Search results for "Tandem Repeat Sequence"

showing 10 items of 39 documents

STR analysis of artificially degraded DNA--results of a collaborative European exercise.

2003

Degradation of human DNA extracted from forensic stains is, in most cases, the result of a natural process due to the exposure of the stain samples to the environment. Experiences with degraded DNA from casework samples show that every sample may exhibit different properties in this respect, and that it is difficult to systematically assess the performance of routinely used typing systems for the analysis of degraded DNA samples. Using a batch of artificially degraded DNA with an average fragment size of approx. 200 bp a collaborative exercise was carried out among 38 forensic laboratories from 17 European countries. The results were assessed according to correct allele detection, peak heig…

DNA FragmentationBiologyPolymerase Chain ReactionPathology and Forensic Medicinelaw.inventionchemistry.chemical_compoundlawGenotypeHumansCooperative BehaviorAlleleAllelesPolymerase chain reactionGeneticsClinical Laboratory TechniquesDNADNA FingerprintingEuropeSTR analysisDNA profilingchemistryTandem Repeat SequencesMicrosatelliteLawDNATaq polymerase
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Tandem repeats lead to sequence assembly errors and impose multi-level challenges for genome and protein databases

2019

AbstractThe widespread occurrence of repetitive stretches of DNA in genomes of organisms across the tree of life imposes fundamental challenges for sequencing, genome assembly, and automated annotation of genes and proteins. This multi-level problem can lead to errors in genome and protein databases that are often not recognized or acknowledged. As a consequence, end users working with sequences with repetitive regions are faced with ‘ready-to-use’ deposited data whose trustworthiness is difficult to determine, let alone to quantify. Here, we provide a review of the problems associated with tandem repeat sequences that originate from different stages during the sequencing-assembly-annotatio…

FOS: Computer and information sciencesBioinformatics[SDV]Life Sciences [q-bio]Sequence assemblyGenomics[SDV.BC]Life Sciences [q-bio]/Cellular BiologyComputational biologyBiologyGenome03 medical and health sciencesAnnotation0302 clinical medicineTandem repeatGeneticsAnimalsSurvey and SummaryDatabases ProteinGeneComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciencesEnd user572: BiochemieDNASequence Analysis DNAGenomics[SDV.BIBS]Life Sciences [q-bio]/Quantitative Methods [q-bio.QM]WorkflowComputingMethodologies_PATTERNRECOGNITIONGadus morhuaTandem Repeat SequencesScientific Experimental Error[INFO.INFO-BI]Computer Science [cs]/Bioinformatics [q-bio.QM]Databases Nucleic Acid030217 neurology & neurosurgery
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Analysis of artificially degraded DNA using STRs and SNPs—results of a collaborative European (EDNAP) exercise

2005

Recently, there has been much debate about what kinds of genetic markers should be implemented as new core loci that constitute national DNA databases. The choices lie between conventional STRs, ranging in size from 100 to 450 bp; mini-STRs, with amplicon sizes less than 200 bp; and single nucleotide polymorphisms (SNPs). There is general agreement by the European DNA Profiling Group (EDNAP) and the European Network of Forensic Science Institutes (ENFSI) that the reason to implement new markers is to increase the chance of amplifying highly degraded DNA rather than to increase the discriminating power of the current techniques. A collaborative study between nine European and US laboratories…

Forensic GeneticsGeneticsAnalysis of VarianceGenotypeDNA Degradation NecroticSingle-nucleotide polymorphismAmpliconBiologyDNA FingerprintingPolymerase Chain ReactionPolymorphism Single NucleotidePathology and Forensic MedicineEuropeBloodDNA profilingTandem Repeat SequencesGenetic markerHumansMicrosatelliteMultiplexDegraded dnaMini strsSalivaLawForensic Science International
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Data for nine autosomal STRs markers from Valencia (East Mediterranean coast of the Iberian Peninsula)

2000

Nine STRs loci have been typed in a sample from Valencia, a population from the East Mediterranean coast of the Iberian Peninsula.

Genetic MarkersQuality ControlPopulationPolymerase Chain ReactionLinkage DisequilibriumWhite PeoplePathology and Forensic MedicineGene FrequencyPeninsulaHumansEast mediterraneaneducationValenciaInterneteducation.field_of_studygeographygeography.geographical_feature_categorybiologyMediterranean Regionbiology.organism_classificationDNA FingerprintingArchaeologySpainTandem Repeat SequencesPopulation dataLawSoftwareForensic Science International
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Validation and casework testing of the BioPlex-11 for STR typing of telogen hair roots.

