Search results for "Targeted therapie"

showing 4 items of 14 documents

Met inhibition revokes IFNγ-induction of PD-1 ligands in MET-amplified tumours

2019

BACKGROUND: Interferon-induced expression of programmed cell death ligands (PD-L1/PD-L2) may sustain tumour immuneevasion. Patients featuring MET amplification, a genetic lesion driving transformation, may benefit from anti-MET treatment. We explored if MET-targeted therapy interferes with Interferon-gamma modulation of PD-L1/PD-L2 in MET-amplified tumours.METHODS: PD-L1/PD-L2 expression and signalling pathways downstream of MET or Interferon-gamma were analysed in MET-amplified tumour cell lines and in patient-derived tumour organoids, in basal condition, upon Interferon-gamma stimulation, and after anti-MET therapy.RESULTS: PD-L1 and PD-L2 were upregulated in MET-amplified tumour cells up…

Programmed cell deathCancer ResearchCancer immunotherapyMET-amplified tumoursB7-H1 AntigenArticleInterferon-gammaTargeted therapiesDownregulation and upregulationInterferonCell Line TumorNeoplasmsHumansMedicineMet inhibitionMolecular Targeted TherapySTAT1Kinase activityReceptorProtein Kinase InhibitorsJanus KinasesReceptors InterferonOncogenebiologyPD-1 ligandsbusiness.industryLiver NeoplasmsOncogenesProto-Oncogene Proteins c-metProgrammed Cell Death 1 Ligand 2 ProteinOrganoidsSTAT1 Transcription FactorOncologybiology.proteinCancer researchOncology; Cancer Research; Met inhibition; IFNγ-induction;PD-1 ligands; MET-amplified tumoursTumor EscapeSignal transductionColorectal NeoplasmsbusinessIFNγ-inductionSignal Transductionmedicine.drug
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OPALINE Study: Observational Study in a Real-World Setting of the Systemic Treatment of Progressive Unresectable or Well-Differentiated Metastatic Pa…

2017

International audience

[SDV.MHEP.EM] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismpnet[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismComputingMilieux_MISCELLANEOUStargeted therapies
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Targeting Phosphatases and Kinases: How to Checkmate Cancer

2021

Metastatic disease represents the major cause of death in oncologic patients worldwide. Accumulating evidence have highlighted the relevance of a small population of cancer cells, named cancer stem cells (CSCs), in the resistance to therapies, as well as cancer recurrence and metastasis. Standard anti-cancer treatments are not always conclusively curative, posing an urgent need to discover new targets for an effective therapy. Kinases and phosphatases are implicated in many cellular processes, such as proliferation, differentiation and oncogenic transformation. These proteins are crucial regulators of intracellular signaling pathways mediating multiple cellular activities. Therefore, altera…

cancer stem cellkinaseQH301-705.5PopulationPhosphataseDiseaseReviewBiologyMetastasisphosphatasecancer stem cell kinase phosphatase phosphatase and kinase inhibitors targeted therapiesCell and Developmental BiologyCancer stem cellmedicineBiology (General)educationphosphatase and kinase inhibitorseducation.field_of_studyKinaseCancerCell Biologymedicine.diseasetargeted therapiesCancer cellCancer researchDevelopmental BiologyFrontiers in Cell and Developmental Biology
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Nueva era en el combate contra el cáncer: Evolución y cáncer: progreso y tratamiento de tumores

2013

L’oncohematologia ha assistit en les últimes dècades a importants avenços en el tractament i supervivència. La toxicitat derivada de la quimioteràpia clàssica i la necessitat de millorar l’eficàcia i tolerància als tractaments han fet que es desencadene un allau d’activitat investigadora que constantment proporciona fruits. Gran part d’aquest esforç s’ha enfocat al desenvolupament de «teràpies diana» dirigides contra determinades alteracions funcionals, clau per a la supervivència de la cèl·lula tumoral. Així es pretén aconseguir major eficàcia en el tractament i que el pacient pague un menor cost en toxicitat. Avenços en altres disciplines de l’oncologia com l’epidemiologia, la cirurgia i …

evolucióncribado poblacionalmecanismes de resistènciafàrmacs antidianaresistance mechanismsbiologia; evolució; genèticaterapias dirigidas; fármacos anti diana; inhibidores tirosin kinasa; mecanismos de resistencia; cribado poblacionalevolutiontyrosine kinase inhibitorsteràpies dirigides; fàrmacs antidiana; inhibidors tirosina-cinasa; mecanismes de resistència; cribatge poblacionalinhibidores tirosin kinasageneticsteràpies dirigidesanti-target drugsbiologygenèticascreeningtargeted therapies; anti-target drugs; tyrosine kinase inhibitors; resistance mechanisms; screeningcribatge poblacionalmecanismos de resistenciatargeted therapiesevolución; biología; medicinaterapias dirigidasevoluciófármacos anti dianabiologíabiology; evolution; geneticsmedicinabiologiainhibidors tirosina-cinasa
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