Search results for "Tatin"

showing 10 items of 700 documents

Fluvastatin stabilizes the blood–brain barrier in vitro by nitric oxide-dependent dephosphorylation of myosin light chains

2006

Inhibition of the 3-hydroxy-3-methylglutaryl-coenzyme-A reductase and the downstream mevalonate pathway is in part responsible for the beneficial effects that statins exert on the cardiovascular system. In this study we aimed at analysing the stabilizing effects of fluvastatin on the blood-brain barrier (BBB) integrity, using an in vitro co-culture model of ECV304 and C6, or primary bovine endothelial cells and rat astrocytes. Fluvastatin dose-dependently (1-25 micromol/l) increased barrier integrity as analysed by measurements of transendothelial electrical resistance (TEER). This effect (117.4+/-2.6% at 25 micromol/l) was significantly reduced by the nitric oxide (NO) synthase inhibitor L…

IndolesMyosin Light ChainsMyosin light-chain kinaseGeranylgeranyl pyrophosphatePhosphataseFarnesyl pyrophosphateBiologyNitric OxideBlood–brain barrierAntioxidantsCapillary PermeabilityFatty Acids MonounsaturatedDephosphorylationMiceCellular and Molecular Neurosciencechemistry.chemical_compoundElectric ImpedancemedicineAnimalsDrug InteractionsEnzyme InhibitorsFluvastatinCells CulturedPharmacologyAnalysis of VarianceMicroscopy Confocalomega-N-MethylarginineDose-Response Relationship DrugEndothelial CellsBiological TransportMolecular biologyCoculture TechniquesRatsmedicine.anatomical_structurechemistryBiochemistryBlood-Brain BarrierAstrocytesModels AnimalCattleMevalonate pathwayFluvastatinmedicine.drugNeuropharmacology
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Fluvastatin prevents glutamate-induced blood-brain-barrier disruption in vitro.

2008

Abstract Glutamate is an important excitatory amino acid in the central nervous system. Under pathological conditions glutamate levels dramatically increase. Aim of the present study was to examine whether the HMG-CoA inhibitor fluvastatin prevents glutamate-induced blood-brain-barrier (BBB) disruption. Measurements of transendothelial electrical resistance (TEER) were performed to analyze BBB integrity in an in vitro co-culture model of brain endothelial and glial cells. Myosin light chain (MLC) phosphorylation was detected by immunohistochemistry, or using the in-cell western technique. Intracellular Ca 2+ and reactive oxygen species (ROS) levels were analyzed using the fluorescence dyes …

IndolesMyosin Light ChainsTime FactorsIntracellular SpaceGlutamic AcidBiologymedicine.disease_causeNitric OxideReceptors N-Methyl-D-AspartateGeneral Biochemistry Genetics and Molecular BiologyNitric oxideCell LineFatty Acids Monounsaturatedchemistry.chemical_compoundBAPTAmedicineElectric ImpedanceAnimalsGeneral Pharmacology Toxicology and PharmaceuticsPhosphorylationFluvastatinDose-Response Relationship DrugGlutamate receptorEndothelial CellsGeneral MedicineCell biologyRatsOxidative StresschemistryBiochemistryBlood-Brain BarrierApocyninNMDA receptorCalciumNAD+ kinaseReactive Oxygen SpeciesOxidative stressFluvastatinmedicine.drugSignal TransductionLife sciences
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Analysis and modulation of the inflammatory response through lung agression related to bacterial infection and mechanical ventilation

2015

Despite major advances since decades in the management of ventilated patients, ventilator-associated pneumonia (VAP) continues to complicate the course of approximately 28% of the patients receiving mechanical ventilation (MV). Among patients hospitalized in intensive care units, the risk of pneumonia is 3- to 10- fold increased in MV patients. However, MV is often the only way to care for critically ill patients with respiratory failure. It has now been clearly demonstrated that MV, in particular adverse ventilatory strategies could activate lung cells, thus leading to a proinflammatory response, even in the absence of pathogen. This is the biotrauma paradigm, which accounts, at least in p…

InflammationVentilation mécaniqueLésions pulmonairesStatinsPneumonie acquise sous ventilation mécaniquePneumonia[SDV.BC]Life Sciences [q-bio]/Cellular BiologyLung injuryStatinesProne positionMechanical ventilationDécubitus ventralAlarminesPneumonieAlarminsVentilator-associated pneumoniaInfection[SDV.BC] Life Sciences [q-bio]/Cellular Biology
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The influence of innovation stimulus, facilitating factors and support mechanisms on innovation performance in Brazilian small textile firms

2020

The ability of the organisation to develop new products is crucial in rapidly changing business environments, and thus, customer demographics, consumers' expectations and buying behaviour play an important role. Hence, exploring the antecedents of innovation performance becomes more important. This study analyses the relationships among innovation stimulus, facilitating factors and support mechanisms for innovative performance in the textile industry of Santa Catarina, Brazil. The sample included 128 respondents, who were all employees of five small and medium-sized textile firms; data were analysed using structural equation modelling. We found that innovation stimulus, facilitating factors…

Innovation facilitating factorsTextile industryVisionEconomics and EconometricsInnovation stimulusDemographicsInnovation performanceInnovation support mechanismsbusiness.industryStrategy and ManagementInnovation facilitating factors; Innovation performance; Innovation stimulus; Innovation support mechanisms; Textile industryStimulus (physiology)Structural equation modelingManagement of Technology and InnovationBusinessBusiness and International ManagementIndustrial organizationTextile industry
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Cholesterol Dependence of Collagen and Echovirus 1 Trafficking along the Novel α2β1 Integrin Internalization Pathway

