsHumansPropidium iodideCytotoxicity030304 developmental biologyPharmacology0303 health sciencesbiologyBufalinCell Cycle Checkpointsbiology.organism_classificationBufonidaeMolecular Docking SimulationTubulinchemistryBiochemistryApoptosis030220 oncology & carcinogenesisCancer cellbiology.proteinAmphibian VenomsJournal of ethnopharmacology
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Tumor cell specific toxicity of Inula helenium extracts.

2006

The aim of the research program was to identify botanical extracts with antineoplastic activity. In this respect extracts prepared from Inula helenium roots showed a remarkable activity. As evidenced by the MTT assay, the Inula helenium extract revealed a highly selective toxicity toward four different tumor cell lines (HT-29, MCF-7, Capan-2 and G1), but a much lower toxicity against healthy human peripheral blood lymphocytes (PBLs) from two donors. The extract-induced death of tumor cells was studied extensively by electron microscopy. There was a remarkable similarity of morphological alterations observed in the four cell lines: patchy chromatin condensations, cytoplasmic vesiculation, sw…

Programmed cell deathCell SurvivalContext (language use)Plant RootsLethal Dose 50Cell Line TumorToxicity Tests AcuteHumansMTT assayLymphocytesAnnexin A5CytotoxicityPharmacologyInulabiologyMutagenicity TestsPlant Extractsbiology.organism_classificationMolecular biologyAntineoplastic Agents PhytogenicApoptosisCell cultureImmunologyInulaHT29 CellsHeleniumPhytotherapy research : PTR
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Cytotoxicity of 40 Egyptian plant extracts targeting mechanisms of drug-resistant cancer cells

2019

Abstract Background The multidrug resistance (MDR) phenotype encounters a major challenge to the success of established chemotherapy in cancer patients. We hypothesized that cytotoxic medicinal plants with novel phytochemicals can overcome MDR and kill MDR-cells with similar efficacy as drug sensitive cells. Purpose We evaluated plant extracts from an unexplored ecosystem in Egypt with unusual climate and nutrient conditions for their activity against sensitive and multidrug-resistant cancer cell lines. Material and methods/study design Methylene chloride: methanol (1:1) and methanol: H2O (7:3) extracts of 40 plants were prepared resulting in a sum of 76 fraction containing compounds with v…

Programmed cell deathCell SurvivalPhytochemicalsPharmaceutical ScienceApoptosisCentaureaWithaniaPulicariaMagnoliopsida03 medical and health sciences0302 clinical medicineCell Line TumorNeoplasmsDrug DiscoveryHumansCytotoxic T cellViability assayCytotoxicity030304 developmental biologyMembrane Potential MitochondrialPharmacology0303 health sciencesPlants MedicinalbiologyPlant ExtractsChemistryWithaniabiology.organism_classificationAntineoplastic Agents PhytogenicMolecular biologyDrug Resistance MultipleMultiple drug resistanceComplementary and alternative medicineDrug Resistance NeoplasmCell culture030220 oncology & carcinogenesisCancer cellMolecular MedicineEgyptReactive Oxygen SpeciesPhytotherapyPhytomedicine
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Polyglutamine toxicity induces rod photoreceptor division, morphological transformation or death in Spinocerebellar ataxia 7 mouse retina

2010

In neurodegenerative disorders caused by polyglutamine (polyQ) expansion, polyQ toxicity is thought to trigger a linear cascade of successive degenerative events leading to neuronal death. To understand how neurons cope with polyQ toxicity, we studied a Spinocerebellar ataxia 7 (SCA7) mouse which expresses polyQ-expanded ATXN7 only in rod photoreceptors. We show that in response to polyQ toxicity, SCA7 rods go through a range of radically different cell fates, including apoptotic and non-apoptotic cell death, cell migration, morphological transformation into a round cell or, most remarkably, cell division. The temporal profile of retinal remodeling indicates that some degenerative pathways …

Programmed cell deathCell divisionProliferationPopulationMice TransgenicNerve Tissue ProteinsBiologylcsh:RC321-571Mice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCell MovementRetinal Rod Photoreceptor CellsmedicineAnimalsSpinocerebellar AtaxiasNeurodegenerationeducationlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCell ShapeComputingMilieux_MISCELLANEOUSSpinocerebellar ataxia 7030304 developmental biologyAtaxin-7Mice Knockout0303 health sciencesRetinaeducation.field_of_studyPhotoreceptorCell DeathRetinal DegenerationNeurodegenerationRetinalmedicine.diseaseRemodelingMice Inbred C57BLmedicine.anatomical_structureNeurologyProteotoxicitychemistryNerve DegenerationSpinocerebellar ataxia[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Apoptosis Regulatory ProteinsPeptidesPolyglutamineNeuroscience030217 neurology & neurosurgery
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Comparative analysis of stress responses of H9c2 rat cardiomyoblasts following treatment with doxorubicin and tBOOH