2005

A new STR typing strategy has been developed allowing the simultaneous amplification and subsequent analysis of 11 polymorphic systems with amplicon sizes smaller than 270 bp. The multiplex amplification reaction includes six STR loci from the European standard set of loci (ESS) for DNA databases (D3S1358, D8S1179, D21S11, THO1, FGA and VWA) as well as four additional STR systems selected for their robustness (D2S1338, D12S391, TPOX and D5S818) together with the sex-specific locus amelogenin. After PCR amplification, the multiplex reaction is splitted into two sets of STR multiplexes by using biotin labelled primers only for one set. Using streptavidin-coated Sepharose beads five STR system…

GeneticsHeterozygoteAmelogeninSTR multiplex systemElectrophoresis CapillaryLocus (genetics)BiologyAmpliconDNA FingerprintingPolymerase Chain ReactionPathology and Forensic Medicinelaw.inventionDental Enamel ProteinslawTandem Repeat SequencesMicrosatelliteHumansMultiplexTypingAmelogeninLawHair FolliclePolymerase chain reactionForensic science international
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Signature of recent historical events in the European Y-chromosomal STR haplotype distribution

2005

Previous studies of human Y-chromosomal single-nucleotide polymorphisms (Y-SNPs) established a link between the extant Y-SNP haplogroup distribution and the prehistoric demography of Europe. By contrast, our analysis of seven rapidly evolving Y-chromosomal short tandem repeat loci (Y-STRs) in over 12,700 samples from 91 different locations in Europe reveals a signature of more recent historic events, not previously detected by other genetic markers. Cluster analysis based upon molecular variance yields two clearly identifiable sub-clusters of Western and Eastern European Y-STR haplotypes, and a diverse transition zone in central Europe, where haplotype spectra change more rapidly with longi…

GeneticsHistoryChromosomes Human YGenotypeDemographic historyHaplotypeContrast (statistics)BiologyPolymorphism Single NucleotideHaplogroupy DNA typingEastern europeanEuropePrehistoric demographyHaplotypesGenetic markerEvolutionary biologyTandem Repeat SequencesGeneticsMicrosatelliteHumansGenetics (clinical)
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Y-chromosomal STR haplotypes in Sicily

2006

Eight Y-chromosomal short tandem repeats (STRs)-DYS19, DYS389-I, DYS389-II, DYS390, DYS391, DYS392, DYS393 and DYS385 - were typed in a population sample (n = 255) of unrelated Sicilian males from nine different towns on the main island and from the island of Pantelleria. (c) 2005 Elsevier Ireland Ltd. All rights reserved.

GeneticshaplotypeChromosomes Human YPopulation sampleshort tandem repeatHaplotypePopulation geneticsY-chromosome; Short tandem repeats; Haplotype; SicilyBiologyY chromosomeDNA Fingerprintinglanguage.human_languageWhite PeoplePathology and Forensic MedicineGenetics PopulationShort tandem repeatsHaplotypesTandem Repeat SequenceslanguageMicrosatelliteHumansLawSicilianY-chromosomeSicily
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AML-associated Flt3 kinase domain mutations show signal transduction differences compared with Flt3 ITD mutations

2005

Activating mutations of Flt3 are found in approximately one third of patients with acute myeloid leukemia (AML) and are an attractive drug target. Two classes of Flt3 mutations occur: internal tandem duplications (ITDs) in the juxtamembrane and point mutations in the tyrosine kinase domain (TKD). We and others have shown that Flt3-ITD induced aberrant signaling including strong activation of signal transducer and activator of transcription 5 (STAT5) and repression of CCAAT/estradiol-binding protein α (c/EBPα) and Pu.1. Here, we compared the signaling properties of Flt3-ITD versus Flt3-TKD in myeloid progenitor cells. We demonstrate that Flt3-TKD mutations induced autonomous growth of 32D ce…

