Search results for "Th1 Cells"

showing 10 items of 115 documents

Transcriptional targeting of dendritic cells in gene gun-mediated DNA immunization favors the induction of type 1 immune responses

2003

Cutaneous dendritic cells (DC) are pivotal for the elicitation of antigen-specific immune responses following gene gun-mediated biolistic transfection of the skin. We transcriptionally targeted transgene expression to DC using vectors containing the murine fascin promoter (pFascin) to control antigen production and compared the immune response elicited with conventional DNA immunization using plasmid constructs with the ubiquitously active CMV promoter (pCMV). Biolistic transfection with pFascin initiated a marked type 1 immune response characterized by the occurrence of a large population of IFN-gamma-producing T helper (Th) cells in spleen and draining lymph nodes. Consistently, immunoglo…

Transcription GeneticGenetic VectorsCancer VaccinesDNA vaccinationGene gunImmune systemAntigenGenes ReporterNeoplasmsDrug DiscoveryGeneticsCytotoxic T cellMolecular BiologyPharmacologybiologyDendritic CellsTransfectionBiolisticsTh1 CellsIsotypeMolecular biologybiology.proteinMolecular MedicineAntibodyCell DivisionSpleenPlasmidsMolecular Therapy
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Regulatory T Cells More Effectively Suppress Th1-Induced Airway Inflammation Compared with Th2

2011

Abstract Asthma is a syndrome with different inflammatory phenotypes. Animal models have shown that, after sensitization and allergen challenge, Th2 and Th1 cells contribute to the development of allergic airway disease. We have previously demonstrated that naturally occurring regulatory T cells (nTregs) can only marginally suppress Th2-induced airway inflammation and airway hyperresponsiveness. In this study, we investigated nTreg-mediated suppression of Th2-induced and Th1-induced acute allergic airway disease. We demonstrate in vivo that nTregs exert their suppressive potency via cAMP transfer on Th2- and Th1-induced airway disease. A comparison of both phenotypes revealed that, despite …

TransgeneImmunologyMice TransgenicInflammationT-Lymphocytes RegulatoryMiceTh2 CellsIn vivoImmunitymedicineAnimalsImmunology and AllergyPotencyCells CulturedSensitizationAsthmaInflammationMice KnockoutMice Inbred BALB Cbusiness.industryTh1 Cellsrespiratory systemmedicine.diseasePhenotypeCoculture TechniquesImmunity Innaterespiratory tract diseasesDisease Models Animalmedicine.anatomical_structureAcute DiseaseImmunologyFemaleDisease SusceptibilityBronchial Hyperreactivitymedicine.symptombusinessThe Journal of Immunology
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Th0 to Th1 switch of CD4 T cell clones specific from the 16-kDa antigen of Mycobacterium tuberculosis after successful therapy: lack of involvement o…

2005

Abstract In this study, we have examined the influence of HLA-DR molecules and the structure of the epitope repertoire of the 16-kDa protein of Mycobacterium tuberculosis on the acquisition of the cytokine secretion pattern of CD4 T cell clones, obtained from tuberculous patients before and after anti-mycobacterial therapy. Our data indicate that TB patients have a predominant Th0 response against the 16-kDa protein and its epitopes and that healing, induced by anti-mycobacterial therapy, is associated with a shift toward a predominant Th1 phenotype. Moreover, both HLA-DR molecules restricting the clone specificity and the nature of the recognized epitope do not play any role in the generat…

TuberculosisImmunologyMolecular Sequence DataEpitopes T-LymphocyteBiologyEpitopeCell LineMycobacterium tuberculosisAntigenHLA-DRmedicineImmunology and AllergyHumansTuberculosisAmino Acid SequenceAntigens BacterialCell DifferentiationHLA-DR AntigensMycobacterium tuberculosisTh1 Cellsbiology.organism_classificationmedicine.diseaseVirologyPhenotypeClone CellsPhenotypeImmunologyCytokine secretionClone (B-cell biology)Immunology letters
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Molecular mechanisms of primary and secondary mucosal immunity using avian infectious bronchitis virus as a model system

2007

Although mucosal immune responses are critical for protection of hosts from clinical illness and even mortality caused by mucosal pathogens, the molecular mechanism of mucosal immunity, which is independent of systemic immunity, remains elusive. To explore the mechanistic basis of mucosal protective immunity, gene transcriptional profiling in mucosal tissues was evaluated after the primary and secondary immunization of animals with an attenuated avian infectious bronchitis virus (IBV), a prototype of Coronavirus and a well-characterized mucosal pathogen. Results showed that a number of innate immune factors including toll-like receptors (TLRs), retinoic-acid-inducible gene-1 (RIG-1), type I…

