Search results for "TRES"

showing 10 items of 6927 documents

A scoring system for biliary atresia: Is this the right one?

2014

medicine.medical_specialtyScoring systemHepatologyBiliary atresiabusiness.industryGastroenterologymedicinemedicine.diseasebusinessSurgeryDigestive and Liver Disease
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Prevention of Atherosclerosis by Interference with the Vascular Nitric Oxide System

2009

Nitric oxide (NO) produced by endothelial NO synthase (eNOS) represents an anti-atherosclerotic principle. NO bioavailability is decreased in atherosclerosis due to increased NO inactivation by reactive oxygen species and reduced NO synthesis. Various types of vascular pathophysiology are associated with oxidative stress, with NADPH oxidases as the major source of reactive oxygen species. These inactivate NO. Also, oxidative stress is likely to be the main cause for oxidation of the essential NOS cofactor, tetrahydrobiopterin (BH(4)). A lack of BH(4) leads to eNOS uncoupling (i.e., uncoupling of oxygen reduction from NO synthesis in eNOS). Based on these pathomechanisms, the therapeutic pot…

medicine.medical_specialtySepiapterinNitric Oxide Synthase Type IIImedicine.drug_classGTP cyclohydrolase INitric Oxidemedicine.disease_causeRenin inhibitorNitric oxidechemistry.chemical_compoundEnosInternal medicineDrug DiscoverymedicineAnimalsHumansHypolipidemic AgentsPharmacologybiologyArteriesTetrahydrobiopterinAtherosclerosisbiology.organism_classificationNitric oxide synthaseOxidative StressTreatment OutcomeEndocrinologychemistrybiology.proteinOxidative stressSignal Transductionmedicine.drugCurrent Pharmaceutical Design
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A novel component of the metabolic syndrome: The oxidative stress

2009

The metabolic syndrome (MS) represents a cluster of cardiovascular (CV) risk factors associated to CV disease and type 2 diabetes. It is still under debate whether MS is a mere aggregation of risk factors or it represents a clinical entity with visceral obesity as underlying pathophysiological trigger. The publication of several diagnostic criteria of MS by scientific associations or experts panels reflects this uncertainty in understanding the real nature of MS. Besides the metabolic disturbances of MS, as visceral obesity, hypertriglyceridemia, low HDL cholesterol, hypertension and hyperglycemia, novel mechanisms of arterial damage have been identified. This paper reviews the evidence sho…

medicine.medical_specialtySettore MED/09 - Medicina InternaEndocrinology Diabetes and MetabolismMedicine (miscellaneous)Blood lipidsType 2 diabetesDiseaseBiologyBioinformaticsmedicine.disease_causeRisk FactorsInternal medicineDiabetes mellitusmedicineAnimalsHumansDyslipidemiasInflammationMetabolic SyndromeNutrition and DieteticsHypertriglyceridemiaCardiovascular riskmedicine.diseaseObesityOxidative StressEndocrinologyDiabetes Mellitus Type 2Cardiovascular DiseasesObesity AbdominalHypertensionMetabolic syndromeCardiology and Cardiovascular MedicineOxidative stressNutrition, Metabolism and Cardiovascular Diseases
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Matrix metalloproteinases in metabolic syndrome.

2011

Metabolic syndrome is commonly accompanied by an elevated cardiovascular risk with high morbidity and mortality. The alterations of the arterial vasculature begin with endothelial dysfunction and lead to micro- and macrovascular complications. The remodeling of the endothelial basal membrane, that promotes erosion and thrombosis, has a multifactorial pathogenesis that includes leukocyte activation, increased oxidative stress and also an altered matrix metalloproteinases (MMPs) expression. MMPs are endopeptidases which degrade extracellular matrix proteins, such as collagen, gelatins, fibronectin and laminin. They can be secreted by several cells within the vascular wall, but macrophages are…

medicine.medical_specialtySettore MED/09 - Medicina InternaMatrix metalloproteinaseGlobal HealthPathogenesisExtracellular matrixRisk FactorsDiabetes mellitusInternal medicineInternal MedicinemedicineHumansEndothelial dysfunctionMetabolic Syndromebiologybusiness.industryMETABOLIC SYNDROME MATRIX METALLOPROTEASES CARDIOVASCULAR RISKmedicine.diseaseMatrix MetalloproteinasesSurvival RateFibronectinOxidative StressEndocrinologyCardiovascular Diseasesbiology.proteinMorbidityMetabolic syndromebusinessDyslipidemia
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Halitosis: new insight into a millennial old problem.