2011

Abstract Cardiotoxicity is the major dose-limiting adverse effect of anthracyclines and is hypothesized to result from damage induced by reactive oxygen species (ROS) or inhibition of topoisomerase II. Here, we comparatively analyzed the effect of doxorubicin and the organic peroxide tertiary-butylhydroperoxide (tBOOH) on stress responses of rat cardiomyblast cells (H9c2). Moreover, we investigated the impact of serum factors and the novel prototypical protein kinase CK2 inhibitor resorufin on the sensentivity of H9c2 cells exposed to doxorubicin or tBOOH. Measuring cell viability by use of the WST assay as well as cell cycle progression and apoptotic death by FACS-based methods, we found t…

Programmed cell deathDNA damageCell SurvivalAntineoplastic AgentsApoptosisBiologyPharmacologyAntioxidantsCell Linetert-ButylhydroperoxidemedicineAnimalsDoxorubicinViability assayCytotoxicitychemistry.chemical_classificationReactive oxygen speciesCardiotoxicityDose-Response Relationship DrugKinaseCell BiologyMolecular biologyAcetylcysteineRatsOxidative StresschemistryDoxorubicinReactive Oxygen SpeciesMyoblasts Cardiacmedicine.drug
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Toxicity of a dental adhesive compared with ionizing radiation and zoledronic acid

2015

Background: To determine the toxicity of aqueous dilutions of a universal self-priming dental adhesive (DA) and comparing these with those elicited by exposure to ionizing radiation (IR), Zoledronic acid (Z) treatment and the synergic effects of the combined treatment with IR+Z. Material and Methods: The genotoxic effect of DA was determined by the increase in the frequency of micronuclei in cytokinesis-blocked in cultured human lymphocytes before and after exposure to 2Gy of X-rays. The cytotoxic effect was studied by using the MTT cell viability test in normal prostate cell lines (PNT2) after exposure to different X-ray doses (0Gy-20Gy). The cell lines divided into different groups and tr…

Programmed cell deathDental CementsOdontologíaPharmacologymedicine.disease_causeZoledronic AcidIonizing radiationToxicologyPolymethacrylic AcidsRadiation IonizingToxicity TestsmedicineCytotoxic T cellHumansViability assayLymphocytesGeneral DentistryCells CulturedOral Medicine and PathologyDiphosphonatesChemistryResearchImidazoles:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludIn vitroOtorhinolaryngologyToxicityMicronucleus testUNESCO::CIENCIAS MÉDICASSurgeryGenotoxicity
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Toxic effects induced by vanadium on sea urchin embryos

2020

Vanadium, a naturally occurring element widely distributed in soil, water and air, has received considerable interest because its compounds are often used in different applications, from industry to medicine. While the possible medical use of vanadium compounds is promising, its potential harmful effects on living organisms are still unclear. Here, for the first time, we provide a toxicological profile induced by vanadium on Paracentrotus lividus sea urchin embryos, reporting an integrated and comparative analysis of the detected effects reflecting vanadium-toxicity. At the morphological level we found a dose-dependent induction of altered phenotypes and of skeletal malformations. At the mo…

Programmed cell deathEmbryo NonmammalianEnvironmental EngineeringHealth Toxicology and Mutagenesis0208 environmental biotechnologyVanadium-stressVanadiumchemistry.chemical_elementApoptosis02 engineering and technology010501 environmental sciences01 natural sciencesParacentrotus lividusDevelopmental abnormalityCellular stress responseHeat shock proteinAutophagyAnimalsHumansEnvironmental ChemistrySettore BIO/06 - Anatomia Comparata E Citologia0105 earth and related environmental sciencesHeat shock proteinsbiologyChemistryAutophagyPublic Health Environmental and Occupational HealthVanadiumGeneral MedicineGeneral Chemistrybiology.organism_classificationPollution020801 environmental engineeringCell biologyApoptosisParacentrotus lividus embryosToxicityParacentrotusChemosphere
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Cell cooperation in coelomocyte cytotoxic activity of Paracentrotus lividus coelomocytes