animal diseasesRespiratory Tract DiseasesLymphocyte Activationmedicine.disease_causeDC dendritic cellMucosal immunityCXCR chemokine (C-X-C motif) receptorCCR chemokine (C-C motif) receptorOligonucleotide Array Sequence AnalysisCoronavirusbiologyReverse Transcriptase Polymerase Chain ReactionAcquired immune systemSpecific Pathogen-Free OrganismsCytokinesAntibodyAvian infectious bronchitis virusCoronavirus InfectionsIBV infectious bronchitis virusInfectious bronchitis virusImmunologychemical and pharmacologic phenomenaArticlePrimary and secondary immunityMolecular mechanismIBVTranscriptional regulationImmune systemImmunitymedicineAnimalsIFN interferonTLR toll-like receptorImmunity MucosalPoultry DiseasesInnate immune systemGeneral VeterinaryGene Expression ProfilingComplement System ProteinsTh1 Cellsbiochemical phenomena metabolism and nutritionCTL cytotoxic T lymphocytebiology.organism_classificationIg immunoglobulinIL interleukinMucosal immunologyImmunologybiology.proteinRNAbacteriaImmunizationChickensVeterinary Immunology and Immunopathology
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Pathogenesis of inflammatory bowel disease: transcription factors in the spotlight.

1998

See article on page 477 Dysregulated cytokine production by mucosal lymphocytes and macrophages has been implicated in the pathogenesis of both Crohn’s disease and ulcerative colitis, the two major forms of human inflammatory bowel disease (IBD).1 Over the past few years, various murine models of chronic intestinal inflammation resembling IBD have been discovered which have provided important clues as to the nature of this dysregulation and to its possible treatment with cytokines.2 Thus, in studies of several of the models most closely resembling Crohn’s disease it has been shown that production of large amounts of Th1-type cytokines—for example, interferon γ, by T cells is a major and ess…

business.industrymedicine.medical_treatmentGastroenterologyNF-kappa BGene ExpressionDiseaseTh1 CellsNFKB1medicine.diseaseInflammatory Bowel DiseasesUlcerative colitisInflammatory bowel diseasedigestive system diseasesPathogenesisCytokineImmunologymedicineCommentaryCytokinesHumansbusinessTranscription factorTransforming growth factorTranscription FactorsGut
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Azithromycin Differentially Alters TCR-Activated Helper T Cell Subset Phenotype and Effector Function

2020

In addition to their antibiotic activities, azithromycin (AZM) exhibits anti-inflammatory effects in various respiratory diseases. One of the potent anti-inflammatory mechanisms is through inhibition of CD4+ helper T (Th) cell effector function. However, their impact on specific Th subset is obscure. Herein, we demonstrate the cellular basis of phenotypic and functional alterations associated with Th subsets following AZM treatment in vitro. Using well-characterized Th subset specific chemokine receptors, we report significant suppression of T cell receptor (TCR)-stimulated hyperactivated CCR4+CXCR3+ (Th0) expansion compared to CCR4-CXCR3+ (Th1-like) and CCR4+CXCR3- (Th2-like) cells. Intere…

lcsh:Immunologic diseases. Allergy0301 basic medicineReceptors CCR4Receptors CXCR3Receptor expressionImmunologyReceptors Antigen T-CellBiologyCXCR303 medical and health sciencesChemokine receptorInterferon-gamma0302 clinical medicineDownregulation and upregulationCell MovementT-Lymphocyte SubsetsImmunology and AllergyHumansIFN-γInterleukin 4Cells CulturedOriginal Researchanti-inflammatoryazithromycinCD4+ helper T cellsCXCR3EffectorCell growthT-cell receptorIL-4apoptosisCell DifferentiationBacterial InfectionsTh1 CellsHealthy VolunteersCell biologyAnti-Bacterial Agents030104 developmental biologyCCR4Interleukin-4lcsh:RC581-607030215 immunologyFrontiers in Immunology
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IL-12 Expands and Differentiates Human Vγ2Vδ2 T Effector Cells Producing Antimicrobial Cytokines and Inhibiting Intracellular Mycobacterial Growth

2019

While IL-12 plays a key role in differentiation of protective CD4+ Th1 response, little is known about mechanisms whereby IL-12 differentiates other T-cell populations. Published studies suggest that predominant Vγ2Vδ2 T cells in humans/nonhuman primates (NHP) are a fast-acting T-cell subset, with capacities to rapidly expand and produce Th1 and cytotoxic cytokines in response to phosphoantigen (E)-4-hydroxy-3-methyl-but-2-enyl pyrophosphate (HMBPP) produced by Mycobacterium tuberculosis (Mtb) or others. However, whether IL-12 signaling pathway mediates fast-acting and Th1 or anti-microbial features of Vγ2Vδ2 T cells remains poorly defined. Here, we show that IL-12, but not other IL-12 fami…

lcsh:Immunologic diseases. AllergyCells1.1 Normal biological development and functioningproliferationImmunologyLymphocyte ActivationV gamma 2V delta 2 T cellsVaccine Related03 medical and health sciencesPhosphatidylinositol 3-Kinases0302 clinical medicineRare DiseasesUnderpinning researchT-Lymphocyte SubsetsImmunology and AllergyTuberculosis2.1 Biological and endogenous factorsHumansAetiologyIntraepithelial LymphocytesCells Cultured030304 developmental biologyOriginal Researchanti-tuberculosis0303 health sciencesCulturedVγ2Vδ2 T cellsTumor Necrosis Factor-alphaInflammatory and immune systemCorrectionCell DifferentiationMycobacterium tuberculosisdifferentiationSTAT4 Transcription FactorTh1 CellsInterleukin-12Organophosphates3. Good healthInfectious DiseasesGood Health and Well BeingMedical MicrobiologyIL-12Infectionlcsh:RC581-607Proto-Oncogene Proteins c-akt030215 immunologySignal TransductionFrontiers in Immunology
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Dendritic cell activation by combined exposure to anti-CD40 plus interleukin (IL)-12 and IL-18 efficiently stimulates anti-tumor immunity

