Search results for "Thymidine"

showing 10 items of 83 documents

Strong immunogenic potential of a B7 retroviral expression vector: generation of HLA-B7-restricted CTL response against selectable marker genes.

1998

The stimulation of a specific immune response is an attractive goal in cancer therapy. Gene transfer of co-stimulatory molecules and/or cytokine genes into tumor cells and the injection of these genetically modified cells leads to tumor rejection by syngeneic hosts and the induction of tumor immunity. However, the development of host immune response could be either due to the introduced immunomodulatory genes or due to vector components. In this study, human renal cell carcinoma cell lines were modified by a retrovirus to express the co-stimulatory molecule B7-1 together with the hygromycin/thymidine kinase fusion protein (HygTk) as positive and negative selection markers. These B7-1-transd…

Genetic MarkersT cellGenetic VectorsLymphocyte ActivationHLA-B7 AntigenImmune systemRetrovirusAntigens NeoplasmGeneticsmedicineTumor Cells CulturedHumansMolecular BiologyCarcinoma Renal CellSelectable markerExpression vectorbiologyHistocompatibility Antigens Class IGene Transfer TechniquesGenetic TherapyAcquired immune systembiology.organism_classificationMolecular biologyKidney Neoplasmsmedicine.anatomical_structureRetroviridaeCell cultureThymidine kinaseCancer researchB7-1 AntigenMolecular MedicineT-Lymphocytes CytotoxicHuman gene therapy
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Apoptosis induced by (E)-5-(2-bromovinyl)-2'-deoxyuridine in varicella zoster virus thymidine kinase-expressing cells is driven by activation of c-Ju…

2003

The molecular mode of cell killing by the antiviral drug (E)-5-(2-bromovinyl-2'-deoxyuridine (BVDU) was studied in Chinese hamster ovary (CHO) cells stably transfected with the thymidine kinase gene (tk) of varicella zoster virus (CHO-VZVtk). The colony-forming ability of the cells was reduced to <1% at a concentration of approximately 1 microM BVDU, whereas for nontransfected cells or cells transfected with tk gene of herpes simplex virus type 1 (CHO-HSVtk), a 1000-fold higher dose was required to achieve the same response. BVDU inhibited thymidylate synthase in CHO-VZVtk but not in CHO-HSVtk and control cells. On the other hand, the drug was incorporated into DNA of VZVtk- and HSVtk-expre…

Herpesvirus 3 HumanFas Ligand ProteinFas-Associated Death Domain ProteinApoptosisCHO CellsBiologyTransfectionAntiviral AgentsThymidine KinaseFas ligandchemistry.chemical_compoundNecrosisCricetinaeCytotoxic T cellAnimalsSimplexvirusAdaptor Proteins Signal TransducingPharmacologyCaspase 8GenomeMembrane GlycoproteinsChinese hamster ovary cellCell CycleJNK Mitogen-Activated Protein KinasesTransfectionDNAThymidylate SynthaseMolecular biologyCaspase 9Transcription Factor AP-1Cell killingchemistryBromodeoxyuridineApoptosisThymidine kinaseCaspasesMolecular MedicineMitogen-Activated Protein KinasesCarrier ProteinsBromodeoxyuridineMolecular pharmacology
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Application of a 5-bromo-2′-deoxyuridine ELISA for measuring the lymphoproliferative response to human cytomegalovirus in HIV-1-infected patients

2002

Assessment of the lymphoproliferative response to human cytomegalovirus (HCMV) may help to identify human immunodeficiency virus (HIV)-1-infected patients at high risk of developing HCMV end-organ disease. The tritiated thymidine ([3H]-TdR)-incorporation assay is the gold standard for measuring lymphoproliferative responses, though it is unsuitable as a routine laboratory procedure. An alternative non-radioactive technique, a 5-bromo-2'-deoxyuridine (BrdU) enzyme-linked immunosorbent assay, was applied for measuring T-cell proliferation in response to HCMV. Stimulation of either 1 x 10(5) or 5 x 10(4) peripheral blood mononuclear cells (PBMCs)/well with 10 PFU/well (before inactivation) of …

Human cytomegalovirusCellular immunityvirusesCytomegalovirusEnzyme-Linked Immunosorbent AssayBiologyLymphocyte ActivationPeripheral blood mononuclear cellViruschemistry.chemical_compoundVirologymedicineHumansAcquired Immunodeficiency SyndromeAIDS-Related Opportunistic Infectionsvirus diseasesmedicine.diseaseVirologyDeoxyuridineBromodeoxyuridinechemistryCytomegalovirus InfectionsHIV-1Indicators and ReagentsThymidineLymphoproliferative responseBromodeoxyuridineJournal of Virological Methods
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A Combined Experimental and Theoretical Approach to the Photogeneration of 5,6-Dihydropyrimidin-5-yl Radicals in Nonaqueous Media