2013

We have previously shown that soluble collagen and a human pathogen, echovirus 1 (EV1) cluster α2β1 integrin on the plasma membrane and cause their internalization into cytoplasmic endosomes. Here we show that cholesterol plays a major role not only in the uptake of α2β1 integrin and its ligands but also in the formation of α2 integrin-specific multivesicular bodies (α2-MVBs) and virus infection. EV1 infection and α2β1 integrin internalization were totally halted by low amounts of the cholesterol-aggregating drugs filipin or nystatin. Inhibition of cholesterol synthesis and accumulation of lanosterol after ketoconazole treatment inhibited uptake of collagen, virus and clustered integrin, an…

IntegrinsNystatinFluorescent Antibody TechniqueBiochemistryCollagen receptorchemistry.chemical_compoundBINDINGMolecular Cell BiologyInternalizationLipid raftREQUIRESmedia_common0303 health sciencesMicroscopy ConfocalMultidisciplinarybiologyQRIMMUNODEFICIENCY-VIRUS TYPE-1RNA REPLICATIONCellular StructuresExtracellular MatrixEnterovirus B Human3. Good healthCell biologyProtein TransportCholesterolENTRYCytochemistryMedicineMembranes and Sortinglipids (amino acids peptides and proteins)CollagenIntegrin alpha2beta1Research ArticleSignal TransductionViral EntryEndosomeSciencemedia_common.quotation_subjecteducationIntegrinLOW-DENSITY-LIPOPROTEINMicrobiologyFilipinClathrinGPI-ANCHORED PROTEINS03 medical and health sciencesVirologyCell Line TumorCell AdhesionHumansFilipinBiology030304 developmental biology030306 microbiologyCell MembraneVirus Uncoatingta1182TRANSPORTLIPID RAFTSMicroscopy ElectronSubcellular Organelleschemistrybiology.protein3111 BiomedicineChromatography Thin LayerCELL-MEMBRANESViral Transmission and InfectionPLoS ONE
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Cathepsin D in the malignant progression of neoplastic diseases

1992

Recent studies suggest that aspartic proteinase Cathepsin D may be implicated in the process of tumor invasion and metastasis. In fact several in vitro observations showed that this proteinase may facilitate the spread of neoplastic cells through different mechanisms related to its proteolytic activity by acting at different levels of the metastatic cascade. Cathepsin D may promote tumor cell proliferation by acting as an autocrine mitogen through the activation of latent forms of growth factors or by interacting with growth factor receptors. The enzyme was also shown to be able to degrade in vitro extracellular matrix and to activate latent precursors forms of other proteinases involved in…

InvasionMetastasiCathepsin DPepstatin
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Step-by-step diagnosis and management of the nocebo/drucebo effect in statin-associated muscle symptoms patients: a position paper from the Internati…

2022

Statin intolerance is a clinical syndrome whereby adverse effects (AEs) associated with statin therapy [most commonly statin-associated muscle symptoms (SAMS)] result in the discontinuation of therapy and consequently increase the risk of adverse cardiovascular outcomes. However, complete statin intolerance occurs in only a small minority of treated patients (estimated prevalence of only 3-5%). Many perceived AEs are misattributed (e.g. physical musculoskeletal injury and inflammatory myopathies), and subjective symptoms occur as a result of the fact that patients expect them to do so when taking medicines (the nocebo/drucebo effect)-what might be truth even for over 50% of all patients wit…

KardiologiDrucebo effect Nocebo effect SAMS Statin intoleranceMusclesSAMSNocebo effectLipidsQPRSStatin intolerance.Muscular DiseasesStatin intolerancePhysiology (medical)Drucebo effectHumansOrthopedics and Sports MedicineCardiac and Cardiovascular SystemsHydroxymethylglutaryl-CoA Reductase Inhibitorsdrucebo effect ; nocebo effect ; SAMS ; statin intoleranceJournal of Cachexia Sarcopenia and Muscle
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Identification of an endothelial cell binding site on kininogen domain D3

1995

High and low molecular mass kininogen, two multidomain plasma proteins, bind to endothelial cells, platelets, and neutrophils in the intravascular compartment. The specific cell attachment site on their common heavy chain is mediated by domain-3, a cystatin-like structure with inhibitory capacity for papain-like proteinases (Jiang, Y., Müller-Esterl, W., and Schmaier, A. H. (1992) J. Biol. Chem. 267, 3712-3717). In this report, the domain-3 cell binding site is determined by an antibody-directed strategy. The epitope of monoclonal antibody HKH15, which binds to domain-3 and blocks the binding of kininogens to platelets and endothelial cells, was mapped using seven synthetic peptides, which …

Kininogen bindingBlotting WesternMolecular Sequence DataBiotinBinding CompetitiveBiochemistryEpitopeEpitopesHumansAmino Acid SequenceBinding siteMolecular BiologyKininogenBinding SitesMolecular massKininogensChemistryAntibodies MonoclonalCell BiologyMolecular biologyEndothelial stem cellBiochemistryBiotinylationEndothelium VascularCystatinPeptidesJournal of Biological Chemistry
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PEP-19 and LPI camstatins are RNA-binding proteins

2010

LPIRNA-binding proteinscamstatinPEP-19
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La simvastatina riduce le lipoproteine aterogene in pazienti con coronaropatia prematura: effetti delle statine “beyond-cholesterol”

2002

La simvastatina le lipoproteine cholesterol
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