2011

The problem of halitosis has been reported since ancienttimes. References have been found in papyrus manuscriptsdating back to 1550 BC. Hippocrates advised that any girlshould have pleasant breath, making sure always to washher mouth with wine, anise and dill seeds [1].More than50 years ago, Blackburn [2] investigated halitosis in a case-series of 73 patients affected by leukemia. He found apeculiar odor of the breath resembling that of a freshlyopened corpse. This characteristic smell is not associatedwith clinical involvement of the gum, mouth, or upperrespiratory or alimentary tract. He associated this particularsmell with the hematologic disease. Nearly 15 years ago,the role of cadaveri…

medicine.medical_specialtySettore MED/09 - Medicina InternaPopulationDentistryOral HealthOral hygienechemistry.chemical_compoundBad breathInternal MedicinemedicineHumansPutrefactioneducationeducation.field_of_studyCadaverinebusiness.industryHalitosisOral HygieneDermatologySettore MED/45 - Scienze Infermieristiche Generali Cliniche E PediatrichechemistryOdorEmergency MedicineEtiologyPutrescinemedicine.symptombusinessMouth DiseasesInternal and emergency medicine
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Protein oxidation in metabolic syndrome

2013

Purpose: Oxidative stress plays a pivotal role in the pathogenesis of the metabolic syndrome and in the progression of its complications. Carbonylated proteins are a stable marker of severe oxidative stress because damage to the protein structure is irreversible and may cause an inhibition of their enzymatic activity or an increased susceptibility to proteolysis. There are few data regarding protein oxidation in metabolic syndrome, although elevated levels of carbonyl groups are often detected in subjects with obesity, diabetes mellitus, hypertension or dyslipidemia, well-known components of the metaboic syndrome. In particular, obesity, insulin resistance and diabetes mellitus are frequent…

medicine.medical_specialtySettore MED/09 - Medicina InternaProtein CarbonylationBiologyProtein oxidationmedicine.disease_causeProtein CarbonylationInsulin resistanceDiabetes mellitusInternal medicinemedicineAnimalsHumansMetabolic SyndromeInterleukin-6Tumor Necrosis Factor-alphaGeneral Medicinemedicine.diseaseOxidative Stressprotein oxidation metabolic syndromeEndocrinologyResistinMetabolic syndromeInsulin ResistanceDyslipidemiaOxidative stress
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Therapy of erectile dysfunction (ED) with sildenafil improves quality of life (QoL) and partnership (QoP)

2004

According to Maslow [1] and others, higher levels of individual and social functioning can only be achieved and maintained, when more basic and particularly physiological levels of functioning are satisfactory and healthy (Fig. 1). Satisfactory partnership and sexuality are core aspects of human wellbeing through the complete course of adulthood. Sexual activity is a major determinant of sexual satisfaction [2]. In men and women, sexual dysfunction as a disorder of sexual desire, arousal, or orgasm, and/or sexual pain results in personal distress and reduced quality of life and interpersonal relationships [3, 4, 5, 6]. Erectile dysfunction (ED) as the inability to attain or maintain penile …

medicine.medical_specialtySildenafilbusiness.industrymedia_common.quotation_subjectPersonal distressHuman sexualityOrgasmmedicine.diseasechemistry.chemical_compoundSexual desireSexual dysfunctionErectile dysfunctionchemistryQuality of lifePhysical therapyMedicinemedicine.symptombusinessClinical psychologymedia_common
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Uncoupling Protein 2 as genetic risk factor for systemic lupus erythematosus: association with malondialdehyde levels and intima media thickness