2016

The chemical fate of radical intermediates is relevant to understand the biological effects of radiation and to explain formation of DNA lesions. A direct approach to selectively generate the putative reactive intermediates is based on the irradiation of photolabile precursors. But, to date, radical formation and reactivity have only been studied in aqueous media, which do not completely mimic the micro environment provided by the DNA structure and its complexes with proteins. Thus, it is also important to evaluate the photogeneration of nucleoside-based radicals in nonaqueous media. The attention here is focused on the independent generation of 5,6-dihydropyrimidin-5-yl radicals in organic…

KetoneTHYMIDINEDNA damageRadicalReactive intermediate010402 general chemistryPhotochemistryHydrogen atom abstraction01 natural sciencesQUIMICA ORGANICAAQUEOUS-SOLUTIONSQUIMICA ANALITICASTRAND SCISSIONReactivity (chemistry)REPAIRchemistry.chemical_classificationAqueous solution010405 organic chemistryOrganic ChemistryINDEPENDENT GENERATION0104 chemical sciences56-DIHYDROTHYMID-5-YLDNA-DAMAGEchemistry2ND-ORDER PERTURBATION-THEORYRADIATIONFlash photolysisHYDROGEN-ATOM ABSTRACTIONThe Journal of Organic Chemistry
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COMPLEMENT-DEPENDENT B-CELL ACTIVATION BY COBRA VENOM FACTOR AND OTHER MITOGENS?

1974

It has been proposed that two distinct signals are required for the triggering of the precursors of antibody-forming bone marrow-derived cells (B cells): (a) the binding of antigen or of a mitogen to the corresponding receptor sites on B-cell membranes and (b) the interaction of activated C3 with the C3 receptor of B lymphocytes. There is growing evidence that B-cell mitogens and T (thymus-derived cell)-independent antigens are capable of activating the alternate pathway of the complement system (bypass). Therefore, the effect of another potent bypass inducer was investigated with regard to B-cell activation and the role of C3. Purified, pyrogen-free cobra venom factor was mitogenic for bot…

LipopolysaccharidesErythrocytesT-LymphocytesImmunologyHemolytic Plaque TechniqueMice Inbred StrainsLymphocyte ActivationTritiumArticleMiceAntigenPolysaccharidesLectinsConcanavalin AEscherichia coliAnimalsImmunology and AllergyCells CulturedImmune adherence reactionAntigens BacterialB-LymphocytesSheepbiologyVenomsPokeweed mitogenSnakesComplement System ProteinsMolecular biologyImmune Adherence ReactionComplement systemKineticsCell cultureConcanavalin AAntibody Formationbiology.proteinMitogensAntibodyFetal bovine serumThymidineJournal of Experimental Medicine
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Regression of fibrosis after HBV antiviral therapy. Is cirrhosis reversible?

2014

Long-lasting HBV-DNA suppression is considered to be the best surrogate end-point of antiviral therapy in patients with hepatitis B virus (HBV) related chronic hepatitis or cirrhosis, and it is a prerequisite to prevent liver-related complications and improve survival. Treatment with oral antiviral drugs in patients with HBV cirrhosis is effective in restoring liver function and improving survival even in those with decompensated cirrhosis. These agents are generally well-tolerated for long-term treatment, and several evidences have demonstrated that they are able to reverse liver fibrosis and prevent the occurrence of HCC.

Liver Cirrhosismedicine.medical_specialtyHepatitis B virusCirrhosisCarcinoma HepatocellularGuanineLiver fibrosisOrganophosphonatesAdministration Oralmedicine.disease_causeGastroenterologyAntiviral AgentsFibrosisInternal medicinemedicineHumansIn patientTenofovirHepatitis B virusTelbivudineHepatologybusiness.industryAdeninecirrhosisLiver NeoplasmsAntiviral therapyNucleosidesmedicine.diseaseHepatitis BFibrosisdigestive system diseasesLamivudineHepatocellular carcinomaLiver functionbusinessThymidine
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Lack of the Cell-Cycle Inhibitor p27Kip1 Results in Selective Increase of Transit-Amplifying Cells for Adult Neurogenesis

2002

The subventricular zone (SVZ) is the largest germinal layer in the adult mammalian brain and comprises stem cells, transit-amplifying progenitors, and committed neuroblasts. Although the SVZ contains the highest concentration of dividing cells in the adult brain, the intracellular mechanisms controlling their proliferation have not been elucidated. We show here that loss of the cyclin-dependent kinase inhibitor p27Kip1 has very specific effects on a population of CNS progenitors responsible for adult neurogenesis. Using bromodeoxyuridine and [3H]thymidine incorporation to label cells in S phase and cell-specific markers and electron microscopy to identify distinct cell types, we compared th…

MaleCell typePopulationSubventricular zoneApoptosisCell CountCell Cycle ProteinsMice TransgenicBiologyMicechemistry.chemical_compoundNeuroblastLateral VentriclesSpheroids CellularIn Situ Nick-End LabelingmedicineAnimalsARTICLEProgenitor celleducationCells CulturedNeuronseducation.field_of_studyStem CellsTumor Suppressor ProteinsGeneral NeuroscienceCell CycleNeurogenesisCell DifferentiationImmunohistochemistryCell biologyMice Inbred C57BLmedicine.anatomical_structureBromodeoxyuridinenervous systemchemistryStem cellCell DivisionCyclin-Dependent Kinase Inhibitor p27BromodeoxyuridineThymidineThe Journal of Neuroscience
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Modulation of induced reversion frequency by nucleotide pool imbalance as a tool for mutant characterization.