2007

The coelomic fluid from the sea urchin Paracentrotus lividus contains several coelomocyte types including amoebocytes and uncoloured spherulocytes involved in immune defences. In the present paper, we show a Ca(2+)-dependent cytotoxic activity for the unfractionated coelomocytes assayed in vitro, with rabbit erythrocytes and the K562 tumour cell line. In a plaque-forming assay, whole coelomocyte preparations as well as density gradient separated coelomocyte populations revealed that cell populations enriched in uncoloured spherulocytes, exerted high cytotoxic activity by releasing lysins in the presence of amoebocytes. This cooperative effect could be dependent on soluble factors released b…

Programmed cell deathErythrocytesPhysiologyCytotoxicityCell CommunicationCell SeparationBiochemistryParacentrotus lividusbiology.animalCentrifugation Density GradientAnimalsHumansCytotoxic T cellCytotoxicityMolecular BiologySea urchinCoelomocyteCoelomocyte cooperationInnate immunityCell DeathEchinodermbiologyAnatomybiology.organism_classificationIn vitroCell biologyParacentrotus lividusCell cultureParacentrotusRabbitsCoelomocyteK562 CellsComparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology
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Loss of ATM sensitizes against O6-methylguanine triggered apoptosis, SCEs and chromosomal aberrations.

2003

A critical pre-cytotoxic and -apoptotic DNA lesion induced by methylating carcinogens and chemotherapeutic drugs is O6-methylguanine (O6MeG). The mechanism by which O6MeG causes cell death via apoptosis is only partially understood. The current model ascribes a role to DNA replication and mismatch repair, which converts O6MeG into a critical distal lesion (presumably a DNA double-strand break) that is finally responsible for genotoxicity and apoptosis. Here we analysed whether the PI3-like kinase ATM is involved in this process. ATM is a major player in recognizing and signaling DNA breaks, but most reports are limited to ionizing radiation. Comparing mouse ATM knockout fibroblasts (ATM-/-)…

Programmed cell deathGuanineDNA damageApoptosisCell Cycle ProteinsAtaxia Telangiectasia Mutated ProteinsBiologyProtein Serine-Threonine Kinasesmedicine.disease_causeBiochemistryMicemedicineCytotoxic T cellAnimalsMolecular BiologyChromosome AberrationsMice KnockoutTumor Suppressor ProteinsCell BiologyTransfectionMolecular biologyDNA-Binding ProteinsCell killingApoptosisDNA mismatch repairSister Chromatid ExchangeGenotoxicityDNA repair
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Synthesis of Triazenoazaindoles: a New Class of Triazenes with Antitumor Activity

2011

Despite improvements in the treatment and prevention of cancer, the number of new diagnoses continues to rise; this has fuelled substantial interest in the development of new and effective chemotherapeutic agents. Compounds of the triazene class, such as dacarbazine, have been used in the clinical management of many cancer types including brain, leukemia, and melanoma. A new compound class bearing a triazenoazaindole scaffold was synthesized with the aim of identifying new antiproliferative agents. Compounds 5 a-g and 6 a-c were screened against a panel of human tumor cell lines, and two of them, 5 e and 5 f, showed cytotoxicity (GI(50) range: 2.2-8.2 μM) in all cell lines. These two compou…

Programmed cell deathIndolesToxicology and Pharmaceutics (all)DacarbazineAntineoplastic AgentsAntiproliferative activityPharmacologyEGF receptorsDrug Screening AssaysBiochemistryCell LineFlow cytometryCell Line TumorNeoplasmsDrug DiscoveryTriazenoazaindolemedicineHumansTriazeno derivativesGeneral Pharmacology Toxicology and PharmaceuticsCytotoxicityPharmacologyAntitumor agentsTumorEpidermal Growth Factormedicine.diagnostic_testChemistryMelanomaOrganic ChemistryCancerAntitumorTriazenoazaindoles; Dacarbazine; Antitumor Activitymedicine.diseaseErbB ReceptorsDacarbazineApoptosisCell cultureMolecular MedicineDrug Screening Assays AntitumorAntitumor ActivityTriazenesTriazenoazaindolesAntiproliferative activity; Antitumor agents; EGF receptors; Triazeno derivatives; Antineoplastic Agents; Cell Line Tumor; Dacarbazine; Drug Screening Assays Antitumor; Humans; Indoles; Neoplasms; Receptor Epidermal Growth Factor; Triazenes; Pharmacology Toxicology and Pharmaceutics (all); Organic Chemistry; Molecular MedicineReceptormedicine.drugChemMedChem
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