2020

BACKGROUND Increased oxidative stress potentially leads to accelerated atherosclerosis and, consequently, cardiovascular diseases, the main cause of death in systemic lupus erythematous (SLE). To gain insight into these mechanisms, we studied the association of uncoupling protein (UCP) 2 genetic variants, gene involved in the mitochondrial production of reactive oxygen species, and oxidative stress with SLE and the presence of atherosclerosis. METHODS Genetic analysis of the UCP2 -866G/A and UCP2 Ins/Del polymorphisms was performed in 45 SLE patients and 36 healthy controls by RFLP-PCR. Oxidation status was determined by measuring malondialdehyde (MDA) levels. Presence of subclinical athero…

medicine.medical_specialtySingle-nucleotide polymorphism030204 cardiovascular system & hematologymedicine.disease_causeCarotid Intima-Media Thickness03 medical and health scienceschemistry.chemical_compound0302 clinical medicineGeneticRisk FactorsMalondialdehydeInternal medicineGenotypemedicineHumansUncoupling proteinUncoupling Protein 2030212 general & internal medicineAlleleskin and connective tissue diseaseschemistry.chemical_classificationReactive oxygen speciesbusiness.industryMalondialdehydeLupus erythematosus systemicEndocrinologychemistryIntima-media thicknessCardiology and Cardiovascular MedicinebusinessCardiovascular diseases.Oxidative stressMinerva Cardioangiologica
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Chronic heart damage following doxorubicin treatment is alleviated by lovastatin.

2014

The anticancer efficacy of anthracyclines is limited by cumulative dose-dependent early and delayed cardiotoxicity resulting in congestive heart failure. Mechanisms responsible for anthracycline-induced heart damage are controversially discussed and effective preventive measures are preferable. Here, we analyzed the influence of the lipid lowering drug lovastatin on anthracycline-induced late cardiotoxicity three month after treatment of C57BL/6 mice with five low doses of doxorubicin (5×3mg/kg BW; i.p.). Doxorubicin increased the cardiac mRNA levels of BNP, IL-6 and CTGF, while the expression of ANP remained unchanged. Lovastatin counteracted these persisting cardiac stress responses evoke…

medicine.medical_specialtyStatinCardiotonic AgentsAnthracyclinemedicine.drug_classBiologymedicine.disease_causeDNA MitochondrialInternal medicineNatriuretic Peptide Brainpolycyclic compoundsmedicineAnimalsDoxorubicinHSP70 Heat-Shock ProteinsLovastatinPharmacologyCardiotoxicityEjection fractionAntibiotics AntineoplasticInterleukin-6Gene Expression ProfilingMyocardiumConnective Tissue Growth Factormedicine.diseaseFibrosisCardiotoxicityMice Inbred C57BLEndocrinologyDoxorubicinHeart failureFemaleLovastatinOxidative stressmedicine.drugDNA DamagePharmacological research
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Infrared thermal imaging in the diagnosis of musculoskeletal injuries: a systematic review and meta-analysis.

2014

OBJECTIVE. Musculoskeletal injuries occur frequently. Diagnostic tests using ionizing radiation can lead to problems for patients, and infrared thermal imaging could be useful when diagnosing these injuries. CONCLUSION. A systematic review was performed to determine the diagnostic accuracy of infrared thermal imaging in patients with musculoskeletal injuries. A meta-analysis of three studies evaluating stress fractures was performed and found a lack of support for the usefulness of infrared thermal imaging in musculoskeletal injuries diagnosis.

medicine.medical_specialtyStress fracturesSoft Tissue Injuriesbusiness.industryInfrared RaysPoison controlGeneral Medicinemedicine.diseaseFractures BoneRisk FactorsThermographyMeta-analysisInjury preventionThermographyMedical imagingmedicineInfrared thermal imagingPhysical therapyPrevalenceHumansRadiology Nuclear Medicine and imagingIn patientPractice Patterns Physicians'businessMuscle SkeletalAJR. American journal of roentgenology
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