2008

Despite as yet limited clinical effectiveness, dendritic cell (DC)-based immunotherapy remains a promising approach for the treatment of cancer, but requires further improvement in its immunostimulatory effectiveness. Potent anti-tumor immunity often depends on the induction of type 1 (T(H)1) immune responses. Therefore, we combined different DC maturation stimuli that are known to induce T(H)1 immunity [anti-CD40, interleukin (IL)-12, IL-18], with the aim to trigger a T(H)1 driven anti-tumor CTL response. When compared with untreated DC or DC treated with anti-CD40 alone, DC matured with anti-CD40 plus IL-12 and IL-18 expressed significantly more IFN-gamma and IL-12, induced enhanced CD8(+…

medicine.medical_treatmentAntineoplastic AgentsDermatologyCD8-Positive T-LymphocytesModels BiologicalBiochemistryMiceImmune systemAntigens NeoplasmmedicineAnimalsCD40 AntigensAntigen-presenting cellMolecular BiologyMice Inbred BALB Cbusiness.industryInterleukin-18InterleukinDendritic CellsImmunotherapyDendritic cellTh1 CellsInterleukin-12Tumor antigenMice Inbred C57BLImmune SystemImmunologyInterleukin 12ImmunotherapybusinessCD8Experimental Dermatology
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Dynamics of gut mucosal and systemic Th1/Th2 cytokine responses in interferon-gamma and interleukin-12p40 knock out mice during primary and challenge…

2009

Cryptosporidium parvum is an intracellular parasite causing enteritis which can become life-threatening in the immunocompromised host. CD4+ T cells and interferon (IFN)-gamma play dominant roles in host immune response to infection. However, effector mechanisms that are responsible for recovery from infection are poorly understood. In the present study we analyzed mice deficient in IFN-gamma or interleukin (IL)-12 in parallel to C57BL/6 wild type mice as models for murine cryptosporidiosis. Our results identified IFN-gamma as the key cytokine in the innate as well as adaptive immunity during primary and also challenge C. parvum infection. Furthermore, both Th1 and Th2 cytokines appear to co…

medicine.medical_treatmentImmunologyCryptosporidiosisBiologyInterferon-gammaMiceImmune systemTh2 CellsImmunityIleummedicineImmunology and AllergyAnimalsInterferon gammaCryptosporidium parvumMice KnockoutInterleukin-12 Subunit p40Interleukin-18InterleukinHematologyTh1 CellsAcquired immune systembiology.organism_classificationMice Inbred C57BLCytokineCryptosporidium parvumGene Expression RegulationGastric MucosaOrgan SpecificityImmunologyInterleukin 12medicine.drugImmunobiology
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Effects of Th1 and Th2 cytokines on cytokine production and ICAM-1 expression on synovial fibroblasts

1995

OBJECTIVES--To investigate the influence of the Th1 and Th2 lymphokines interleukins (IL)-4 and IL-13, interferon gamma (IFN gamma), and several monokines on the adhesion of mononuclear cells to synovial fibroblasts and intercellular adhesion molecule-1 (ICAM-1) expression and cytokine production of synovial fibroblasts in patients with osteoarthritis. METHODS--Synovial fibroblasts were isolated from patients with osteoarthritis and stimulated with IL-1 beta, IL-4, IL-6, IL-10, IL-12, IL-13, tumour necrosis factor alpha (TNF alpha), and IFN gamma. Subsequently, we determined the production of IL-1 alpha, IL-1 beta, IL-6, IL-10, IL-12, IFN alpha and TNF alpha, and the expression of ICAM-1 ly…

medicine.medical_treatmentImmunologyIntercellular Adhesion Molecule-1BiologyGeneral Biochemistry Genetics and Molecular BiologyTh2 CellsRheumatologyOsteoarthritisCell AdhesionmedicineHumansImmunology and AllergyInterferon gammaCell adhesionCells CulturedInterleukin-13Interleukin-6Cell adhesion moleculeSynovial MembraneLymphokineReceptors InterleukinFibroblastsTh1 CellsIntercellular Adhesion Molecule-1Molecular biologyRecombinant ProteinsReceptors Interleukin-4Cytokinemedicine.anatomical_structureImmunologyCytokinesTumor necrosis factor alphaInterleukin-4Synovial membraneResearch Articlemedicine.drug
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