1987

Addition of thymidine (TdR) or deoxycytidine (CdR) to the culture medium during posttreatment incubation affected the frequency of mutagen-induced reversion in three hypoxanthine-guanine phosphoribosyl transferase-deficient mutants of V79 Chinese hamster cells. With two of the mutants (E20 and I3), reversions induced by N-ethylnitrosourea, ethyl methanesulfonate, and methyl methanesulfonate were enhanced by TdR and were either decreased (E20) or not affected (I3) by CdR. With the third mutant (E21), alkylating agent-induced reversions were enhanced by CdR and decreased by TdR. Finally, 6-amino-2-hydroxypurine induced reversions were enhanced by TdR in mutant I3 and were decreased by TdR or …

MaleEthyl methanesulfonateEpidemiologyHealth Toxicology and MutagenesisMutantReversionMutagenesis (molecular biology technique)BiologyDeoxycytidineCell Linechemistry.chemical_compoundCricetulusDeoxyadenosineCricetinaeAnimalsGenetics (clinical)DeoxyadenosinesNucleotidesPoint mutationFibroblastsMethyl methanesulfonatechemistryBiochemistryMutationThymidineMutagensThymidineEnvironmental and molecular mutagenesis
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Conditions for the Enhancing Effect of Protease Inhibitors on the Concanavalin A Induced Thymidine Response of Murine Lymphocytes

1983

Incorporation of [&lt;sup&gt;3&lt;/sup&gt;H]-thymidine – [&lt;sup&gt;3&lt;/sup&gt;H]-TdR – into concanavalin A (Con A) stimulated murine splenocytes and thymocytes was found to be enhanced by addition of certain concentrations of phenyl-methyl-sulfonylfluoride (PMSF), di-isopropylfluorophosphate (DFP), N-α-tosyl-&lt;i&gt;L&lt;/i&gt;-lysyl-&lt;i&gt;L&lt;/i&gt;-chloromethylketone (TLCK), and soybean trypsin inhibitor (SBTI). No enhancement could be observed when mononuclear cells of the peripheral blood were used, and a medium enhancement when thymocytes were applied. Furthermore, no enhancing effect of the protease inhibitors (PI) on the Con A response of murine splenocytes could be observed…

MaleIsoflurophateTime Factorsmedicine.medical_treatmentImmunologyBiologyLymphocyte ActivationTosyllysine Chloromethyl KetoneMicechemistry.chemical_compoundPhagocytosisConcanavalin AmedicineAnimalsImmunology and AllergyProtease InhibitorsMice Inbred BALB CProteaseDose-Response Relationship DrugMurine splenocytesDrug SynergismGeneral MedicineMolecular biologyKineticschemistryBiochemistryConcanavalin AImmunologybiology.proteinThymidineSpleenThymidineInternational Archives of Allergy and Immunology
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Proliferative response of cells of the dentogingival junction to mechanical stimulation

2001

SUMMARY The aim of this research was to study the proliferative response of junctional epithelium (JE) and gingival connective tissue (GCT) to mechanical stimulation in vivo with regard to the potential occurrence of apical migration of JE and loss of GCT attachment during orthodontic tooth movement. Elastic bands were inserted between the maxillary first and second molars of male rats aged 8 weeks, which were pulse-labelled with 3 H-thymidine and subsequently killed in groups, together with labelled control animals (a total of 98 rats) after periods of 1‐168 hours. Autoradiographs were prepared from plastic mesiodistal sections, and parameters of cell proliferation for JE and GCT of the pa…

MaleMolarPathologymedicine.medical_specialtyTooth Movement TechniquesPeriodontal LigamentEpithelial AttachmentGingivaJunctional epitheliumConnective tissueOrthodonticsStimulationTritiumStatistics NonparametricOrthodontic AppliancesCell MovementPressuremedicineAnimalsPeriodontal fiberConnective Tissue CellsAnalysis of VarianceHyperplasiaChemistryAnatomyHyperplasiaEpithelial Attachmentmedicine.diseaseMolarRatsMajor duodenal papillamedicine.anatomical_structureAutoradiographyStress MechanicalRadiopharmaceuticalsCell DivisionThymidineThe European Journal of Orthodontics
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