ImmunologyApoptosisBiologymedicine.disease_causeBiochemistryCell Linefluids and secretionsProto-Oncogene Proteinshemic and lymphatic diseasesSTAT5 Transcription FactormedicineAnimalsHumansPoint MutationMyeloid CellsPhosphorylationProtein kinase BProtein kinase CMutationPoint mutationAutophosphorylationIntracellular Signaling Peptides and ProteinsReceptor Protein-Tyrosine Kinaseshemic and immune systemsCell BiologyHematologyMilk ProteinsStaurosporineMolecular biologyProtein Structure TertiaryDNA-Binding ProteinsMuridaefms-Like Tyrosine Kinase 3Leukemia MyeloidTandem Repeat SequencesAcute Diseaseembryonic structuresFms-Like Tyrosine Kinase 3Mutagenesis Site-DirectedTrans-ActivatorsSignal transductionTyrosine kinaseSignal TransductionTranscription FactorsBlood
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Bis(1H-indol-2-yl)methanones are effective inhibitors of FLT3-ITD tyrosine kinase and partially overcome resistance to PKC412A in vitro.

2009

Inhibition of the mutated fms-like tyrosine kinase 3 (FLT3) receptor tyrosine kinase is a promising therapeutic strategy in acute myeloid leukaemia (AML). However, development of resistance to FLT3 tyrosine kinase inhibitors (TKI), such as PKC412A, has been described recently. This observation may have an increasing impact on the duration of response and relapse rates in upcoming clinical trials employing FLT3-TKI. Herein we investigated two representatives of a novel class of FLT3-TKI: Bis(1H-indol-2-yl)methanones. Both compounds effectively induced apoptosis in FLT3-internal tandem duplicate (ITD)-transfected murine myeloid cells and in primary FLT3-ITD positive blasts. Combination of bot…

MAPK/ERK pathwayIndolesmedicine.drug_classAntineoplastic AgentsApoptosisTransfectionTyrosine-kinase inhibitorReceptor tyrosine kinaseCell Linefluids and secretionshemic and lymphatic diseasesmedicineTumor Cells CulturedHumansProtein kinase Bbiologyhemic and immune systemsHematologyStaurosporineIn vitroLeukemia Myeloid Acutefms-Like Tyrosine Kinase 3Drug Resistance NeoplasmTandem Repeat SequencesTrk receptorembryonic structuresFms-Like Tyrosine Kinase 3Cancer researchbiology.proteinTyrosine kinasepsychological phenomena and processesBritish journal of haematology
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Inheritance patterns of ATCCT repeat interruptions in spinocerebellar ataxia type 10 (SCA10) expansions

2017

Spinocerebellar ataxia type 10 (SCA10), an autosomal dominant cerebellar ataxia disorder, is caused by a non-coding ATTCT microsatellite repeat expansion in the ataxin 10 gene. In a subset of SCA10 families, the 5'-end of the repeat expansion contains a complex sequence of penta- and heptanucleotide interruption motifs which is followed by a pure tract of tandem ATCCT repeats of unknown length at its 3'-end. Intriguingly, expansions that carry these interruption motifs correlate with an epileptic seizure phenotype and are unstable despite the theory that interruptions are expected to stabilize expanded repeats. To examine the apparent contradiction of unstable, interruption-positive SCA10 e…

Male0301 basic medicineMolecular biologyInheritance Patternslcsh:MedicineGene ExpressionArtificial Gene Amplification and ExtensionPolymerase Chain ReactionDatabase and Informatics MethodsSequencing techniquesAutosomal dominant cerebellar ataxiaMedicine and Health SciencesDNA sequencinglcsh:ScienceGeneticsMovement DisordersMultidisciplinaryNeurodegenerative DiseasesGenomicsPedigreePhenotypeNeurologyMutation (genetic algorithm)Spinocerebellar ataxiaFemaleSequence AnalysisResearch ArticleBioinformaticsBiologyAtaxin-1003 medical and health sciencesSequence Motif AnalysisMicrosatellite RepeatGeneticsmedicineHumansSpinocerebellar AtaxiasRepeated SequencesAlleleAllelesSequence (medicine)EpilepsyBase SequenceBiology and life scienceslcsh:RDideoxy DNA sequencingGenetic Variationmedicine.diseaseResearch and analysis methodsMolecular biology techniques030104 developmental biologyTandem Repeat Sequence AnalysisAtaxinMutationlcsh:QAtaxiaTrinucleotide repeat expansionMicrosatellite RepeatsPLOS ONE
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